Scripts 2011

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    US threatened – More Like the GMO or GE companies – ‘retaliation’ to bully EU into accepting biotech crops, cable shows
    Reacting to a French pledge to represent the “common interest” in considering biotech foods, a former US ambassador recommended publishing a “retaliation list” of European locations where genetically modified organisms (GMOs) were being grown in hopes that activists would destroy them and “cause some pain” for officials, a leaked diplomatic cable shows.–In a confidential communication dated Dec. 14, 2007 and released by WikiLeaks on Sunday, then-US Ambassador to France Craig Roberts Stapleton recommended creating the list if France and the EU continued to ban biotech seeds.–“Mission Paris recommends that that the [United States government] reinforce our negotiating position with the EU on agricultural biotechnology by publishing a retaliation list when the extend ‘Reasonable Time Period’ expires,” Stapleton wrote. “Europe is moving backwards not forwards on this issue with France playing a leading role, along with Austria, Italy and even the [European] Commission.”
    Stapelton added that the US should create a list “that causes some pain across the EU, since this is a collective responsibility, but that also focuses in part on the worst culprits.” –He continued: “The list should be measured rather than vicious and must be sustainable over the long term, since we should not expect an early victory.” The former US official added that France’s “High Authority” on agricultural biotech was particularly offensive because it sought to “roll back established science-based decision making.” He added that a bill considered by the French National Assembly should be rebuked by the publication “of a registry identifying the cultivation of GMOs at the parcel levels” … “given the propensity for activists to destroy GMO crops in the field.”–The document would appear to expose a high-ranking US official advocating a selective leak of otherwise confidential information to achieve a European political objective on behalf of US private industry. —The law that was considered in France would have made farmers and biotech firms liable for pollen drift of their modified crops — a move that “could make any biotech planting impossible in practical terms,[U1]” the Stapleton wrote.
    It was essentially the same principle the US employs for environmental pollution: the polluter must pay. GMO firms, however, are given exception to those regulations in North America.
    The spread of modified genes into the wild is of particular concern to critics of biotech food crops, who cite studies linking GM seeds to organ damage and infertility in animals. Most Monsanto seeds are modified to resist pesticides such as Roundup, which has been shown to cause cancer and genetic mutations in humans. It is still unclear whether genetically modified foods pose health risks, but they have been adopted in soaring quantities in the United States. “Soybeans and cotton genetically engineered with herbicide-tolerant traits have been the most widely and rapidly adopted GE crops in the U.S., followed by insect-resistant cotton and corn,” according to the US Department of Agriculture (USDA).
     
    Researchers at the University of Arkansas found in August that canola, a modified rapeseed used mainly for oil, had managed to sustain itself in the wilds of North Dakota. Up to 80 percent of the plants they tested had genes that were modified to resist herbicide. It was the first time modified crops had been discovered growing in the wild[U2], the BBC noted.
    Monsanto’s genetically modified corn, currently banned across the EU, was also found growing in Ireland, the Irish Department of Agriculture said. –Stapleton was appointed Ambassador to France in 2005 by President George W. Bush. His wife is a cousin of President George H.W. Bush. Stapelton was replaced as ambassador in July 2009, when President Barack Obama named Charles Rivkin to the post.
    Europe continues biotech resistance
    It’s not clear from the release if the US went ahead with its plan for the “retaliation list,” but Stapelton was certainly right on whether the US should expect an early victory in European public opinion. In December, more than one million Europeans signed a petition demanding the EU halt the approval of new genetically modified crops. The petition was later dismissed by the EU Commission on procedural grounds.–In the last 12 years only two organisms have been licensed for seeding across Europe, and one of them was a potato that triggered the recent mass petition against the crops. The number one multinational biotech firm in the world, Monsanto, isn’t happy about that.-But like other US business interests, Monsanto hasn’t been sitting around whining about policy backlash foreign or domestic. The Nation’s Jeremy Scahill revealed in September that the world’s top producer of genetically modified seeds hired US security contractor Blackwater to “infiltrate activist groups organizing against the multinational biotech firm.” The leaked cable makes no mention of Blackwater mercenaries operating in Europe. Other cables released in recent days showed that, behind the scenes, Spain has been a key ally of the US in defending genetically modified crops.
    Spanish Secretary of State and Deputy Minister Josep Puxeu contacted US officials to ask for support after his country came under pressure to ban their Monsanto-developed MON810 corn crop, a cable revealed. “He asked that the USG maintain pressure on Brussels to keep agricultural biotechnology an option for Member States and requested that the USG work together with Spain in this endeavor.” Another cable sent to the Vatican on Nov. 19, 2009 indicated that Pope Benedict XVI also supports genetically modified crops, but will not admit it in public. “Vatican officials remain largely supportive of genetically modified crops as a vehicle for protecting the environment while feeding the hungry, but — at least for now — are unwilling to challenge bishops who disagree,” it explained.
     
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    Coffee and liver diseases.
    Fitoterapia. 2010 Jul;81(5):297-305-Authors: Muriel P, Arauz J
    Coffee consumption is worldwide spread with few side effects. Interestingly, coffee intake has been inversely related to the serum enzyme activities gamma-glutamyltransferase, and alanine aminotransferase in studies performed in various countries. In addition, epidemiological results, taken together, indicate that coffee consumption is inversely related with hepatic cirrhosis; however, they cannot demonstrate a causative role of coffee with prevention of liver injury. Animal models and cell culture studies indicate that kahweol, diterpenes and cafestol (some coffee compounds) can function as blocking agents by modulating multiple enzymes involved in carcinogenic detoxification; these molecules also alter the xenotoxic metabolism by inducing the enzymes glutathione-S-transferase and inhibiting N-acetyltransferase. Drinking coffee has been associated with reduced risk of hepatic injury and cirrhosis, a major pathogenic step in the process of hepatocarcinogenesis, thus, the benefit that produces coffee consumption on hepatic cancer may be attributed to its inverse relation with cirrhosis, although allowance for clinical history of cirrhosis did not completely account for the inverse association. Therefore, it seems to be a continuum of the beneficial effect of coffee consumption on liver enzymes, cirrhosis and hepatocellular carcinoma. At present, it seems reasonable to propose experiments with animal models of liver damage and to test the effect of coffee, and/or isolated compounds of this beverage, not only to evaluate the possible causative role of coffee but also its action mechanism. Clinical prospective double blind studies are also needed.– PMID: 19825397 [PubMed – indexed for MEDLINE]
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    Pollutants in boys’ blood tied to lower growth
    NEW YORK (Reuters Health) – Russian boys exposed to unusually high levels of environmental pollutants are smaller than their peers, a new study reports.-After following nearly 500 boys for three years, an international group of researchers found that those with the highest levels of polychlorinated biphenyls (PCBs) in their blood were nearly three centimeters (more than an inch) shorter than boys from the same region with the lowest amount of PCBs in their bodies. Boys with the highest exposures also averaged two points lower in body mass index (BMI), a measure of weight relative to height.–The authors found a similar pattern in boys with the highest exposure to the pollutant dioxin.–“You’re always a little surprised to see such a dramatic effect,” study author Jane Burns of the Harvard School of Public Health told Reuters Health, but the findings are “consistent” with some other research about the effect of these chemicals.–Burns explained that the boys included in the study have much higher exposures to these pollutants than the general U.S. population, likely a result of their proximity to a chemical plant that generated dioxin as a byproduct. But a small number of people in the U.S. and other developed countries live in regions with exposures that match – or even exceed – those seen here, she added.—PCBs were once used in everything from appliances and fluorescent lighting to insulation and insecticides. While the chemicals were banned in the 1970s as potential health hazards, they remain a public-health concern because they linger in the environment and accumulate in the fat of fish, mammals and birds.–Research has linked PCBs to an elevated risk of cancer, type 2 diabetes and other ailments. A study of children in Taiwan also found that those exposed in utero to PCBs from contaminated cooking oil were shorter than their peers. Dioxins are toxic substances formed by burning — for example in waste incinerators or forest fires — and in some industrial processes. Airborne dioxins are deposited onto plants, soils and water, and they enter the food chain when ingested by livestock and fish. Dioxin exposure has been shown to lead to both higher cancer rates and changes in birth rates resulting in more female babies and fewer males.—To further investigate the impact of high exposures to PCBs and dioxins on growth, Burns and her colleagues took blood samples from 499 boys ages 8 and 9 living in Chapaevsk, Russia, an area known to be highly contaminated.–Indeed, the highest levels of PCBs and dioxins in the boys’ blood far exceeded those found in the average U.S. population, Burns noted.—Prenatal exposure to PCBs, but not dioxins, has been linked to low birth weight. And along with differences in the prepubescent boys’ height and weight, the authors found that those with the highest blood levels of PCBs were growing significantly more slowly than boys with the least exposure – by about 0.2 centimeters a year over the three-year study period.–The findings appear in the journal Pediatrics.–It’s not entirely clear why PCBs or dioxins might affect growth, Burns said in an interview. “We’re not really sure of what the mechanisms are.” Research suggests dioxins interfere with genes that regulate normal development, while PCBs disrupt the regulation of thyroid hormones, which could impact growth.
    SOURCE: http://link.reuters.com/vab24r Pediatrics, online December 27, 2010.
     
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    [U1]This is what should have been here in Canada and The USA–instead we have given the Agro Corporates free and and clear access to dump-contaminate and cause chaos and havoc with creation–if this was enforced then Monsanto would have been the ones who would have lost the farms–Not the farmers they stripped and stole there lands through litigations and unjust rulings making the farmer at fault when in reality the Agro Companies like this would have been the ones to Pay—this kind of Agro business should have never been allowed to even begin to flourish or released–The Supreme court ruling that favoured this should not have transpired
    [U2]DING DING DING!!!! DANGER DANGER —this is all we need to have our healthy foods mixed with this Genetic Abomination
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    Show of the Week January 7 2011
    USDA Certified Organics Dirty Little Secret- Neotame
    The Vermont Resolution for Food Sovereignty
    NOTICE!-DO NOT DISCARD
    Multi Studies and Solutions (1) Isoquercitrin (2) Uptake of Antioxidants in kidneys and liver and heart (3) Minimizing radiation exposure to intestine (4) Wolfberry or Goji Berry on intestinal Cancers (5) Probiotic-Prebiotic and Oils protection
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    A) USDA Certified Organics Dirty Little Secret- Neotame
     
    Just when we thought that buying “Organic” was safe, we run headlong into the deliberate poisoning of our organic food supply by the FDA [U1] in collusion with none other than the folks who brought us Aspartame. NutraSweet, a former Monsanto asset, has developed a new and improved version of this neurotoxin called Neotame. —Neotame has similar structure to aspartame — except that, from it’s structure, appears to be even more toxic than aspartame. This potential increase in toxicity will make up for the fact that less will be used in diet drinks. Like aspartame, some of the concerns include gradual neurotoxic and immunotoxic damage from the combination of the formaldehyde metabolite (which is toxic at extremely low doses) and the excitotoxic amino acid. (Holisticmed.com) –But surely, this product would be labeled! NOT SO!!! For this little gem, no labeling required. And it is even included in USDA Certified Organic food. —The food labeling requirements required for aspartame have now been dropped for Neotame, and no one is clear why this was allowed to happen. Neotame has been ruled acceptable, and without being included on the list of ingredients, for:
    USDA Certified Organic food items.
    Certified Kosher products with the official letter k inside the circle on labels. (Janet Hull)
    Let me make this perfectly clear. Neotame does not have to be included in ANY list of ingredients! So, if you buy processed food, whether USDA Certified Organic or not, that food most likely will contain Neotame because it is cost-effective, and since no one knows it is there, there is no public backlash similar to what is happening with Aspartame. A win/win situation! but that’s not all. Just love chowing down on that delicious steak? Well, that cow most likely will have been fed with feed containing…..you guessed it…..Neotame! A product called “Sweetos,” which is actually composed of Neotame, is being substituted for molasses in animal feed. —“Sweetos is an economical substitute for molasses. Sweetos guarantees the masking of unpleasant tastes and odor and improves the palatability of feed. This product will be economical for farmers and manufacturers of cattle feed. It can also be used in mineral mixture,” said Craig Petray, CEO, The NutraSweet Company, a division of Searle, which is a part of Monsanto. (Bungalow Bill)—- Why would we feed animals food that is so distasteful that we would have to mask the unpleasantness with an artificial sweetener? Most animals will not eat spoiled, rancid feed[U2] . They know by the smell that it is not good. Enter Sweetos (Neotame). Just cover up the unpleasant tastes and odors, and you can feed them anything you want to, courtesy of the oh, so considerate folks at Monsanto and company. But of course, Monsanto is no longer associated with NutraSweet. In the time-honored tradition of covering its assets, Monsanto has a proven track record of spinning off controversial portions of its company that generate too much scrutiny, such as it did with the Solutia solution.
     
    Says the Farm Industry News, “Monsanto, which has long resided in the crosshairs of public scorn and scrutiny, appears to have dodged at least one bullet by spinning off its industrial chemical business into a separate entity called Solutia a couple of years ago. Solutia has since been hammered by lawsuits regarding PCB contamination from what were once called Monsanto chemical plants in Alabama and other states” (Source Watch) –So what is the solution to this problem? Buy Local Chemical FREE food, know your local farmer, and don’t buy processed foods whether they are labeled “Organic” or not. This requires a drastic change in lifestyle that most will not want to make. For those who choose to ride the wheel of chance by succumbing to this genocidal adulteration of our food supply by those who stand to profit from our sickness and early demise, my only comment is….it is your choice. But for those of us who have decided to fight this battle one bite at a time by hitting these GLOBALIST in the pocketbook where it hurts
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    MOST LIKELY TO SECEDE:
    The Vermont Resolution for Food Sovereignty
    Time to get serious about Food Sovereignty.
    WHEREAS All people are endowed by their Creator with certain inalienable rights, and among these are life, liberty and the pursuit of happiness; and WHEREAS Food is human sustenance and is the fundamental prerequisite to life; and WHEREAS The basis of human sustenance rests on the ability of all people to save seed, grow, process, consume and exchange food and farm products; and WHEREAS We the People of Vermont, have an obligation to protect these rights as is the Common and Natural Law; and in recognition of the State’s proud agricultural heritage; and the necessity of agricultural, ecological and economic diversity and sustainability to a free and healthy Society;
    THEREFORE, Be it resolved, that We The People, stand on our rights under the 10th Amendment to the US Constitution and reject such Federal decrees, statutes, regulations or corporate practices that threaten our basic human right to save seed, grow, process, consume and exchange food and farm products within the State of Vermont; and, Be it further resolved, that We The People, shall resist any and all infringements upon these rights, from whatever sources that are contrary to the rights of the People of the State of Vermont
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    NOTICE–DO NOT DISCARD
     
     
    ü You should have a packet that looks like this from Senator Johnson which was hand carried by her two weeks ago.
     
    ü A number of us helped distribute her letter to you because we represent hundreds of your constituents who are aware of a critical issue going on outlined in Senator Johnson’s letter.
     
    ü We believe it’s imperative we take an active role in our local government since Arizona is being sanctioned in the billions at the end of this month by the EPA for what we believe is based on erroneous statistics. A letter will be delivered to you in a few days giving proof of this statement.
     
    ü Additionally, EPA claims Arizona falls into the “Extreme Pollution Level” category. Interestingly, there are 11 other states within this same category, yet Arizona is the ONLY state being sanctioned!
     
    ü Also interestingly, every registered vehicle in Arizona MUST meet EPA’s Vehicle Emissions Standards. Arizona motorists spend millions annually for this requirement. Has something failed in this process?
     
    ü The Arizona Republic (9/4/10) states, “The particles, about one-seventh the size of a human hair, can invade the lungs and blood stream, aggravate asthma and other breathing problems, and can cause respiratory and heart ailments especially among children.”
     
    ü These symptoms are exactly the result of a Geo-Engineering Program that has been going on in Arizona and globally, as portrayed in the documentary, “What in the World Are They Spraying” DVD in your packet.
     
    ü Essentially, Arizona should NOT be financially burdened more than it already is for an unwarranted claim; illness described in EPA’s “Particulate Matter” description does NOT come from our road dust; and, Arizona should NOT be singled out for sanctions in the billions.
     
