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Fountain of Youth in Bile- Longevity Molecule Identified
(Bile Acids are Organic Acids that are a constituent of Bile – they occur primarily as Salts. Chemically, Bile Acids are classified as Steroids)
Cholesterol is excreted from the body by Bile Acids. Bile Acids are the body’s only significant mechanism for the elimination of excess Cholesterol from the body. Bile Acids solubilize Cholesterol in the Bile, preventing the precipitation of Cholesterol in the Gallbladder.
Bile Acids facilitate the digestion of Triglycerides from dietary Fats. Bile Acids function as emulsifying agents that render the Fatty Acids from dietary Triglycerides accessible to Pancreatic Lipase.
Bile Acids facilitate the intestinal absorption of Fat-Soluble Vitamins.
Immune System-Bile Acids may suppress Helicobacter pylori.
New research has identified the role of a bile acid, called lithocholic acid, in extending the lifespan of normally aging yeast—ScienceDaily (Sep. 15, 2010) — The human quest for longer life may be one step closer, thanks to research from Concordia University. Published in the journal Aging, a new study is the first to identify the role of a bile acid, called lithocholic acid (LCA), in extending the lifespan of normally aging yeast. The findings may have significant implications for human longevity and health, as yeast share some common elements with people. “Although we found that LCA greatly extends yeast longevity, yeast do not synthesize this or any other bile acid found in mammals,” says senior author Vladimir Titorenko, Concordia University Research Chair in Genomics, Cell Biology and Aging and a professor in the Department of Biology. “It may be that yeast have evolved to sense bile acids as mildly toxic molecules and respond by undergoing life-extending changes. It is conceivable that the life-extending potential of LCA may be relevant to humans as well.”
Over 19,000 small molecules screened
Titorenko and colleagues screened more than 19,000 small molecules to test their ability to extend yeast-lifespan. Under both normal and stressed conditions, LCA had a major impact.–“Our findings imply that LCA extends longevity by targeting two different mechanisms,” says first author Alexander Goldberg, a Concordia doctoral student. “The first takes place regardless of the number of calories and involves the day-to-day or housekeeping proteins. The second system occurs during calorie-restriction and involves stressor proteins.”—“Regardless of their triggers both of these mechanisms work to suppress the pro-aging process,” he continues.– Bile acids may be beneficial to health—“Although we have an overall idea how LCA works to extend longevity in yeast, we still need to determine if this is the case for other species,” says Titorenko. “We do know from previous studies, however, that bile acids are beneficial to health and longevity. For example, they have shown to accumulate in the serum of long living mice and play a role in improving rodent liver and pancreatic function.”–“This leads us to believe that bile acids have potential as pharmaceutical agents for the treatment of diabetes, obesity and various metabolic disorders, all of which are age-related,” continues Titorenko. “They may indeed offer hope for a healthy aging life.”—This study was funded by the Canadian Institutes of Health Research, the Natural Sciences and Engineering Research Council of Canada, the Canada Foundation for Innovation and the Concordia University Chair Fund.—Story Source-The above story is reprinted from materials provided by Concordia University. –Journal References-Alexander A. Goldberg, Vincent R. Richard, Pavlo Kyryakov, Simon D. Bourque, Adam Beach, Michelle T. Burstein, Anastasia Glebov, Olivia Koupaki, Tatiana Boukh‐Viner, Christopher Gregg, Mylène Juneau, Ann M. English, Vladimir I. Titorenko and David Y. Thomas. Chemical genetic screen identifies lithocholic acid as an anti‐aging compound that extends yeast chronological life span in a TOR independent manner, by modulating housekeeping longevity assurance processes. Aging, 2010; 2 (7): 393 [link]–Alexander A. Goldberg, Pavlo Kyryakov, Simon D. Bourque and Vladimir I. Titorenko. Xenohormetic, hormetic and cytostatic selective forces driving longevity at the ecosystemic level. Aging, 2010; 2 (8): 461-470 [link]
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Show Of The Week January 13 2012
 
Taurine increases bile acid pool size and reduces bile saturation index In the hamster
Dietary Taurine Enhances Cholesterol Degradation and Reduces Serum and Liver Cholesterol
High Dietary Taurine Reduces Apoptosis and Atherosclerosis in the Left Main Coronary Artery
Leaked– US to Start ‘Trade Wars’ with Nations Opposed to Monsanto, GMO Crops
How to Oxygenate yourself
Hydrogen Peroxide Benefits and Data
 
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Taurine increases bile acid pool size and reduces bile saturation index In the hamster
S Bellentani,
M Pecorari,
P Cordoma,
P Marchegiano,
F Manenti,
E Bosisio,
E De Fabiani and
G Galli
+ Author Affiliations
Chair of Gastroenterology, University of Modena, Italy.
Abstract
There is evidence that increased availability of taurine enhances the proportion of taurine-conjugated bile acids in bile. To explore the possibility that taurine treatment could also influence hepatic cholesterol and bile acid metabolism, we fed female hamsters for 1 week and measured both the biliary lipid content and the microsomal level of the rate-limiting enzymes of cholesterol and bile acid synthesis. In these animals the cholesterol 7 alpha-hydroxylase activity was significantly greater in respect to controls (P less than 0.05). The total HMG-CoA reductase activity, as well as that of the active form, was similarly increased. The stimulation of 7 alpha-hydroxycholesterol synthesis was associated with an expansion of the bile acid pool size in taurine-fed animals. Taurine feeding was observed to induce an increase in bile flow as well as in the rate of excretion of bile acids, whereas the secretion rate of cholesterol in bile was decreased. As a consequence, the saturation index was significantly lower in taurine-fed animals (P less than 0.05). The possible mechanisms through which taurine exhibits the modification of the enzyme activities and of the biliary lipid composition are discussed.
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Dietary Taurine Enhances Cholesterol Degradation and Reduces Serum and Liver Cholesterol Concentrations in Rats Fed a High-Cholesterol Diet1
Hidehiko Yokogoshi*2, Hideki Mochizuki*, Ken Nanami*, Yuko Hida*, Fuyuko Miyachi† and Hiroaki Oda†
; * ; School of Food and Nutritional Sciences, The University of Shizuoka, Shizuoka 422-8526, Japan and; † ; Department of Applied Biological Sciences, Nagoya University, Nagoya 464-8601, Japan
2To whom correspondence and reprint requests should be addressed.
