Reply To: Scripts 2013

Forums Herbalist Scripts Scripts 2013 Reply To: Scripts 2013

#2665
AvatarEK
Keymaster

Scientists Uncover How Grapefruits Provide a Secret Weapon in Medical Drug Delivery
May 21, 2013 — Lipids (right panel first three tubes) derived from grapefruit. GNVs can efficiently deliver a variety of therapeutic agents, including DNA, RNA (DIR-GNVs), proteins and anti-cancer drugs (GNVs-Drugs) as demonstrated in this study.—Grapefruits have long been known for their health benefits, and the subtropical fruit may revolutionize how medical therapies like anti-cancer drugs are delivered to specific tumor cells.—University of Louisville researchers have uncovered how to create nanoparticles using natural lipids derived from grapefruit, and have discovered how to use them as drug delivery vehicles. UofL scientists Huang-Ge Zhang, D.V.M., Ph.D., Qilong Wang, Ph.D., and their team today (May 21, 2013), published their findings in Nature Communications.—-“These nanoparticles, which we’ve named grapefruit-derived nanovectors (GNVs), are derived from an edible plant, and we believe they are less toxic for patients, result in less biohazardous waste for the environment, and are much cheaper to produce at large scale than nanoparticles made from synthetic materials,” Zhang said.—-The researchers demonstrated that GNVs can transport various therapeutic agents, including anti-cancer drugs, DNA/RNA and proteins such as antibodies. Treatment of animals with GNVs seemed to cause less adverse effects than treatment with drugs encapsulated in synthetic lipids.—“Our GNVs can be modified to target specific cells — we can use them like missiles to carry a variety of therapeutic agents for the purpose of destroying diseased cells[F1],” he said. “Furthermore, we can do this at an affordable price.”—The therapeutic potential of grapefruit derived nanoparticles was further validated through a Phase 1 clinical trial for treatment of colon cancer patients. So far, researchers have observed no toxicity in the patients who orally took the anti-inflammatory agent curcumin encapsulated in grapefruit nanoparticles.——–The UofL scientists also plan to test whether this technology can be applied in the treatment of inflammation related autoimmune diseases like rheumatoid arthritis.—A Common Sense Approach—–Zhang said he began this research by considering how our ancestors selected food to eat. –“The fruits and vegetables we buy from the grocery today were passed down from generation to generation as favorable and nutritious for the human body. On the flip side, outcomes were not favorable for our ancestors who ate poisonous mushrooms, for example,” he said. “It made sense for us to consider eatable plants as a mechanism to create medical nanoparticles as a potential non-toxic therapeutic delivery vehicle.”[F2]—In addition to grapefruit, Zhang and his team analyzed the nanoparticles from tomatoes and grapes. Grapefruits were chosen for further exploration because a larger quantity of lipids can be derived from this fruit.–Story Source–The above story is reprinted from materials provided by University of Louisville.
************************************************************************
Herbals and Foods For Anti Cancer for Intestines
Water extracts of tree Hypericum sps. protect DNA from oxidative and alkylating damage and enhance DNA repair in colon cells.
Food Chem Toxicol. 2013 Jan;51:80-6
Authors: Ramos AA, Marques F, Fernandes-Ferreira M, Pereira-Wilson C
Abstract
Diet may induce colon carcinogenesis through oxidative or alkylating DNA damage. However, diet may also contain anticarcinogenic compounds that contribute to cancer prevention. DNA damage prevention and/or induction of repair are two important mechanisms involved in cancer chemoprevention by dietary compounds. Hypericum sps. are widely used in traditional medicine to prepare infusions due to their beneficial digestive and neurologic effects. In this study, we investigated the potential of water extracts from three Hypericum sps. and some of their main phenolic compounds to prevent and repair oxidative and alkylating DNA damage in colon cells. The results showed that water extracts of Hypericum perforatum-( St John’sWort), Hypericum androsaemum- (Tutsan), Hypericum undulatum- ( Wavy St John’s Wort ), quercetin and rutin have protective effect against oxidative DNA damage in HT29 cells. Protective effect was also observed against alkylating DNA damage induced by methyl-methanesulfonate ( MSM), except for H. androsaemum. With regard to alkylating damage repair H. perforatum, H. androsaemum and chlorogenic acid increased repair of alkylating DNA damage by base excision repair pathway. No effect was observed on nucleotide excision repair pathway. Antigenotoxic effects of Hypericum sps. may contribute to colon cancer prevention and the high amount of phenolic compounds present in Hypericum sps. play an important role in DNA protective effects.-PMID: 23000446 [PubMed – in process]
Remedy—Simplest way of utilizing any form of St John’s would be in tea form —after a meal—would increase protection and assist in the reversing of damage to colon area—in making the tea use 1 tablespoon in a 2-3 pint pot of distilled water—bring to a boil —rolling boil is good or when starting shut off stove after the boiling occurs this will reduce heat and allow to simmer—drank before bed with other herbs like lemon balm or Melissa or lavender may for some induce relaxation and sleep
*************************************************************************
Apigenin and its impact on gastrointestinal cancers.
Mol Nutr Food Res. 2013 Jan;57(1):126-44
Authors: Lefort ÉC, Blay J
Abstract
Apigenin (4′,5,7-trihydroxyflavone, 5,7-dihydroxy-2-(4-hydroxyphenyl)-4H-1-benzopyran-4-one) is a flavonoid found in many fruits, vegetables, and herbs, the most abundant sources being the leafy herb parsley and dried flowers of chamomile. Present in dietary sources as a glycoside, it is cleaved in the gastrointestinal lumen to be absorbed and distributed as apigenin itself. For this reason, the epithelium of the gastrointestinal tract is exposed to higher concentrations of apigenin than tissues at other locations. This would also be true for epithelial cancers of the gastrointestinal tract. We consider the evidence for actions of apigenin that might hinder the ability of gastrointestinal cancers to progress and spread. Apigenin has been shown to inhibit cell growth, sensitize cancer cells to elimination by apoptosis, and hinder the development of blood vessels to serve the growing tumor. It also has actions that alter the relationship of the cancer cells with their microenvironment. Apigenin is able to reduce cancer cell glucose uptake, inhibit remodeling of the extracellular matrix, inhibit cell adhesion molecules that participate in cancer progression, and oppose chemokine signaling pathways that direct the course of metastasis into other locations. As such, apigenin may provide some additional benefit beyond existing drugs in slowing the emergence of metastatic disease.–PMID: 23197449 [PubMed – in process]
***********************************************************************
Herbal Extract Rhodiola rosea Boosts Fruit Fly Lifespan by Nearly 25 Percent
June 18, 2013 — The herbal extract of a yellow-flowered mountain plant long used for stress relief was found to increase the lifespan of fruit fly populations by an average of 24 percent, according to UC Irvine researchers.—But it’s how Rhodiola rosea, also known as golden root, did this that grabbed the attention of study leaders Mahtab Jafari and Sam Schriner. They discovered that Rhodiola works in a manner completely unrelated to dietary restriction and affects different molecular pathways.—This is significant, said Jafari, associate professor of pharmaceutical sciences, because dietary restriction is considered the most robust method of improving lifespan in laboratory animals, and scientists have been scrambling to identify compounds that can mimic its effects.—“We found that Rhodiola actually increases lifespan on top of that of dietary restriction,” Jafari said. “It demonstrates that Rhodiola can act even in individuals who are already long-lived and healthy. This is quite unlike resveratrol, which appears to only act in overfed or unhealthy individuals.”—The researchers proved this by putting flies on a calorie-restricted diet. It has been shown that flies live longer when the amount of yeast they consume is decreased. Jafari and Schriner expected that if Rhodiola functioned in the same manner as dietary restriction, it would not work in these flies. But it did. They also tested Rhodiola in flies in which the molecular pathways of dietary restriction had been genetically inactivated. It still worked.[F3]—-Not only did Rhodiola improve lifespan an average of 24 percent in both sexes and multiple strains of flies, but it also delayed the loss of physical performance in flies as they aged and even extended the lives of old flies. Jafari’s group previously had shown that the extract decreased the natural production of reactive oxygen species molecules in the fly mitochondria and protected both flies and cultured human cells against oxidative stress.—Jafari and Schriner, an assistant project scientist in Jafari’s laboratory, are not claiming that Rhodiola supplements will enable humans to live longer, but their discovery is enhancing scientific understanding of how supplements believed to promote longevity actually work in the body.–Rhodiola has already shown possible health benefits in humans, such as decreasing fatigue, anxiety and depression; boosting mood, memory and stamina; and preventing altitude sickness. Grown in cold climates at high elevations, the herb has been used for centuries by Scandinavians and Russians to reduce stress. It’s also thought to have antioxidant properties.—Jafari’s research group is currently exploring the plant’s potential to kill cancer cells, improve Alzheimer’s disease and help stem cells grow.—Rhodiola is readily available online and in health food stores. Jafari, though, has analyzed several commercial products and found them to not contain sufficient amounts of the reputed active compounds — such as rosavin and salidroside — that characterize high-quality products.—Story Source-The above story is reprinted from materials provided by University of California – Irvine. –Journal Reference—Samuel E. Schriner, Kevin Lee, Stephanie Truong, Kathyrn T. Salvadora, Steven Maler, Alexander Nam, Thomas Lee, Mahtab Jafari. Extension of Drosophila Lifespan by Rhodiola rosea through a Mechanism Independent from Dietary Restriction. PLoS ONE, 2013; 8 (5): e63886 DOI: 10.1371/journal.pone.0063886
***************************************************************************
Testosterone Therapy May Help Improve Pain in Men With Low Testosterone
June 17, 2013 — Testosterone therapy is associated with decreased pain perception in men with low testosterone levels related to opioid (narcotic) pain relievers (analgesics), a new study finds. —The results were presented Monday at The Endocrine Society’s 95th Annual Meeting in San Francisco.—“In this study, we attempted to determine whether testosterone replacement improves pain perception and tolerance, and quality of life in men with low testosterone levels due to narcotic analgesics,” said the study’s lead author Shehzad Basaria, MD, Medical Director, Section of Men’s Health, Aging, & Metabolism at Brigham & Women’s Hospital, Harvard Medical School, in Boston, MA. “We found that testosterone administration in these men was associated with a greater reduction in several measures of pain sensitivity during laboratory pain testing compared with men who were on placebo.”—Opioids belong to a class of pain-reducing drugs that are used to relieve chronic pain from injuries, surgery and cancer treatment. These drugs include morphine, codeine, fentanyl and oxycodone, and are among the most frequently prescribed medications in the United States today.–In addition to being highly addictive, opioid use is associated with a number of side effects, including suppression of the hypothalamic-pituitary-gonadal axis in both women and men, resulting in decreased testosterone production. Low testosterone, in turn, can result in sexual dysfunction, decreased muscle mass, increased fat mass and decreased quality of life.–Previous animal research has demonstrated that castration of rodents is associated with increased pain perception while testosterone replacement reduces pain perception, suggesting an analgesic effect of this sex steroid. Whether these beneficial effects can be replicated in humans, however, remained unclear.—In this study, investigators found that, compared to placebo, testosterone therapy significantly improved pain perception and tolerance during laboratory pain testing. Testosterone therapy also improved some aspects of quality of life.—“If larger studies confirm these findings, testosterone therapy in this patient population may be beneficial in improving pain perception,” Basaria said.—The study included 84 men ages 18-64 years old with opioid-induced testosterone deficiency. Their average age was 49 years. Of this group, 65 participants completed the study. Investigators randomly assigned participants to receive either testosterone gel, applied to the skin, or placebo, for 14 weeks. Thirty-six men received the testosterone gel, and 29 received a placebo.—At the beginning of the study, and then again at 14 weeks, the investigators assessed pain measures and quality-of-life parameters.—Solvay (now Abbott) Pharmaceuticals, Inc. funded the study.
*******************************************************************
 