    ü Since this Geo-Engineering Program is now being openly discussed to be legalized and outlines toxic heavy metals to be sprayed over humans . . our concerns cannot be passed on as a conspiracy theory! This MUST be addressed. We are counting on each and every one of you to take action. You will receive a follow-up letter in a few days. Thank you for your time.
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    1) Inhibition of influenza virus replication by plant-derived isoquercetin.
    Antiviral Res. 2010 Nov;88(2):227-35
    Authors: Kim Y, Narayanan S, Chang KO
    Influenza virus infects the respiratory system of human and animals causing mild to severe illness which could lead to death. Although vaccines are available, there is still a great need for influenza antiviral drugs to reduce disease progression and virus transmission. Currently two classes (M2 channel blockers and neuraminidase inhibitors) of FDA-approved influenza antiviral drugs are available, but there are great concerns of emergence of viral resistance. Therefore, timely development of new antiviral drugs against influenza viruses is crucial. Plant-derived polyphenols have been studied for antioxidant activity, anti-carcinogenic, and cardio- and neuroprotective actions. Recently, some polyphenols, such as resveratrol and epigallocatechin gallate, showed significant anti-influenza activity in vitro and/or in vivo. Therefore we investigated selected polyphenols for their antiviral activity against influenza A and B viruses. Among the polyphenols we tested, isoquercetin inhibited the replication of both influenza A and B viruses at the lowest effective concentration. In a double treatment of isoquercetin and amantadine, synergistic effects were observed on the reduction of viral replication in vitro. The serial passages of virus in the presence of isoquercetin did not lead to the emergence of resistant virus, and the addition of isoquercetin to amantadine or oseltamivir treatment suppressed the emergence of amantadine- or oseltamivir-resistant virus. In a mouse model of influenza virus infection, isoquercetin administered intraperitoneally to mice inoculated with human influenza A virus significantly decreased the virus titers and pathological changes in the lung. Our results suggest that isoquercetin may have the potential to be developed as a therapeutic agent for the treatment of influenza virus infection and for the suppression of resistance in combination therapy with existing drugs.– PMID: 20826184 [PubMed – indexed for MEDLINE]
    ICan be found in Apples-Cherries- Onion- Bay leaf- Black Tea-Sweet Basil- AllSpice-Black Pepper-Apricot-Black Currant-White Willow-Elderberry—-these can as well be combined for instance you can combine apple and onion in a apple/onion sauce by blending the 2 together ( 2 apples to one onion (red onion has similar polyphenols that of red wine)–you can combo elderberry in an alcohol or honey mix by adding 1/4 cup of elderberry with 1 oz of brandy and 1/2 cup of honey -blend til fused smooth ( about 7 minutes at high speed ) or just use 1/4 cup of elderberry and 1/2 cup-3/4 cup of brandy or alcohol that is clear again blend for 10 minutes at high speed and then pour into a glass bottle-you can mix allspice with pepper when garnishing your foods in light doses when combined–you can combo elderberry and currant in a honey jam by adding equal parts of the currant to the elderberry and then double the volume of honey and blend-while blending add allspice to this about 1/2 tsp and then blend til smooth–these are some examples
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    2) A dietary supplement containing multiple phytochemicals and vitamins elevates hepatorenal and cardiac antioxidant enzymes in the absence of significant serum chemistry and genomic changes.
    Oxid Med Cell Longev. 2010 Mar-Apr;3(2):129-44
    Authors: Bulku E, Zinkovsky D, Patel P, Javia V, Lahoti T, Khodos I, Stohs SJ, Ray SD
    A novel dietary supplement composed of three well-known phytochemicals, namely, Salvia officinalis (sage) extract, Camellia sinensis (oolong tea) extract, and Paullinia cupana (guarana) extract, and two prominent vitamins (thiamine and niacin) was designed to provide nutritional support by enhancing metabolism and maintaining healthy weight and energy. The present study evaluated the safety of this dietary supplement (STG; S=sage; T=tea; G=guarana) and assessed changes in target organ antioxidant enzymes (liver, kidneys and heart), serum chemistry profiles and organ histopathology in Fisher 344 rats. Adult male and female Fisher 344 rats were fed control (no STG) or STG containing (1X and 7X, 1X=daily human dose) diets and sacrificed after 2 and 4 months. Serum chemistry analysis and histopathological examination of three vital target organs disclosed no adverse influence on protein, lipid and carbohydrate profiles, genomic integrity of the liver and/or the tissue architecture. However, analysis of the most important antioxidant components in the liver, kidney and heart homogenates revealed a dramatic increase in total glutathione concentrations, glutathione peroxidase and superoxide dismutase enzyme activities. Concomitantly, oxidative stress levels (malondialdehyde accumulation) in these three organs were less than control. Organ specific serum markers (ALT/AST for the liver; CPK/AST for the heart; BUN/creatinine for kidneys) and the genomic integrity disclosed no STG-induced alteration. Some of the serum components (lipid and protein) showed insignificant changes. Overall, STG-exposed rats were more active, and the results suggest that STG exposure produces normal serum chemistry coupled with elevated antioxidant capacity in rats fed up to seven times the normal human dose and does not adversely influence any of the vital target organs. Additionally, this study reiterates the potential benefits of exposure to a pharmacologically relevant combination of phytochemicals compared to a single phytochemical entity. — PMID: 20716937 [PubMed – indexed for MEDLINE]
    IThis is how synergy can work with a combination of herbs and supplements–this is called Orthomolecular healing-using things of the body or of nature in adeqautes dosing or in combination to rebalance and restore the body to it’s norm
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    3) Berberine inhibits acute radiation intestinal syndrome in human with abdomen radiotherapy.
    Med Oncol. 2010 Sep;27(3):919-25
    Authors: Li GH, Wang DL, Hu YD, Pu P, Li DZ, Wang WD, Zhu B, Hao P, Wang J, Xu XQ, Wan JQ, Zhou YB, Chen ZT
    Radiation-induced acute intestinal symptoms (RIAISs) are the most relevant complication of abdominal or pelvic radiation. Considering the negative impact of RIAIS on patients’ daily activities, the preventive effects of berberine on RIAIS in patients were investigated. Thirty-six patients with seminoma or lymphomas were randomized to receive berberine oral (n = 18) or not (n = 18). Forty-two patients with cervical cancer were randomized to a trial group (n = 21) and control group (n = 21). Radiotherapy used a parallel opposed anterior and posterior. 300-mg berberine was administered orally three times daily in trial groups. Eight patients with RIAIS were treated with 300-mg berberine three times daily from the third to the fifth week. Toxicities, such as fatigue, anorexia/nausea, etc., were graded weekly according to CTC version 2.0. Patients with abdominal/pelvic radiation in the control group showed grade 1 fatigue, anorexia/nausea, colitis, vomiting, proctitis, weight loss, diarrhea and grade 2 anorexia/nausea, fatigue. Only grade 1 colitis, anorexia/nausea, and fatigue were seen in patients of abdominal radiation treated with berberine. Grade 1 fatigue, colitis, anorexia/nausea, and proctitis occurred in patients of pelvic radiotherapy treated with berberine. Pretreatment with berberine significantly decreased the incidence and severity of RIAIS in patients with abdominal/pelvic radiotherapy when compared with the patients of the control group (P < 0.05). RIAIS were reduced in patients with abdominal radiotherapy/pelvic radiation after receiving berberine treatment. Berberine significantly reduced the incidence and severity of RIAIS and postponed the occurrence of RIAIS in patients with abdominal or whole pelvic radiation.
    IThis can be found in Golden Seal ( if you use no more then 10 days on and a week off ) or Oregon Grape Make a tea with these or make an infusion with alcohol or dried in capsule
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    4) Growth inhibition and cell-cycle arrest of human gastric cancer cells by Lycium barbarum polysaccharide.
    Med Oncol. 2010 Sep;27(3):785-90
    Authors: Miao Y, Xiao B, Jiang Z, Guo Y, Mao F, Zhao J, Huang X, Guo J
    Lycium barbarum polysaccharide (LBP) is extracted from the traditional Chinese herb Lycium barbarum, and has potential anticancer activity. However, the detailed mechanisms are largely unknown. The purpose of this study was to observe the anticancer effect of LBP on human gastric cancer, and its possible mechanisms. Human gastric cancer MGC-803 and SGC-7901 cells were treated with various concentrations of LBP for 1-5 days, and cell growth was measured by 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyl tetrazolium bromide assay. Distribution of the cell cycle was analyzed by flow cytometry. Western blotting was used to indicate changes in the level of cyclins and cyclin-dependent kinases (CDKs). LBP treatment inhibited growth of MGC-803 and SGC-7901 cells, with cell-cycle arrest at the G0/G1 and S phase, respectively. We believe that this is the first study to show that LBP arrested different cell lines from the same types of cancer at different phases. The changes in cell-cycle-associated protein, cyclins, and CDKs were consistent with the changes in cell-cycle distribution. This study suggests that induction of cell-cycle arrest participates in the anticancer activity of LBP on gastric cancer cells. PMID: 19669955 [PubMed – indexed for MEDLINE]
    IRemedy–Fused this in either alcohol or honey —Take a 1/4 cup of Goji Berry( wolfberry) and add either brandy-vodka-gin or a good wine at 1 1/2 cup in blender and blend for about 10 minutes at high speed – then pour contents through a sift and allow the mix to drain and pour content in a glass bottle–use 1/2 a teaspoon once or twice a day–this also improves the vision-increases liver support-brain support due to the S.O.D content ( Superoxide Dismutase ) which increases Glutathione( major antioxidant in the body ) –You can as well do this as an elixor By adding 1/4 cup of brandy 1/2-3/4 cup of honey and blend til liquified–then add 1/4 cup of goji berry to this and blend for 7-10 minutes at high speed -when done pour entire conten in a glass bottle and use agai 1/2 tsp 2 times a day or as needed
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    5)The effect of probiotic microorganisms and bioactive compounds on chemically induced carcinogenesis in rats.
    Neoplasma. 2010;57(5):422-8
    Authors: Bertkova I, Hijova E, Chmelarova A, Mojzisova G, Petrasova D, Strojny L, Bomba A, Zitnan R
    Diet interventions and natural bioactive supplements have now been extensively studied to reduce risks of colon cancer, which is one of the major public health problem throughout the world. The objective of our investigation was to study the effects of probiotic, prebiotic, nutritional plant extract, and plant oil on selected biochemical and immunological parameters in rats with colon cancer induced by N,N dimethylhydrazine (DMH). Male and female Wistar albino rats were were fed by a high-fat (HF) diet (10% fat in the diet) and were divided into 9 groups: Control group; PRO group – HF diet supplemented with probiotic Lactobacillus plantarum to provide 3 x 109 c.f.u. of strain/1 ml of medium; PRE group – HF diet supplemented with inulin enriched with oligofructose (2% of HF diet); HES group – HF diet supplemented with plant extract of Aesculus hippocastanum L.( Horse Chestnut ) (1% of HF diet); OIL group – HF diet comprised Linioleum virginale (2% of HF diet); and combination of probiotic microorganisms and bioactive compounds in the groups – PRO-PRE, PRO-HES, PRO-OIL, PRE-OIL. Carcinogenesis was initiated with subcutaneous injection of DMH (20 mg/kg) two times at week interval and dietary treatments were continued for the six weeks. Application of probiotic microorganisms and bioactive compounds in all treated groups significantly decreased the activities of bacterial enzymes (p<0.001), the fecal bile acids concentration (p<0.01; p<0.001) and significantly increased serum TNFalpha level (p<0.001) in comparison to the control rats. The number of coliforms was reduced in PRO, PRO-PRE, PRO-OIL and PRE-OIL groups and significantly higher count of lactobacilli (p<0.05) was observed in PRO-PRE, PRO-OIL and PRE-OIL groups in compare with the controls. In conclusion, the results of this study indicate that probiotic microorganisms and bioactive compounds could exert a preventive effect on colon carcinogenesis induced by DMH.— PMID: 20568896 [PubMed – indexed for MEDLINE]
    ISo this is saying that if you are on a high fat and protein diet this can offset some of the side effects of this diet as well as neutralize cancer causing chemicals from reacting with the colon
     
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    [U1]funny thing alot of us have been saying for a long time that the organics are not safe and the term we should really be using is pesticide free –chemical free–colour free–synthetic free if we are really trying to eat things as pure as possible—what we see is “ORGASMIC” may as well be as soon as people hear the term organic they assume thet have bitten into nirvana-well the truth is that we live in a world where gov’t have investments with corporations and so they legislate to protect there interest NOT OURS so if you have any intentions of buying anything then either buy direct from farmers especially those who have been bullied my the Globalist–
    [U2]Does this make you feel better when you are eating soy?? a garbage food –leftover byproduct and waste which was coined as a health food which is worse then aspartame but when combo’d with aspartame is even more toxic due to the synergy–CHOW DOWN
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    Show of the Week January 10 2011
     
    INTRAVENOUS VITAMIN C SAVES NZ MAN WITH FLU DAMAGED LUNG
    The Herxheimer Reaction – Feeling worse before feeling better
     
    Not All Infant Formulas Are Alike– Differential Effects on Weight Gain
     
    Recipe for Formula making for adult of child
     
    Google map of Bird and Fish Fall out
     
    Healing Autism: No Finer a Cause on the Planet
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    INTRAVENOUS VITAMIN C SAVES NZ MAN WITH FLU DAMAGED LUNG
    January 6, 2010 – 5:40 am
    A 56 year old male was referred to Auckland Hospital ICU on 1 July 2009 with total respiratory failure, for ECMO external oxygenation. The patient had contracted H1N1 Swine flu (confirmed by tests) while on holiday overseas, and had developed what is known as ‘white out’ pneumonia. This refers to x-rays showing no air space in the lungs.–After 20 days of life-sustaining ECMO treatment and other critical care, the patient, who was unconscious by induced coma, had not responded. The ICU team advised the family of the likely outcome and had prepared them for the possibility of the patient’s death.—Family members approached Centre for Advanced Medicine Limited (CAM) for advice on the clinical use of intravenous vitamin C for such cases.—At the family’s request, information was provided to ICU doctors including ISO 9001:2008 registered protocols, safety data, dosages and access to vials of IV vitamin C under CAM’s license for wholesale medicines.—The ICU team agreed to administer intravenous vitamin C according to the family’s wishes. This decision acknowledged the family’s rights, in compliance with the New Zealand Health and Disability Act, 1997.—The patient received intravenous vitamin C starting on the evening of 21 July, continuing until 29 July. 25 grams was provided on the first day increasing over the first three days to 50 grams twice daily which was sustained for a further six days.—By 24 July x-rays indicated increasing lung function and ECMO external oxygenation was discontinued on 26 July. After several days of assisted ventilation and critical care for ongoing secondary conditions, the patient was able to commence his recovery and rehabilitation.—The patient was discharged from hospital on Friday 18 September, and is recovering at home on the farm.–The decision by the Auckland Hospital ICU team to administer adequate dosages of IV vitamin C, and their skillful coordination of ICU procedures, were responsible for the positive medical outcome.—Permission from the patient and his family has been sought by CAM to publish these details on its website and elsewhere in the interests of accuracy. This permission was willingly provided and CAM expresses its thanks, admiration and respect.–CAM welcomes opportunities to provide similar professional support for registered medical practitioners and their patients.
    http://www.camltd.co.nz/h1n1.html
    Spirited debate in Poland against the Vaccines on H1N1
     
    Finnish Minster Against FLU SHOTS
     
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    The Herxheimer Reaction – Feeling worse before feeling better
    The Herxheimer Reaction is a short-term (from days to a few weeks) detoxification reaction in the body. As the body detoxifies, it is not uncommon to experience flu-like symptoms including headache, joint and muscle pain, body aches, sore throat, general malaise, sweating, chills, nausea or other symptoms. –This is a normal — and even healthy — reaction that indicates that parasites, fungus, viruses, bacteria or other pathogens are being effectively killed off. The biggest problem with the Herxheimer reaction is that people stop taking the supplement or medication that is causing the reaction, and thus discontinue the very treatment that is helping to make them better. Although the experience may not make you feel particularly good, the Herxheimer Reaction is actually a sign that healing is taking place.
    What Is The Herxheimer Reaction?
    The Herxheimer Reaction is an immune system reaction to the toxins (endotoxins) that are released when large amounts of pathogens are being killed off, and the body does not eliminate the toxins quickly enough. Simply stated, it is a reaction that occurs when the body is detoxifying and the released toxins either exacerbate the symptoms being treated or create their own symptoms. The important thing to note is that worsening symptoms do not indicate failure of the treatment in question; in fact, usually just the opposite.
    A Rose By Any Other Name
    Technically known as the Jarisch-Herxheimer Reaction, this syndrome goes by many names, including JHR, the Herxheimer Effect, the Herxheimer Response, a Herx Reaction, Herx or Herks. The most common terminology used is the Herxheimer Reaction. It is also often referred to as a healing crisis, a detox reaction, or die-off syndrome.
    History
    The phenomenon was first described by Adolf Jarisch (1860-1902) working in Vienna, Austria, and a few years later by Karl Herxheimer (1861-1942), working in Frankfort, Germany. Both doctors were dermatologists mainly treating syphilitic lesions of the skin. They noticed that in response to treatment, many patients developed not only fever, perspiration, night sweats, nausea and vomiting, but their skin lesions became larger and more inflamed before settling down and healing. Interestingly, they found that those who had the most extreme reactions healed the best and fastest. The patient might be ill for 2-3 days, but then their lesions resolved.
    A Medical Example
    The Herxheimer reaction is caused by the release of toxic chemicals (endotoxins) released from the cell walls of dying bacteria due to effective treatment. The Herxheimer Reaction is well recognized in medical circles and is certainly not confined to the world of natural medicine or supplements. –For example, a recent study report (Feb ’04) on the treatment of Sarcoidosis found that, “. . . without exception, the improving patients are reporting periodic aggravation of their symptoms as an apparent direct response to the antibiotics. In other words, these patients say that their treatment makes them feel much worse before they experience symptom-relief.” The abstract of the study goes on to say, “This phenomenon is known as the Jarisch-Herxheimer Reaction (JHR) and is often referred to informally as Herx. JHR is believed to be caused when injured or dead bacteria release their endotoxins into blood and tissues faster than the body can comfortably handle it. . . . This provokes a sudden and exaggerated inflammatory response . . . . In Sarcoidosis patients, the Herxheimer reaction seems to be a valuable indication that an antibiotic is reaching its target.” In the conclusion, the author states: “In my work with Sarcoidosis patients, it is my experience that recovering MP patients understand and welcome the Herxheimer reactions even when they must endure temporary increased suffering. They accept it as the price that they must pay in order to get well and they even seem to find it gratifying to experience tangible evidence of bacterial elimination. “
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    Not All Infant Formulas Are Alike– Differential Effects on Weight Gain
    ScienceDaily (Dec. 28, 2010) — New findings from the Monell Center reveal that weight gain of formula-fed infants is influenced by the type of formula the infant is consuming. The findings have implications related to the infant’s risk for the development of obesity, diabetes and other diseases later in life.—“Events early in life have long-term consequences on health and one of the most significant influences is early growth rate,” said study lead author Julie Mennella, Ph.D., a developmental psychobiologist at Monell. “We already know that formula-fed babies gain more weight than breast-fed babies. But we didn’t know whether this was true for all types of formula.”–While most infant formulas are cow’s milk-based, other choices include soy-based[U1] and protein hydrolysate-based formulas. Protein hydrolysate formulas contain pre-digested proteins and typically are fed to infants who cannot tolerate the intact proteins in other formulas.—In adults, pre-digested proteins are believed to act in the intestine to initiate the end of a meal, thus leading to smaller meals and intake of fewer calories. Based on this, the authors hypothesized that infants who were feeding protein hydrolysate formulas would eat less and have an altered growth pattern relative to infants feeding cow’s milk-based formula.–In the study, published online in the journal Pediatrics, infants whose parents had already decided to bottle-feed were randomly assigned at two weeks of age to feed either a cow’s milk-based formula (35 infants) or a protein hydrolysate formula (24 infants) for seven months.–Both formulas contained the same amount of calories, but the hydrolysate formula had more protein, including greater amounts of small peptides and free amino acids.—Infants were weighed once each month in the laboratory, where they also were videotaped consuming a meal of the assigned formula. The meal continued until the infant signaled that s/he was full.–Over the seven months of the study, the protein hydrolysate infants gained weight at a slower rate than infants fed cow milk formula. Linear growth, or length, did not differ between the two groups, demonstrating that the differences in growth were specifically attributable to weight.”All formulas are not alike,” said Mennella. “These two formulas have the same amount of calories, but differ considerably in terms of how they influence infant growth.”–When the data were compared to national norms for breast-fed infants, the rate of weight gain of protein hydrolysate infants was comparable to the breast milk standards; in contrast, infants fed cow’s milk formula gained weight at a greater rate than the same breast milk standards.–Analysis of the laboratory meal revealed the infants fed the protein hydrolysate formula consumed less formula during the meal.–“One of the reasons the protein hydrolysate infants had similar growth patterns to breast-fed infants, who are the gold standard, is that they consumed less formula during a feed as compared to infants fed cow’s milk formula” said Mennella. “The next question to ask is: Why do infants on cow’s milk formula overfeed?”–The findings highlight the need to understand the long-term influences of infant formula composition on feeding behavior, growth, and metabolic health. Future studies will utilize measures of energy metabolism and expenditure to examine how the individual formulas influence growth, and how each differs from breastfeeding.–Also contributing to the study, which was funded by the National Institute of Child Health and Human Development, were Monell scientists Gary Beauchamp and Alison Ventura.
    Story Source:–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Monell Chemical Senses Center, via EurekAlert!, a service of AAAS.–Journal Reference: Julie A. Mennella, Alison K. Ventura, and Gary K. Beauchamp. Differential Growth Patterns Among Healthy Infants Fed Protein Hydrolysate or Cow-Milk Formulas. Pediatrics, 2010; DOI: 10.1542/peds.2010-1675
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    Recipe for Formula making for adult of child
    —Get a Whey isolate or Whey Hydroslate 25-28 grams Unflavoured or Neutral or Natural and Add Mct 1-2 tablespoons –and add either vanilla or cocoa And sweeten with either Unpasteurized hone ot Maple syrup or Xylitol or Stevia ad 1-1 ½ cup of water all in a blender—you can substitute the cocoa for even fruit or almond extract or orange extract or add the cocoa and peppermint extract 1-3 drops of any of the extracts or 1 tablespoon of vanilla—blend for 4-5 minutes —this can be used as a pre meal for utilizing this to curb appetite —you can take it In between meals as well as a protein supplement—you can warm this and give it to infants but reduce the gram dosage to about ¼ -1/3 so 28 grams would be about 7-9 grams and reduce the MCT to 1 tablespoon
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    Google map of Bird and Fish Fall out
    http://maps.google.com/maps/ms?ie=UTF8&hl=en&oe=UTF8&msa=0&msid=201817256339889828327.0004991bca25af104a22b
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    FEAT
    “Healing Autism: No Finer a Cause on the Planet
    ______________________________________________________
    January 20, 2001 Search http://www.feat.org/search/news.asp
    [This Sunday Herald (UK) article is another treatment of the same MMR – vaccine story in previous newsletter post.] http://www.sundayherald.com/news/newsi.hts?section=News+Focus&amp;story_id=13820
    Amid claims that the MMR triple vaccine can cause autism, Dr Andrew Wakefield insists in a new report that it should never have got a licence–Five years ago, Dr Andrew Wakefield was approached by parents who told him a tragic story. Their children had been developing normally until the toddlers were given the vaccine for measles, mumps and rubella, MMR. The parents said they then witnessed a regression in their children’s behaviour and speech, as well as the onset of an unpleasant bowel disorder. Presented with similar accounts by numerous sets of parents, the London doctor decided to investigate. What he discovered was to provoke one of the most bitter vaccination controversies ever. Today, he has a paper published in the Journal of Adverse Drug Reactions. It concludes that the vaccine for measles, mumps and rubella should not have been licensed in 1988 because inadequate research had been carried out. The conclusion is backed up by four senior figures in medicine regulation, including Dr Peter Fletcher, a former senior scientific civil servant and assessor to the Committee on Safety of Medicines. They say children given the vaccine in trials should have been monitored for much longer because adverse reactions could have taken several months to show.–They also say there was little information on the effects of giving three live vaccines together, compared with giving them separately. Wakefield’s original theory was published in The Lancet in 1998. He then extended his work and further findings were published in the American Journal of Gastroenterology. His most up-to-date work on 170 children is soon to be published.—Last year, Professor John O’Leary, director of pathology at Coombe Women’s Hospital in Dublin, uncovered evidence pointing to a link between the MMR vaccine and autism in children. His findings, presented to a US Congressional hearing earlier this month, backed up Wakefield’s Lancet study. But, despite having been peer-reviewed, other experts insist there is no evidence to suggest a link. A three-year war has ensued between Department of Health scientists, who are struggling to maintain confidence in the vaccination programme , and researchers such as Wakefield and O’Leary. Speaking from his London home, Wakefield explained that his duty is to his patients, regardless of how much his findings upset the Department of Health. He said: “At first, we were sceptical but the parents’ story was so consistent that we felt we had to investigate. When the parent tells you they believe the problems started after exposure to the MMR vaccine, do you say, ‘That is very interesting but politically it makes me very uncomfortable’? -“I represent patients whose parents say to me, ‘This is what happened to my child: At one point, my child was developing normally, then they encountered the MMR and they developed autism.’ They say their children became badly behaved, developed terrible bowel problems and their communication regressed. They said there is an epidemic of this problem but the medical profession told them they were wrong.–“We had to take their story at face value and to investigate it. What we found, to our surprise, was that the parents were right.” Government scientists have heavily criticised Wakefield and his research. They claim his science has not been reproduced by either himself or other scientists – an allegation he disputes. —The hostilities penetrate down to the hospital where he works, the Royal Free in London. While Wakefield is carrying out his research, Professor Brent Taylor – head of the department of paediatrics and child health at the hospital – does his best to convince the public that the link does not exist. Professor Taylor told the Sunday Herald: “There are no serious side- effects and the MMR does not cause autism and inflammatory bowel disease. Nobody anywhere else in the world has been able to reproduce any of Wakefield’s studies. “Separate vaccines do not provide good protection for children. We do not want a new programme that could result in serious diseases which kill and maim people.”–The publication of a paper in the Journal of Adverse Drug Reactions saying that the MMR should never have been licensed caused a damaging row between the editor and the head of immunisation in Scotland. Dr Ian Jones, director of the Scottish Centre for Infection and Environmental Health, wrote to the editor, Dr John Griffin, suggesting that the journal should not publish the controversial paper. This prompted Griffin to accuse Jones of trying to censor the journal.–Wakefield’s claims that a major government vaccination programme is causing children to become autistic are so damaging that his trust usually bans him from speaking to the media. Whenever the MMR debate is re fuelled by new claims, receptionists at the Royal Free Hospital are put on alert and any journalists asking to speak to the doctor are redirected to the trust’s official spokesman.–But, this weekend, Wakefield has been given permission to discuss his latest research. He is adamant that, despite being the subject of bitter personal criticism, his arguments will be based on his science. He does, however, express regret that the strength of government opposition is hindering the research.–He said: “I don’t want to moan about what has happened to me. We have got to fight this on the science. You go into this sort of thing with your eyes open. Yes, you are taking on your colleagues, the health department and the biggest drug companies in the world but the question you have to ask is, ‘Who do you represent?’ “My responsibility is to the patient and not to the Department of Health. I am not going to decide against investigating something because of the possible consequences for the Department of Health, because it may make things difficult for them. “The frustration is more that we are restricted in our attempts to help the children in the way that we would like to. It is becoming incredibly difficult to get funding for further investigations and clinical trials.”Discussing the paper to be published today, Wakefield points out that while the government scientists had until now directed their criticism at him, they must admit that they are also criticising former members of their own department.He said: “Government scientists were critical of our science but the work was peer-reviewed, unlike other papers, because of the controversial nature of the subject. This paper went to four reviewers who agreed to have their comments published. “We agreed to have that done not knowing what they would say and that is the way that science should be conducted. They have come out being very supportive. These are people who are not anti-vaccine but eminent people involved in drug regulation. The regulators themselves are saying that the vaccine was not safe.”-He added: “If they want to attack me, that’s fine but now they are bringing in four eminent regulators. Are they going to denounce the very valid opinions of their own colleagues?” Wakefield first suggested in 1998 that single vaccines should be made available on the basis of the fears of a link between the MMR and autism and inflammatory bowel disease. He says the findings of the latest report reinforces the need to make mono valent vaccines available now.–He said: “It is absolutely no good to come out with tired rhetoric claiming that the vaccine has been proven to be safe. I am afraid that that is not the case. Until this is resolved, single vaccines must be made available.–“The concerns have been dismissed by the Department of Health but doctors, practice nurses and health visitors are changing their minds. “I have referrals at the moment from paediatricians who believe their patients have been damaged by the MMR. We get this time and time again. The change in attitude is very telling.”–In May last year, Wakefield sent a copy of his latest paper to Dr Liam Donaldson, the Chief Medical Officer for England. He said: “I was giving them the option to provide supplies of monovalent vaccines because I did expect there to be a scare. I did that knowing that they could use it against us by attempting to underm
    ine the paper but it would have been morally wrong just to let it appear on the news stands on Monday. What they chose to do with that information I do not know.”—Wakefield suggests vaccinating against measles at 15 months, against mumps when the child is three years old and against rubella when the child is four.–“What they are saying is that I could be responsible for an epidemic – children would suffer and some may die. But my point of view is pro vaccine. Nobody is saying, ‘Do not protect children against measles.’ This is about the safest way to do it. There is no need for measles to come back if we vaccinate against measles at 15 months. “I have been honest and have published data whether it is positive or negative. Thus far, the parents have been absolutely right and I am obliged to continue to take this to its natural conclusion. In the meantime, I think parents deserve the option of single vaccines.”–This month, the Scottish parliament has come under increasing pressure from MSPs to reintroduce single vaccines. Wakefield hopes that this may become a reality. “The Scottish parliament has a unique opportunity, having devolved from the power of Whitehall, to take important decisions. The people are closer to their representatives, who seem prepared to listen.”
     