The effect of taurine on hypercholesterolemia induced by feeding a high-cholesterol (HC) diet (10g/kg) to rats was examined. When various amounts of taurine (0.25, 0.5, 1, 2.5, 5, 10, 20, 30, 40 or 50 g/kg diet) were supplemented to HC for 2 wk, serum total cholesterol gradually and significantly decreased in a dose-dependent manner and normalized at the dose of 10 g taurine/kg, compared with the control (cholesterol free) diet group. By contrast, serum HDL-cholesterol was elevated by taurine supplementation. The HC diet caused a significant decrease in the concentration of taurine in serum, liver and heart compared to that in the control group, and the effective dose of supplemental taurine to improve its reduction was 2.5 g/kg diet. In the hypercholesterolemic rats fed the HC diet, the excretion of fecal bile acids and hepatic cholesterol 7 α-hydroxylase (CYP7A1) activity and its mRNA level increased significantly, and the supplementation of taurine further enhanced these indexes, indicating an increase in cholesterol degradation. The abundance of mRNA for Apo A-I, one of the main components of HDL, was reduced by HC and recovered by taurine supplementation. Agarose gel electrophoresis revealed that, in hypercholesterolemic rats fed the HC diet, the serum level of the heavier VLDL increased significantly, but taurine repressed this increase and normalized this pattern. Significant correlations were observed between the time- and dose-dependent increases of CYP7A1 gene expression and the decrease of blood cholesterol concentration in rats fed the HC diet supplemented with taurine (time, r = -0.538, P < 0.01, n = 32; dose, r = -0.738, P < 0.001, n = 20). These results suggest that the hypocholesterolemic effects of taurine observed in the hypocholesterolemic rats fed the HC diet were mainly due to the enhancement of cholesterol degradation and the excretion of bile acid.
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High Dietary Taurine Reduces Apoptosis and Atherosclerosis in the Left Main Coronary Artery—-Association With Reduced CCAAT/Enhancer Binding Protein Homologous Protein and Total Plasma Homocysteine but not Lipidemia
Anthony Zulli,
Eza Lau,
Bagus P.P. Wijaya,
Xin Jin,
Komang Sutarga,
Grace D. Schwartz,
Jonathon Learmont,
Peter J. Wookey,
Angelo Zinellu,
Ciriaco Carru,
David L. Hare
+ Author Affiliations
From the Departments of Cardiology (A.Z., E.L., B.P.P.W., X.J., K.S., G.D.S., J.L., P.J.W., D.L.H.) and Medicine (A.Z., D.L.H.), University of Melbourne, Austin Health, Heidelberg, Australia; and the Department of Biomedical Sciences (A.Z., C.C.), University of Sassari Viale S Pietro, Sassari, Italy.
Correspondence to Anthony Zulli, Vascular Laboratory, Department of Cardiology, Austin Health, Heidelberg, Australia. E-mail [email protected]
Abstract
We sought to determine whether taurine could specifically protect against coronary artery disease during an atherogenic diet and whether taurine affects the lipid profile, metabolites of methionine, and endothelial atherogenic systems. Rabbits were fed one of the following diets for 4 weeks: (1) control diet; (2) 0.5% cholesterol+1.0% methionine; or (3) 0.5% cholesterol+1.0% methionine+2.5% taurine. Endothelial function was examined, and the left main coronary artery atherosclerosis was quantified by stereology and semiquantitative immunohistochemistry to determine the endothelial expression of proteins related to the NO, renin-angiotensin, endoplasmic reticulum, and oxidative stress systems, as well as apoptosis. Taurine normalized hyperhomocysteinemia (P<0.05) and significantly reduced hypermethioninemia (P<0.05) but not lipidemia. The intima:media ratio was reduced by 28% (P=0.034), and atherosclerosis was reduced by 64% (P=0.012) and endothelial cell apoptosis by 30% (P<0.01). Endothelial cell CCAAT/enhancer binding protein homologous protein was normalized (P<0.05). Taurine failed to improve hyperlipidemia, endothelial function, or endothelial proteins related to the NO, renin-angiotensin, and oxidative stress systems. Taurine reduces left main coronary artery wall pathology associated with decreased plasma total homocysteine, methionine, apoptosis, and normalization of CCAAT/enhancer binding protein homologous protein. These results elucidate the antiapoptotic and antiatherogenic properties of taurine, possibly via normalization of endoplasmic reticulum stress.
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Leaked– US to Start ‘Trade Wars’ with Nations Opposed to Monsanto, GMO Crops
The United States is threatening nations who oppose Monsanto’s genetically modified (GM) crops with military-style trade wars, according to information obtained and released by the organization WikiLeaks. Nations like France, which have moved to ban one of Monsanto’s GM corn varieties, were requested to be ‘penalized’ by the United States for opposing Monsanto and genetically modified foods. The information reveals just how deep Monsanto’s roots have penetrated key positions within the United States government, with the cables reporting that many U.S. diplomats work directly for Monsanto. The WikiLeaks cable reveals that in late 2007, the United States ambassador to France and business partner to George W. Bush, Craig Stapleton, requested that the European Union along with particular nations that did not support GMO crops be penalized. Stapleton, who co-owned the Dallas/Fort Worth-based Texas Rangers baseball team with Bush in the 1990s, stated–“Country team Paris recommends that we calibrate a target retaliation list that causes some pain across the EU since this is a collective responsibility, but that also focuses in part on the worst culprits. The list should be measured rather than vicious and must be sustainable over the long term, since we should not expect an early victory. Moving to retaliation will make clear that the current path has real costs to EU interests and could help strengthen European pro-biotech voices.”
The Leaked Political Agenda Behind Monsanto’s GMO Crops
The ambassador plainly calls for ‘target retaliation’ against nations who are against using Monsanto’s genetically modified corn, admittedly linked to organ damage and environmental devastation. Amazingly, this is not an isolated case. In similar newly released cables, United States diplomats are found to have pushed GMO crops as a strategic government and commercial imperative. Furthermore, the U.S. specifically targeted advisers to the pope, due to the fact that many Catholic bishops and figureheads have openly denounced GMO crops. In fact, the Vatican has openly declared Monsanto’s GMO crops as a ‘new form of slavery’.[U1] “A Martino deputy told us recently that the cardinal had co-operated with embassy Vatican on biotech over the past two years in part to compensate for his vocal disapproval of the Iraq war and its aftermath – to keep relations with the USG [US government] smooth. According to our source, Martino no longer feels the need to take this approach,” says the cable.—Perhaps the most shocking piece of information exposed by the cables is the fact that these U.S. diplomats are actually working directly for biotech corporations like Monsanto. The cables also highlight the relationship between the U.S. and Spain in their conquest to persuade other nations to allow for the expansion of GMO crops. Not only did the Spanish government secretly correspond with the U.S. government on the subject, but the U.S. government actually knew beforehand how Spain would vote before the Spanish biotech commission reported their decision regarding GMO crops. The cable states–“In response to recent urgent requests by [Spanish rural affairs ministry] state secretary Josep Puxeu and Monsanto, post requests renewed US government support of Spain’s science-based agricultural biotechnology position through high-level US government intervention.”—-Monsanto has undoubtedly infiltrated the United States government in order to push their health-endangering agenda, and this has been known long before the release of these WikiLeaks cables. The U.S. is the only place where Monsanto’s synthetic hormone Posilac is still used in roughly 1/3 of all cows, with 27 nations banning the substance over legitimate health concerns. Despite Monsanto’s best attempts at incognito political corruption, nothing can stop the grassroots anti-Monsanto movement that is taking over cities and nations alike
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How to Oxygenate yourself
Oxygenate—when you see heavy dosing of chem. Trails you may want to use “ Rocket Fuel” as some call it in science or Hydrogen Peroxide—if you have food grade—take 1-3 drops in a 1 ounce container—add distilled water and then attach a mister or a spray nozzle—and every so often give your self a pump this will release or increase Oxygen into the blood stream and take a load off the heart—and increase oxygen circulation throughout the body—
When doing this as well Increase Vitamin C intake ( 5 grams plus) to offset any prooxidant effect of the H2O2
ORRR got to a dollar store and get a Peroxide without stabilzers—and add the whole thing to a one ounce bottle and add a mister to it and apply as been mentioned in the above paragraph—this will again offset any potential issue that may occur as a lack of Oxygen
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Hydrogen Peroxide Benefits and Data
Pure Hydrogen Peroxide, discovered by a French chemist in 1818 was utilized extensively from the early 19th century until the early 20th Century as a treatment of various illnesses such as; asthma, cholera, syphilis, typhoid fever, tuberculosis, ulcers and pertussis. Food Grade Hydrogen Peroxide is approved by the United States Department of Agriculture as it contains no harmful additives.