Statin Myths
#1. Biological Mechanisms Behind Statin Side Effects: Duane Graveline, the M.D. who goes by the nickname “Spacedoc” has done everything he can to inform the public of the dangers of statin drugs. Dr. Graveline is a former NASA astronaut and Air Force flight surgeon. His reasons for taking on the challenge of debunking statins are very personal, because he developed global transient amnesia while taking Lipitor, an unnerving experience that deprives you of all memory of events in your life looking back several decades.
Since then, he has conducted extensive research on the biological mechanisms behind the diverse severe side effects of statin drugs. The link above is but one branch of a vast network of frightening yet informative information about the potentially devastating long-term effects of statin therapy.
#2. Statins and Nerve Damage: The People’s Pharmacy is a wonderful forum that allows patients to share their experience. This is one of many examples of adverse side effects of statin drugs available on that site.
#3. Statins and Pregnancy: Like thalidomide, statin drugs are a class X drug with regard to pregnancy, meaning they are contraindicated and should NOT be taken by pregnant women. They can cause significant damage to the nervous system of a developing embryo.
#4. New York Times Exposes Potential for Misdiagnosis Caused by Statin Use: This article points out some of the severe side effects statins can cause, and illustrates with a poignant story about a woman from Kansas. She had been taking a statin for years to reduce her cholesterol. Over that same time period, she experienced chronic muscle pain which neither she nor her doctor attributed to the statin therapy. It even led to a useless shoulder operation.—Her problem eventually escalated into skin lesions caused by a reaction to toxic protein by-products released by her disintegrating muscles. She was given an antifungal to treat the skin lesions, another misdiagnosis. But the antifungal interacted with the statins to further increase the severity of her muscle disorders. Three months later, she could barely stand, and her pulmonary muscles were so weak she couldn’t breathe. She died shortly thereafter.
#5. The International Network of Cholesterol Skeptics (THINCS): Dr. Uffe Ravnskov has campaigned tirelessly to fight the myth that high cholesterol is damaging to your health. This web page contains valuable information about all the ways in which cholesterol keeps you healthy.
#6. The ASEPSIS Trial: The statin industry continues to claim that statins protect against sepsis because of several retrospective studies that show that those who take statins have less risk than those who don’t.—What these studies are proving is that cholesterol protects from sepsis. The media keep saying that what is needed is a double-blind placebo controlled study, but they already have one they could talk about. It’s just that you can’t find out anything about it except that it was completed in January, 2008. You can read my take on statins and sepsis here.
#7. Statins Increase Pneumonia Risk: Just as for sepsis, the statin industry likes to claim that statins improve your chances against pneumonia. But a double-blind placebo controlled study proved them wrong. The risk for pneumonia that required hospitalization was increased by 61 percent in the statin group compared to the controls.
#8. WebMD Article on Muscle Pain and Weakness: This relatively benign article on WebMD provoked a firestorm of responses; each comment tells the story of another tragedy unfolding. To understand the biological mechanism behind the process by which statin drugs destroy muscles, see this link.—- Here’s a typical comment from that site—-“I was prescribed Crestor 20mg 2 weeks ago with cholesterol level 7.6. First time on any medication. After approx. 4 days I started to experience severe muscle pain, thigh, buttocks, arms, legs to the extent that I can hardly get out of bed in the morning. Have been back to Dr. who advised stopping tablets. Have been off them for 3 days, very little difference. I am hoping these pains will go away soon. I will never take a statin drug again – would rather take the healthy option, diet and exercise and take the risk. Have never felt so bad. Usually very healthy, fit person.”
#9. Statins and Heart Failure: Dr. Peter Langsjoen believes that statin drugs greatly increase your risk of heart failure. I have argued why this might be true here.
#10. JUPITER Trial and Diabetes: The JUPITER trial, which was terminated prematurely after less than two years, was widely heralded for showing that statins reduce the risk of heart attacks in the short term for people with high levels of C-reactive protein but without high cholesterol.—-However, little note was made of the fact that the JUPITER trial also showed a 25 percent increased risk to new-onset diabetes in the treatment group. Since diabetes is a strong risk factor for heart disease, one wonders how the trial would have turned out if it had been allowed to run to completion.
#11. High Cholesterol and Alzheimer’s: This is the article that inspired the Newsweek article, #12 below, with the lead story that statins “protect from” Alzheimer’s — which is the exact opposite of the truth about statins and Alzheimer’s.—-The only relationship between high cholesterol and Alzheimer’s the authors could find was if they looked back 30 years. What they’re not saying is that, in the intervening years, cholesterol levels fell for those who later developed Alzheimer’s. While no one has said exactly why their levels might have fallen, statin drugs are a good bet.
#12. Glowing Newsweek Article on Statins:This article illustrates how thoroughly the statin industry has succeeded in brainwashing the media into believing that black is white. The lead story is that statins protect against Alzheimer’s. If you have read my essay on statins and Alzheimer’s, you will think otherwise.
 