    TOP B
    [U1]MOST are that today rarely do you see a dairy based formula with out soy or aspartame or canola in the mix
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    Show of the Week January 14 2011
     
    Making a Laundry Soap—Clean Soap
     
    Study Probes Obesity Link to Fibromyalgia
     
    Tablet Splitting Is a Highly Inaccurate and Potentially Dangerous Practice
     
    Scientist Shows Link Between Diet and Onset of Mental Illness
     
    HRT=Misery
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    Making a Laundry Soap—Clean Soap
     
     
    Making a Laundry Soap—Clean Soap
    You will need one bar of lye soap
    You will need 1 cup of borax
    You will need ½ cup of baking soda
    And water and a 5 gallon container
     
    Shred the soap with a grader and pour the contents into a pot adequately sized
    With water to take the pulverized soap—then add new water to the 5 gallon container half way with water—then add the liquefied soap water from the pot into the container and whisk together with a whisker—add the 1 cup of borax and add the ½ cup of baking soda ( you can add an essential oil for scent if you like such as lime –lemon –orange etc) whisk again til completely mixed-then allow to set over night—it will cogeal ( solidify like jello) then take a 2 litre ( quart ) container fill with water half way with water—then take the whisker and mix the jello soap till liquid again and pour to top off the container—can be used for clothes using 2-3 oz at a time –can be used as well for doing dishes—Will make 5 gallons for a bout 5 dollar total investment –so for about 2.50 you can make 5 gallons of cleaning soap

    #2079
    AvatarEK
    Keymaster

    Study Probes Obesity Link to Fibromyalgia
    ScienceDaily (Jan. 1, 2011) — Afflicting up to 5 percent of the U.S. population, mostly women, fibromyalgia is characterized by widespread pain and range of function problems. A new study in The Journal of Pain reports there is close association between obesity and disability in fibromyalgia patients.—The purpose of the study, conducted by University of Utah researchers, was to evaluate the relationship between fibromyalgia and obesity. They hypothesized that obesity significantly adds to the disease and disability burden of the condition. Two hundred fifteen fibromyalgia patients were evaluated in the study and given several physical tests to measure strength, flexibility, range of motion, and strength. Heart rates and sleep quality also were assessed.—The authors reported that consistent with previous studies, obesity is common among those with fibromyalgia. Half the study sample was obese and an additional thirty percent were overweight. Also consistent with previous findings, obese patients in this study showed increased pain sensitivity, which was more pronounced in lower body areas. The obese patients also had impaired flexibility in the lower body and reduced strength.–The study concluded that obesity is a common comorbidity of fibromyalgia that may compromise clinical outcomes. The adverse impact of obesity is evidenced by hyperalgesia, disability, impaired quality of life and sleep problems. The authors also noted that recent evidence suggests weight loss improves fibromyalgia symptoms, perhaps resulting from patients adopting healthier lifestyles and taking more positive attitudes toward symptom management, and overall quality of life. Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by American Pain Society.–Journal Reference:–Akiko Okifuji, Gary W. Donaldson, Lynn Barck, Perry G. Fine. Relationship Between Fibromyalgia and Obesity in Pain, Function, Mood, and Sleep. The Journal of Pain, 2010; 11 (12): 1329 DOI: 10.1016/j.jpain.2010.03.006
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    Tablet Splitting Is a Highly Inaccurate and Potentially Dangerous Practice
     
    ScienceDaily (Jan. 7, 2011) — Medical experts have issued a warning about the common practice of tablet splitting, after a study found that nearly a third of the split fragments deviated from recommended dosages by 15 per cent or more.–Their study, published in the January issue of the Journal of Advanced Nursing, points out that the practice could have serious clinical consequences for tablets that have a narrow margin between therapeutic and toxic doses.–And they are calling on manufacturers to produce greater dose options and liquid alternatives to make the practice unnecessary.–Researchers from the Faculty of Pharmaceutical Sciences at Ghent University, Belgium, asked five volunteers to split eight different-sized tablets using three techniques commonly used in nursing homes.–They found that 31 per cent of the tablet fragments deviated from their theoretical weight by more than 15 per cent and that 14 per cent deviated by more than 25 per cent. Even the most accurate method produced error margins of 21 per cent and eight per cent respectively. “Tablet-splitting is widespread in all healthcare sectors and a primary care study in Germany found that just under a quarter of all drugs were split” says study lead Dr Charlotte Verrue.–“It is done for a number of reasons: to increase dose flexibility, to make tablets easier to swallow and to save money for both patients and healthcare providers. However, the split tablets are often unequal sizes and a substantial amount of the tablet can be lost during splitting.”—The five researchers comprised a pharmacy student, researcher and professor, an administrative worker and a laboratory technician, ranging from 21 to 55 years of age. With the exception of the technician, none of the other study participants had tablet-splitting experience. The authors believe this replicated nursing home conditions where splitting is not always performed by professional nurses.–Between them they split tablets into 3,600 separate quarters or halves using a splitting device, scissors and a kitchen knife. The eight different tablets were different shapes and sizes, three were unscored, three had one score line and the others had two.—The drugs were prescribed for a range of health conditions, including Parkinson’s, congestive heart failure, thrombosis and arthritis.—After splitting, each fragment was weighed to see how much they deviated from the theoretical weight.
    Key results included:
    Using a splitting device was the most accurate method. It still produced a 15 to 25 per cent error margin in 13 per cent of cases, but this was lower than the 22 per cent for scissors and the 17 per cent for the knife.
    The splitting device produced a deviation of more than 25 per cent in eight per cent of cases, compared with 19 per cent for the scissors and 17 per cent for the knife.
    Some drugs were much easier to split accurately than others. The easiest to split produced an overall error margin (15 per cent deviation or more) of two percent and the most difficult tablets produced an error margin of 19 per cent.
    “Tablet splitting is daily practice in nursing homes” says Dr Verrue. “However, not all formulations are suitable for splitting and, even when they are, large dose deviations or weight losses can occur. This could have serious clinical consequences for drugs where there is a small difference between therapeutic and toxic doses.—“Based on our results, we recommend use of a splitting device when splitting cannot be avoided, for example when the prescribed dose is not commercially available or where there is no alternative formulation, such as a liquid.–“Staff who are responsible for splitting tablets should receive training to enable them to split as accurately as possible. They should also be made aware of the possible clinical consequences of dose deviations.–“We would also like to see manufacturers introduce a wider range of tablet doses or liquid formulations so that tablet splitting becomes increasingly unnecessary.”Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Wiley – Blackwell, via AlphaGalileo.–Journal Reference–Charlotte Verrue, Els Mehuys, Koen Boussery, Jean-Paul Remon, Mirko Petrovic. Tablet-splitting: a common yet not so innocent practice. Journal of Advanced Nursing, 2011; 67 (1): 26 DOI: 10.1111/j.1365-2648.2010.05477.x
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    Scientist Shows Link Between Diet and Onset of Mental Illness
    ScienceDaily (Dec. 13, 2010) — Changes in diet have been linked to a reduction of abnormal behaviors in mentally ill people or animals, but a Purdue University study shows that diet might also trigger the onset of mental illness in the first place.–Joseph Garner, an associate professor of animal sciences, fed mice a diet high in sugar and tryptophan that was expected to reduce abnormal hair-pulling. Instead, mice that were already ill worsened their hair-pulling behaviors or started a new self-injurious scratching behavior, and the seemingly healthy mice developed the same abnormal behaviors.–“This strain of mouse is predisposed to being either a scratcher or a hair-puller. Giving them this diet brought out those predispositions,” said Garner, whose results were published in the December issue of the journal Nutritional Neuroscience. “They’re like genetically at-risk people.”–Garner studies trichotillomania, an impulse-control disorder in which people pull out their hair. The disorder, which disproportionately occurs in women, is thought to affect between 2 percent and 4 percent of the population. Mice that barber, or pull their hair out, have been shown to have low levels of serotonin activity in the brain. That neurotransmitter is known to affect mood and impulses. Garner hypothesized that increasing serotonin activity in the brain might cure or reduce barbering and possibly trichotillomania.–Serotonin is manufactured in the brain from the amino acid tryptophan, which is consumed in diets. The problem is that tryptophan often doesn’t make it across the barrier between blood and the brain because other amino acids can get through more easily and essentially block the door for tryptophan. Garner modified a mouse diet to increase simple carbohydrates, or sugars, and tryptophan. The sugars trigger a release of insulin, which causes muscles to absorb those other amino acids and gives tryptophan a chance to make it to the brain.–Using eight times as much sugar and four times as much tryptophan, Garner observed a doubling of serotonin activity in the brain. But the mice that barbered did not get better.—“We put them on this diet, and it made them much, much worse,” Garner said.–A second experiment divided the mice into three groups: those that were seemingly normal, others that had some hair loss due to barbering and a group that had severe hair loss. All the mice soon got worse, with conditions escalating over time.–“Three-quarters of the mice that were ostensibly healthy developed one of the behaviors after 12 weeks on the new diet,” Garner said.–Some of the mice developed ulcerated dermatitis, a fatal skin condition thought to be caused by an unidentified pathogen or allergen. Garner saw that the only mice that contracted the condition were the scratchers.—“What if ulcerated dermatitis, like skin-picking, another common behavioral disorder, is not really a skin disease at all?” Garner said. “We now have evidence that it may be a behavioral disorder instead.” When taken off the new diet, the negative behaviors stopped developing in the mice. When control mice were switched to the new diet, they started scratching and barbering.—Garner’s study raises questions of how diet might be affecting other behavioral or mental illnesses such as autism, Tourette syndrome, trichotillomania and skin-picking. He said that before now, a link between diet and the onset of mental disorders hadn’t been shown.–“What if the increase of simple sugars in the American diet is contributing to the increase of these diseases?” Garner said. “Because we fed the mice more tryptophan than in the typical human diet, this experiment doesn’t show that, but it certainly makes it a possibility.”—Garner next wants to refine the experiments to better imitate human dietary habits, including the amount of tryptophan people consume. Internal Purdue funding paid for his work. Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Purdue University, via EurekAlert!, a service of AAAS. Journal Reference–Brett D. Dufour, Olayiwola Adeola, Heng-Wei Cheng, Shawn S. Donkin, Jon D. Klein, Edmond A. Pajor, Joseph P. Garner. Nutritional up-regulation of serotonin paradoxically induces compulsive behavior. Nutritional Neuroscience, 2010; 13 (6): 256 DOI: 10.1179/147683010X12611460764688
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    HRT=Misery
     
    Potential ‘Safe Period’ For Hormone Replacement Use Identified
    ScienceDaily (Feb. 8, 2009) — A new study makes important new findings on the role of hormone use on the risk of breast cancer, confirming that the use of estrogen plus progesterone increases the risk of both ductal and lobular breast cancer far more than estrogen-only; suggesting a two-year “safe” period for the use of estrogen and progesterone; and finding that the increased risk for ductal cancers observed in long-term past users of hormone replacement therapy drops off substantially two years after hormone use is stopped.–Previous studies have shown that hormone replacement therapy after menopause increases the risk of breast cancer and that use of a regimen that includes both estrogen and progesterone is more detrimental for the breast than the use of estrogen alone. But more data from large prospective studies are needed to fully characterize the impact of exogenous hormones (Exogenousor exogeneous) refers to an action or object coming from outside a system. It is the opposite of endogenous, something generated from within the system ) on breast cancer incidence by type of hormone preparation and histology of the cancer.—To investigate the association in more detail, American Cancer Society epidemiologists led by Eugenia E. Calle, PhD, did a prospective study of 68,369 postmenopausal women who were cancer-free at baseline in 1992. They examined the use of estrogen-only and estrogen and progesterone in current and former users of varying duration, and the subsequent risk of developing invasive ductal and lobular carcinoma of the breast. They also looked at whether the risk for each type of breast cancer and each type of hormone regimen varied by body mass index (BMI), stage of disease at diagnosis, and estrogen receptor (ER) and progesterone receptor (PR) status. For the present study, the follow-up period ended on June 30, 2005.—They confirmed the findings from previous work that estrogen and progesterone increases the risk of both ductal and lobular breast cancer far more estrogen alone. They also found the risk associated with use of estrogen and progesterone increases significantly and substantially within three years of beginning hormone use. The data showed no increased risk for women who used estrogen and progesterone for less than two years, potentially identifying a “safe” period for estrogen and progesterone use.—The study also found no increased risk of breast cancer in women who had stopped using estrogen and progesterone two or more years ago, suggesting a window of two to three years for the risks of estrogen and progesterone both to become apparent after initial use and to diminish after cessation. Few estimates of risk within two to three years of initiation and cessation are available, so these findings need replication in other large studies. The study found the use of estrogen and progesterone was associated with a doubling of risk of lobular cancer after three years of use, and a doubling of risk of ductal cancer with 10 years of use. Estrogen-only use was not associated with increased risk of ductal cancer, even after 20 years of use, but was associated with a 50 percent increase in risk of lobular cancer after 10 years of use.—Story Source: The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by American Cancer Society, via EurekAlert!, a service of AAAS. — Journal Reference: –Eugenia E. Calle et al. —Postmenopausal hormone use and breast cancer associations differ by hormone regimen and histologic subtype. Cancer, Published Online: January 20, 2008 DOI: 10.1002/cncr.24101
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    Long-Term Estrogen Therapy Linked To Breast Cancer Risk
    ScienceDaily (May 9, 2006) — Long-term estrogen therapy may be related to a higher risk of breast cancer among postmenopausal women who have had a hysterectomy, according to an article in the May 8 issue of Archives of Internal Medicine, one of the JAMA/Archives journals. —Previous studies have linked the use of hormone therapy to breast cancer among postmenopausal women, but have primarily focused on the hormone combination of estrogen plus progestin, according to background information in the article. Recently released results from the Women’s Health Initiative (WHI), a large clinical trial of hormone therapy, found no significant link between estrogen therapy and breast cancer in women who took the hormone for seven years. —Wendy Y. Chen, M.D., M.P.H., Brigham and Women’s Hospital and Dana Farber Cancer Institute, Boston, and colleagues evaluated women who were part of the Nurses’ Health Study, a group of female nurses that have been followed since 1976. In 1980, 11,508 women from the study were postmenopausal and had had a hysterectomy. Every two years the researchers enrolled all the additional women who become postmenopausal and had a hysterectomy, so 28,835 women were included by the end of the study in 2002. Women were asked by questionnaire every two years if they used hormones and whether they had developed breast cancer. For women who developed breast cancer, the researchers obtained permission to review the women’s medical records, which they used to record the hormone receptor information. Tumors were classified as positive or negative for estrogen receptor or progesterone receptor based on how they responded to specific hormonal therapies. —Throughout the study period, 934 invasive breast cancers developed, 226 among women who had never used hormones and 708 among women who were using estrogen at the time. The longer a woman used estrogen, the higher her risk of breast cancer. Those who had been taking estrogen for fewer than 10 years did not appear to have a higher risk than those who had never taken hormones, but those who had been taking estrogen for more than 20 years had a significantly increased risk. The association was strongest for cancers that were estrogen receptor positive and progesterone receptor positive. The results were similar when the researchers evaluated only women who were older than age 60; only women who had begun estrogen therapy after reaching age 50; and only women who were at least age 50 and had undergone a hysterectomy, even if they had not gone through menopause. —“In conclusion, we found that estrogen therapy was associated with an increased risk of breast cancer with longer-term use,” the authors write. “Although current use of estrogen therapy for less than 10 years was not associated with a statistically significant increase in breast cancer risk, the WHI has shown an increased risk of stroke and deep-vein thrombosis in the same time period. Women who take estrogen therapy for prevention or treatment of osteoporosis typically require longer-term treatment and should thus explore other options, given the increased risk of breast cancer with longer-term use.”—(Arch Intern Med. 2006; 166: 1027-1032. Available pre-embargo to media at http://www.jamamedia.org.) –Editor’s Note: This study was supported by a grant from the National Institutes of Health, Bethesda, Md. —Story Source: The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by JAMA and Archives Journals, via EurekAlert!, a service of AAAS —
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    Estrogen Therapy May Be Associated With Kidney Stones in Postmenopausal Women
     