The use of Hydrogen Peroxide Treatment lost a great deal of its appeal with the introduction of the pharmaceutical industry in the 20th Century. With the increased knowledge of several of the side effects produced by taking today’s prescribed medicines, a lot of individuals are reverting back to the use of the older, conventional cures including Hydrogen Peroxide Treatment, also called Oxygen Therapy.
Hydrogen Peroxide was utilized following the First World War to fight a pneumonia pandemic. It was, and still is, used extensively to treat emphysema and congestive lung problems. The main benefit is due to its ability to increment the oxygen content of blood and body tissue which increases blood flow and raises body temperature.
Some of the medical uses of Food Grade Hydrogen Peroxide contain:
Cuts, Scrapes and Open Sores -This is the old conventional treatment for open cuts and sores. The procedure consists of applying Hydrogen Peroxide to kill any infection.
Acne – Applied with a cotton swob it will dehydrate the acne. A moisturizer ought to be applied afterwards to restore some of the natural moisture of the skin as the Hydrogen Peroxide will dry the area encompassing the acne.
Mouthwash – A mild solution may be used as a mouthwash. This will kill bacteria that causes bad breath as well as help prevent Periodontal disease. Simultaneously it will whiten your teeth.
Take away Ear Wax – A weak solution put in your ear will aid in the removal of set ear wax.
Athletes Feet – You could use a spray bottle to apply to affected areas of the feet. This will kill off the fungus causing the athletes feet.
Colds, Flu – Gargling a weak solution will kill off the germs causing these illnesses.
Some Non-medical, family uses, include:
Stain Remover – Amazing for removing blood stains.
Sanitizer – Utilize it in the kitchen and bathroom to sanitize and clean all surfaces.
Wash Vegetables – Vegetables are sprayed with all types of insecticides and additives to keep them fresh. Rinsing them off with a solution of Hydrogen Peroxide will kill off these harmful additives.
Water Plants – Utilized to provide extra oxygen to the soil. Numerous farmers now spray a weak solution on their crops to remove harmful bacteria and act as a natural insecticide.
Treat Fungus – Wash moldy places with Hydrogen Peroxide.
Hydrogen Peroxide is likely one of the best and least expensive products on the market nowadays that can be utilized in a lot of areas. Everybody should have a bottle on hand at all times and utilize it wherever harmful bacteria is present.
Utilize it with care and utilize it often
Read more: http://www.articlesbase.com/health-articles/benefits-and-many-uses-of-food-grade-hydrogen-peroxide-4201698.html#ixzz1KriOiqjM
Under Creative Commons License: Attribution
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[U1]The Irony Here is that Own Monsanto—so this is a smoke screen to distance themselves as to save Face among there 1 billion catholics—if they knew that there own religion was forming a genocide on the membership they would be outraged—this would disassociate them from the institution
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Show of the Week January 16 2012
 
Does Sugar Feed Cancer?
Cancer Cells Feed On Sugar-Free Diet
Statins linked with small diabetes risk
Rare ‘supersoldier’ ants created in Canadian lab
These Substances may Enhance Hair Growth -if Hair Follicles are still Alive
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Does Sugar Feed Cancer?
ScienceDaily (Aug. 17, 2009) — Researchers at Huntsman Cancer Institute at the University of Utah have uncovered new information on the notion that sugar “feeds” tumors. The findings may also have implications for other diseases such as diabetes. The research is published in the journal Proceedings of the National Academy of Sciences.–“It’s been known since 1923 that tumor cells use a lot more glucose than normal cells. Our research helps show how this process takes place, and how it might be stopped to control tumor growth,” says Don Ayer, Ph.D., a Huntsman Cancer Institute investigator and professor in the Department of Oncological Sciences at the University of Utah.-During both normal and cancerous cell growth, a cellular process takes place that involves both glucose (sugar) and glutamine (an amino acid). Glucose and glutamine are both essential for cell growth,[U1] and it was long assumed they operated independently, but Ayer’s research shows they are inter-dependent. He discovered that by restricting glutamine availability, glucose utilization is also stopped. “Essentially, if you don’t have glutamine, the cell is short circuited due to a lack of glucose, which halts the growth of the tumor cell” Ayer says.–The research, spearheaded by Mohan Kaadige, Ph.D., a postdoctoral fellow in Ayer’s lab, focused on MondoA, a protein that is responsible for turning genes on and off. In the presence of glutamine, MondoA blocks the expression of a gene called TXNIP. TXNIP is thought to be a tumor suppressor, but when it’s blocked by MondoA , it allows cells to take up glucose, which in turn drives tumor growth. Ayer’s research could lead to new drugs that would target glutamine utilization, or target MondoA or TXNIP.–Ayer says the next step in his research is to develop animal models to test his ideas about how MondoA and TXNIP control cell growth. “If we can understand that, we can break the cycle of glucose utilization which could be beneficial in the treatment of cancer,” Ayer says.–Story Source–The above story is reprinted from materials provided by University of Utah Health Sciences, via EurekAlert!, a service of AAAS.