TOP
 
 
**************************************************************************
TOP A
HOME
 
 
Script of the Show July 13 2013
 
PICA—EATING DISORDER FROM MINERAL DEFICIENCY
Formula-feeding linked to metabolic stress and increased risk of later disease
Zanthoxylum Avicennae-Prickly Ash Leaves or Seeds
Getting Enough Sleep Could Help Prevent Type 2 Diabetes
Timing of Calcium and Vitamin D Supplementation May Affect How Bone Adapts to Exercise
Aspirin May Fight Cancer by Slowing DNA Damage
Minerals
**************************************************************************
PICA—EATING DISORDER FROM MINERAL DEFICIENCY
A person afflicted with pica has a persistent craving for a substance that is not commonly considered to be food. The substances that are craved and ingested tend to vary with the person’s age. Children with pica may eat glue, animal droppings, sand, insects, leaves, or gravel. Adolescents and adults may consume clay, laundry detergent, or soil.
Although the cravings associated with pica may be related to vitamin or mineral deficiencies, ingesting non-foods can cause serious medical complications such as intestinal obstruction, intestinal perforation, infections, or lead poisoning
Pica (/ˈpaɪkə/ PY-kə) is characterized by an appetite for substances largely non-nutritive, such as clay, chalk, dirt, or sand.[1] For these actions to be considered pica, they must persist for more than one month at an age where eating such objects is considered developmentally inappropriate. There are different variations of pica, as it can be from a cultural tradition, acquired taste or a neurological mechanism such as an iron deficiency, or chemical imbalance. It can lead to intoxication in children which can result in an impairment in both physical and mental development. In addition, it can also lead to surgical emergencies due to an intestinal obstruction as well as more subtle symptoms such as nutritional deficiencies and parasitosis.[2] Pica has been linked to mental disorders and they often have psychotic comorbidity. Stressors such as maternal deprivation, family issues, parental neglect, pregnancy, poverty, and a disorganized family structure are strongly linked to pica.
Signs and symptoms
Chalky stone composed of kaolinite with traces of quartz ingested by a person with pica.
Pica is the consumption of substances with no significant nutritive value such as earth or ice. Subtypes are characterized by the substance eaten for example:
Amylophagia (consumption of starch)
Coprophagy (consumption of feces)
Geophagy (consumption of soil, clay, or chalk)
Hyalophagia (consumption of glass)
Consumption of dust or sand has been reported among iron-deficient patients.
Lithophagia (a subset of geophagia, consumption of pebbles or rocks)[5]
Mucophagia (consumption of mucus)
Odowa (soft stones eaten by pregnant women in Kenya)[6]
Consumption of paint.
Pagophagia (pathological consumption of ice)
Self-cannibalism (rare condition where body parts may be consumed; see also Lesch-Nyhan syndrome)
Trichophagia (consumption of hair or wool)
Urophagia (consumption of urine)
Xylophagia (consumption of wood or paper)
This pattern of eating should last at least one month to fit the diagnosis of pica.
Diagnosis
There is no single test that confirms pica. However, because pica can occur in people who have lower than normal nutrient levels and poor nutrition (malnutrition), the health care provider should test blood levels of iron and zinc. Hemoglobin can also be checked to test for anemia. Lead levels should always be checked in children who may have eaten paint or objects covered in lead-paint dust. The health care provider should test for infection if the person has been eating contaminated soil or animal waste
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~
 
People who consume animal products usually do not get afflicted with this unless there are other imbalances —this is usually those on vegetarian Diet —will consume things that are unusal—this is again a sign of starvation—lack of nutrients or minerals—so one way to avoid this is to ge adequate nutrients—if not in Diet then in supplementation—or in the consumption of veges or fruits that are fermented to yield a higher mineral and nutrient uptake due to the fibre being broken down releasing the vitality of it’scompliment—People who crave clay usually are trying to fulfill mineral loss—In some cases a Iron ( gotten from meats) and zinc ( animal Protein) restored to the diet will sometimes restore a person to normal—those who consume ice ( pagophagia)—are usually iron deficient—In some cases cannibalism has ben recorded as well or different animals from cats and dogs to mice ( while they are living)—The asian Diet is low in calcium and as a result leach the bones with vinegar to obtain there calcium levels—Magnesium deficiency will result in higher salt use —and Potassium deficiency will be a result of diuretic use –so if you utilize diuretics then make sure you use a dandelion tea it puts potassium back into the system—Minerals Like chromium if there is a deficiency the cravings will be for a pop ( soft drink)and high consumption of grain related carbs—
Cannibalism —would be an extreme case of pica as result of high mineral deficiency
 
Animals can show signs of this by consuming bones –wood-and cattle will sometimes gnaw on each others horns as well especially if they have a phosphorus need—
History
The term pica originates from the Latin word for magpie, a bird that is reputed for its unusual eating behaviors where they are known to eat almost anything. In 13th century Latin work, pica was referenced by the Greeks and Romans; however, it was not addressed in medical texts until 1563. In the southern United States in the 1800s, geophagia was a common practice among the slave population.[Geophagia is a form of pica in which the person consumes earthly substances such as clay, and is particularly prevalent to augment a mineral-deficient diet. Research on eating disorders in the 16th century to the 20th century suggests that during that time in history, pica was regarded more as a symptom of other disorders rather than its own specific disorder. Even today, what can be classified as pica behavior is a normative practice in some cultures as part of magical beliefs, healing methods, or religious ceremonies.
 