    ScienceDaily (Oct. 12, 2010) — Use of estrogen therapy is associated with an increased risk of developing kidney stones in postmenopausal women, according to a report in the October 11 issue of Archives of Internal Medicine, one of the JAMA/Archives journals.– “Nephrolithiasis [kidney stones] is a common condition that affects 5 percent to 7 percent of postmenopausal women in the United States,” according to background information in the article. “Because the process of kidney stone formation is influenced by a variety of lifestyle and other health-related factors, the true impact of estrogen therapy on the risk of kidney stone formation is difficult to infer from observational studies.” — Using data from the national Women’s Health Initiative study, Naim M. Maalouf, M.D., of the University of Texas Southwestern Medical Center, Dallas, examined data from two trials: 10,739 postmenopausal women with hysterectomy who received either an estrogen-only treatment or matching placebo and 16,608 postmenopausal women without hysterectomy who received either an estrogen plus progestin treatment or matching placebo. Data were collected for an average of 7.1 years in the estrogen-only trial and 5.6 years for the estrogen plus progestin trial. — A total of 335 cases of kidney stones were reported in the active treatment groups, while 284 cases occurred in the placebo groups. The beginning demographic characteristics and risk factors for kidney stones were similar in the two groups, and the authors found that estrogen therapy was associated with a significant increase in risk of kidney stones. The corresponding annualized incidence rate per 10,000 women per year was 39 in the treatment group and 34 in the placebo group. Development of kidney stones was five times more common in women with a history of kidney stones at the beginning of the study, but was not significantly altered by estrogen therapy. In this trial, estrogen therapy increased the risk of development of kidney stones irrespective of age, ethnicity, body mass index, prior hormone therapy use or use of coffee or thiazide diuretics.The authors conclude that their results “indicate that estrogen therapy increases the risk of nephrolithiasis in healthy postmenopausal women. The mechanisms underlying this higher propensity remain to be determined. In view of the sizable prevalence of nephrolithiasis in this segment of the population, these findings need to be considered in the decision-making process regarding postmenopausal n use.”—Story Source:–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by JAMA and Archives Journals.–Journal Reference: N. M. Maalouf, A. H. Sato, B. J. Welch, B. V. Howard, B. B. Cochrane, K. Sakhaee, J. A. Robbins. Postmenopausal Hormone Use and the Risk of Nephrolithiasis: Results From the Women’s Health Initiative Hormone Therapy Trials. Archives of Internal Medicine, 2010; 170 (18): 1678 DOI: 10.1001/archinternmed.2010.342
     
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    Show of the Week January 17 2011
     
    Garlic– Recipe for Garlic and Uses- Garlic & Lecithin Recipe
     
    Dietary zinc and prostate cancer survival in a Swedish cohort1
    Digested green tea compounds show dementia and cancer benefits
     
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    Garlic
     
    English _ Garlic.
    Ayurvedic _ Lashuna, Rasona,
    Yavaneshta, Ugragandha, Mahaushadh,
    Arishta.
    Unani _ Seer, Lahsun.
    Siddha/Tamil _ Ullippoondu, Vellaippondu.
    Action _ Antibiotic, bacteriostatic, fungicide, anthelmintic, antithrombic, hypotensive, hypoglycaemic, hypocholesterolaemic. Also used for upper respiratory tract infections and catarrhal conditions.
    Key application _ As a supportive to dietary measures for elevated levels
    of lipids in blood; as a preventive measure for age-dependent vascular
    changes. (German Commission E, ESCOP, WHO, The British Herbal
    Pharmacopoeia.) Also as an antimicrobial. (The British Herbal
    Pharmacopoeia). Garlic has been shown to be effective in respiratory
    infections and catarrhal conditions. (The British Herbal Compendium.)
    The Ayurvedic Pharmacopoeia of India indicates the use of the bulb as
    a brain tonic in epilepsy and psychic disorders. Heavy consumption of garlic prior to surgery led to increased clotting time or reduced platelet aggregation (in human case reports). Garlic tablets at a dose of 400 mg twice daily for 12 weeks reduced platelet aggregation 59% compared with placebo in 80 patients (in human clinical study). (Francis Brinker.) Garlic cloves are high in sulphurcontaining amino acids known as alliin (no taste, no smell, no medicinal action). With crushing or chewing alliin comes into contact with the enzyme alliinase. Alliinase, in less than 6 s, transforms alliin into allicin (strongly medicinal), which breaks down into a number of sulphur compounds including ajoene, vinyldithin and diallyl disulfide, and trisulfide. The antibiotic effect is attributed to allicin; hypoglycaemic effect to allicin and allylprophyldisulphide (also to S-allyl Cysteine sulfoxide); anticarcinogenic activity to diallyl monosulfide; platelet aggregation inhibitory effect to diallyl-di- and tri-sulphides. Ajoene inactivated human gastric lipase, which is involved in digestion and absorption of dietary fats. Diallyltetra, penta-, hexa- and heptasulphides are potential antioxidants. AlliumleptophyllumWall. is equated with Vana Lashuna, Jangali Lahsun. Dosage _1 Bulb=3 g
    Garlic inhibits the activity of 5-Lipoxygenase.
    Lipids–Garlic inhibits the production of Prostaglandin F2 alpha (PGF2 alpha). Garlic inhibits the production of Prostaglandin I2 (PGI2). Garlic suppresses the production of Thromboxane A2 (TXA2).
    Minerals–Garlic helps to prevent the cellular damage caused by excessive Arsenic ingestion.
    Garlic facilitates the removal of accumulated Cadmium from the body. Garlic facilitates the removal of accumulated Lead from the body. Garlic facilitates the removal of accumulated Mercury from the body.
    Nucleic Compounds–Garlic inhibits the incorporation of Thymidine into the Deoxyribonucleic Acid (DNA) of many types of Cancer Cells (and thereby inhibits the ability of Thymidine to function as a growth factor for some types of Cancer Cells
    Garlic increases Glutathione Reductase levels.
    Garlic activates Nitric Oxide Synthase (NOS).
    Hormones—Garlic increases plasma Luteinizing Hormone (LH) levels (due to the Diallyl Disulfide content of Garlic).
    Garlic increases Testosterone levels (due to the Diallyl Disulfide content of Garlic increasing Luteinizing Hormone (LH) levels.
    Neurotransmitters–Garlic increases plasma Norepinephrine levels
    Garlic protects Chromosomes from the damage caused by exposure to Gamma-Rays
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    Aged garlic extract delays the appearance of infarct area in a cerebral ischemia model, an effect likely conditioned by the cellular antioxidant systems.
    Phytomedicine. 2010 Mar;17(3-4):241-7
    Authors: Aguilera P, Chánez-Cárdenas ME, Ortiz-Plata A, León-Aparicio D, Barrera D, Espinoza-Rojo M, Villeda-Hernández J, Sánchez-García A, Maldonado PD
    Experimental evidence has shown that some garlic-derived products have a protective effect against ischemic brain injury. The present study was designed to investigate the effect of aged garlic extract (AGE), establish the therapeutic window, and determine its protective mechanism in a cerebral ischemia model. Animals were subjected to middle cerebral artery occlusion (MCAO) for 2h and treated with 1.2ml/kg body wt.(i.p.) of AGE 30min before, at the beginning of (0R), or 1h after reperfusion. The 0R treatment significantly reduced the size of the infarct area after 2h of reperfusion. Repeated doses subsequent to the 0R treatment (at 1, 2, or 3h after reperfusion) had no effect on the temporal window of protection. The protective 0R treatment with AGE prevented the increase in nitrotyrosine and the decrease in total superoxide dismutase, glutathione peroxidase, and extracellular superoxide dismutase activities induced by MCAO. These data indicate that AGE delays the effects of ischemia/reperfusion-induced neuronal injury. However, this treatment itself was not associated with a noticeable improvement in the neurological outcome, or with an effect on the inflammatory response. We conclude that the neuroprotective effect of AGE in the 0R treatment might be associated with control of the free-radical burst induced by reperfusion, preservation of antioxidant enzyme activity, and the delay of other pathophysiological processes.–PMID: 19577455 [PubMed – indexed for MEDLINE]
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    Recipe for Garlic and Uses—Take 2 bulbs of garlic and add to blender ( peeled) the add 1 ½ cup of vinegar ( any will do ) then blend for 10 minutes and strain Put into glass container—then take 2 bulbs of garlic ( peeled) and add to a oil or fat again blend for about 10 minutes –strain the mix so only the oil comes out add to glass container –then take these specific vitamins –zinc ( use approximately 100mgs ) Selenium ( 1 mg ) B1 ( 600mgs) then add equal parts of the oil and the vinegar mixes of garlic ( do not use the whole amounts approximately 1-2 oz when doing this of each of the mixes ) blend for 5 minutes then pour this into a seperate container –and use ¼ tsp increments
    RThis will have sugar regulating effects—chelating effects ( removing- lead cadmium-mercury-arsenic) Anti Cancer Impact- Strength and endurance increase-anti inflammatory effect-reproductive aid-healing factor-Brain functioning improvement—heart health-liver health-pancreatic support-wound healing properties-Protects against alcohol poisoning—anti glycating of cells-Anti Aging-Antioxidant-Anti Microbial-Anti Bacterial-Anti Fungal—Cholesterol regulating
    RThe Garlic vinegar can be utilized as well independently as a tonic –healant—in cooking –as a protectant against differing pathogens in foods-again as a digestive aid-cholesterol impacting and as well has all the other attributes associated with vinegar and garlic-use 1 teaspon as neded or 1 X 3 aday
    RThe garlic oil can as well be utilized like the vinegar –use it ½ tsp increments
    RGarlic & Lecithin Recipe—you will need lecithin( sunflower or egg yolk not soy) 1 table spoon—1 whole bulb of garlic-1/4 cup of garlic vinegar-1/4 cup of garlic oil—then add zinc citrate 300mgs ( open capsule ) 600mgs of B1 ( open capsule) selenium 1 mg ( open either 5 capsules at 200 mcg strength or 10 capsules at 100mg strength ) if you like add cq 10 to this 200 mgs ( again depending on capsule strength open the appropriate amount ) and again you can increase these or decrease as you see fit—add all to blender and blend for 10 minutes at medium to high speed—add garlic oil or garlic vinegar if needed if to thick—when done pour into a glass bottle and use ½ tsp several times a day—the impact will be noticeable on the legs a renewed strength will be noticed—this will again impact the Norepinephrine levels of the brain ( flight or fright—clear thinking—cognitive ability-sex drive) cellular health and increases immune health—liver health—cholesterol regulating anti cancer—anti fungal-anti viral-anti bacterial-anti microbial—general tonic as well—can be used on a spread –in yogurt—on foods –taken straight—use ½ tsp several times a day
    RThese can be spread on your eggs –integrated in your cooking oils can be used in soups and have the benefits as well as protective properties-mixed in salads or salad dressings
    RThe garlic mix with the combo of supplements and vinegar can be used as a supplement—a tonic—immune restorer—immune supporter and again ¼ tsp 1 X3 a day or as needed
     
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    Dietary zinc and prostate cancer survival in a Swedish cohort1,2,3
     
    1. Mara M Epstein, Julie L Kasperzyk, Ove Andrén, Edward L Giovannucci, Alicja Wolk, Niclas Håkansson, Swen-Olof Andersson,
    2. Jan-Erik Johansson, Katja Fall, and Lorelei A Mucci
    + Author Affiliations
    1. 1From the Departments of Epidemiology (MME, JLK, ELG, KF, and LAM) and Nutrition (ELG), Harvard School of Public Health, Boston, MA; the Channing Laboratory, Department of Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, MA (MME, JLK, ELG, and LAM); the Örebro University Hospital, Örebro, Sweden (OA, S-OA, and J-EJ); The Institute of Environmental Medicine, Karolinska Institutet, Stockholm, Sweden (AW and NH); the Department of Genetics and Pathology, Uppsala University Hospital, Uppsala, Sweden (KF); and The Centre for Public Health Services, University of Iceland, Reykjavik, Iceland (KF and LAM).
    · ↵2 Supported by the National Institutes of Health research training grant R25CA098566 (to MME).
    · ↵3 Address reprint requests and correspondence to MM Epstein, Channing Laboratory, Brigham and Women’s Hospital, 181 Longwood Avenue, 3rd Floor, Boston, MA 02115. E-mail: [email protected]
    Abstract
    Background: Zinc is involved in many essential cellular functions, including DNA repair and immune system maintenance. Although experimental evidence supports a role for zinc in prostate carcinogenesis, epidemiologic data are inconsistent; no data on cancer-specific survival have been reported. –Objective: Our objective was to determine whether dietary zinc assessed near the time of prostate cancer diagnosis is associated with improved disease-specific survival. –Design: This population-based cohort consists of 525 men aged <80 y from Örebro County, Sweden, with a diagnosis of prostate cancer made between 1989 and 1994. Study participants completed self-administered food-frequency questionnaires, and zinc intake was derived from nutrient databases. Cox proportional hazards regression was used to estimate multivariate hazard ratios (HRs) and 95% CIs for time to death from prostate cancer as well as death from all causes through February 2009 by quartile (Q) of dietary zinc intake. Models were also stratified by disease stage at diagnosis (localized or advanced). –Results: With a median follow-up of 6.4 y, 218 (42%) men died of prostate cancer and 257 (49%) died of other causes. High dietary zinc intake was associated with a reduced risk of prostate cancer–specific mortality (HRQ4 vs Q1: 0.64; 95% CI: 0.44, 0.94; P for trend = 0.05) in the study population. The association was stronger in men with localized tumors (HR: 0.24; 95% CI: 0.09, 0.66; P for trend = 0.005). Zinc intake was not associated with mortality from other causes. -Conclusion: These results suggest that high dietary intake of zinc is associated with lower prostate cancer–specific mortality after diagnosis, particularly in men with localized disease
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    Digested green tea compounds show dementia and cancer benefits
    Digested polyphenol compounds from green tea could protect the brain against developing Alzheimer’s and other forms of dementia,
    The in vitro study, published in Phytomedicine, confirmed that post-digestion, extracts of the phytochemical rich drink shows protective effects for dementia, and could play an important role in protecting the body against cancer. –The researchers investigated whether the protective properties of green tea – which have previously been shown to be present in the undigested, freshly brewed form of the drink – were still ‘active’ once the drink had been digested. –Lead researcher Dr Ed Okello from Newcastle University, U.K.said that just because a consumed food is generally accepted to contain health-boosting properties; it should not be assumed that such compounds will ever be absorbed in the body. –“What was really exciting about this study was that we found when green tea is digested by enzymes in the gut, the resulting chemicals are actually more effective against key triggers of Alzheimer’s development than the undigested form of the tea,” explained Okello –“In addition to this, we also found the digested compounds had anti-cancer properties, significantly slowing down the growth of the tumour cells which we were using in our experiments,” added Okello.
    Dementia
    Alzheimer’s disease – the most common form of dementia – is a progressive and irreversible neurodegenerative disorder associated with cognitive dysfunction. The authors noted that “mounting evidence” suggests that beta-amyloid peptides in conjunction with free radical species (such as hydrogen peroxide) in the brain play a significant role in the development and pathogenesis of Alzheimer’s. Many previous studies, have suggested that both black and green teas possess protective properties, which have been mainly attributed to their polyphenol content. —Green tea is high in flavan-3-ols, which are believed to be efficient scavengers of highly reactive free radical species, and have been shown to exhibit anti-carcinogenic; hypocholesterolaemic and neuroprotective properties . –Okello said that although research has identified certain compounds as beneficial for health, and in many cases has identified foods with high concentrations of such compounds, he explained that “what happens during the digestion process is crucial to whether these foods are actually doing us any good.” “Flavan-3-ols have been reported to possess properties beneficial to health, [but] they are known to undergo significant metabolism and conjugation in the gastrointestinal tract,” said the researchers. –“It is unknown how such metabolism and conjugation may influence the putative properties of these polyphenols, hence the focus of our study on a digested green tea extract,” they explained.
    Digested extract
    Green tea extract – from brewed Temple of Heaven, Gunpowder China tea, purchased by the researchers from a local store – was subjected to a simulated gastrointestinal digestion and a ‘colon-available’ extract (CAGTE) was prepared and assessed for its potential protective effects against the damaging effects of hydrogen peroxide and beta-amyloid on neuronal cells in the brain – which are believed to play a role in the development of dementia. –The CAGTE, which represents green tea phytochemicals potentially available after upper gastrointestinal digestion, was found to be depleted in flavan-3-ols when compared to the pre-digested green tea extract; yet was still found to protect cells in a brain neurone model from both hydrogen peroxide and beta-amyloid toxicity. –“At high concentrations, CAGTE exhibited direct anti-proliferative effects, in line with the reputed anti-cancer properties of green tea polyphenols,” wrote the authors. –“The CAGTE, which effectively lacked flavan-3-ols, had a protective effect on beta-amyloid induced toxicity in vitro at 0.03–0.125 μg/ml … This figure is much lower than the 20 μg/ml reported for effective protection in whole green tea extracts,” said the researchers.
    Next step
    He said that the next step in the research is to investigate whether the beneficial compounds found in the digestion model are produced during digestion in human volunteers that consume green tea polyphenols – for which the team has already secured funding from the Biotechnology and Biological Sciences Research Council (BBSRC).
    Source: Phytomedicine–Published online ahead of print, doi: 10.1016/j.phymed.2010.11.004 –“In vitro protective effects of colon-available extract of Camellia sinensis (tea) against hydrogen peroxide and beta-amyloid (Aβ(1–42)) induced cytotoxicity in differentiated PC12 cells” –Authors: E.J. Okello, G.J. McDougall, S. Kumar, C.J. Seal
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    Show of the Week Jan 21- 2011
     
    Calorie Restricted Diet Prevents Pancreatic Inflammation And Cancer
     
    How Calorie-Restricted Diets Fight Obesity and Extend Life Span
     
    Fueling the Body on Fat Critical Tuning Dial for Controlling Energy Found
     
    Key Enzyme In Fat Absorption Discovered
     
    How Progesterone & Estrogen Increases Breast Cancer Risk
     
    BRCA1 Gene Found To Inhibit Two Sex Hormones, Not Just One
    Anti Estrogenic Antidote
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    Calorie Restricted Diet Prevents Pancreatic Inflammation And Cancer
    ScienceDaily (Apr. 17, 2008) — Prevention of weight gain with a restricted calorie diet sharply reduced the development of pancreatic lesions that lead to cancer in preclinical research reported April 15 by researchers from The University of Texas at Austin and The University of Texas M. D. Anderson Cancer Center at the American Association for Cancer Research annual meeting.—The research sheds light on the connection between obesity, calorie intake and pancreatic cancer by comparing a calorie restricted diet, an overweight diet and an obesity-inducing diet in a strain of mice that spontaneously develops pancreatic lesions that lead to cancer.—“Obesity is a known risk factor for pancreatic cancer, but the mechanism underlying that relationship is unknown,” said senior author Stephen D. Hursting, Ph.D., professor in M. D. Anderson’s Department of Carcinogenesis and Chair of the Division of Nutritional Sciences at the University of Texas. “Our findings indicate that calorie restriction hinders development of pancreatic cancer, which could have implications for prevention and treatment of pancreatic tumors caused by chronic inflammation and obesity.”–The group’s analysis points to a connection between calorie intake and a protein called Insulin-like Growth Factor (IGF) -1, with obesity increasing and calorie restriction decreasing levels of IGF-1. IGF-1 is an important growth factor known to stimulate the growth of many types of cancer cells. Inflammatory signaling proteins also were found to be reduced in the blood of the calorie-restricted mice.—“Mice on the heavier diets had significantly more lesions and larger lesions than those on the restricted calorie diet,” said first author Laura Lashinger, Ph.D., a post-doctoral fellow in Hursting’s laboratory. The strain of mice, developed by Susan Fischer, professor in M. D. Anderson’s Department of Carcinogenesis, spontaneously develops lesions associated with pancreatitis – inflammation of the pancreas. These lesions develop into pancreatic cancer and virtually all of these mice die within six to eight months.-The researchers fed the calorie restricted group a diet that was 30 percent lower in calories than that consumed by the overweight group and 50 percent lower than the obese group. Only 7.5 percent of mice on the calorie-restricted diet developed pancreatic lesions at the end of the experiment, and these lesions were so small that none exhibited symptoms of illness. For mice on the overweight diet, 45 percent developed lesions, as did 57.5 percent of those on the obesity-inducing diet. Lesions were also much larger in the overweight and obese mice than the calorie restricted mice.—While calorie restriction has been shown to have an anti-cancer effect in multiple species and for a variety of tumor types, its impact had not been well-studied in a model of pancreatic cancer. Pancreatic cancer is the fourth leading cause of cancer death and remains mostly intractable to existing treatments.–The decline in blood levels of inflammatory proteins in the calorie restricted mice makes sense, Lashinger notes, because fat tissue is a major source of inflammatory factors such as cytokines.–[U1]The research was funded by grants from the National Institute of Health and the University of Texas, and is a collaboration between M. D. Anderson’s Department of Carcinogenesis, based at the The Virginia Harris Cockrell Cancer Research Center at M. D. Anderson’s Science Park – Research Division in Smithville, Texas, and the University of Texas at Austin Department of Nutritional Sciences.–Co-authors with Lashinger, Hursting and Fischer are Lauren Malone, Elizabeth Daniels, Nicole Smith, and Susan Perkins of the UT Department of Nutritional Sciences and Amy Pavone of M. D. Anderson’s Department of Carcinogenesis.–Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of Texas M. D. Anderson Cancer Center
     
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    How Calorie-Restricted Diets Fight Obesity and Extend Life Span
     