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Cancer Cells Feed On Sugar-Free Diet
ScienceDaily (Jan. 10, 2012) — Cancer cells have been long known to have a “sweet tooth,” using vast amounts of glucose for energy and for building blocks for cell replication.—Now, a study by a team of researchers at Johns Hopkins and elsewhere shows that lymph gland cancer cells called B cells can use glutamine in the absence of glucose for cell replication and survival, particularly under low-oxygen conditions, which are common in tumors.—Writing in the Jan. 4, 2012, edition of Cell Metabolism, Anne Le, M.D., and a team of investigators collaborating with the Johns Hopkins Brain Science Institute, say the finding is critical for developing innovative cancer therapies because it offers “proof of concept” evidence that curbing the growth of B cell cancers can be accomplished by inhibiting a glutamine enzyme called glutaminase.–Le notes that although little is known about glutamine’s role in the growth of B cell cancer, the amino acid circulates in the blood at the highest level among the 20 amino acids that do so.–The tricarboxylic acid cycle (TCA or Krebs cycle) is classically regarded as a pathway for glucose oxidation. However, the experiments by Le and the team show that B cells oxidize glutamine when glucose is absent.—The study also found that when oxygen is scarce, there is enhanced conversion of glutamine to glutathione, an important agent for controlling the accumulation of oxygen-containing chemically reactive molecules that cause damage to normal cells.—When the investigators used a glutaminase inhibitor, cancerous growth of B cells was stopped in petri dishes.—“The flexibility of the TCA cycle in using both glutamine and glucose pathways may be important for cancer cells to proliferate and survive, especially under the low-oxygen and nutrient-deprived conditions often encountered in the tumor microenvironment,” says Le.–Now, perhaps, scientists can exploit that survival strategy to stop cancer, according to former Johns Hopkins scientist Chi Dang, M.D., now at the Abramson Cancer Center at the University of Pennsylvania. “A broader and deeper understanding of cancer cell metabolism and cancer cells’ability to reprogram biochemical pathways under metabolic stress can be a rich ground for therapeutic approaches targeting tumor metabolism,” he says.—In addition to Le, an assistant professor in the Department of Pathology at the Johns Hopkins University School of Medicine, other researchers from Johns Hopkins who participated in this study include Sminu Bose, Arvin Gouw, Joseph Barbi, Takashi Tsukamoto, Camilo J. Rojas and Barbara Slusher. The Johns Hopkins Brain Science Institute, where Tsukamoto, Rojas and Slusher are faculty, is pursuing the development of new glutaminase inhibitor drugs.—Story Source-The above story is reprinted from materials provided by Johns Hopkins Medicine. –Journal Reference–Anne Le, Andrew N. Lane, Max Hamaker, Sminu Bose, Arvin Gouw, Joseph Barbi, Takashi Tsukamoto, Camilio J. Rojas, Barbara S. Slusher, Haixia Zhang, Lisa J. Zimmerman, Daniel C. Liebler, Robbert J.C. Slebos, Pawel K. Lorkiewicz, Richard M. Higashi, Teresa W.M. Fan, Chi V. Dang. Glucose-Independent Glutamine Metabolism via TCA Cycling for Proliferation and Survival in B Cells. Cell Metabolism, 2012; 15 (1): 110 DOI: 10.1016/j.cmet.2011.12.009
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Statins linked with small diabetes risk
Whether statins can make blood sugar rise enough that someone crosses the threshold to diabetes has been confusing. -A new side-effect seems to be emerging for those cholesterol-lowering wonder drugs called statins: They may increase some people’s chances of developing Type 2 diabetes.–A study adds to the evidence, finding a modest risk among older women who used a variety of statins. It’s a puzzling link, and specialists say people who most need statins because of a high risk for a heart attack should stick with the drugs.-“What I fear here is that people who need and will benefit from statins will be scared off of using the drugs because of reports like this,” says Dr. Steven Nissen, cardiology chairman at the Cleveland Clinic, who wasn’t involved with the research. “We don’t want these drugs in the water supply, but we want the right people treated[U2]. When they are, this effect is not a significant limitation.”–But more and more doctors are urging otherwise healthy people to use the pills as a way to prevent heart disease[U3]. For them, the findings add another potential complication as they consider whether to tackle their cholesterol with diet and exercise alone or add a medication.-“The statin should not be seen as the magic pill,”[U4] says Dr. Yunsheng Ma of the University of Massachusetts Medical School, who led Monday’s study of postmenopausal women.
Heart risks of diabetes
Statins are one of the most widely prescribed drugs, and among the most touted with good reason. They can dramatically lower so-called “bad” LDL cholesterol. Studies make clear that they save lives when used by people who already have heart disease.–What’s debated is how much the drugs help people who don’t yet have cardiovascular disease but whose chances are higher because of other factors such as smoking or high blood pressure — or diabetes.—In fact, long-term diabetes is so heart-risky that the American Diabetes Association urges fairly aggressive statin use by many diabetics. For everyone else, Nissen says the general rule is statins help people who have at least a 10 per cent chance of a heart attack in the next 10 years, something a doctor can calculate.
All drugs have side-effects that are important to consider while deciding whether they’re a good bet for an individual. Statins have long been known to cause muscle pain that on rare occasions becomes a serious breakdown of muscle that can lead to kidney failure, even death.—Not to mention RNA damage—and lobotomizing the brain as well—But whether statins can make blood sugar rise enough that someone crosses the threshold to diabetes has been confusing.–After all, some of the same risks for heart disease — such as being overweight and sedentary — also increase the odds of developing Type 2 diabetes. And Ma says too many statin users wrongly assume the pills will let them eat whatever they want.
Ma’s team examined a huge government study that tracked the health of postmenopausal women for many years. They culled the records of more than 153,000 women who didn’t have diabetes when they enrolled in the Women’s Health Initiative in the 1990s. Just 7 per cent were taking statins at the time. Fast forward to 2005: Nearly 10 per cent of the statin users had developed diabetes as a needed note of caution for women, saying there’s less certainty about the drugs’ overall effects in them. Stay tuned: Her own research aims to narrow down which statin users are more likely to experience a blood-sugar jump.