*************************************************************************
Formula-feeding linked to metabolic stress and increased risk of later disease
June 5th, 2013 in Chemistry / Biochemistry
New evidence from research suggests that infants fed formula, rather than breast milk, experience metabolic stress that could play a part in the long-recognized link between formula-feeding and an increased risk of obesity, type 2 diabetes and other conditions in adult life. —New evidence from research suggests that infants fed formula, rather than breast milk, experience metabolic stress that could play a part in the long-recognized link between formula-feeding and an increased risk of obesity, type 2 diabetes and other conditions in adult life. Credit: iStockphoto/Thinkstock—New evidence from research suggests that infants fed formula, rather than breast milk, experience metabolic stress that could play a part in the long-recognized link between formula-feeding and an increased risk of obesity, type 2 diabetes and other conditions in adult life.[F4] The study appears in ACS’ Journal of Proteome Research.—Carolyn Slupsky and colleagues explain that past research showed a link between formula-feeding and a higher risk for chronic diseases later in life. Gaps exist, however, in the scientific understanding of the basis for that link. The scientists turned to rhesus monkeys, stand-ins for human infants in such research, that were formula-fed or breast-fed for data to fill those gaps.
Their analysis of the monkeys’ urine, blood and stool samples identified key differences between formula-fed and breast-fed individuals. It also produced hints that reducing the protein content of infant formula might be beneficial in reducing the metabolic stress in formula-fed infants[F5]. “Our findings support the contention that infant feeding practice profoundly influences metabolism in developing infants and may be the link between early feeding and the development of metabolic disease later in life,” the study states.— The article is titled “Early Diet Impacts Infant Rhesus Gut Microbiome, Immunity, and Metabolism.”–More information: Early Diet Impacts Infant Rhesus Gut Microbiome, Immunity, and Metabolism, J. Proteome Res., Article ASAP. DOI: 10.1021/pr4001702—- Abstract
Epidemiological research has indicated a relationship between infant formula feeding and increased risk of chronic diseases later in life including obesity, type-2 diabetes, and cardiovascular disease. The present study used an infant rhesus monkey model to compare the comprehensive metabolic implications of formula- and breast-feeding practices using NMR spectroscopy to characterize metabolite fingerprints from urine and serum, in combination with anthropometric measurements, fecal microbial profiling, and cytokine measurements. Here we show that formula-fed infants are larger than their breast-fed counterparts and have a different gut microbiome that includes higher levels of bacteria from the Ruminococcus genus and lower levels of bacteria from the [F6]Lactobacillus genus. In addition, formula-fed infants have higher serum insulin coupled with higher amino acid levels, while amino acid degradation products were higher in breast-fed infants. Increases in serum and urine galactose and urine galactitol were observed in the second month of life in formula-fed infants, along with higher levels of TNFα, IFN-γ, IL-1β, IL-4, and other cytokines and growth factors at week 4. These results demonstrate that metabolic and gut microbiome development of formula-fed infants is different from breast-fed infants and that the choice of infant feeding may hold future health consequences.—-Provided by American Chemical Society
**********************************************************************
Zanthoxylum Avicennae-Prickly Ash Leaves or Seeds
[Study on the chemical components, antimicrobial and antitumor activities of the essential oil from the leaves of Zanthoxylum avicennae].
Zhong Yao Cai. 2012 Aug;35(8):1263-7
Authors: Zhang DS, Zhong QX, Song XM, Liu WJ, Wang J, Zhang QY
Abstract
OBJECTIVE: To study the chemical constituents, antimicrobial activity and antitumor activity of the essential oil from Zanthoxylum avicennae.
METHODS: The essential oil from the leaves of Zanthoxylum avicennae was extracted by steam distillation. The components of the essential oil were separated and identified by GC-MS.–RESULTS: 72 components were identified and accounted for 98.15% of the all peak area. The essential oil exhibited strong antitumor activity against K-562 human tumor cell lines with IC50 of 1.76 microg/mL. It also exhibited moderate antimicrobial activity against three bacteria.—CONCLUSION: The essential oil of Zanthoxylum avicennae contains various active constituents. This result provides scientific reference for the pharmacological further research of Zanthoxylum avicennae.–PMID: 23320360 [PubMed – indexed for MEDLINE]
*************************************************************************
Getting Enough Sleep Could Help Prevent Type 2 Diabetes
June 18, 2013 — Men who lose sleep during the work week may be able to lower their risk of developing Type 2 diabetes by getting more hours of sleep, according to Los Angeles Biomedical Research Institute (LA BioMed) research findings presented today at The Endocrine Society’s 95th Annual Meeting in San Francisco.—The study by Peter Liu, MD, PhD, an LA BioMed lead researcher, found that insulin sensitivity, the body’s ability to clear glucose (blood sugar) from the bloodstream, significantly improved after three nights of “catch-up sleep” on the weekend in men with long-term, weekday sleep restrictions.—“We all know we need to get adequate sleep, but that is often impossible because of work demands and busy lifestyles,” said Dr. Liu. “Our study found extending the hours of sleep can improve the body’s use of insulin, thereby reducing the risk of Type 2 diabetes in adult men. –Insulin is a hormone that regulates a person’s blood sugar level. The body of a patient with Type 2 diabetes cannot effectively use the insulin it produces, or it becomes “resistant” to insulin. Retaining the body’s sensitivity to insulin reduces the risk of developing Type 2 diabetes, a chronic illness that is the seventh leading cause of death in the U.S.–Other research had demonstrated the harmful effects of experimental sleep restriction on insulin sensitivity in healthy, normal sleepers. The new study provides information about people who lose sleep during the week — often because of jobs and busy lifestyles — but “catch up” on their sleep on the weekends.-“The good news is that by extending the hours they sleep, adult men — who over a long period of time do not get enough sleep during the working week — can still improve their insulin sensitivity,” Liu said.-Liu and researchers from the University of Sydney in Australia studied 19 non-diabetic men, with an average age of 28.6 years, who for six months or longer (average, 5.1 years) self-reported inadequate sleep during the workweek. On average, the men received only 6.2 hours of sleep each work night. But they regularly caught up on their sleep on the weekends, sleeping an extra 37.4 percent, or 2.3 hours, per night, the authors reported. Their reported sleep times were verified by actigraphy, in which each man wore a small device on his wrist that monitored sleep-wake cycles.—The men spent three nights in a sleep lab on each of two separate weekends. The researchers randomly assigned the men to two of three sleep conditions: (1) 10 hours of sleep, (2) six hours of sleep or (3) 10 hours in bed, in which noises during deep sleep aroused them into shallow sleep without waking them. The six hours of sleep tested persistent sleep restriction.—On the fourth morning, the research staff drew the men’s blood to measure their blood sugar and insulin levels to calculate insulin sensitivity. Each individual had the same food intake during the study visits, so that diet would not influence the results, Liu said.—When the men slept 10 hours a night on each of three nights of catch-up sleep, their insulin sensitivity was much better than when they had persistent sleep restriction, the scientists found. Their insulin resistance test score also improved (decreased) with sleep extension.—Story Source-The above story is reprinted from materials provided by Los Angeles Biomedical Research Institute at Harbor-UCLA Medical Center (LA BioMed).
*********************************************************************
Timing of Calcium and Vitamin D Supplementation May Affect How Bone Adapts to Exercise
June 18, 2013 — Taking calcium and vitamin D before exercise may influence how bones adapt to exercise, according to a new study. The results will be presented on Tuesday at The Endocrine Society’s 95th Annual Meeting in San Francisco.—“The timing of calcium supplementation, and not just the amount of supplementation, may be an important factor in how the skeleton adapts to exercise training,” said study lead author Vanessa D. Sherk, PhD, postdoctoral research fellow at the University of Colorado Anschutz Medical Campus. “Further research, however, is needed to determine whether the timing of calcium supplementation affects the skeletal adaptations to exercise training.”—Previous research has shown that a year of intense training is associated with substantial decreases in bone mineral density among competitive road cyclists. Experts believe that this kind of exercise-induced bone loss could be related to the loss of calcium during exercise. As blood calcium levels drop, the parathyroid gland produces excess parathyroid hormone, which can mobilize calcium from the skeleton.—In this study, investigators found that an exercise-induced decrease in blood calcium occurred whether calcium supplements were taken before or after exercising. Pre-exercise supplementation, however, resulted in less of a decrease. Although not statistically significant, parathyroid hormone levels increased slightly less among cyclists who took calcium before exercising.—“These findings are relevant to individuals who engage in vigorous exercise and may lose a substantial amount of calcium through sweating,” Sherk said. “Taking calcium before exercise may help keep blood levels more stable during exercise, compared to taking the supplement afterwards, but we do not yet know the long-term effects of this on bone density.”—The timing of calcium supplementation did not cause a difference in blood levels of a compound that is a biological indicator of bone loss. Both the before- and after-exercise groups exhibited 50-percent increases in the level of this compound, called CTX, for collagen type-1 C-telopeptide.—Study participants included 52 men aged 18 to 45 years. Investigators randomly assigned participants to take 1,000 milligrams of calcium and 1,000 international units of vitamin D either 30 minutes before or one hour after exercise. The exercise comprised a simulated 35-kilometer time trial, and participants wore skin patches to absorb sweat.—Investigators measured blood levels of calcium and parathyroid hormone before and immediately after exercise. They also measured CTX before and 30 minutes after exercise. They used pre- and post-body weight, adjusted for fluid intake, combined with the calcium measured in the sweat from the skin patches, to estimate the amount of calcium lost through the skin during exercise.–The National Institutes of Health and the American College of Sports Medicine funded the study.—Story Source-The above story is reprinted from materials provided by Endocrine Society, via Newswise.
Special Note— if anyone was going to try this then you would not use 1000mgs of Calcium but rather 300mmgs with magnesium 150mgs—or you would utilize a creatine with the calcium or even a carbonated calcium —this would insure absorption and usage —there test were flawed because there deliver system would not work due to the overloading of calcium—even comboing it with gelatin as well—then you would be chelating the minerals into the system
*************************************************************************
Aspirin May Fight Cancer by Slowing DNA Damage
June 18, 2013 — Aspirin is known to lower risk for some cancers, and a new study led by a UC San Francisco scientist points to a possible explanation, with the discovery that aspirin slows the accumulation of DNA mutations in abnormal cells in at least one pre-cancerous condition.—-“Aspirin and other non-steroidal anti-inflammatory drugs, which are commonly available and cost-effective medications, may exert cancer-preventing effects by lowering mutation rates,” said Carlo Maley, PhD, a member of the UCSF Helen Diller Family Comprehensive Cancer Center, and an expert on how cancers evolve in the body over time. In the study, published June 13 in the online journal PLOS Genetics, Maley, working with gastroenterologist and geneticist Brian Reid, MD, PhD, of the Fred Hutchinson Cancer Research Center, analyzed biopsy samples from 13 patients with a pre-cancerous condition called Barrett’s esophagus who were tracked for six to 19 years. In an “observational crossover” study design, some patients started out taking daily aspirin for several years, and then stopped, while others started taking aspirin for the first time during observation. The goal was to track the rate of mutations in tissues sampled at different times.—The researchers found that biopsies taken while patients were on an aspirin had on average accumulated new mutations about 10 times more slowly than biopsies obtained during years when patients were not taking aspirin.[F7]—“This is the first study to measure genome-wide mutation rates of a pre-malignant tissue within patients for more than a decade, and the first to evaluate how aspirin affects those rates,” Maley said.—Gender and ethnic distribution of study patients reflected the known demographics of esophageal cancer, which predominantly affects, white, middle-aged and elderly men, he said. Barrett’s esophagus only occasionally progresses to esophageal cancer.—Cancers are known to accumulate mutations over time much more rapidly than normal tissue, and different mutations arise in different groups of cells within the same tumor. The acquisition of key mutations ultimately allows tumor cells to grow out of control, and diversity within a tumor may foster drug resistance, a phenomenon that is a major focus of Maley’s research.—-Maley plans to test a hypothesis that may explain the results — that aspirin’s lowering of mutation rates is due to the drug’s effect of reducing inflammation. Inflammation, a response of the immune system, in recent years has been recognized as a hallmark of cancer. Maley said that less inflammation may result in less production within pre-cancerous tissue of oxidants known to damage DNA, and may dampen growth-stimulating signaling.—For the duration of the study, the rate of accumulation of mutations measured in the biopsied tissue between time points was slow, even when patients were not taking aspirin, with the exception of one patient. While mutations accumulated at a steady rate, the vast majority of mutations arose before the abnormal tissue was first detected in the clinic, the researchers concluded.—These findings are consistent with the fact that although Barrett’s esophagus is a significant risk factor for esophageal cancer, the vast majority of cases do not progress to cancer, Maley said.
In the one patient who later went on to develop cancer, a population of cellular “clones” with a great number of mutations emerged shortly before he started taking aspirin.—More studies are needed to further explore the link between non-steroidal anti-inflammatory drugs, mutation rates and the development of invasive cancer, Maley said. He plans to continue studying Barrett’s esophagus and esophageal cancer, and to expand his research to investigate lung cancer.–Rather than aiming to kill the most tumor cells, it may be better to try to halt or slow growth and mutation. Current drug treatments for cancer may in many cases hasten the emergence of cancer that is more difficult to eradicate, according to Maley. The capability to mutate frequently allows tumors to become resistant to drug treatment, he said. A better-adapted mutant can begin to spin off a population of genetic clones that survives and grows, while poorly adapted tumor cells die off.[F8]–Additional authors from the Fred Hutchinson Cancer Research Center include Xiaohong Li, PhD, Carissa Sanchez, PhD, Patricia Galipeau, PhD, Thomas Paulson, PhD, Patricia Blount, PhD, Thomas Vaughan, PhD, and Cassandra Sather, PhD. Amitabh Srivastava, MD, and Robert Odze, MD, from Harvard University; Rumen Kostadinov, PhD, from the University of Pennsylvania; and Mary Kuhner, PhD from the University of Washington also were members of the research team and authors of the study.—Story Source-The above story is reprinted from materials provided by University of California, San Francisco (UCSF). The original article was written by Jeffrey Norris. –Journal Reference-Rumen L. Kostadinov, Mary K. Kuhner, Xiaohong Li, Carissa A. Sanchez, Patricia C. Galipeau, Thomas G. Paulson, Cassandra L. Sather, Amitabh Srivastava, Robert D. Odze, Patricia L. Blount, Thomas L. Vaughan, Brian J. Reid, Carlo C. Maley. NSAIDs Modulate Clonal Evolution in Barrett’s Esophagus. PLoS Genetics, 2013; 9 (6): e1003553 DOI: 10.1371/journal.pgen.1003553
***********************************************************************
Minerals—-
% Average Human Body Content
(70 kg person)
Macrominerals (Essential)
Oxygen 65.4 43 kg
Carbon 18.2 12 kg
Hydrogen 9.5 6.3 kg
Nitrogen 3 2 kg
Calcium 1.67 1.1 kg
Phosphorus 1.14 750 grams
Potassium 0.342 225 grams
Sulfur 0.228 150 grams
Chloride 0.152 100 grams
Sodium 0.137 90 grams
Magnesium 0.053 35 grams
Silicon 0.046 30 grams
 