    Fruits and vegetables are a key part of calorie-restricted diets, which may increase longevity.–ScienceDaily (Dec. 29, 2009) — Scientists searching for the secrets of how calorie-restricted diets increase longevity are reporting discovery of proteins in the fat cells of human volunteers that change as pounds drop off. The proteins could become markers for monitoring or boosting the effectiveness of calorie-restricted diets — the only scientifically proven way of extending life span in animals.–Their study appears online in ACS’ Journal of Proteome Research.–Edwin Mariman and colleagues note that scientists have long known that sharply restricting intake of calories while maintaining good nutrition makes animals live longer and stay healthier. Recent studies suggest that people may gain similar benefits. But scientists know little about how these diets work in humans, particularly their effects on cells that store fat.–The new study focused on proteins in abdominal subcutaneous fat cells from a group of overweight people before and after they went on a five-week-long calorie-restricted diet. The volunteers each lost an average of 21 pounds[U2]. Scientists identified changes in the levels of 6 proteins as the volunteers shed pounds, including proteins that tell the body to store fat. These proteins could serve as important markers for improving or tracking the effectiveness of therapies involving calorie-restricted diets, they say.-Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by American Chemical Society, via EurekAlert!, a service of AAAS.-Journal Reference–Bouwman et al. The Physiologic Effects of Caloric Restriction Are Reflected in the <i>in Vivo</i> Adipocyte-Enriched Proteome of Overweight/Obese Subjects. Journal of Proteome Research, 2009; 8 (12): 5532 DOI: 10.1021/pr900606m
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    Fueling the Body on Fat Critical Tuning Dial for Controlling Energy Found
    ScienceDaily (Jan. 5, 2011) — Researchers have found what appears to be a critical tuning dial for controlling whole body energy, according to a new report in the January issue of Cell Metabolism, a Cell Press publication. When energy levels within cells drop, it sets off a series of events designed to increase the amount of calorie-rich dietary fat that the body will absorb.—This energy reset mechanism is surely critical for survival under natural conditions of scarcity to ensure a steady supply of fuel, the researchers say. Today, many of us who enjoy a Western diet loaded with fat might do better if we could find a way to turn the activity of the so-called AMPK-SRC-2 pathway down.–“Thousands of years ago, this would have been crucial,” said Bert O’Malley of Baylor College of Medicine. “Now it’s trouble because we eat so much fatty food.”[U3]–Earlier studies had shown the enzyme AMPK to be an ancient energy sensor. The enzyme causes cells to consume less energy in the form of ATP and to produce more. AMPK also drives appetite.–The new work shows that AMPK also allows for the optimal absorption of THE MOST ENERGY-RICH FUEL from the diet: fat[U4]. That effect of AMPK depends on its activation of SRC-2, a master control gene whose job is to switch other genes on.—When SRC-2 springs into action, it controls genes that lead to the secretion of bile from the gall bladder into the intestine. “You need bile to emulsify and absorb fat,” O’Malley explained.- Mice lacking SRC-2 fail to absorb fat normally, they report. Those deficiencies can be corrected by restoring bile acids to the gut.[U5]-Together with earlier work, the findings present a “pretty picture” in which SRC-2 is involved in absorbing and storing fat. SRC-2 is also known to play a role in releasing stored glucose from the liver. “It’s all about energy accretion, storage and delivery,” O’Malley says.–This process takes place on a daily basis even when there is already plenty of fat stored in the body. “It’s designed to get in more fat,” he says. “Over evolutionary time, when you didn’t know when the next meal would be, you really couldn’t get enough fat. Now, our next meal is at the corner McDonald’s.”–The discovery reveals a key mechanism linking the cellular energy state with the whole-body energy state and may ultimately have important clinical implications, the researchers say.–“Obesity is all about fat absorption and storage,” O’Malley said. “If you could turn that down, you could have a major effect on a disease that is slowly killing the population.” He says his team is now conducting studies in search of SRC-2 inhibitors that might do exactly that.—Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Cell Press, via EurekAlert!, a service of AAAS.–Journal Reference–Atul R. Chopra, Ramakrishna Kommagani, Pradip Saha, Jean-Francois Louet, Christina Salazar, Junghun Song, Jaewook Jeong, Milton Finegold, Benoit Viollet, Franco DeMayo, Lawrence Chan, David D. Moore, Bert W. O’Malley. Cellular Energy Depletion Resets Whole-Body Energy by Promoting Coactivator-Mediated Dietary Fuel Absorption. Cell Metabolism, 2011; 13 (1): 35-43 DOI: 10.1016/j.cmet.2010.12.001
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    Key Enzyme In Fat Absorption Discovered
    ScienceDaily (Mar. 18, 2009) — Scientists at the Gladstone Institutes of Cardiovascular Disease (GICD) have found that a key enzyme involved in absorbing fat may also be a key to reducing it. The enzyme, acyl CoA: [U6]monoacylglycerol acyltransferase 2 or Mgat2 is found in the intestines and plays an important part in the uptake of dietary fat by catalyzing a critical step in making triglyceride, a kind of fat. –Triglyceride accounts for nearly one-third of the fat eaten by people in developed countries.–Researchers in the laboratory of Robert V. Farese, Jr. MD, found that mice that were genetically modified to lack Mgat2 remain normal on a low-fat diet. However, when fed a high-fat diet that is similar to that eaten by many Americans, the mice do not get fat and do not develop other symptoms of obesity, such as glucose intolerance, hypercholesterolemia, and fatty livers. The mice eat the same number of calories as other mice, and the calories are fully absorbed. –“Because mice that lack this enzyme do not gain weight on a high-fat diet, it is an intriguing target for future interventions to prevent weight gain and the problems associated with that extra weight,” said Dr. Farese.–The mechanism of action, the researchers identified was that the lack of Mgat2 may reduce the uptake of fat in the small intestine and delay its entry into the blood. This process may dissociate fat from carbohydrate absorption and insulin secretion and ultimately lower the amount of fat stored and used. How this happens is not clear. One possibility is that the absorbed fat is partitioned more to tissues where it is burned up.–“Differences in Mgat2 expression may contribute to the propensity of some people to gain weight from diets rich in fat,” said Eric Yen, PhD, lead author of the study. “Our findings suggest that inhibiting this enzyme in the small intestine might be an effective way to treating metabolic diseases that result from excessive fat intake.”–Results of their study were published in the current issue of the journal Nature Medicine.–Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Gladstone Institutes, via EurekAlert!, a service of AAAS
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    How Progesterone & EstrogenIncreases Breast Cancer Risk
    ScienceDaily (Jan. 18, 2011) — Researchers have identified how the hormones progesterone and estrogen interact to increase cell growth in normal mammary cells and mammary cancers, a novel finding that may explain why postmenopausal women receiving hormone replacement therapy with estrogen plus progestin are at increased risk of breast cancer.–The discovery that both estrogen and progesterone must be present for the increased production of the protein amphiregulin, which binds to mammary cells and promotes cell growth, could lead to new treatment methods for the disease, said Sandra Haslam, director of Michigan State University’s Breast Cancer and the Environment Research Center and lead researcher on the project.–The study, funded by the Department of Defense’s Breast Cancer Research Program and published in Hormones and Cancer, looked at why progesterone combined with estrogen may contribute to increased breast cancer risk. In the study, researchers used both the native hormone, progesterone, and a synthetic compound, progestin — obtaining the same results.—The finding might help explain earlier results from the groundbreaking Women’s Health Initiative showing the risk of breast cancer is significantly greater for postmenopausal women who received hormone replacement therapy with combined estrogen plus progestin compared to women receiving estrogen alone.–“Also, breast cancers that develop in women receiving estrogen plus progestin are more invasive and deadlier,” Haslam said. “What is the progestin doing to increase the risk of tumor growth?”–Along with co-investigator Anastasia Kariagina, a colleague in the College of Human Medicine and Department of Physiology, Haslam identified the protein amphiregulin and its receptor as one potential culprit.—“Amphiregulin — acting through its receptor, epidermal growth factor receptor — along with progesterone leads to the activation of intracellular pathways that regulate cell growth,” Haslam said. “When activated, this promotes normal cell growth and the growth of tumors.”—The study was performed in rats because breast cancers in rats contain receptors for estrogen and progesterone — similar to the human breast — and tumor growth is hormone-dependent, as are the majority of human breast cancers. The research team also confirmed the same phenomenon in human breast cancer cell cultures.–In addition, the research team found that Iressa, a cancer drug that blocks the epidermal growth factor receptor, effectively stopped the proliferation caused by amphiregulin. While those studies were done only in cell cultures and not on tumors growing in animals, the results are promising, Haslam said.—“The results indicate that the interactions between estrogen, progesterone and epidermal growth factor receptor pathways may be considered relevant targets for the treatment of hormone-dependent breast cancers,” she said. “This may be especially important in premenopausal breast cancer because women produce their own estrogen and progesterone.–“A combined approach of inhibiting both the hormones and the epidermal growth factor receptor may be beneficial for some women in treating hormone-dependent breast cancer.”–Story Source:–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Michigan State University.–Journal Reference-Anastasia Kariagina, Jianwei Xie, Jeffrey R. Leipprandt, Sandra Z. Haslam. Amphiregulin Mediates Estrogen, Progesterone, and EGFR Signaling in the Normal Rat Mammary Gland and in Hormone-Dependent Rat Mammary Cancers. Hormones and Cancer, 2010; 1 (5): 229 DOI: 10.1007/s12672-010-0048-0
     
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    BRCA1 Gene Found To Inhibit Two Sex Hormones, Not Just One
     
    ScienceDaily (Jan. 26, 2006) — It’s been known that the breast cancer susceptibility gene BRCA1 regulates use of estrogen in breast and other cells, but now researchers at Georgetown University Medical Center have discovered that it also controls activity of a second sex steroid hormone, progesterone. —The findings, conducted in cell culture and in mice and reported by the researchers in the January issue of Molecular Endocrinology, could help explain why women who have mutations in their BRCA1 gene are susceptible to a number of different “hormone-dependent” cancers, including those of the breast, endometriun and cervix. —It also has implications for ordinary cancers that arise because a normal BRCA1 gene is under-expressed, said the study’s principal investigator, Eliot Rosen, MD, PhD, professor of oncology, cell biology, and radiation medicine at the Lombardi Comprehensive Cancer Center. —For example, he says that up to 40 percent of breast tumors are deficient in BRCA1, “and it may be that some patients could benefit not only from an anti-estrogen therapy, like tamoxifen, but also from an anti-progesterone agent. —“We don’t know if that is true yet, of course, but it is certainly worth investigating, given our findings,” Rosen said. –The BRCA1 gene and a second gene, BRCA2, were discovered to be breast cancer susceptibility genes in 1994 and 1995, respectively. Women who inherit faulty copies of one of these genes have up to an 80 percent increased risk of developing breast cancer by age 70, and are also more likely to be diagnosed with ovarian cancer. –Rosen and his research team undertook the study to understand why loss of the BRCA1 gene results in cancers in tissues that are dependent on hormones. They focused on the progesterone hormone, in part, because of the observation that women who use hormone replacement therapy that includes both estrogen and progestin (a synthetic form of progesterone) are at greater risk of developing breast cancer than women who use only estrogen replacement. [U7]–The use of progesterone in the breast is tightly regulated and is primarily activated when growth in cells is needed, such as during the female menstrual cycle and to support a pregnancy. A cell’s use of progesterone and other such hormones is controlled by specific receptor proteins, located inside cells, which bind on to the hormone. This process activates the receptor, which then migrates to the cell nucleus to stimulate gene expression. —To find out what role BRCA1 played in progesterone receptor signaling, the Lombardi research team conducted a series of experiments. In one set of cell culture studies in the laboratory, they used breast cancer cells that were responsive to progesterone, and then genetically manipulated them to either over or under-express the BRCA1 gene in order to assess the gene’s effect on progesterone receptor signaling. –They also used mice in which the BRCA1 gene was partially deleted, but only in breast tissue. The animals were treated with estrogen, or progesterone, or both, and response of the mammary gland was compared with that of normal mice. –In this way, the researchers concluded that BRCA1 interacts physically with the progesterone receptor, and stops it from activating other genes. It does this even in the absence of the progesterone hormone, and, thus, acts as a strong check on errant growth. —“But in mice deficient in BRCA1, we found that estrogen plus progesterone has a particularly large effect in stimulating the growth of mammary epithelial cells − an effect much greater than the effects of either hormone used alone,” Rosen said. —The study was funded by grants from the Susan G. Komen Breast Cancer Foundation and the National Cancer Institute. Contributing to the study were Yongxian Ma, MD; Pragati Katiyar, MS; Laudette P. Jones, PhD; Saijun Fan, MD, PhD; Yiyu Zhang, MD; and Priscilla A. Furth, MD. –Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Georgetown University Medical Center

    #2080
    AvatarEK
    Keymaster

    My Comment Here—the issue is not about the estrogens and progesterones but rather the bodies ability to utilize them and remove them—the herbalist and other alternative health and healers knew this of yesteryear and would require women to do blood tonics and organ supporting herbs that would alleviate the load of these hormones—In todays times when foods consumed are highly hormonal –Soy – Vege oils ( estrogen imbalancing )-olive oils ( which can increase testosterone) peas ( estrogenic)– and chemicals that are highly xenoestrogenic like dish soaps and laundry soaps and cleaners and pesticide on or in the foods and hormone produced milk and meats—the system may not necessarily lack the gene but rather being so overwhelmed with the load that there is not enough to compensate for the imbalances—the old ways maybe tougher but the work –the thought and suggestions here would be to eliminate the foods causing the overload and to potential fast or reduce drastically consumption and increase foods that are going to reinforce the organs and the gene production to assist in balancing out what is overloaded
     
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    Anti Estrogenic Antidote
    Because of the overloading of the endocrine and lymphatic system of estrogen three has been a number of reproductive cancers occurring at a higher then normal rate…..if you look up the stats this didn’t happen til the 60’s when cancers of all types started to become more apparent in the USA and Canada…. What changed? The exposure to pesticides had definitely increased, the exposure to toxic pollutants had definitely increased, the removal of foods from the diet which were maligned to be a problem but in fact were not, and new food and dietary guidelines were introduced that were in fact cancer causing, such as soy and margarine, food additives and chemical preservatives, plastics that contained foods and detergents which are xeno-estrogenic characteristics. This means these are synthetic estrogens that will in fact react the same way as plant estrogens but in a higher concentrated levels that will bind to what are known as receptor sites ….these are areas of the body that are triggered by hormones and enzymes which cause a chemical reaction or trigger a signal in the body to perform whatever function that is required. These estrogens coming from with out, are extremely dangerous because of the way they accumulate in the body, and the reaction they cause in the body. In the case of estrogen it will lead to cancer, there are over 50 years of research an data to validate this, and to this day the end result has been the same since we have introduced highly estrogenic foods into the diet, males are becoming more mentally and physically dysfunctional, and women are breaking down more rapidly then there predecessors did. By being exposed to dish soap, latex, fuel oils, carpeting, clothing , fabric softeners, clothing made with synthethics, exhaust fumes from all petro products, including the Idea of biodiesel or bio fuels, this to will exacerbrate a bad condition already existing, and will compound the effect that much more…
     
    So how do we heal this condition? How do we reverse the effect? Can we reverse the effect ?…there are herbs, supplements, and vitamins that actually regulate estrogen and remove the excess out of the body, via liver. And through other means of expulsion from the body. There are foods we can eat as well as foods to avoid that will assist us in the regulating of the overloading of the hormones in our system. I will give a list of foods herbs and supplements that will assist in this endeavor to keep us hormonally balanced :….Some of these will be used as a tea, others will be consumed and others used as a supplement….and in some cases used together to remove whatever is afflicting us hormonally….
     
    Vitamins:
    Zinc
    Iodine
    Selenium
    Magnesium
    Sulfur
     
     
     
    Herbs that can assist in the removal of estrogen from the body
    Watercress
    Turmeric
    Garlic
    Onion
    Shallots
    Parsely
    Mint
    Celery Root
    Rosemary
    Siberian Ginseng
    Gota Kola
    Thyme
    Milk Thistle
    Dandelion
    Lady’s Thistle
    Summer and Winter Savoury
    Ginger and Galangal
    Balm of Gilead ( balsam poplar )
    Perilla
    Olive Leaf
    Oregano
     
    Supplements that will remove estrogen or block it’s effect
    NAC
    L Cysteine
    Methionine
    Taurine
     
     
    Foods that can remove or block estrogen:
    Nuts
    Seeds
    Omega 9 oils
    Avocado
    Rice Bran Oil
    Wheat Germ Oil
    Papaya
    Pineapple
    Berries
    Citrus fruits ( Bioflavonoids )
    Oats and Barley ( Whole )
    Broccoli and Broccoli Sprouts
    Onion Sprouts
    Cabbage
    Cauliflower
    Kohl Rabi
    Kale
    Parsley
    Celery Root
    Grass fed Animals ( grain fed will encourage hormone imbalancing due to the excess of omega 6 in the fat rather then 3 ( grass fed cows have 15 times more omega 3 then fish )
    Ocean caught fish ( make sure you increase garlic or sulfur based supplement with Vitamin C to offset any potential mercury or lead poisoning of the fish.
    Ground flax seed ( due to the fibre content of the lignans which will bind to the estrogens ) Make sure you grind it fresh not store bought
     
    Herbal Teas
    Any tea that will support the liver and the endocrine system should be used to assist in the removal of any unwanted toxin or increase the antioxidant levels so that the body is able to be more efficient in the removing of unwanted pathogens or hormones
    Schizandra berry
    Fo Ti Eng
    Rehmannia Root
    Jiagolan
    Milk thistle
    Nettle
    Dandelion
    Burdock
    Sarsparilla
    Perilla
    Parsley
    White Pine
    Birch
    Siberian ginseng
    Self Heal
    Make sure all teas are drunk without any processed sugar or artificial sweetners…Use Xylitol from birch trees or Unpasteurized honey
     
    ®®RAW MILK ( not pasteurized ) due to the CLA and Vitamin D content
    ®®Grass fed Cow’s Butter
    ®®Seed and Nut Milk
    ®®Almonds ( due to the phosphorous in it to assist the liver in detoxing the estrogens out)
    ®®Walnuts ( omega 3 which block estrogen conversion of omega 6 )
    ®®Pumpkin Seed ( due to the zinc content in regulating hormones )
     
    http://augmentinforce.50webs.com/ANTI%20ESTROGEN%20SOLUTIONS.htm#ANTI ESTROGEN SOLUTIONS
     
     
    TOP E
    [U1]Let’s take a look here, there is an issue with this conclusion—see they did a calorie restricted diet has nothing to do with fat or protein or carb or even other nutrients –calorie restricted —this implies a reduction in everything—so the misleading end of this is to get the reader to see that it is FAT that is the problem—it may very well be —but NOT NECESSARILY–what usually triggers the IGF is sugar ( which increases insulin ) and proteins ( arginine-aspartic acid–precursors to GH and whey protein–not to mention zinc-magnesium-and potassium )—the point here is that IGF is as well utilized to break down fatty acids as well—so again in these type of article pay attention to how they will show you a specific but then lead you to a bad conclusion!!
    [U2]21 pounds in 5 weeks is about all anyone should be losing in this time frame–the avg lost should be about 3lbs per week anything more can be a rebounder—it can come back and with more pounds as well–this was avg just over 4 .2 lbs
    [U3]Again look at this comment here—we eat to much fatty foods—what FAT—since we have an abundant of choices here –vegetable oils ( soy) canola oils -margarines-fih oils with mercury–these are fats as well and People do consume alot of these as well—and then end result is always illnesss the FAT that they are talking is which one?? and can they then explore is it the fat or the components in the fats which maybe the culprit? again see this for what it is —
    [U4]Hmm seems to me that the energy of source for the body isssssss F-A-T
    [U5]This maybe the resolution to getting rid of the fat storage or to increase fat efficiency getting Bile salts
    [U6]Coenzyme A combines with Acetic Acid (derived from endogenous Pyruvic Acid) to form Acetyl Coenzyme A.
     
    Nucleic Compounds–Coenzyme A facilitates the repair of the body’s endodgenous Deoxyribonucleic Acid (DNA).-Coenzyme A facilitates the repair of the body’s endodgenous Ribonucleic Acid (RNA).
     