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Rare ‘supersoldier’ ants created in Canadian lab
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McGill Unversity researchers were able to create supersoldier ants, which are a biological anomaly, in the lab. (Alex Wilder/alexanderwilder.com)
As It Happens on super soldier ants8:09
Canadian researchers have discovered how to make a certain kinds of ants develop into “supersoldiers”[U5] with huge oblong heads and giant vicious jaws.–The findings are significant because they show there is dormant genetic potential that can be invoked by changes in the environment and locked in place for a very long time, said lead author Ehab Abouheif, a McGill University biology professor, whose research was published Friday in the journal Science.–The authors suggest that hanging on to ancestral developmental toolkits can be an important way for organisms to evolve new physical traits.–“Birds with teeth, snakes with fingers and humans with ape-like hair – these are ancestral traits that pop regularly in nature,”[U6] Abouheif said. “But for the longest time in evolutionary theory, these ancestral traits were thought to go nowhere … the Barnum and Bailey of evolution. So they’ve been an unappreciated source of evolutionary variation.”—Typically, supersoldier ants are biological anomalies that occur rarely in nature and only in limited geographical regions. But the McGill researchers found these supersoldiers in unexpected regions and also created them by manipulating hormones.—Pheidole (big-headed) ant colonies contain millions of ants, including minor workers and soldiers. Depending on the food ants are fed, certain hormones are triggered in the ant larvae and they either develop into soldiers or minor workers.–Then there are supersoldier ants that block their nest entrance with their extra-large heads and fight with invading ants during army ant raids.–Abouheif and his team unexpectedly found supersoldier ants in the Pheidole species in Long Island where they aren’t normally seen. They were then able to artificially induce them in the lab by by dabbing the larvae with juvenile hormone, indicating that environmental cues can switch on the genetic machinery that produces supersoldiers.[U7]–“The kind of environmental stressors that evoke this dormant potential are there all the time, so when the need arises natural selection can take hold of the potential and actualize it,” said Abouheif.—“So what we’re showing is that environmental stress is important for evolution because it can facilitate the development of novel phenotypes. Any time you have a mismatch between the normal environment of the organism and its genetic potential you can release them – and these things can be locked in place for 30 to 65 million year
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[U1]Glutamine Breaks down into 5 different amino acids and is the most abundant amino in the oesophagus-and intestine and stomach regions—it will converto to gaba-glutationie-glycine-glutamic acid and ghb—the glycine converts to glucagons( sugar ) which is required fro brain and liver and cellular energy
[U2]“the right People/”” this is amazing these drugs are so damaging from the genetic code to the arterial integrity-RNA DNA damage—brain lobotomizing—who are these “ right people?
[U3]This is unbelievable and the ignorance is what this report is showing the medical field is doing to healthy individuals to compromise there health
[U4]First we are going to use a s preventative and now we are not to look at it as a magical pill
[U5]Welcome to Canada EH!!! The land of Biodiversity and death—we export Nuclear energy ( bombs) food ( GMO WHEAT AND CANOLA ..ETC ) andd Now Pestilence–
[U6]Genetic engineering experiments—anomalies created in a lab
[U7]In other words they created these ants and put them on Long Island and were able to manipulate the Genetic code of these bugs—meaning we are now heading for a new Bio Warfare –we will just create a Super bug to inflict a people or Undesirables we do not want—we will give an ant a poison bite a butterfly with a knoxious sting –a flea to carry MRSA—etc.
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Show of the Week January 20 2012
Antioxidant, antiradical and antimutagenic activities of phenolic compounds present in maple products
Effects of Maple (Acer) Plant Part Extracts on Proliferation, Apoptosis and Cell Cycle Arrest of Human Tumorigenic and Non-tumorigenic Colon Cells
Recipe for Maple Syrup Extract
Ingested Maple Syrup Evokes a Possible Liver-Protecting Effect—Physiologic and Genomic Investigations with Rats
Fungus Contaminant in Corn, Peanuts, Soybeans- Chlorophyll Effective Against Aflatoxin
An aqueous birch leaf extract of Betula pendula (tea or extract ) inhibits the growth and cell division of inflammatory lymphocytes
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Antioxidant, antiradical and antimutagenic activities of phenolic compounds present in maple products
(1) Research Research Laboratory in Sciences Applied to Food, Canadian Irradiation Center (CC), INRS-Institut Armand-Frappier, 531 Boulevard des Prairies, Laval, QC., H7V 1B7, CANADA–(2) Department of Food Science and Agricultural Chemistry, McGill University, 21, 111 Lakeshore, Ste-Anne de Bellevue, QC, H9X 3V9, CANADA
AbstractThe phenolic compounds in maple sap and syrup were extracted at different periods of the season and were separated to collect the glycosylated compounds and the aglycone compounds. The antioxidant and antiradical activities of each phenolic compound were studied using the thiobarbituric acid reactive substances (TBARS) assay and the N,N-diethyl-p-phenylenediamine (DPD) decoloration test to measure the free radical scavenging. The results showed that in general the phenolic compounds had a good antioxidant and antiradical properties. The glycosylated compounds from maple sap and maple syrup showed a better activity than the aglycones.[U1] The antimutagenic effects of each phenolic compounds from maple sap and syrup were also investigated as the inhibition of SOS induction by chemical agents in Salmonella typhimurium TA1535/pSK1002 containing the fusion gene umuC-lacZ. Induction of the SOS gene (umuC) expression was assayed by measuring accumulated β-galactosidase activity using a modified Umu test. The antimutagenic properties were studied per se and after metabolisation by S9 fraction. The results showed that an optimum of antimutagenic properties of the glycosylated metabolites phenolic compounds from sap and syrup was observed at 75% of the season for the sap and at 25% of the season for the syrup. A higher antimutagenic activity was observed at 25% and 100% of the season for aglycones present in syrup and at 75% of the season for aglycones present in sap.
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Effects of Maple (Acer) Plant Part Extracts on Proliferation, Apoptosis and Cell Cycle Arrest of Human Tumorigenic and Non-tumorigenic Colon Cells
Phenolic-enriched extracts of maple sap and syrup, obtained from the sugar and red maple species (Acer saccharum Marsh, A. rubrum L., respectively), are reported to show anticancer effects. Despite traditional medicinal uses of various other parts of these plants by Native Americans, they have not been investigated for anticancer activity. Here leaves, stems/twigs, barks and sapwoods of both maple species were evaluated for antiproliferative effects against human colon tumorigenic (HCT-116, HT-29, Caco-2) and non-tumorigenic (CCD-18Co) cells. Extracts were standardized to total phenolic and ginnalin-A (isolated in our laboratory) levels. Overall, the extracts inhibited the growth of the colon cancer more than normal cells (over two-fold), their activities increased with their ginnalin-A levels, with red > sugar maple extracts. The red maple leaf extract, which contained the highest ginnalin-A content, was the most active extract (IC50 = 35 and 16 µg/mL for extract and ginnalin-A, respectively). The extracts were not cytotoxic nor did they induce apoptosis of the colon cancer cells. However, cell cycle analyses revealed that the antiproliferative effects of the extracts were mediated through cell cycle arrest in the S-phase. The results from the current study suggest that these maple plant part extracts may have potential anticolon cancer effects.
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Ingested Maple Syrup Evokes a Possible Liver-Protecting Effect—Physiologic and Genomic Investigations with Rats
Yuki WATANABE1), Asuka KAMEI1), Fumika SHINOZAKI1), Tomoko ISHIJIMA1)2), Kota IIDA2), Yuji NAKAI1)2), Soichi ARAI1)3) and Keiko ABE1)2
1) Project on Health and Anti-aging, Kanagawa Academy of Science and Technology
2) Graduate School of Agricultural and Life Science, The University of Tokyo
3) General Research Institute, Tokyo University of Agriculture
Rats fed a 20%-maple syrup diet (maple syrup group) for 11 d showed significantly lower values of the hepatic function markers than those fed a 20%-sugar mix syrup diet (control). The reason was suggested by a DNA microarray analysis which revealed that the expression of genes for the enzymes of ammonia formation were down-regulated in the liver of the maple syrup group.