 
Microminerals Essentiality
Iron 0.00638 4,200 mg Essential
Fluoride 0.00395 2,600 mg Possibly Essential
Zinc 0.00365 2,400 mg Essential
Rubidium 0.000532 350 mg Probably Essential
Strontium 0.000486 320 mg Possibly Essential
Lead 0.000243 160 mg Toxic
Copper 0.000137 90 mg Essential
Aluminium 0.0000988 65 mg Toxic
Cadmium 0.0000608 40 mg Toxic
Barium 0.0000344 22 mg Possibly Essential
Cobalt 0.0000304 20 mg Essential
Vanadium 0.0000304 20 mg Probably Essential
Iodine 0.0000228 15 mg Essential
Tin 0.0000228 15 mg Possibly Essential
Selenium 0.0000228 15 mg Essential
Arsenic 0.0000228 15 mg Possibly Essential
Manganese 0.0000198 13 mg Essential
Mercury 0.0000190 12.5 mg Toxic
Nickel 0.0000167 11 mg Possibly Essential
Molybdenum 0.0000122 8 mg Essential
Chromium 0.0000091 6 mg Essential
Bismuth 0.0000045 3 mg Probably Toxic
Lithium 0.0000038 2.5 mg Probably Essential
Uranium 90 mcg Toxic
Boron Probably Essential
Germanium Probably Essential
Bromine Possibly Essential
Gold Probably Non Essential
Silver Probably Non Essential
Beryllium Probably Toxic
Antimony Slightly Toxic
Thallium Toxic
 
Forms of Minerals (Generally)
 
Mineral Form Mineral Bonded With Comments
Inorganic Salts: Phosphates-Sulfates-Chlorides Important for Electrolyte balance.
 
Organic Acid Salts-Ascorbates-Acetates-Citrates Natural and more effectively absorbed by the Digestive System than Inorganic Salts
.
Amino Acid Chelates-Amino Acids The best form of absorption of Minerals. Specific Minerals chelate best with specific Amino Acids.
 