    These Substances Enhance the Function of Coenzyme A–Amino Acids-By exchanging across sub-cellular membranes, Acetyl-L-Carnitine (ALC) serves as a pool of acetyl groups to regenerate Acetyl-Coenzyme A from free Coenzyme A.
    Lipids–Caprylic Acid is rapidly reduced to Coenzyme A in the Liver.
    Vitamins–Metabolites of Vitamin B5 (in its Pantethine form) are essential components of the Coenzyme A molecule
     
    [U7]Interpret this accurately they are not saying you will not get cancer using estrogen but you will get it more then likely if combining this with progesterone or it’s synthethic progestrin—so in other word we are overloading these receptor sites with an already overloaded system and the genes that regulate these hormones are either inadequate or are not present—so then the alternative would be to reduce the overload-not increase the hormones—by blocking the receptors with more hormones all that is happening is a delaying of a cancer nothing more
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    TOP F
    HOME
     
    Show of the Week January 24- 2011
     
    99% of Pregnant Women in US Test Positive for Multiple Chemicals Including Banned Ones-Research
    Dangerous Antidepressants
     
    OAT VINEGAR
     
    Recipe for Oat Vinegar
     
    Vinegar
     
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    99% of Pregnant Women in US Test Positive for Multiple Chemicals Including Banned Ones-Research
     
    ScienceDaily (Jan. 16, 2011) — The bodies of virtually all U.S. pregnant women carry multiple chemicals, including some banned since the 1970s and others used in common products such as non-stick cookware, processed foods and personal care products, according to a new study from UCSF. The study marks the first time that the number of chemicals to which pregnant women are exposed has been counted.–Analyzing data for 163 chemicals, researchers detected polychlorinated biphenyls (PCBs), organochlorine pesticides, perfluorinated compounds (PFCs), phenols, polybrominated diphenyl ethers (PBDEs), phthalates, polycyclic aromatic hydrocarbons (PAHs) and perchlorate in 99 to 100 percent of pregnant women. Among the chemicals found in the study group were PBDEs, compounds used as flame retardants now banned in many states including California, and dichlorodiphenyltrichloroethane ( DDT), an organochlorine pesticide banned in the United States in 1972.—Bisphenol A (BPA), which makes plastic hard and clear, and is found in epoxy resins that are used to line the inside of metal food and beverage cans, was identified in 96 percent of the women surveyed. Prenatal exposure to BPA has been linked to adverse health outcomes, affecting brain development and increasing susceptibility to cancer later in life, according to the researchers. Findings will be published in Environmental Health Perspectives on Jan. 14. The study was not designed to identify direct connections to adverse health outcomes.”It was surprising and concerning to find so many chemicals in pregnant women without fully knowing the implications for pregnancy,” said lead author Tracey Woodruff, PhD, MPH, director of the UCSF Program on Reproductive Health and the Environment.—“Several of these chemicals in pregnant women were at the same concentrations that have been associated with negative effects in children from other studies. In addition, exposure to multiple chemicals that can increase the risk of the same adverse health outcome can have a greater impact than exposure to just one chemical,” said Woodruff, an associate professor in the UCSF Department of Obstetrics and Gynecology and Reproductive Sciences.–Exposure to chemicals during fetal development has been shown to increase the risk of adverse health consequences, including preterm birth and birth defects[U1], childhood morbidity, and adult disease and mortality according to the research team. In addition, chemicals can cross the placenta and enter the fetus,[U2] and in other studies, a number of chemicals measured in maternal urine and serum have been found in amniotic fluid, cord blood and meconium, they state.–The researchers analyzed data for 268 pregnant women from the National Health and Nutritional Examination Survey (NHANES) 2003-2004, a nationally representative sample of the U.S. population.–“Our findings indicate several courses of action. First, additional research is needed to identify dominant sources of exposure to chemicals and how they influence our health, especially in reproduction,” said Woodruff. “Second, while individuals can take actions in their everyday lives to protect themselves from toxins, significant, long-lasting change only will result from a systemic approach that includes proactive government policies.[U3]”–Co-authors of the study are Ami R. Zota and Jackie M. Schwartz of the Program on Reproductive Health and the Environment, UCSF Department of Obstetrics and Gynecology and Reproductive Sciences.–Funding for the study was provided by the Pew Charitable Trusts and a grant from the Passport Science Innovation Fund.–Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of California – San Francisco.–Journal Reference–Tracey J. Woodruff, Ami R. Zota, Jackie M. Schwartz. Environmental Chemicals in Pregnant Women in the US: NHANES 2003-2004. Environmental Health Perspectives, 2011; DOI: 10.1289/ehp.1002727 —
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    Dangerous Antidepressants
    The Institute for Safe Medication Practices (ISMP) recently published a study in the journal PLoS One highlighting the worst prescription drug offenders that cause patients to become violent.
    Among the top-ten most dangerous are the antidepressants Pristiq (desvenlafaxine), Paxil (paroxetine) and Prozac (fluoxetine).
    Concerns about the extreme negative side effects of many popular antidepressant and antipsychotic drugs have been on the rise, as these drugs not only cause severe health problems to users, but also pose a significant threat to society. The ISMP report indicates that, according to the U.S. Food and Drug Administration’s (FDA) Adverse Event Reporting System, many popular drugs are linked even to homicides.
    Most of the drugs in the top ten most dangerous are antidepressants, but also included are an insomnia medication, an attention-deficit hyperactivity disorder (ADHD) drug, a malaria drug and an anti-smoking medication.
    As reported in Time, the top ten list is as follows:
    [10.] Desvenlafaxine (Pristiq) – An antidepressant that affects serotonin and noradrenaline. The drug is 7.9 times more likely to be associated with violence than other drugs.
    [9.] Venlafaxine (Effexor) – An antidepressant that treats anxiety disorders. The drug is 8.3 times more likely to be associated with violence than other drugs.
    [8.] Fluvoxamine (Luvox) – A selective serotonin reuptake inhibitor (SSRI) drug that is 8.4 times more likely to be associated with violence than other drugs.
    [7.] Triazolam (Halcion) – A benzodiazepine drug for insomnia that is 8.7 times more likely to be associated with violence than other drugs.
    [6.] Atomoxetine (Strattera) – An ADHD drug that is 9 times more likely to be associated with violence than other drugs.
    [5.] Mefoquine (Lariam) – A malaria drug that is 9.5 times more likely to be associated with violence than other drugs.
    [4.] Amphetamines – This general class of ADHD drug is 9.6 times more likely to be associated with violence than other drugs.
    [3.] Paroxetine (Paxil) – An SSRI antidepressant drug that is 10.3 times more likely to be associated with violence than other drugs. It is also linked to severe withdrawal symptoms and birth defects.
    [2.] Fluoxetine (Prozac) – A popular SSRI antidepressant drug that is 10.9 times more likely to be associated with violence than other drugs.
    [1.] Varenicline (Chantix) – An anti-smoking drug that is a shocking 18 times more likely to be associated with violence than other drugs.
     
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    OAT VINEGAR ( WILD OAT )
    BACKGROUND: The present study focused on the antioxidant activities of aged oat (Avena sativa L.) vinegar. The antioxidant activities of oat and vinegar have been proved by many previous research studies. It should be noted that oat vinegar, as a novel seasoning, has antioxidant activity.
    RESULTS: Oat vinegar showed stronger radical scavenging activities, reducing power, and inhibition of lipid peroxidation than rice vinegar. The concentrations of polyphenols and flavonoids in oat vinegar were higher than those in rice vinegar. Ethyl acetate extract of oat vinegar possessed the most varieties of phenolic acids and showed the strongest antioxidant activity compared with ethanol and water extracts. At suitable doses of oat vinegar, the malondialdehyde value was decreased, activities of superoxide dismutase and glutathione peroxidase were promoted, and hepatic damage induced by (60)Co gamma-irradiation was ameliorated in aging mice.
    CONCLUSION: Oat vinegar manifested antioxidant activity which was stronger than that of rice vinegar in vitro and the same as that of vitamin E in vivo.
     
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    Antioxidant capacity of oat (Avena sativa L.) extracts. 2. In vitro antioxidant activity and contents of phenolic and tocol antioxidants.
    Emmons CL, Peterson DM, Paul GL.
    Cereal Crops Research Unit, Agricultural Research Service, U.S. Department of Agriculture, 501 Walnut Street, Madison, Wisconsin 53705, USA.
    Abstract
    Oat milling fractions were examined for concentrations of total phenolics, tocols, and phenolic acids and in vitro antioxidant activity to determine their potential as dietary antioxidants. Methanolic extracts of pearling fractions, flour and aspirations from flaking, and trichomes had high, intermediate, and low antioxidant activities, respectively, evaluated by the beta-carotene bleaching method. Pearling fractions were also highest in total phenolics and tocols. p-Hydroxybenzoic acid, vanillic acid, caffeic acid, vanillin, p-coumaric acid, and ferulic acid were identified and quantified by HPLC. Three avenanthramides and an unidentified ferulate derivative were also detected. Total phenolic content was significantly correlated with antioxidant activity, and regression equations that predicted antioxidant activity from phenolic and tocol concentrations were calculated. Antioxidant activity, evaluated by beta-carotene bleaching, was correlated with measures of oxygen radical absorbance capacity and low-density lipoprotein oxidation. These data indicate a potential for oat products, especially those enriched in outer layers of the groat, to contribute to dietary intakes of -antioxidant phytonutrients.
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    Potential health benefits of Wild Oats antioxidants avenanthramides
    Meydani M.
    Vascular Biology Laboratory, Jean Mayer USDA Human Nutrition Research Center on Aging at Tufts University, Boston, MA 02111, USA. [email protected]
    Abstract
    Oats are known to be a healthy food for the heart due mainly to their high beta-glucan content. In addition, they contain more than 20 unique polyphenols, avenanthramides, which have shown strong antioxidant activity in vitro and in vivo. The polyphenols of oats have also recently been shown to exhibit anti-inflammatory, antiproliferative, and anti-itching activity, which may provide additional protection against coronary heart disease, colon cancer, and skin irritation.–PMID: 19941618 [PubMed – indexed for MEDLINE]
     
    CRecipe for Oat Vinegar—add a cup of groats ( oats with the shell ) in a jar and add vinegar to this and let sit for 10 days on a fridge—this will have fermented the grain with all of it’s content) strain and pour rest into a GLASS container and use as a vinegar on salads or even straight up as a tonic or antioxidant aid
     
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    Vinegar
    Can be made from any liquid that is capable of being converted into alcohol in a two-step process. The fruit juice or other liquid contains sugar, which is converted into alcohol and carbon dioxide gas by the actions of yeast enzymes. The alcohol thus formed combines with atmospheric oxygen by the action of Acetobacter bacteria, forming acetic acid and water. Organic acids and esters derived from the fruit or other source material are also present and are responsible for the flavour and aroma variations of vinegar. Table vinegar contains approximately 4 percent acetic acid. These bacteria work together symbiotically, producing enough acetic acid to prevent invasion by other organisms.–Despite its ancient origin, the technology of vinegar production advanced slowly, improvements consisting principally of better methods of aeration. The Orleans process, best-known of the old methods, used a barrel of about 50 gallons (200 l) capacity. A mash consisting of wine or other alcoholic liquid was poured into the barrel, and a small amount of vinegar containing a mass of vinegar bacteria, called mother of vinegar, was added to start the reaction. One or two small air holes drilled above the liquid level exposed the surface to aeration. The finished vinegar was drawn off through a wooden spigot near the bottom. Care was taken in refilling the barrel with the new charge of raw ingredients to avoid breaking up the surface film of bacteria.—Early in the 18th century, a Dutch technologist, Hermann Boerhaave, found that the rate of acid production in the vinegar process was directly proportional to the amount of surface exposed to air. Thus, subsequent methods attempted to introduce more air into the casks. In the 20th century, continuous aeration—air bubbles pumped through the mash—was developed.–Vinegar’s principal uses are the flavouring of foods and the preservation, or pickling, of meat products, fish, fruit, and vegetables. For use as a condiment, vinegar is often flavoured with garlic, onions, tarragon, or other herbs and spices. Mixed with oil and seasonings it becomes a classic cold sauce—vinaigrette—used as a dressing on vegetable salads and served as a sauce with cold cooked vegetables, meats, and fish. Vinegar is also a common ingredient in marinades and is widely used in the pickling of cucumbers and other vegetables.
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    TOP F
    [U1]NOWWWWW you have to ask the Question the big one, Why are women getting Vaccinated and or the children or Fetuses in the womb, woman are being purposely infected???? and a MULTITUDE OF CHEMICALS WILL INCREASE ADVERSE HEALTH OUTCOMES????!!!!! DING DING DING –Are We seeing This!!!
    [U2]Is this what we want —A violation of fetal Life by having this cross through the placenta!!
    [U3]This should not be allowed in the Gov’t’s hands at all—this should be with those of us who have a geniune interest in preerving the Human Race from Gov’t who can easily be bought out by Special interest who mass produce thes toxic waste as well as those who would infect the populace with more infectious disease–the Gov’t has no credibility NONE!!
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    TOP G
    HOME
     
    Show of the Week Jan 28- 2011
     
    Obamacare Bill suggests data chip implants
     
    Montrealers Are Feeding Fish Prozac- Research Shows Influence on Brain Activity While Long-Term Consequences Are Unclear
     
    CRN– IND guidance could run against spirit of DSHEA–Article 13 American Style
     
    Inflammation and Headache Recipes
    Council for Responsible Nutrition, Washington, DC
     
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    Obamacare Bill suggests data chip implants
     
    Coverage under Obamacare will require an implantable microchip.
    There’s a pretty starling thing in the bill that 95% of Americans won’t like.
    The Obama Health care bill under Class II (Paragraph 1, Section B) specifically includes ‘‘(ii) a class II device that is implantable.” Then on page 1004 it describes what the term “data” means in paragraph 1, section B:
    14 ‘‘(B) In this paragraph, the term ‘data’ refers to in
    15 formation respecting a device described in paragraph (1),
    16 including claims data, patient survey data, standardized
    17 analytic files that allow for the pooling and analysis of
    18 data from disparate data environments, electronic health
    19 records, and any other data deemed appropriate by the
    20 Secretary”
    What exactly is a class II device that is implantable?
    Lets see…
    Approved by the FDA, a class II implantable device is a “implantable radiofrequency
    transponder system for patient identification and health information.” The purpose of a class II device is to collect data in medical patients such as “claims data, patient survey data, standardized analytic files that allow for the pooling and analysis of data from disparate data environments, electronic health records, and any other data deemed appropriate by the Secretary.”
    This sort of device would be implanted in the majority of people who opt to become covered by the public health care option. With the reform of the private insurance companies, who charge outrageous rates, many people will switch their coverage to a more affordable insurance plan. This means the number of people who choose the public option will increase. This also means the number of people chipped will be plentiful as well. The adults who choose to have a chip implanted are the lucky (yes, lucky) ones in this case. Children who are “born in the United States who at the time of birth is not otherwise covered under acceptable coverage” will be qualified and placed into the CHIP or Children’s Health Insurance Program (what a convenient name). With a name like CHIP it would seem consistent to have the chip implanted into a child. Children conceived by parents who are already covered under the public option will more than likely be implanted with a chip by the consent of the parent. Eventually everyone will be implanted with a chip. And with the price and coverage of the public option being so competitive with the private companies, the private company may not survive.
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    Montrealers Are Feeding Fish Prozac; Research Shows Influence on Brain Activity While Long-Term Consequences Are Unclear
    ScienceDaily (Jan. 22, 2011) — Around one in four Montrealers take some kind of anti-depressant, and according to new research, the drugs are passing into the waterways and affecting fish.–The findings are internationally significant as the city’s sewage treatment system is similar to that in use in other major cities, and moreover, it is reputed to be the third largest treatment system in the world. Lead by Dr. Sébastien Sauvé at the University of Montreal’s Department of Chemistry and André Lajeunesse, a PhD candidate, the research team found that the drugs accumulate in fish tissues and are affecting the fish’s brain activity.—The Saint Lawrence is a major international waterway that connects the Atlantic Ocean to the Great Lakes, and it surrounds the island of Montreal. Sauvé has been looking at the chemical pollution of the water system for years. “Montreal has a very basic sewage system — the city basically only removes solids, there’s no disinfecting of the water,” he explained. “In any case, the chemical structure of anti-depressants makes them extremely difficult to remove from sewage, even with the most sophisticated systems available.”–“We know that antidepressants have negative side effects on human beings,” Sauvé said, “but we don’t know how exactly how these chemicals are affecting the fish, and by extension, the Saint Lawrence River’s ecosystem.” Despite a lack of information about the possible toxicity brought from these substances, the research group suggests an interesting tool to track the early biological effects of antidepressants. “Since the acute toxicity of antidepressants is less probable toward aquatic organisms, chronic toxicity remained possible. In this way, the suggested biomarker involved in the serotonin regulation in the brain may represent a promising means of determining subtle biological effects to fish,” explained Lajeunesse. Chronic toxicity means harm resulting from long-term exposure, whereas acute relates to more immediate harm following a single high-dose incident. Serotonin is an important chemical that plays a role in feelings of happiness — it’s sometimes referred to as the “happy hormone.”–Sauvé was quick to point out that there is no immediate danger to humans. “The amount of anti-depressants being released into our river works out to roughly the equivalent of a grain of salt in an Olympic-size swimming pool,” he said. “That’s not enough to affect people, should they are brave enough to go fishing out there — I’d be more worried about the trace metals! Nevertheless, we are seeing an impact on the river’s ecosystem, which should concern cities everywhere.” Further research by other teams will look at exactly what the consequences might be.–This research received funding from the Chemical Management Plan — Health Canada, the St. Lawrence Action Plan and the Canadian Foundation for Innovation. It was published online by Chemosphere on Jan. 5, 2011. Christian Gagnon, François Gagné, and Séverine Louis at Environment Canada and Patrick Čejka at the Montreal Sewage Treatment Plant contributed to the findings.
    Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of Montreal, via EurekAlert!, a service of AAAS.-Journal Reference-André Lajeunesse, Christian Gagnon, François Gagné, Séverine Louis, Patrick Čejka, Sébastien Sauvé. Distribution of antidepressants and their metabolites in brook trout exposed to municipal wastewaters before and after ozone treatment – Evidence of biological effects. Chemosphere, 2011; DOI: 10.1016/j.chemosphere.2010.12.026
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    CRN– IND guidance could run against spirit of DSHEA–Article 13 American Style
    As the Food and Drug Administration (FDA) prepares final guidance on Investigational New Drugs (INDs) a leading trade group has warned of the damaging effect on nutrient research that could follow if the application of INDs is not modified from the current draft guidance.
    The Washington DC-based Council for Responsible Nutrition (CRN) submitted comments to the FDA highlighting why nutrient research could sometimes be applied in both the pharma and food spheres.–A demarcation between the two would defeat the intention of the 1994 Dietary Supplement and Health Education Act (DSHEA), it said, and called for certain types of research o be exempt from NDI requirements.
    “While CRN agrees that a product’s intended use should dictate its regulatory category, we question the rationale for allowing a product’s regulatory category to drive scientific inquiry,” the CRN’s vice president of scientific and regulatory affairs, Duffy MacKay wrote in a nine-page submission.
    MacKay wrote his group was concerned, “some aspects of the Draft Guidance may have the unintended consequence of acting in opposition to the spirit” of DSHEA, and create, “unnecessary obstacles to the continued scientific study of dietary supplements.”
    He added that DSHEA had authorized the establishment of the Office of Dietary Supplements (ODS) at the National Institutes of Health (NIH) to study the benefits of dietary supplements in maintaining health and preventing chronic disease and so ‘dietary supplements’ had been mandated to be studied in a pharma and nutra ways.
    “The potential for a dietary supplement to have a dual definition, which is dependent on its intended use, has resulted in much confusion amongst the research community.”
    Nutrients versus drugs
    Industry is concerned some nutrients like omega-3s or probiotics may require IND’s if they are to be studied, and that there will be a bias to disease endpoints as there are no validated endpoints for wellness.
    “For example, an investigator interested in evaluating a yogurt product from the grocery store that contains beneficial bacteria for its ability to prevent constipation in a healthy population would be required to file an IND. This seems incongruent with the intent of the IND…”
    MacKay highlighted further clarity was needed because:
    · Much nutrient research was academia, not commercial driven and academia need to be alerted to the guidance and its implications for their work, especially when an IND is required.
    · Cost and proprietary science concerns would see companies pulling out of the scientific process if INDs are required for certain trials.
    · IND approaches across various sectors are not unified and an IND policy in the Center for Food Safety and Applied Nutrition (CFSAN). The current system may see an inundation of INDs.
    · Many nutrients possess a variability that does not fit the IND model.
    · Obtaining an IND does not necessarily improve study safety or quality.
    He pointed out a grey area in that DSHEA does not permit a, “product that does not contain an article authorized for investigation as a new drug, antibiotic, or biological for which substantial clinical investigations have been instituted and for which existence of such investigations has been made public.”
    But the FDA has indicated a citizen’s petition, such as the one currently being appraised for a hormone produced by Canadian firm Ovos Natural Health, could be a way to have nutrients that have been subject to drug research classified as dietary supplements, via the New Dietary Ingredients route.
    MacKay called for the FDA to specify financial penalties for conducting trials without INDs.
    **************************************************************************
    Inflammation and Headache Recipes
     
    Headaches—Body aches—Aches in places where you have aches
    Lets make a remedy—Lets start out with something conventional and give it a boost
    Aspirin 81 mg or a generic that is 81mg –Uneneterocoated—1 caplet with with 100-200 mgs of magnesium and 1/8 tsp of cayenne pepper or 1 capsule at 500mgs—take together
     
    Take White Willow Extract or tincture put into a glass container and add 1 ½ oz to ½ oz of cayenne tincture—Mix Well and then take ¼-1/2 tsp
     
    Dlpa 500 -750 mgs with 200 mgs of magnesium citrate
     
    Bay Leaf Extract with Cayenne Pepper 1:1 ratio ( equal parts say ½ oz to a ½ oz ) mix well and take ¼ -1/2 tsp
     
    White Willow or FeverFew 1/8 cup with 6-8 leaves of Bay Leaf in a 2 pint pot of water—drink a 4-6 oz serving several times a day
     
    Breatheing in the Essential oil of lavender
     
    Body Ache
    Using Vicks as a body Rub or make your own cream by adding Peppermint 8 drops—Camphor 8 drops —Eucalyptus 8 drops and Wintergreen 8 drops with a red or black pepper extract 1 oz and fuse in bees wax and coconut oil ½ cup of coconut oil and ¼ cup of wax or less if you want a lotion –add 1 oz of alcohol and heat together in a double broiler or a ban marie heat til all soft –wne at this point then take a blender and pour content in glass container—allow for cooling then apply this liberally and even on chest cavity for congestion the apply this where pain exist
     
    Using Pine oil 4 drops and Camphor oil 4 drops and mix with 4 oz of peanut oil and massage in pained areas or areas of intense out break
     
    Take a bath in Magnesium sulfate or magnesium chloride1 cup per bath –will alleviate pain
     
    Some of these can apply to menstrual pain as well—body aches—prostrate inflammation—leg problems—circulatory problems—muscle joint pain Back aches and the list goes on
     
     
    TOP G
     
    **********************************************************************************************
    TOP H
    HOME
    Show Of the Week January 31- 2011
     
    Nutrients that can Induce Sleep
     
    Some things that can carry Melatonin
     
    Tryptophan (Trp) Content of Various Foods
     
    Stages of Sleep
     
     
    Nutrients That Can Induce Sleep
    5-HTP (5-Hydroxytryptophan) increases total Sleep time.
    Acetyl-L-Carnitine improves the quality of Sleep and reduces the optimal number of hours of Sleep required.
    Phenylalanine reduces the amount of Sleep time required.
    Tryptophan (500 – 4,000 mg per night) reduces the amount of time taken to fall asleep (Sleep latency) when taken one to two hours prior to retiring.
     