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Recipe for Maple Syrup Extract—
if you cannot tap a tree then buy a maple syrup ( real maple syrup not a pancake syrup) pout it into a pot and at low heat simmer it –make sure you are there with the stuff or it will overflow—ask me how I know?—then with a wooden spoon stir it —when you get to about half way—stop the heat and pour it back in the container you got it from unless the container is plastic then use glass—take out another small portion 4 oz and pour it back into the pot—turn the heat on and continue letting it be warm for another 5 min or so.—then stop the stove and pour into a glass container—it will have almost gone frothy by then before you pour it in the container- allow to sit and it will crystallize –as a bonus you can ad essential oils or tinctures to this and when it crystallizes the fused oil will also be noticeable
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Fungus Contaminant in Corn, Peanuts, Soybeans- Chlorophyll Effective Against Aflatoxin
ScienceDaily (Dec. 29, 2009) — A new study has found that chlorophyll and its derivative chlorophyllin are effective in limiting the absorption of aflatoxin in humans. [U2]Aflatoxin is produced by a fungus that is a contaminant of grains including corn, peanuts and soybeans; it is known to cause liver cancer — and can work in concert with other health concerns, such as hepatitis.—-Levels of aflatoxin are carefully regulated in the United States, but are often found in the food supplies of developing nations, especially those with poor storage facilities.–OSU scientist George Bailey, a distinguished professor of environmental and molecular toxicology, pioneered studies of aflatoxin in China, where he found that in one region, one out of every 10 adults died from liver cancer.–But what has the science world particularly intrigued with this follow-up study is the methodology used by the researchers — a new “Phase 0” approach that safely tests low levels of carcinogens in human volunteers to measure the total aflatoxin exposure and to determine the effect of dietary chlorophlls on reducing this exposure.—Results of the study were just published in the journal Cancer Prevention Research.–Bailey and several other researchers, including lead author Carole Jubert, were part of the recent study. The journal also included a perspective written by a pair of Johns Hopkins researchers — Thomas Kensler and John Groopman — who praise the methodology and suggest that these Phase 0 “microdosing” studies should be expanded.–They wrote: “…microdosing studies with carcinogens have the potential to provide important insights into chemopreventive interventions and to enhance the overall clinical development and safety evaluation of preventive agents.”–The Phase 0 study “…may open the door for all kinds of new research,” said Jubert, a former researcher in Bailey’s lab at OSU’s Linus Pauling Institute. Jubert now works for Life Microsystems, an OSU spinoff company that hopes to continue work with natural products grown in Oregon, including pure chlorophylls.–“The technology is not particularly difficult,” she added. “It’s just a novel approach to evaluate toxin exposure in humans.”–In their study, Jubert and her colleagues gave very low doses of aflatoxin labeled with carbon-14 isotopes as a tracer to four human volunteers. They then gave the volunteers the same doses of aflatoxin along with doses of either chlorophyll or chlorophyllin, which previously had been shown to reduce carcinogen bioavailability in trout and rats. Using an accelerator mass spectrometer, they measured the rate of aflaxtoxin bioavailability. This technique is extremely sensitive, the researchers say, allowing measurement of minute amounts of any labeled compound.–Their research revealed rapid absorption of aflatoxin, which was significantly limited after the chlorophyll and chlorophyllin treatments.–“The beauty of this kind of ‘Phase 0’ study is the use of ultra-sensitive technology and ‘microdoses’ of environmental carcinogens to study toxicokinetics within the human body,” said John Mata, an OSU pharmacologist and second author on the study. “These measurements can be important because they allow us to better design future studies to understand the effects of dietary constituents on cancer risk.——–“In this case, clearly the results merit further study,” Mata added. “We showed that aflatoxin is absorbed quite rapidly and that chlorophyll and chlorophyllin have an ameliorating effect, preventing the toxin from getting into the bloodstream. Further studies can more precisely explore the interactions, as well as dosage levels.”—Jubert and Mata also have tested the feasibility of using similar technology on human exposure to other toxins, including smokers who ingest carcinogens through cigarette smoke.–Mata, a professor in OSU’s College of Veterinary Medicine, is a pharmacologist who previously worked in the drug industry. He said Phase 1 studies are designed to see if a compound is safe; Phase 2 expands the scope of the project, and Phase 3 looks at the compounds’ efficacy. Phase 0 represents a new concept — a way to measure the kinetics of a drug by using extremely small doses that pose little risk to the volunteers.–In this case, the amount of radiation given the human volunteers was equal to that you would encounter from a one-hour airplane ride; the level of aflatoxin administered was 1/30th the amount the Food and Drug Administration allows in a peanut butter sandwich.–Story Source-The above story is reprinted from materials provided by Oregon State University, via EurekAlert!, a service of AAAS.
Simple Remedy—Use chlorophyll to reduce this or juice high amounts of peppermint –parsley-dandelion-watercress and drink glass amounts to offset this
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An aqueous birch leaf extract of Betula pendula (tea or extract ) inhibits the growth and cell division of inflammatory lymphocytes.
J Ethnopharmacol. 2011 Jul 14;136(3):444-51
Authors: Gründemann C, Gruber CW, Hertrampf A, Zehl M, Kopp B, Huber R
Abstract
ETHNOPHARMACOLOGICAL RELEVANCE: Leaf extracts of Betula pendula have been traditionally used for the treatment of patients with rheumatoid arthritis (RA) or osteoarthritis. –AIM OF THE STUDY: We investigated the anti-proliferative capacity of an aqueous leaf extract of Betula pendula (BPE) on human primary lymphocytes in vitro, because activated lymphocytes play a major role in the initiation and maintenance of RA.–MATERIALS AND METHODS: Lymphocyte proliferation and cell division was measured by the activity of mitochondrial dehydrogenases and by using the membrane-permeable dye carboxyfluorescein diacetate succinimidyl ester (CFSE), respectively. Apoptosis was analyzed by surface staining of phosphatidylserine and intracellular activation of effector caspases 3 and 7 in comparison to the drug methotrexate using flow cytometric and photometrical analysis. In addition, the impact of the extract on cell cycle distribution was investigated by propidium iodide staining of DNA. For the bioassays BPE concentrations of 10-160 μg/mL were investigated. A phytochemical analysis, using LC-MS and HPLC, was conducted to identify the polyphenolic constituents of the birch leaf extract.