 
TOP A
 
**************************************************************************
 
[F1]Methods such as these can be done with almost any fat ( lipid) —and directing them would be what you would sompile in the lipid ( fat)
[F2]Creating a Nanoparticle delivery or liposomal delivery to get things into the cells would be a good concept—with cells salt is one way and mixing a salt with some of these herbals may in fact potentiate them better
[F3]Rhodiola Rosea acts as an Antioxidant and Life Extending even on a restricted diet
[F4]This would have to do with the soy proteins in the formulas which are low on the protein scale for uptake and digestibility—the strain and stress on the digestive system would cause the colon to become corrupt with bad bacteria
[F5]Would be better to give a child a higher profile of protein then the soy and rice in the formulas—they are low on the bv and are excessively difficult t break down even for an adult so the excretment would be a direct result of the proteins passing through thedigestive system and causing damage topancreas-liver and stomach
[F6]Gut flora affects brain functionality in the utmost way—so damaging this will effect emotional and mental up the way
[F7]Make sure if you use an aspirin it does not have yellow coluring agents-synthetic flavours—natural or artificial flavours or polymers—if necessary buy a a supply for animals will be cleaner—and mix this with magnesium and choline as well to insure effect and safety
[F8]This is why today you need more then one remedy and a lifestyle change due to the genetics of cancer and capacity to mutate and adapt—this is how morgellons works as well they are finding more and more that cancer tumours are carrying crystal like components and by offsetting the inflammation this can allow the body to remove the nano particles out through antioxidant free radical scavenging or by chelation
 
 
******************************************************************************************************************************************************
TOP B
HOME
 
 
Script of the Show July 20 2013
Curcuminoids and resveratrol as anti-Alzheimer agents.
RUSSIA Suggests that WAR May Be Necessary To Stop MONSANTO
Over 30 Million Bees Found Dead In Elmwood Canada
Monsanto to pull the plug on GM lobbying and activities in Europe
 
 
*************************************************************************
Curcuminoids and resveratrol as anti-Alzheimer agents.
Taiwan J Obstet Gynecol. 2012 Dec;51(4):515-25
Authors: Villaflores OB, Chen YJ, Chen CP, Yeh JM, Wu TY
Abstract
Alzheimer disease (AD) is by far the most common cause of dementia globally. This neurodegenerative disorder of the brain is chronic and progressive, characterized clinically by the deterioration in the key symptoms of behavioral and cognitive abilities. Treatment options for this disease currently are limited. Deposition of amyloid-β and tau hyperphosphorylation are cardinal pathologic features of AD that lead to the formation of neuronal plaques and neurofibrillary tangles, respectively. In addition to mounting research on herbal compounds for the treatment of AD, curcuminoids and resveratrol appear to be beneficial as anti-AD agents. Curcuminoids (curcumin and demethoxycurcumin) and resveratrol possess unique properties that make them especially worthy of further studies. This review article revisits and presents the current research done on the potential of the curcuminoids curcumin and demethoxycurcumin and the polyphenolic compound resveratrol as anti-AD compounds.—PMID: 23276553 [PubMed – indexed for MEDLINE]
Recipe
Let’s Make our own version of this—Lets take some wine – 1 .5 cups-some tumeric -1/4 cup and pepper ( black) 1/8 cup and some deep bluish purple raisons -1/2 cup and 2 cups of water—put everything in either a high speed blender or a pressure cooker— and proceed to process—high speed blender 15 minutes at high speed—pressure cooker 1 hour —-when done stop devices ( either one) allow pressure cooker to cool down before opening—take either extract from either device and pour through a filter and then wring out what is in the filter ( sheet—handkerchief-pillow case) then take the filtered stuff and put in pot and simmer down to about ½-1/4 above the bottom—when cooled off the add to container and use ¼ tsp increments several times a day
Will get the benefits for the liver-blood-brain—circulation—antioxidants-anti fungal –antibacterial-immune enhancing—
*************************************************************************
 