    Antioxidants—Butylated Hydroxytoluene (BHT) reduces the amount of Sleep time that is required for optimal health.
     
    Hormones—-Supplemental, exogenous Dehydroepiandrosterone (DHEA) (50 mg per day) improves the quality of Sleep (especially in people aged 40 and over).
    Human Growth Hormone (hGH) helps to regulate and facilitate Sleep:
    Endogenous hGH is released during Slow-Wave Sleep and helps to regulate REM Sleep. Exogenous, supplemental hGH has been demonstrated to restore Slow-Wave Sleep and REM Sleep patterns to normal. Supplemental, exogenous Melatonin decreases the relative proportion of the Drowsiness and Stage 1 Sleep and increases the relative proportion of Stage 2, Slow-Wave and Rapid Eye Movement Sleep Supplemental Melatonin greatly reduces the number of movements during Sleep, reduces the time taken to fall asleep after retiring (Sleep latency), reduces the body’s core temperature during Sleep and improves the subjective quality of Sleep without increasing next-morning Drowsiness.
     
    Progesterone helps to restore normal Sleep cycles.
    Exogenous Vasopressin improves the quality of Sleep, increases total Sleep time and increases the quantity of Slow-Wave Sleep.
     
    Lipids
     
    Exogenous Prostaglandin D2 (PGD2) infused into the Cerebral Ventricles (in a clinical setting) induces natural Sleep.
     
    Minerals
     
    Magnesium (when consumed just prior to retiring) improves the quality of Sleep. Potassium improves the quality of Sleep and reduces the frequency of awakenings after the onset of Sleep.
     
    Neurotransmitters
     
    Acetylcholine helps to maintain Sleep. It controls the amount of sensory input that reaches the Brain during Sleep and increases the stimulus barrier (increasing the threshold of sensory inputs before wakiing occurs) during Sleep. Optimal Acetylcholine allows sleepers to remain asleep through minor noises and other disturbances. Light sleepers are likely to have sub-optimal Acetylcholine.
     
    Supplemental, exogenous Gamma Aminobutyric Acid (GABA) (1,000 – 3,000 mg administered sublingually at night) facilitates Sleep. research
     
    Smart Drugs
     
    Diapid alleviates some Sleep disorders.
    Dimethylaminoethanol (DMAE) reduces the amount of Sleep required by approximately one hour per night (this effect occurs after six weeks of continuous DMAE use):
    DMAE makes it easier for most people to get fall asleep Sleep at night.
    DMAE users experience a sounder Sleep and after six weeks of continuous DMAE use, most people report waking earlier and having a clearer mind upon waking.
     
    Gamma-Hydroxybutyrate (GHB) facilitates both Rapid Eye Movement (REM) Sleep and Slow-Wave (deep or non-REM) Sleep. research
    Minaprine improves the quality of Sleep (when taken early in the day):
    Minaprine users report awakening with reduces tiredness in the morning.
    Caution: Minaprine should not be consumed at night as it can prevent Sleep.
     
    Vitamins
     
    Choline improves the quality of Sleep. It helps to keep an individual asleep after they have fallen asleep (via its role as a precursor for Acetylcholine, the Neurotransmitter that helps to prevent unnecessary interruptions to Sleep from non-threatening stimuli).
    Inositol (1,000 – 10,000 mg taken at bedtime) improves the quality of Sleep (by assisting Vitamin B3 to bind to the Benzodiazepine Receptors in the Brain).
    Vitamin B3 (200 – 1,000 mg of the Niacinamide form of Vitamin B3 taken at bedtime) improves the quality of Sleep (by binding to the Benzodiazepine Receptors of the Brain).
    Vitamin B6 improves the quality of Sleep (by functioning as a cofactor for the production of Serotonin).
    Vitamin B12 improves the quality of Sleep (by increasing endogenous Melatonin levels early in the night and reducing Melatonin levels at then end of a night’s Sleep).
    Vitamin E improves the quality of Sleep.
     
    These Foods/Herbs Improve the Quality of Sleep
     
    Fruits—Grapefruit (juice consumed at night) improves the quality of Sleep.
     
    Herbs—-Astragalus improves the quality of Sleep (it has been demonstrated to increase Sleep time in animal studies).
    Catuaba reputedly improves the quality of Sleep.
    Damiana helps some people to Sleep better (according to anecdotal reports).
    Essiac (a mixture of Herbs – primarily Sheep Sorrel) improves the quality of Sleep (according to anecdotal reports).
    Ginkgo biloba rectifies impaired Sleep quality in persons using Tricyclic Antidepressants and may also improve Sleep quality in normal, healthy persons: Ginkgo biloba reduces the duration of Stage 1 Sleep in persons using Tricyclic Antidepressants and may also reduce the duration of Stage 1 Sleep in normal, healthy persons. Ginkgo biloba enhances the quality of Slow-Wave Sleep (deep Sleep) in persons using Tricyclic Antidepressants and may also enhance the quality of Slow-Wave Sleep in normal, healthy persons.
     
    Lavender (oil used in Aromatherapy) and tincture or tea improves the quality of Sleep.
    Passion Flower induces a restful Sleep free from frequent awakenings and disturbances (by means of its constituent Harmala Alkaloids sedating the Central Nervous System).
    Siberian Ginseng normalizes Sleep patterns.
    Valerian improves the quality of Sleep.
    Vanillin or Vanilla assist in the relaxation and inducing of sleep
    ++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++++
    Recipes for Sleep—Glycine 500mgs + Inositol 500mgs 30 min before bed
    Taurine 500mgs + magnesium—Gaba 500mgs + Niacinamide 250-500mgs
    Melatonin- Trytophan 500mgs + Niacin 50 mgs Vanilla 10 drops + Lavender tincture 10 drops in 2 oz of water—Passion flower 10 drops + St John’s wort
    Valerian tincture + Passion Flower10 drops of each in water—Utilize an of these combinations 30 minutes before going to bed and do not Mix Under any circumstance with anything from the pharmacy that is drug related
     
    Some things that can carry Melatonin
     
    Melatonin: Natural food and non-food sources of melatonin
     
    Melatonin is a molecule synthesized in the brain by the pineal gland and in the gastrointestinal tract. Melatonin is also synthesized in other organisms including plants.
    Melatonin is believed to be responsible for the synchrony of circadian rhythm, modulating sleep patterns with day and night.
    Melatonin is an antioxidant and protects tissues from oxidative damage by free radical elements. Further more, melatonin induces synthesis of endigenous antioxidants such as superoxide dismtase (SOD).
    Several researches indicate that melatonin protects the gastrointestinal tract from irritation, reduces stress-induced lesion formation and heals ulcer.
    Below are lists containing natural plant sources of melatonin. Melatonin content is in nanogram (ng) per gram of plant sample

    #2081
    AvatarEK
    Keymaster

    Melatonin source
    Melatonin content (ng/g)
    Huang-qin
    7,110
    St. John’s wort, flower
    4,390
    Fever few, green leaf
    2,450
    Fever few, gold leaf
    1,920
    St. John’s wort, leaf
    1,750
    White mustard seed
    189
    Black mustard seed
    129
    Wolf berry seed
    103
    Fenugreek seed
    43
    Sunflower seed
    29
    Fennel seed
    28
    Alfalfa seed
    16
    Green cardamom seed
    15
    Tart cherry fruit (Montmorency)
    15
    Flax seed
    12
    Anise seed
    7
    Coriander seed
    7
    Celery seed
    7
    Poppy seed
    6
    Milk thistle seed
    2
    Tart cherry fruit (Balaton)
    2
    Melatonin source
    Melatonin content [ng/g]
    Feverfew, fresh leaf
    > 1,300
    Feverfew, dried leaf
    >7,000
    Almond seed
    39
    Pimpinella peregrina, dried root
    38
    Sunflower seed
    29
    Fennel seed
    28
    Lemon verbena, young plant
    22
    Balm mint, young plant
    16
    Green cardamom seed
    15
    Artcherry, Montmorency, fruit
    15–18
    Anise seed
    7
    Tall fescue seed
    5
    oat seed
    1.8
    Indian corn seed
    1.3
    Rice seed
    1
    Red radish root tuber
    0.6
    Japanese radish , stem and leaves
    0.6
    Tomato fruit
    0.5
    Ginger tuber
    0.5
     
     
    In using this chart keep in mind this is nanogram which means 1 billionth of a mg
    So that implies a small amount—so in order to get any benefit from these sources you would have to consume them in adequate amounts st johns is 4,390 per gram 0r 1000-mgs so if you took 5 grams you would get over 2 mgs which is what the pill is usually between 1-3 miligrams
    ****************************************************************************
    Tryptophan (Trp) Content of Various Foods
     
     
    Food↓
    Protein
    [g/100 g of food]↓
    Tryptophan
    [g/100 g of food]↓
    Tryptophan/Protein [%]↓
    egg, white, dried
    81.10
    1.00
    1.23
    spirulina, dried
    57.47
    0.93
    1.62
    cod, atlantic, dried
    62.82
    0.70
    1.11
    soybeans, raw
    36.49
    0.59
    1.62
    pumpkin seed
    33.08
    0.57
    1.72
    cheese, Parmesan
    37.90
    0.56
    1.47
    Caribou
    29.77
    0.46
    1.55
    sesame seed
    17.00
    0.37
    2.17
    cheese, cheddar
    24.90
    0.32
    1.29
    sunflower seed
    17.20
    0.30
    1.74
    pork, chop
    19.27
    0.25
    1.27
    Turkey
    21.89
    0.24
    1.11
    Chicken
    20.85
    0.24
    1.14
    Beef
    20.13
    0.23
    1.12
    Salmon
    19.84
    0.22
    1.12
    lamb, chop
    18.33
    0.21
    1.17
    perch, Atlantic
    18.62
    0.21
    1.12
    Egg
    12.58
    0.17
    1.33
    wheat flour, white
    10.33
    0.13
    1.23
    baking chocolate, unsweetened
    12.9
    0.13
    1.23
    Milk
    3.22
    0.08
    2.34
    rice, white
    7.13
    0.08
    1.16
    oatmeal, cooked
    2.54
    0.04
    1.16
    potatoes, russet
    2.14
    0.02
    0.84
     
    Stages of Sleep
    Usually sleepers pass through five stages: 1, 2, 3, 4 and REM (rapid eye movement) sleep. These stages progress cyclically from 1 through REM then begin again with stage 1. A complete sleep cycle takes an average of 90 to 110 minutes. The first sleep cycles each night have relatively short REM sleeps and long periods of deep sleep but later in the night, REM periods lengthen and deep sleep time decreases.
    Stage 1 is light sleep where you drift in and out of sleep and can be awakened easily. In this stage, the eyes move slowly and muscle activity slows. During this stage, many people experience sudden muscle contractions preceded by a sensation of falling.
    In stage 2, eye movement stops and brain waves become slower with only an occasional burst of rapid brain waves. When a person enters stage 3, extremely slow brain waves called delta waves are interspersed with smaller, faster waves. In stage 4, the brain produces delta waves almost exclusively. Stages 3 and 4 are referred to as deep sleep or delta sleep, and it is very difficult to wake someone from them. In deep sleep, there is no eye movement or muscle activity. This is when some children experience bedwetting, sleepwalking or night terrors. In 2008 the sleep profession in the US eliminated the use of stage 4. Stages 3 and 4 are now considered stage 3.
    In the REM period, breathing becomes more rapid, irregular and shallow, eyes jerk rapidly and limb muscles are temporarily paralyzed. Brain waves during this stage increase to levels experienced when a person is awake. Also, heart rate increases, blood pressure rises, males develop erections and the body loses some of the ability to regulate its temperature. This is the time when most dreams occur, and, if awoken during REM sleep, a person can remember the dreams. Most people experience three to five intervals of REM sleep each night.
    Infants spend almost 50% of their time in REM sleep. Adults spend nearly half of sleep time in stage 2, about 20% in REM and the other 30% is divided between the other three stages. Older adults spend progressively less time in REM sleep.
    As sleep research is still a relatively young field, scientists did not discover REM sleep until 1953 when new machines were developed to monitor brain activity. Before this discovery it was believed that most brain activity ceased during sleep. Since then, scientists have also disproved the idea that deprivation of REM sleep can lead to insanity and have found that lack of REM sleep can alleviate clinical depression although they do not know why. Recent theories link REM sleep to learning and memory.
    Stage
    Frequency (Hz)
    Amplitude (micro Volts)
    Waveform type
    awake
    15-50
    <50
     
    pre-sleep
    8-12
    50
    alpha rhthym
    1
    4-8
    50-100
    theta
    2
    4-15
    50-150
    splindle waves
    3
    2-4
    100-150
    spindle waves and slow waves
    4
    0.5-2
    100-200
    slow waves and delta waves
    REM
    15-30
    <50
     
    The waveform during REM has low amplitudes and high frequencies., just like the waking state. Early researchers actually called it “paradoxial sleep”.
    According to the results of a study completed by Statistics Canada, the amount of sleep a person gets each night depends on a variety of factors, including gender, marital and employment statuses. More…
    The functions of many organ systems are linked to the sleep cycle.
    Endocrine system—Most hormone secretion is controlled by the circadian clock or in response to physical events. Sleep is one of the events that modify the timing of secretion for certain hormones. Many hormones are secreted into the blood during sleep. For example, scientists believe that the release of growth hormone is related in part to repair processes that occur during sleep. Follicle stimulating hormone and luteinizing hormone, which are involved in maturational and reproductive processes, are among the hormones released during sleep. In fact, the sleep-dependent release of luteinizing hormone is thought to be the event that initiates puberty. Other hormones, such as thyroid-stimulating hormone, are released prior to sleep.— Both sleep and circadian effects interact to produce the overall rhythmic pattern of the pituitary and pituitary-dependent hormones. Some of the 24-h hormonal rhythms depend on the circadian clock (ACTH, cortisol and melatonin), or are sleep related (prolactin and TSH). GH secretion is influenced by the first slow wave sleep (SWS) episode at the beginning of the night. Pulses of prolactin and GH are positively linked to increases in delta wave activity, i.e. deepest phases of sleep, occurring primarily during the first third of the night. Pulses of TSH and cortisol are related to superficial phases of sleep. As a result of the consolidation of the sleep period, the wake-sleep transition is associated with physiological changes with the endocrine system being part of the adaptive mechanism to reduce physical activity during sleep—-
    Renal system—Kidney filtration, plasma flow, and the excretion of sodium, chloride, potassium, and calcium all are reduced during sleep. These changes cause urine to be more concentrated during sleep. —There is also sleep-related increase in plasma aldosterone levels; an increase in prolactin secretion. There is increased parathyroid hormone release during sleep, which may affect calcium excretion. In general, the following are reduced during sleep: glomerular filtration rate, renal plasma flow, filtration fraction, and the excretion of sodium, chloride, potassium, and calcium. Smaller quantities of more concentrated urine are excreted during NREM sleep than during wakefulness; during REM sleep urine excretion is reduced and concentrated to a greater extent than during NREM sleep.
    Alimentary activity–In a person with normal digestive function, gastric acid secretion is reduced during sleep. In those with an active ulcer, gastric acid secretion is actually increased and swallowing occurs less frequently.