RESULTS: Leaf extracts of Betula pendula inhibited the growth and cell division (CD8(+): 40 μg/mL: 45%; 80 μg/mL: 60%; 160 μg/mL: 87%) (CD4(+): 40 μg/mL: 33%; 80 μg/mL: 54%; 160 μg/mL: 79%) of activated, but not of resting T lymphocytes in a significant dose-dependent manner. The inhibition of lymphocyte proliferation due to apoptosis induction (compared to untreated control: 40 μg/mL: 163%; 80 μg/mL: 240%; 160 μg/mL: 348%) and cell cycle arrest was comparable to that of methotrexate. LC-MS analyses showed that the extract contains different quercetin-glycosides.–CONCLUSION: Our results give a rational basis for the use of Betula pendula leaf extract for the treatment of immune disorders, like rheumatoid arthritis, by diminishing proliferating inflammatory lymphocytes.–PMID: 21619918 [PubMed – indexed for MEDLINE]
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[U1]Definition of AGLYCONE: an organic compound (as a phenol or alcohol) combined with the sugar portion of a glycoside
[U2]There is copper in this and this is anti mold-antifungal and antibacterial
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Show of the Week January 23 2012
 
First Link Between Potentially Toxic PFCs in Office Air and in Office Workers’ Blood
Short, Sharp Shock Treatment for E. Coli
Dietary DHA Linked to Male Fertility
Pesticide Illnesses and GM Soybeans-Ban on Aerial Spraying Demanded in Argentina
Kidney Stones—how to get rid of them
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First Link Between Potentially Toxic PFCs in Office Air and in Office Workers’ Blood
ScienceDaily (Jan. 18, 2012) — In a first-of-its-kind study, scientists are reporting that the indoor air in offices is an important source of worker exposure to potentially toxic substances released by carpeting, furniture, paint and other items. Their report, which documents a link between levels of these so-called polyfluorinated compounds (PFCs) in office air and in the blood of workers, appears in ACS’ journal Environmental Science & Technology.—-Michael McClean and colleagues explain that PFCs, used in water-repellent coatings on carpet and furniture, may have adverse effects on human health. The substances are widespread in the environment and in humans around the world. Scientists know that potential sources of exposure include food, water, indoor air, indoor dust and direct contact with PFC-containing objects. But the link between levels in air and blood had not been explored previously, so McClean’s group set out to fill that gap with a study of 31 office workers in Boston.—They found concentrations of a PFC called fluorotelomer alcohol (FTOH) in office air that were 3-5 times higher than those reported in previous studies of household air, “suggesting that offices may represent a unique and important exposure environment.” In addition, the study found a strong link between concentrations of FTOH in office air and perfluorooctanoic acid (a metabolite of FTOH) in the blood of office workers. The results also suggested that workers in newly renovated office buildings may receive considerably higher doses of PFCs than workers in older buildings.–The authors acknowledge funding from the National Institute of Environmental Health Sciences. –Story Source–The above story is reprinted from materials provided by American Chemical Society. —Note: Materials may be edited for content and length. For further information, please contact the source cited above.—-Journal Reference-Alicia J. Fraser, Thomas F. Webster, Deborah J. Watkins, Jessica W. Nelson, Heather M. Stapleton, Antonia M. Calafat, Kayoko Kato, Mahiba Shoeib, Verónica M. Vieira, Michael D. McClean. Polyfluorinated Compounds in Serum Linked to Indoor Air in Office Environments. Environmental Science & Technology, 2012; 46 (2): 1209 DOI: 10.1021/es2038257
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Short, Sharp Shock Treatment for E. Coli
ScienceDaily (Jan. 11, 2012) — A short burst of low voltage alternating current can effectively eradicate E. coli bacteria growing on the surface of even heavily contaminated beef, according to a study published in the International Journal of Food Safety, Nutrition and Public Health. The technique offers an inexpensive and easy to implement approach to reducing the risk of food poisoning, which can occur despite handlers complying with hygiene standards.—Food poisoning is a serious public-health issue, especially with the emergence of lethal and highly virulent strains of Escherichia coli (E. coli O157:H7, for example). Infection with this bacterium causes serious diarrhea, dehydration, kidney problems and can lead to serious long-term problems or even be fatal in children, the elderly and people with pre-existing health problems. Tens of thousands of people are affected by E. coli infection each year through eating contaminated beef and other food products. The US Centers for Disease Control and Prevention (CDC) estimates that about 2500 people are hospitalized and there are several dozen deaths each year.—Now, Ajit Mahapatra and colleagues at Fort Valley State University, in Georgia and Virginia Tech have demonstrated that applying a low-voltage alternating current to beef samples inoculated with large numbers of the potentially lethal E. coli O157:H7 can almost completely deactivate the bacterium, which is usually present on the surface of contaminated meat. The team points out that the level of contamination used in their tests far exceeded the contamination that would be seen in commercial carcasses after slaughter.—Previous researchers had demonstrated that electricity can kill bacteria effectively. The study by Mahapatra and colleagues proves efficacy against E. coli O157:H7 at low voltage and low alternating current. It offers a quick and easy way to decontaminate at-risk, but otherwise safe beef without recourse to microbicidal chemicals or other more complicated treatment processes.—Story Source-The above story is reprinted from materials provided by Inderscience Publishers, via EurekAlert!, a service of AAAS. –Note: Materials may be edited for content and length. For further information, please contact the source cited above.—Journal Reference-Donna L. Harris, Ajit K. Mahapatra, Baron L. Jones, Govind Kannan. Efficacy of low-voltage AC for inactivating surface adherent Escherichia coli O157:H7 on beef. International Journal of Food Safety, Nutrition and Public Health, 2011; 4 (2/3/4): 214 DOI: 10.1504/IJFSNPH.2011.044624
SPECIAL NOTE—Remedy—So it would appear running A small cuurent of electricity to you foods may in fact assist in the prevention of pathogens that may infact creat a proble—so a good zapper or even a stunner set to low may in fact kill off any thing in the meats you are about to prepare
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Dietary DHA Linked to Male Fertility