RUSSIA Suggests that WAR May Be Necessary To Stop MONSANTO
Posted on June 13, 2013
Russian President Vladimir Putin commented that war might be a necessary result of the Monsanto Protection Act. did a little digging around, and it turns out the story checks out… The shocking minutes relating to President Putin’s meeting last month with US Secretary of State John Kerry reveal the Russian leaders “extreme outrage” over the Obama regimes continued protection of global seed and plant bio-genetic giants Syngenta and Monsanto in the face of a growing “bee apocalypse” that the Kremlin warns “will most certainly” lead to world war. -According to these minutes, released in the Kremlin today by the Ministry of Natural Resources and Environment of the Russian Federation (MNRE), Putin was so incensed over the Obama regimes refusal to discuss this grave matter that he refused for three hours to even meet with Kerry, who had traveled to Moscow on a scheduled diplomatic mission, but then relented so as to not cause an even greater rift between these two nations.—At the center of this dispute between Russia and the US, this MNRE report says, is the “undisputed evidence” that a class of neuro-active insecticides chemically related to nicotine, known as neonicotinoids, are destroying our planets bee population, and which if left unchecked could destroy our world’s ability to grow enough food to feed its population.—So grave has this situation become, the MNRE reports, the full European Commission (EC) this past week instituted a two-year precautionary ban (set to begin on 1 December 2013) on these “bee killing” pesticides following the lead of Switzerland, France, Italy, Russia, Slovenia and Ukraine, all of whom had previously banned these most dangerous of genetically altered organisms from being used on the continent.–Two of the most feared neonicotinoids being banned are Actara and Cruiser made by the Swiss global bio-tech seed and pesticide giant Syngenta AG which employs over 26,000 people in over 90 countries and ranks third in total global sales in the commercial agricultural seeds market.–Important to note, this report says, is that Syngenta, along with bio-tech giants Monsanto, Bayer, Dow and DuPont, now control nearly 100% of the global market for genetically modified pesticides, plants and seeds.—Also to note about Syngenta, this report continues, is that in 2012 it was criminally charged in Germany for concealing the fact that its genetically modified corn killed cattle, and settled a class-action lawsuit in the US for $105 million after it was discovered they had contaminated the drinking supply of some 52 million Americans in more than 2,000 water districts with its “gender-bending” herbicide Atrazine.—To how staggeringly frightful this situation is, the MNRE says, can be seen in the report issued this past March by the American Bird Conservancy (ABC) wherein they warned our whole planet is in danger, and as we can, in part, read: “As part of a study on impacts from the world’s most widely used class of insecticides, nicotine-like chemicals called neonicotinoids, American Bird Conservancy (ABC) has called for a ban on their use as seed treatments and for the suspension of all applications pending an independent review of the products’ effects on birds, terrestrial and aquatic invertebrates, and other wildlife.—“It is clear that these chemicals have the potential to affect entire food chains. The environmental persistence of the neonicotinoids, their propensity for runoff and for groundwater infiltration, and their cumulative and largely irreversible mode of action in invertebrates raise significant environmental concerns,” said Cynthia Palmer, co-author of the report and Pesticides Program Manager for ABC, one of the nation’s leading bird conservation organizations.—ABC commissioned world renowned environmental toxicologist Dr. Pierre Mineau to conduct the research. The 100-page report, “The Impact of the Nation’s Most Widely Used Insecticides on Birds,” reviews 200 studies on neonicotinoids including industry research obtained through the US Freedom of Information Act. The report evaluates the toxicological risk to birds and aquatic systems and includes extensive comparisons with the older pesticides that the neonicotinoids have replaced. The assessment concludes that the neonicotinoids are lethal to birds and to the aquatic systems on which they depend.—“A single corn kernel coated with a neonicotinoid can kill a songbird,” Palmer said. “Even a tiny grain of wheat or canola treated with the oldest neonicotinoid — called imidacloprid — can fatally poison a bird. And as little as 1/10th of a neonicotinoid-coated corn seed per day during egg-laying season is all that is needed to affect reproduction.”—The new report concludes that neonicotinoid contamination levels in both surface- and ground water in the United States and around the world are already beyond the threshold found to kill many aquatic invertebrates.”
Quickly following this damning report, the MRNE says, a large group of group of American beekeepers and environmentalists sued the Obama regime over the continued use of these neonicotinoids stating: “We are taking the EPA to court for its failure to protect bees from pesticides. Despite our best efforts to warn the agency about the problems posed by neonicotinoids, the EPA continued to ignore the clear warning signs of an agricultural system in trouble.”—And to how bad the world’s agricultural system has really become due to these genetically modified plants, pesticides and seeds, this report continues, can be seen by the EC’s proposal this past week, following their ban on neonicotinoids, in which they plan to criminalize nearly all seeds and plants not registered with the European Union, and as we can, in part, read:—“Europe is rushing towards the good ol days circa 1939, 40… A new law proposed by the European Commission would make it illegal to “grow, reproduce or trade” any vegetable seeds that have not been “tested, approved and accepted” by a new EU bureaucracy named the “EU Plant Variety Agency.”[F1]–It’s called the Plant Reproductive Material Law, and it attempts to put the government in charge of virtually all plants and seeds. [F2]Home gardeners who grow their own plants from non-regulated seeds would be considered criminals under this law.”—This MRNE report points out that even though this EC action may appear draconian, it is nevertheless necessary in order to purge the continent from continued contamination of these genetically bred “seed monstrosities.”–[F3]-Most perplexing in all of this, the MRNE says, and which led to Putin’s anger at the US, has been the Obama regimes efforts to protect pesticide-producer profits over the catastrophic damaging being done to the environment, and as the Guardian News Service detailed in their 2 May article titled “US rejects EU claim of insecticide as prime reason for bee colony collapse” and which, in part, says:–“The European Union voted this week for a two-year ban on a class of pesticides, known as neonicotinoids, that has been associated with the bees’ collapse. The US government report, in contrast, found multiple causes for the collapse of the honeybees.”-To the “truer” reason for the Obama regimes protection of these bio-tech giants destroying our world, the MRNE says, can be viewed in the report titled “How did Barack Obama become Monsanto’s man in Washington?” and which, in part, says:—“After his victory in the 2008 election, Obama filled key posts with Monsanto people, in federal agencies that wield tremendous force in food issues, the USDA and the FDA: At the USDA, as the director of the National Institute of Food and Agriculture, Roger Beachy, former director of the Monsanto Danforth Center. As deputy commissioner of the FDA, the new food-safety-issues czar, the infamous Michael Taylor, former vice-president for public policy for Monsanto. Taylor had been instrumental in getting approval for Monsanto’s genetically engineered bovine growth hormone.”—Even worse, after Russia suspended the import and use of an Monsanto genetically modified corn following a study suggesting a link to breast cancer and organ damage this past September, the Russia Today News Service reported on the Obama regimes response:
“The US House of Representatives quietly passed a last-minute addition to the Agricultural Appropriations Bill for 2013 last week – including a provision protecting genetically modified seeds from litigation in the face of health risks.–The rider, which is officially known as the Farmer Assurance Provision, has been derided by opponents of biotech lobbying as the “Monsanto Protection Act,” as it would strip federal courts of the authority to immediately halt the planting and sale of genetically modified (GMO) seed crop regardless of any consumer health concerns.—The provision, also decried as a “biotech rider,” should have gone through the Agricultural or Judiciary Committees for review. Instead, no hearings were held, and the piece was evidently unknown to most Democrats (who hold the majority in the Senate) prior to its approval as part of HR 993, the short-term funding bill that was approved to avoid a federal government shutdown.”—On 26 March, Obama quietly signed this “Monsanto Protection Act” into law thus ensuring the American people have no recourse against this bio-tech giant as they fall ill by the tens of millions, and many millions will surely end up dying in what this MRNE report calls the greatest agricultural apocalypse in human history as over 90% of feral (wild) bee population in the US has already died out, and up to 80% of domestic bees have died out too.
****************************************************************************
Over 30 Million Bees Found Dead In Elmwood Canada
July 1, 2013 by Arjun
Shortly after 50,000 bees were found dead in an Oregon parking lot (read more here), a staggering 37 million bees have been found dead in Elmwood, Ontario, Canada. Dave Schuit, who runs a honey operation in Elmwood has lost 600 hives. He is pointing the finger at the insecticides known as neonicotinoids, which are manufactured by Bayer CropScience Inc. This also comes after a recent report released by the British Beekeepers Association (BBKA) that recorded its largest loss of honeybees ever. You can read more about that here. The European Union has stepped forward, having banned multiple pesticides that have been linked to killing millions of bees. You can view the studies and read more about that here.—The loss comes after the planting of corn. Neonicotinoid pesticides are used to coat corn seed with air seeders. This results in having the pesticide dust blown into the air when planted. The death of millions of pollinators was studied by Purdue University. They discovered that Bees exhibited neurotoxic symptoms. They analyzed dead bees and found that traces of thiamethoxam/clothiandin were present in each case. The only major source of these compounds are seed treatments of field crops. You can view that study here (1).—Bee deaths are increasing exponentially. An international team of scientists led by Holland’s Utrecht University has concluded that, “large scale prophylaxic use in agriculture, their high persistence in soil and water, and their uptake by plants and translocation to flowers, neonicotinoids put pollinators at risk. This is some of the research that led to the European Unions ban of the pesticides, as mentioned and referenced earlier.—Can we really debate this much longer? The evidence linking pesticides to bee deaths is overwhelming. It’s not only bees, but an array of other insects as well. The last thing we need is for Monsanto and other corps to use another reason to manufacture and develop fake food! One reason that has been used for justification of GMO’s is a food shortage, and we all know how critical bees are to our food supply. There is a huge conflict of interest here, the pesticides used to spray the crops that are killing the bees are developed by biotech corporations like Monsanto.
Sources:
http://www.thepost.on.ca/2013/06/19/bees-dying-by-the-millions
(1) http://extension.entm.purdue.edu/publications/E-53.pdf
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3250423/?tool=pubmed
http://www.nature.com/nature/journal/vaop/ncurrent/full/nature11585.html
****************************************************************************
Monsanto to pull the plug on GM lobbying and activities in Europe
Posted on03 June 2013.
Monsanto plans to halt lobbying activity for its genetically modified (GM) crop varieties in Europe, the agriculture and biotechnology giant has confirmed.
US-based biotechnology company Monsanto will drop lobbying activities in Europe, and does not plan any new applications for the approval of genetically modified seeds in the EU, a spokesperson has said.–The moves comes on the back of low demand from farmers and ‘stiff opposition’ from the general public, according to the German spokeswoman Ursula Luettmer-Ouazane.—“As long as there’s not enough demand from farmers for these products and the public at large doesn’t accept the technology, it makes no sense to fight against windmills,” she confirmed.—Indeed, Monsanto officials added that the GM giant will only focus on expansion and market penetration of its GM products in areas that provide broad support – meaning that activities will continue in Portugal, Spain, and Eastern Europe where its MON810 maize is more widely grown and accepted.—Monsanto’s confirmation comes almost 18 months after an announcement that German chemical and biotech firm BASF would be pulling out of the European market due to a lack of consumer and industry acceptance. Other companies including Bayer AG’s CropScience unit and Syngenta have also largely moved out of the European market.–Monsanto is also currently under growing pressure in the USA and Asia, after the discovery of a non-approved, experimental wheat strain was found growing in Oregon.–The finding of the experimental wheat, which the company said it finished trials of several years previously, has led to uncertainty in the wheat export market with Japan imposing an outright ban , while a number of other markets – including the EU – said to be closely monitoring and assessing the situation.
TOP B
[F1]This legislation is a set up to further control access to foods and eventually will lead to wars as well due to the restrictions of what can and cannot be grown—the legislation should have regulated the Seeds of Biotech not the seeds that have been utilized historically
[F2]Unbelievable this will be europes undoing since corpations run Gov’t this will be just another way for the agro globalist to run a nation by either starvation or regulation—this will cause a shortage and a hike in cost to eat
[F3]This legislation has taken the power of the seed out of the farmers hands into the gov’t –the farming industry has now lost it’s only leverage
 
 
******************************************************************************************************************************************************
TOP C
HOME
 
SCRIPT OF THE SHOW JULY 27 2013
 
Fighting Cancer with Fats
Ketogenic diet effective against metastatic cancer
Long-term effects of a ketogenic diet in obese patients
Morgellons-Biowarefare-with Nano Technolgy
The Global Threat of Persistent Organic Pollutants
**************************************************************************
 