ScienceDaily (Jan. 9, 2012) — Who knew that male fertility depends on sperm-cell architecture? A University of Illinois study reports that a certain omega-3 fatty acid is necessary to construct the arch that turns a round, immature sperm cell into a pointy-headed super swimmer with an extra long tail.—“Normal sperm cells contain an arc-like structure called the acrosome that is critical in fertilization because it houses, organizes, and concentrates a variety of enzymes that sperm use to penetrate an egg,” said Manabu Nakamura, a U of I associate professor of biochemical and molecular nutrition.—The study shows for the first time that docosahexaenoic acid (DHA) is essential in fusing the building blocks of the acrosome together. “Without DHA, this vital structure doesn’t form and sperm cells don’t work,” said Timothy Abbott, a doctoral student who co-authored the study.–Men concerned about their fertility may wonder what foods contain DHA. Marine fish, such as salmon or tuna, are excellent sources of this omega-3 fatty acid.[U1]–The scientists became intrigued with DHA’s role in creating healthy sperm when they experimented with “knockout” mice that lack a gene essential to its synthesis. “We looked at sperm count, shape, and motility, and tested the breeding success rate. The male mice that lacked DHA were basically infertile,” Nakamura said.—But when DHA was introduced into the mice’s diet, fertility was completely restored. “It was very striking. When we fed the mice DHA, all these abnormalities were prevented,” he said.—The scientists then used confocal laser scanning (3D) microscopy to look at thin slices of tissue in progressive stages of a sperm cell’s development. By labeling enzymes with fluorescence, they could track their location in a cell.–“We could see that the acrosome is constructed when small vesicles containing enzymes fuse together in an arc. But that fusion doesn’t happen without DHA,” he said.–In the absence of DHA, the vesicles are formed but they don’t come together to make the arch that is so important in sperm cell structure, he noted.–Nakamura finds the role this omega-3 fatty acid plays in membrane fusion particularly exciting. Because DHA is abundant in specific tissues, including the brain and the retina as well as the testes, the scientists believe their research findings could also impact research relating to brain function and vision.–“It’s logical to hypothesize that DHA is involved in vesicle fusion elsewhere in the body, and because the brain contains so much of it, we wonder if deficiencies could play a role, for example, in the development of dementia. Any communication between neurons in the brain involves vesicle fusion,” he noted.–The Illinois scientists will continue to study sperm; meanwhile, Nakamura has sent some of his DHA-deficient knockout mice to other laboratories where scientists are studying DHA function in the brain and the retina.–This work was supported in part by a CONACyT Mexico fellowship award.–Story Source-The above story is reprinted from materials provided by University of Illinois College of Agricultural, Consumer and Environmental Sciences. The original article was written by PhyllisPicklesimer. -Journal Reference-M. Roqueta-Rivera, T. L. Abbott, M. Sivaguru, R. A. Hess, M. T. Nakamura. Deficiency in the Omega-3 Fatty Acid Pathway Results in Failure of Acrosome Biogenesis in Mice. Biology of Reproduction, 2011; 85 (4): 721 DOI: 10.1095/biolreprod.110.089524
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Pesticide Illnesses and GM Soybeans-Ban on Aerial Spraying Demanded in Argentina
Coalition of doctors, health professionals and researchers demand ban on aerial spraying based on evidence documenting increase in pesticide-related illnesses since the introduction of GM soya. Dr Eva Sirinathsinghji——A network of 160 physicians, health workers and researchers in Argentina are demanding a ban on aerial spraying of pesticides based on increases in cancer and a range of pesticide-related illnesses since the introduction of genetically modified (GM) soybeans. These illnesses affect development, reproduction, the skin, as well as the immune, respiratory, neurological, and endocrine systems.–The network met in August 2010 [1] and again in 2011[2]. Following the 2010
meeting an important report was compiled linking agrochemical exposure to significant increase in illnesses including birth defects and cancers in and around areas where agrochemicals are used. Increasing illness parallels the introduction and spread of GM soybeans tolerant to Monsanto’s glyphosate-based herbicide, Roundup. —-The striking link between illness and pesticide use presented at the first meeting led the Network Of Physicians Of Drop-Sprayed Towns to propose a complete ban on all aerial sprayings of pesticides, a complete ban of pesticides of toxicological types Ia (defined as extremely hazardous) and Ib (highly hazardous), and a ban on other pesticide use within 1 kilometre of residential areas. They also questioned the current agro-industrial and transgenic production model, stating that other options for agro-ecological production should be promoted and developed by the State University.–The second meeting called on local and national government to ensure people’s right to good health instead of the rights of agribusiness and private proprietors that currently prevail, and repeated their call for a complete ban on aerial spraying.—The Argentinean communities’ fight for judicial, healthcare and governmental
recognition for health problems associated with pesticide use goes back 10 years. A summary compiled by non-governmental organisations and community residents is contained in the ‘Declaration of Caroya’. It documents [3] the “reduction in the average age and height” of residents in crop-sprayed towns “due to malnutrition”, and “decrease of the body’s natural defences.” In addition, “birth defects, mutagenesis, miscarriages, depression and suicide, disorders of the central nervous system and other neurological pathologies; disabilities, spina bifida, lupus, leukemia and other types of cancers; chloracne and other skin problems; asthma, allergies, and other respiratory and lung-related problems; male sterility and impotence; hormonal disruption and other hormonal disorders; diminished childhood development; prolonged febrile syndrome without focus; children’s increased vulnerability to pollutants; anemia, multiple sclerosis, cerebral ischemia, death..[U2]”—Alarming increases in birth defects, cancers, and other illnesses Public health officials have ignored ‘alarming notes coming from healthcare officials’ and consequently, very few epidemiological studies have been conducted. However the physicians (most of whom have been serving the same populations for over 25 years) have striking observational data that correlated with presentations and stories from people in the communities for a range of diseases associated with agrochemical exposure. They highlighted a systematic link between the unusual observations of recent years with this exposure. –The province of Chaco, where Dr Maria del Carmen Serveso heads a hospital intensive care unit, presented a devastating overview of rising illnesses in many towns. The illnesses include renal failure, birth defects in children of young mothers, cancers even in very young people, miscarriages, difficulty in becoming pregnant, respiratory problems and acute allergies; [U3]all linked by health teams to chemical contamination from agroindustrial farming recently imposed on the area replacing small-scale cotton farming and native forests. Respiratory illnesses were found to correlate with paraquat exposure.– One of the few epidemiological studies performed by Dr Gladys Trombotto, a geneticist from the University of Cordoba Maternity and Neonatal Unit, assessed 110 000 births over 10 years, and found a 2- and 3-fold increase in congenital and musculoskeletal defects respectively between 1971 and 2003 [4]. Child cancer data presented by Dr Otaño backed up what other physicians found in their own observations – incidence of birth defect rates increased significantly. He recorded 15.7 cases/100 000 in 2007 compared with the pre-existent level of 10.5 cases/100 000 in 1985—. The strong correlation of pesticide exposure with incidence of illness was exemplified by the observation made in 2005 by members of the Córdoba’s Ituzaingó neighbourhood, showing a higher rate of cancer in residents living closer to the fields that are sprayed. — The doctors suspect that the scale of damage to human health caused by agrochemicals is not fully recognized. The numbers of miscarriages may be higher than recorded levels, and other neurological and psychological problems are not currently being assessed. Preliminary and small-scale tests of children under a year of age suggest the existence of such illnesses in agrochemical use zones. [U4]
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Kidney Stones—How to Get rid of them!

Life Force Energy