Fighting Cancer with Fats
Barrow researcher explores innovative cancer treatment
For Barrow Researcher Adrienne C. Scheck, PhD, diets are far more than a health fad: Dr. Scheck is researching the potential of a therapy known as the ketogenic diet to complement traditional treatments for brain cancer such as radiation and chemotherapy.—Since the 1920s, the ketogenic diet has been used to control epilepsy, but its popularity has waxed and waned. It is now used almost exclusively in children whose seizures do not respond to conventional drug therapies. The highly regimented diet calls for nearly 80 percent of a patient’s calorie intake to come from fat and less than 1 percent from carbohydrates, with the remainder coming from protein.—-Researchers were spurred to investigate the ketogenic diet as a cancer therapy because cancer cells rely primarily on glucose to fuel their metabolism. The diet causes the body to enter a state called ketosis, where fat-derived chemicals called ketones are burned in response to the absence of carbohydrates. It has been theorized that because tumor cells cannot use ketones for energy, this metabolic change would starve tumor tissue, stunting its growth and improving survival rates for cancer patients.–While not ruling out the metabolic explanation, Dr. Scheck’s research has uncovered other ways that the diet acts on tumors.–Dr. Scheck and her team in the Barrow Neuro-Oncology Research Laboratory observed that live tumor models subjected to the ketogenic diet had fewer reactive oxygen species present in and around the cancerous tissue.–“Reactive oxygen species are created during normal cellular metabolism and are important in the control of many aspects of cell growth—too many or too few are bad for the cell,” says Dr. Scheck. “The metabolism of tumors is higher than that of normal tissue—a feature that may allow them to adapt to increases in reactive oxygen species and contribute to their resistance against chemotherapy.”—Dr. Scheck hypothesizes that by helping to reduce the number of reactive oxygen species tumor cells are accustomed to, the diet may enhance the effectiveness of chemotherapy.[F1]—The results of Dr. Scheck’s latest study also suggest that the diet affects gene expression in tumor cells. “We are seeing that the genetic profile of the tumor cells exposed to ketone bodies is shoved more toward the gene expression of normal cells,” says Dr. Scheck.—How the above mechanisms work together to slow cancer growth is not yet clearly defined, but the results of their combined action are reason to take notice. Dr. Scheck’s team reports that a ketogenic diet, combined with traditional therapies such as radiation and chemotherapy, has the potential to slow the growth of living cancer cells and significantly increase survival time.[F2]—Nonetheless, the ketogenic diet has its drawbacks. Dr. Scheck cautions that this treatment is very different from diets that people manage on their own. Rather, she says, it is a therapy that requires total compliance
********************************************************************
Ketogenic diet effective against metastatic cancer
A research team from the Hyperbaric Biomedical Research Laboratory at the University of South Florida has finally found what the nutritional healing community has been crying hoarse about – that a ketogenic diet kills cancer cells. The study, “The Ketogenic Diet and Hyperbaric Oxygen Therapy Prolong Survival in Mice with Systemic Metastatic Cancer,” was published online on June 5, 2013 in PLOS ONE.—-In the study, mice with advanced metastatic cancer were fed either a standard high carbohydrate diet or carbohydrate-restricted ketogenic diet. Mice on both diets also received hyperbaric oxygen therapy, which uses a special chamber to increase the amount of oxygen in the tissues.—The ketogenic diet forces a physiological shift in substrate utilization from glucose to fatty acids and ketone bodies for energy. Normal healthy cells readily adapt to using ketone bodies for fuel, but cancer cells lack this metabolic flexibility, and thus become selectively vulnerable to reduced glucose availability. Solid tumors also have areas of low oxygen, which promotes tumor growth and metastatic spread. –Hyperbaric oxygen therapy involves breathing 100 percent oxygen at elevated barometric pressure, saturating the tumors with oxygen. When administered properly, both the ketogenic diet and hyperbaric oxygen therapy are non-toxic and even protect healthy tissues while simultaneously damaging cancer cells.–The research shows the effects of combining two nontoxic adjuvant cancer therapies, the ketogenic diet and hyperbaric oxygen therapy, in a mouse model of late-stage, metastatic cancer. While both therapies slowed disease progression independently, animals receiving the combined ketogenic diet and hyperbaric oxygen therapy lived 78 percent longer than mice fed a standard high-carbohydrate diet.—Metastasis, the spreading of cancer from the primary tumor to distant spots, is responsible for over 90 percent of cancer-related deaths in humans. A lack of available therapies effective against metastatic disease remains the largest obstacle in finding a cure for cancer.
How you can incorporate this into your lifestyle to not let cancer cells grow:
Restrict carbohydrates and replace them with healthy fats available in fish oil, nuts, seeds, extra virgin olive oil (raw) and healthy protein upasteurized milk, yogurt with fat ,kefir with fat,nuts, eggs, fish ghee-coconut oils-mct oils-tallow-lard. Concentrate on low glycemic index, high fibre carbohydrates in small quantities like apples-peaches-plums –pears-apricots.
When eating fruit, combine with spices-such as cinnamon-clove-cardamon-tumeric-galangal-ginger, as examples to avoid fructose related sugar spikes in the blood. The vitamins and minerals in fruits are anti-cancer hence fruits and fruit fibre are very important. Go for low GL foods like apple, papaya and fruits like pineapples with anti-inflammatory qualities. Consume raw, consume juices made from fresh veges home made and unpasteurized-if you use fruit juices then add the minerals zinc or chromium or manganese-or magnesium to them—or a full spectrum of minerals
To adapt hyperbaric oxygen therapy for prevention and to oxygenate your organs, practice deep breatheing techniques every day for 20-30 min. This will flush out the toxins from the blood and re-energise you while increasing oxygen in the blood flow, hence killing tumour cells in your body.
Remember, a strong immune system will fight cancer and tumour cells, and a ketogenic diet combined with oxygenating your body goes a long way in building immunity.
****************************************************************************
Long-term effects of a ketogenic diet in obese patients
Hussein M Dashti, MD PhD FICS FACS,1 Thazhumpal C Mathew, MSc PhD FRCPath,4 Talib Hussein, MB ChB,5 Sami K Asfar, MB ChB MD FRCSEd FACS,1 Abdulla Behbahani, MB ChB FRCS FACSI PhD FICS FACS,1 Mousa A Khoursheed, MB ChB FRCS FICS,1 Hilal M Al-Sayer, MD PhD FICS FACS,1 Yousef Y Bo-Abbas, MD FRCPC,2 and Naji S Al-Zaid, BSc PhD3
Author information ► Copyright and License information ►
This article has been cited by other articles in PMC.
Go to:
Abstract
BACKGROUND:
Although various studies have examined the short-term effects of a ketogenic diet in reducing weight in obese patients, its long-term effects on various physical and biochemical parameters are not known.
OBJECTIVE:
To determine the effects of a 24-week ketogenic diet (consisting of 30 g carbohydrate, 1 g/kg body weight protein, 20% saturated fat, and 80% polyunsaturated and monounsaturated fat) in obese patients.
PATIENTS AND METHODS:
In the present study, 83 obese patients (39 men and 44 women) with a body mass index greater than 35 kg/m2, and high glucose and cholesterol levels were selected. The body weight, body mass index, total cholesterol, low density lipoprotein (LDL) cholesterol, high density lipoprotein (HDL) cholesterol, triglycerides, fasting blood sugar, urea and creatinine levels were determined before and after the administration of the ketogenic diet. Changes in these parameters were monitored after eight, 16 and 24 weeks of treatment.
RESULTS:
The weight and body mass index of the patients decreased significantly (P<0.0001). The level of total cholesterol decreased from week 1 to week 24. HDL cholesterol levels significantly increased, whereas LDL cholesterol levels significantly decreased after treatment. The level of triglycerides decreased significantly following 24 weeks of treatment. The level of blood glucose significantly decreased. The changes in the level of urea and creatinine were not statistically significant.
CONCLUSIONS:–The present study shows the beneficial effects of a long-term ketogenic diet. It significantly reduced the body weight and body mass index of the patients. Furthermore, it decreased the level of triglycerides, LDL cholesterol and blood glucose, and increased the level of HDL cholesterol. Administering a ketogenic diet for a relatively longer period of time did not produce any significant side effects in the patients. Therefore, the present study confirms that it is safe to use a ketogenic diet for a longer period of time than previously demonstrated.
Keywords: Diet, Ketosis, Obesity
Obesity has become a serious chronic disease in both developing and developed countries. Furthermore, it is associated with a variety of chronic diseases (
The following popper user interface control may not be accessible. Tab to the next button to revert the control to an accessible version.
Therefore, the purpose of the present study was to investigate the long-term effects of a ketogenic diet on obesity and obesity-associated risk factors in a large population of obese patients.
Go to:
PATIENTS AND METHODS

Life Force Energy