    #2082
    AvatarEK
    Keymaster

    The Organic Elite Surrenders to Monsanto
    by Ronnie Cummins
    “The policy set for GE alfalfa will most likely guide policies for other GE crops as well. True coexistence is a must.” — Whole Foods Market, Jan. 21, 2011
    In the wake of a 12-year battle to keep Monsanto’s Genetically Engineered (GE) crops from contaminating the nation’s 25,000 organic farms and ranches, America’s organic consumers and producers are facing betrayal. A self-appointed cabal of the Organic Elite, spearheaded by Whole Foods Market , Organic Valley , and Stonyfield Farm , has decided it’s time to surrender to Monsanto. Top executives from these companies have publicly admitted that they no longer oppose the mass commercialization of GE crops, such as Monsanto’s controversial Roundup Ready alfalfa, and are prepared to sit down and cut a deal for “coexistence” with Monsanto and USDA biotech cheerleader Tom Vilsack. —In a cleverly worded, but profoundly misleading email sent to its customers last week, Whole Foods Market, while proclaiming their support for organics and “seed purity,” gave the green light to USDA bureaucrats to approve the “conditional deregulation” of Monsanto’s genetically engineered, herbicide-resistant alfalfa. Beyond the regulatory euphemism of “conditional deregulation,” this means that WFM and their colleagues are willing to go along with the massive planting of a chemical and energy-intensive GE perennial crop, alfalfa; guaranteed to spread its mutant genes and seeds across the nation; guaranteed to contaminate the alfalfa fed to organic animals; guaranteed to lead to massive poisoning of farm workers and destruction of the essential soil food web by the toxic herbicide, Roundup; and guaranteed to produce Roundup-resistant superweeds that will require even more deadly herbicides such as 2,4 D to be sprayed on millions of acres of alfalfa across the U.S.
    In exchange for allowing Monsanto’s premeditated pollution of the alfalfa gene pool, WFM wants “compensation.” In payment for a new broad spectrum attack on the soil’s crucial ability to provide nutrition for food crops and to sequester dangerous greenhouse gases (recent studies show that Roundup devastates essential soil microorganisms that provide plant nutrition and sequester climate-destabilizing greenhouse gases), WFM wants the Biotech Bully of St. Louis to agree to pay “compensation” (i.e. hush money) to farmers “for any losses related to the contamination of his crop.”
    In its email of Jan. 21, 2011 WFM calls for “public oversight by the USDA rather than reliance on the biotechnology industry,” even though WFM knows full well that federal regulations on Genetically Modified Organisms (GMOs) do not require pre-market safety testing, nor labeling; and that even federal judges have repeatedly ruled that so-called government “oversight” of Frankencrops such as Monsanto’s sugar beets and alfalfa is basically a farce. At the end of its email, WFM admits that its surrender to Monsanto is permanent: “The policy set for GE alfalfa will most likely guide policies for other GE crops as well True coexistence is a must.” –Why Is Organic Inc. Surrendering?
    According to informed sources, the CEOs of WFM and Stonyfield are personal friends of former Iowa governor, now USDA Secretary, Tom Vilsack, and in fact made financial contributions to Vilsack’s previous electoral campaigns. Vilsack was hailed as “Governor of the Year” in 2001 by the Biotechnology Industry Organization, and traveled in a Monsanto corporate jet on the campaign trail. Perhaps even more fundamental to Organic Inc.’s abject surrender is the fact that the organic elite has become more and more isolated from the concerns and passions of organic consumers and locavores. The Organic Inc. CEOs are tired of activist pressure, boycotts, and petitions. Several of them have told me this to my face. They apparently believe that the battle against GMOs has been lost, and that it’s time to reach for the consolation prize. The consolation prize they seek is a so-called “coexistence” between the biotech Behemoth and the organic community that will lull the public to sleep and greenwash the unpleasant fact that Monsanto’s unlabeled and unregulated genetically engineered crops are now spreading their toxic genes on 1/3 of U.S. (and 1/10 of global) crop land.—WFM and most of the largest organic companies have deliberately separated themselves from anti-GMO efforts and cut off all funding to campaigns working to label or ban GMOs. The so-called Non-GMO Project , funded by Whole Foods and giant wholesaler United Natural Foods (UNFI) is basically a greenwashing effort (although the 100% organic companies involved in this project seem to be operating in good faith[U1]) to show that certified organic foods are basically free from GMOs (we already know this since GMOs are banned in organic production), while failing to focus on so-called “natural” foods, which constitute most of WFM and UNFI’s sales and are routinely contaminated with GMOs. —From their “business as usual” perspective, successful lawsuits against GMOs filed by public interest groups such as the Center for Food Safety; or noisy attacks on Monsanto by groups like the Organic Consumers Association, create bad publicity, rattle their big customers such as Wal-Mart, Target, Kroger, Costco, Supervalu, Publix and Safeway; and remind consumers that organic crops and foods such as corn, soybeans, and canola are slowly but surely becoming contaminated by Monsanto’s GMOs.—Whole Food’s Dirty Little Secret: Most of the So-Called “Natural” Processed Foods and Animal Products They Sell Are Contaminated with GMOs–The main reason, however, why Whole Foods is pleading for coexistence with Monsanto, Dow, Bayer, Syngenta, BASF and the rest of the biotech bullies, is that they desperately want the controversy surrounding genetically engineered foods and crops to go away. Why? Because they know, just as we do, that 2/3 of WFM’s $9 billion annual sales is derived from so-called “natural” processed foods and animal products that are contaminated with GMOs. We and our allies have tested their so-called “natural” products (no doubt WFM’s lab has too) containing non-organic corn and soy, and guess what: they’re all contaminated with GMOs, in contrast to their certified organic products, which are basically free of GMOs, or else contain barely detectable trace amounts.–Approximately 2/3 of the products sold by Whole Foods Market and their main distributor, United Natural Foods (UNFI) are not certified organic, but rather are conventional (chemical-intensive and GMO-tainted) foods and products disguised as “natural.”—Unprecedented wholesale and retail control of the organic marketplace by UNFI and Whole Foods, employing a business model of selling twice as much so-called “natural” food as certified organic food, coupled with the takeover of many organic companies by multinational food corporations such as Dean Foods, threatens the growth of the organic movement.—Covering Up GMO Contamination: Perpetrating “Natural” Fraud—Many well-meaning consumers are confused about the difference between conventional products marketed as “natural,” and those nutritionally/environmentally superior and climate-friendly products that are “certified organic.” —Retail stores like WFM and wholesale distributors like UNFI have failed to educate their customers about the qualitative difference between natural and certified organic, conveniently glossing over the fact that nearly all of the processed “natural” foods and products they sell contain GMOs, or else come from a “natural” supply chain where animals are force-fed GMO grains in factory farms or Confined Animal Feeding Operations (CAFOs).—A troubling trend in organics today is the calculated shift on the part of certain large formerly organic brands from certified organic ingredients and products to so-called “natural” ingredients. With the exception of the “grass-fed and grass-finished” meat sector, most “natural” meat, dairy, and eggs are coming from animals reared on GMO grains and drugs, and confined, entirely, or for a good portion of their lives, in CAFOs. —Whole Foods and UNFI are maximizing their profits by selling quasi-natural products at premium organic prices. Organic consumers are increasingly left without certified organic choices while genuine organic farmers and ranchers continue to lose market share to “natural” imposters. It’s no wonder that less than 1% of American farmland is certified organic, while well-intentioned but misled consumers have boosted organic and “natural” purchases to $80 billion annually-approximately 12% of all grocery store sales. The Solution: Truth-in-Labeling Will Enable Consumers to Drive So-Called “Natural” GMO and CAFO-Tainted Foods Off the Market[U2]—There can be no such thing as “coexistence” with a reckless industry that undermines public health, destroys biodiversity, damages the environment, tortures and poisons animals, destabilizes the climate, and economically devastates the world’s 1.5 billion seed-saving small farmers. There is no such thing as coexistence between GMOs and organics in the European Union. Why? Because in the EU there are almost no GMO crops under cultivation, nor[U3] GM consumer food products on supermarket shelves. And why is this? Because under EU law, all foods containing GMOs or GMO ingredients must be labeled. Consumers have the freedom to choose or not to choose GMOs; while farmers, food processors, and retailers have (at least legally) the right to lace foods with GMOs, as long as they are safety-tested and labeled. Of course the EU food industry understands that consumers, for the most part, do not want to purchase or consume GE foods. European farmers and food companies, even junk food purveyors like McDonald’s and Wal-Mart, understand quite well the concept expressed by a Monsanto executive when GMOs first came on the market: “If you put a label on genetically engineered food you might as well put a skull and crossbones on it.”—The biotech industry and Organic Inc. are supremely conscious of the fact that North American consumers, like their European counterparts, are wary and suspicious of GMO foods. Even without a PhD, consumers understand you don’t want your food safety or environmental sustainability decisions to be made by out-of-control chemical companies like Monsanto, Dow, or Dupont – the same people who brought you toxic pesticides, Agent Orange, PCBs, and now global warming. Industry leaders are acutely aware of the fact that every single industry or government poll over the last 16 years has shown that 85-95% of American consumers want mandatory labels on GMO foods. Why? So that we can avoid buying them. GMO foods have absolutely no benefits for consumers or the environment, only hazards. This is why Monsanto and their friends in the Bush, Clinton, and Obama administrations have prevented consumer GMO truth-in-labeling laws from getting a public discussion in Congress.
    Although Congressman Dennis Kucinich (Democrat, Ohio) recently introduced a bill in Congress calling for mandatory labeling and safety testing for GMOs, don’t hold your breath for Congress to take a stand for truth-in-labeling and consumers’ right to know what’s in their food. Especially since the 2010 Supreme Court decision in the so-called “Citizens United” case gave big corporations and billionaires the right to spend unlimited amounts of money (and remain anonymous, as they do so) to buy media coverage and elections, our chances of passing federal GMO labeling laws against the wishes of Monsanto and Food Inc. are all but non-existent. Perfectly dramatizing the “Revolving Door ” between Monsanto and the Federal Government, Supreme Court Justice Clarence Thomas, formerly chief counsel for Monsanto, delivered one of the decisive votes in the Citizens United case, in effect giving Monsanto and other biotech bullies the right to buy the votes it needs in the U.S. Congress.–With big money controlling Congress and the media, we have little choice but to shift our focus and go local. We’ve got to concentrate our forces where our leverage and power lie, in the marketplace, at the retail level; pressuring retail food stores to voluntarily label their products; while on the legislative front we must organize a broad coalition to pass mandatory GMO (and CAFO) labeling laws, at the city, county, and state levels.
    The Organic Consumers Association, joined by our consumer, farmer, environmental, and labor allies, has just launched a nationwide Truth-in-Labeling campaign to stop Monsanto and the Biotech Bullies from force-feeding unlabeled GMOs to animals and humans.
    Utilizing scientific data, legal precedent, and consumer power the OCA and our local coalitions will educate and mobilize at the grassroots level to pressure giant supermarket chains (Wal-Mart, Kroger, Costco, Safeway, Supervalu, and Publix) and natural food retailers such as Whole Foods and Trader Joe’s to voluntarily implement “truth-in-labeling” practices for GMOs and CAFO products; while simultaneously organizing a critical mass to pass mandatory local and state truth-in-labeling ordinances – similar to labeling laws already in effect for country of origin, irradiated food, allergens, and carcinogens. If local and state government bodies refuse to take action, wherever possible we must attempt to gather sufficient petition signatures and place these truth-in-labeling initiatives directly on the ballot in 2011 or 2012. If you’re interesting in helping organize or coordinate a Millions Against Monsanto and Factory Farms Truth-in-Labeling campaign in your local community, sign up here: http://organicconsumers.org/oca-volunteer/
    To pressure Whole Foods Market and the nation’s largest supermarket chains to voluntarily adopt truth-in-labeling practices sign here, and circulate this petition widely: http://www.organicconsumers.org/articles/article_22309.cfm
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    New Probiotic Combats Inflammatory Bowel Disease
    ScienceDaily (Feb. 1, 2011) — You know the probiotics in your peach yogurt are healthful, but now it appears they may also be a powerful treatment for disease. A genetically tweaked version of a common probiotic found in yogurt and cheese appears to be an effective therapy for inflammatory bowel diseases such as Crohn’s disease and ulcerative colitis. It may also prove to be useful in colon cancer, another disease triggered by inflammation.—Northwestern Medicine researchers deleted a gene in the probiotic Lactobacillus acidophilus and fed the new form to mice with two different models of colitis. After 13 days of treatment, the novel probiotic strain nearly eliminated colon inflammation in the mice and halted progression of their disease by 95 percent.–“This opens brand new avenues to treat various autoimmune diseases of the gut, including inflammatory bowel disease and colon cancer, all which can be triggered by imbalanced inflammatory immune responses,” said Mansour Mohamadzadeh, associate professor of medicine at Northwestern University Feinberg School of Medicine and lead investigator of the study. He also is a member of the Robert H. Lurie Comprehensive Cancer Center of Northwestern University.–The study will be published Jan. 31 in the Proceedings of the National Academy of Sciences.–While the origin of these bowel diseases is not known, Crohn’s disease and ulcerative colitis are two chronically relapsing diseases in which sufferers have an ongoing tissue inflammation that alters the functioning of the intestine. The diseases affect more than 1 million people in the United States and can cause weight loss, diarrhea, abdominal pain and cramping and gastrointestinal bleeding. Current drug treatment is not completely effective and patients can relapse, Mohamadzadeh said.–“Such gene targeting in a probiotic bacteria such as Lactobacillus acidophilus offers the possibility of a safe, drug-free treatment in the near future,” he said.–In the study, the modified Lactobacillus acidophilus entered the gut, which is akin to a battlefield of friendly fire with immune cells attacking the intestine. The Lactobacillus acidophilus acted as the gut’s peacekeeping force, calming the overstimulated immune cells. The probiotic restored intestinal peace by mobilizing messenger immune cells, called dendritic cells. The dendritic cells, in turn, enhanced the production of other functional immune cells, regulatory T-cells that rebalanced intestinal and systemic inflammation.–“They essentially calm everything down and restore it to normal,” Mohamadzadeh explained. The next step will be a clinical trial with the new form of Lactobacillus acidophilus. Mohamadzadeh and his colleagues at the Lurie Cancer Center are currently researching the effect of the new Lactobacillus acidophilus on colon cancer. Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Northwestern University, via EurekAlert!, a service of AAAS.
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    More Evidence Cancer Drug Avastin May Cause Harm
    Increased risk of death seen in review of 16 trials
     
    TUESDAY, Feb. 1 (HealthDay News) — A new review finds more evidence that the beleaguered cancer drug Avastin may harm patients.–An analysis of previously published studies found that the drug, when used in combination with chemotherapy or biological therapy, actually increased patient deaths from adverse events, compared with using chemotherapy or biologics alone. –This new information should change the way patients and practitioners think about the drug, said Dr. Shenhong Wu, senior author of the report, published Feb. 2 in the Journal of the American Medical Association. –“I believe the risk-benefit ratio has changed,” said Wu, assistant professor of medicine at Stony Brook University Medical Center in Stony Brook, N.Y. “Physicians need to think about this.”—Avastin (bevacizumab) was okayed in 2008 for use in conjunction with chemotherapy to treat breast cancer under the U.S. Food and Drug Administration’s accelerated approval program. Approval was based on one clinical trial in patients with metastatic HER2-negative breast cancer that found a benefit in terms of cancer recurrence — but not overall survival — and was contingent on further data to confirm the results. –Three subsequent studies failed to find an overall survival benefit and, in fact, showed less impressive improvements in survival involving no progression of cancer. —After reviewing all four studies, the FDA in December recommended revoking approval of Avastin to fight breast cancer. The recommendation didn’t affect use of Avastin for advanced colon, lung, kidney and brain cancer. –Wu and his colleagues looked at 16 completed randomized controlled trials on Avastin involving more than 10,000 patients with different forms of cancer. —Some 2.5 percent of patients taking Genentech-made Avastin died compared with 1.7 percent of those taking chemotherapy alone, a 46 percent increased risk, the investigators found. –The rate was about the same across tumor types but not across different chemotherapy regimens. The risk was increased among those receiving chemotherapy containing taxanes or platinum agents but not other chemo compounds. –The most common causes of death were hemorrhage, followed by low white-blood-cell count and gastrointestinal perforations. –Dr. Roman Perez-Soler, chairman of oncology and chief of medical oncology at Montefiore Medical Center in New York City, a lung cancer expert, pointed out that lung cancer is one of the few malignancies in which Avastin has prolonged survival and thus might still have a place in this armamentarium. –But an accompanying journal editorial raises other considerations, including the cost ($50,000 a year for the drug alone, plus the cost of monitoring and chemo) and a cumbersome delivery method (frequent injections for years). —The author of that editorial acknowledged that Avastin works well in some patients, but said it’s impossible to predict in which patients and for how long benefits will last.
    SOURCES: Shenhong Wu, M.D., Ph.D., assistant professor, medicine, Stony Brook University Medical Center, Stony Brook, N.Y.; Roman Perez-Soler, M.D., chairman, oncology, and chief, medical oncology, Montefiore Medical Center, New York City; Feb. 2, 2011, Journal of the American Medical Association
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    Probiotics protect against bacterial infection
    6/20/2007- A newly characterised strain of probiotic bacteria may have potential to kill Listeria monocytogenes, an often-lethal pathogen in pregnant women, Irish researchers report.
    The research, published in the Proceedings of the National Academy of Sciences, could see pregnant women and a number of other high-risk groups receiving the probiotic to protect them from potential infection.
    Lead author Sinead Corr from University College Cork told NutraIngredients.com: “Our results clearly demonstrate the ability of certain probiotic bacteria to protect against potentially fatal illnesses. More specifically we have shown a role for bacteriocins in protecting against the potentially fatal foodborne pathogen Listeria monocytogenes. “As probiotics mechanisms are specific, thorough understanding of their beneficial effects are required. Understanding these mechanisms will enable their use in prevention and treatment of specific illnesses. This study clearly demonstrates how probiotic bacteria may help to improve the health of consumers.” Probiotic products containing ‘friendly’ bacteria are now well accepted by consumers in many European countries, with putative benefits highlighted for gut and immune health. Some of the researchers in this new study previously reported that a mixture of five Lactobacillus probiotic strains may reduce food poisoning by salmonella. In that instance, the benefits for gut health were reported to be due to the probiotic bacteria adhering to the walls of the intestine, which inhibits the ability of the pathogenic Salmonella to stick and colonise the gut, thereby reducing the infection. The new study offers an alternative method of protection, with a specific strain of Lactobacillus salivarius named UCC118 capable of producing an antibiotic-like compound called a bacteriocin. When the researchers tested UCC118 in mice infected with two strains of food-borne Listeria monocytogenes, EGDe and LO28, they found that the mice were protected against infection. When the mice were given a mutant form of the probiotic that was unable to produce the Abp118 bacteriocin, no protection against infection was observed.
    Also, “Lb. salivarius UCC118 did not offer any protection when mice were infected with a strain of L. monocytogenes expressing the cognate Abp118 immunity protein AbpIM, confirming that the antimicrobial effect is a result of direct antagonism between Lb. salivarius and the pathogen, mediated by the bacteriocin Abp118,” reported the researchers. “The results of the UCC work clearly demonstrate a role for bacteriocins in protecting the host against potentially lethal infections. The study is the first to clearly demonstrate a mechanism by which probiotic bacteria may act to help improve the health of consumers,” said a release from the university. Most foods containing probiotic bacteria are found in the refrigerated section of supermarkets as the bacteria is destroyed by heat and other processing conditions. This has given the dairy sector, already used to handling live bacteria for the manufacture of yoghurt, a major advantage in probiotic foods – probiotic drinking yoghurts are currently the fastest growing dairy product in Europe.
    But increasing research has focused on expanding protecting probiotics during processing and expanding the food categories available to prebiotics. Such an avenue of research has led companies like Cell Biotech from Korea using a dual-coating to protect probiotics against oxygen, acid, moisture and high temperatures for use in emerging new product categories such as breakfast cereals and smoothies. Other approaches are also being explored, with scientists looking at improving probiotic viability by using whey protein gel particles, or prebiotic fibres.
    Source: Proceedings of the National Academy of Sciences
    May 1, 2007, Volume 104, Number 18, Pages 7617-7621
    “Bacteriocin production as a mechanism for the antiinfective activity of Lactobacillus salivarius UCC118”
    Authors: S.C. Corr, Y. Li, C.U. Riedel, P.W. O’Toole, C. Hill, and C.G.M. Gahan
     
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    Diabetic Recipe—what we give here are protocols and changes in diet and as well as a utilization of several things in particular—the things we give are
    The Green drink of Dandelion Parsley and Watercress— ( Video on how to make — http://www.youtube.com/user/HerbsPlusBeadWorks?feature=mhum#p/u/63/GZws7VeUtds )drunk as much as you want ( initially for those who are loaded with sugar this drink will taste like sewer water ( as I have been told many a time) but by usually the third day everyone seems to either enjoy it or tolerate it better) this is due to the fact it is removing the excesses of sugar and toxic waste and renewing the body of nutrients and minerals and regenerates the liver and kidneys and indirectly the pancreas–The other thing we have everyone due is to use Bitter Melon or Karella or Nopal and Juniper Berry—Cinnamon Bark-Peppermint as a tea or as a capsule or as a food—the Bitter Melon and the Karella has a hormone in it that actually can regenerate the Pancreas—so Does the Nopal—the cinnamon –the juniper berry and peppermint have insulin regulating/or producing ( juniper berry) to assist the pancreas in working with a lot less stress—the enzymes are increased and we suggest Pancreatin enzymes or companies such as Megazyme or Infy Zyme due to there ability to break down foods and to reduce inflamed pancreas as well as assist in the regeneration—and as well we have seen in conjunction with this Alpha lipoic acid 200mg in combo with Acetyl carnitine 400mg bring sugar down below 5 ( measured in canada would be the equivalent to about 50 in the usa) the big thing is to lay off of BREADS and Carbs that are starched based and Foods that are high in the glycemic ranges like bananas –Grapes- Watermelon-Raisons—No Pork products either due to there sugars- NO SOY ( Kills Pancreas and Thyroid) and use MCT as a fat for quick and easy utilization of fat and energy with over taxing pancreas or liver–
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    TOP
    [U1]this is not entirely true the standards that are set for food production and levels of contamination aare not according to the standards set–this to is a false sense of security–best to grow your own
    [U2]This is not going to happen –with the EFSA in Europe and agenda 21 progressing and bills C-36 and bills 510 in the USA unless the efsa is totally undermined and broken this legislation will initiate this new way or profitting for those in the drug industry and the food cartels as well
    [U3]This is not entirely true each member of the EU haa jurisdiction on there farms in Spain they grow GMO and push to market it in Europee so does Portugal –In france the can gorw it but the farmers have to pay for any cross contamination problems—germany does grow GMO as well and So does the UK –what is true is the labeling of GMO
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    HOME
     
    Show of the Week February 7, 2011
     
     
    Coffee Human and Plant Protecting Properties 1 & 2
     
    HPV Vaccine Works for Boys- Study Shows First Clear Benefits[U1]
     
    Recipe for Bioflavonoid Extract
     
    Limonene
     
    Nobiletin
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    1) Roasting Coffee Beans a Dark Brown Produces Valued Antioxidants, Scientists Find
    Researchers have found that the prevailing antioxidants present in dark roasted coffee brew extracts result from the green beans being browned under high temperatures—ScienceDaily (Feb. 3, 2011) — Food scientists at the University of British Columbia have been able to pinpoint more of the complex chemistry behind coffee’s much touted antioxidant benefits, tracing valuable compounds to the roasting process.—Lead author Yazheng Liu and co-author Prof. David Kitts found that the prevailing antioxidants present in dark roasted coffee brew extracts result from the green beans being browned under high temperatures.—Their findings will appear in a forthcoming issue of Food Research International.–Liu and Kitts analyzed the complex mixture of chemical compounds produced during the bean’s browning process, called the “Maillard reaction.” The term refers to the work by French chemist Louis-Camille Maillard who in the 1900s looked at how heat affects the carbohydrates, sugars and proteins in food, such as when grilling steaks or toasting bread.—Antioxidants aid in removing free radicals, the end products of metabolism which have been linked to the aging process.—“Previous studies suggested that antioxidants in coffee could be traced to caffeine or the chlorogenic acid found in green coffee beans, but our results clearly show that the Maillard reaction is the main source of antioxidants,” says Liu, an MSc student in the Faculty of Land and Food Systems (LFS).
    “We found, for example, that coffee beans lose 90 per cent of their chlorogenic acid during the roasting process,” says Kitts, LFS food science professor and director of the Food, Nutrition and Health program.
    The UBC study sheds light on an area of research that has yielded largely inconsistent findings. While some scientists report increased antioxidant activity in coffee made from dark roasted beans, others found a decrease. Yet other theories insist that medium roast coffees yield the highest level of antioxidant activity.
    Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of British Columbia.
     
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    2) Proteins in Unroasted Coffee Beans May Become Next-Generation Insecticides
    ScienceDaily (Apr. 1, 2010) — Scientists in Brazil are reporting for the first time that coffee beans contain proteins that can kill insects and might be developed into new insecticides for protecting food crops against destructive pests.–Their study, which suggests a new use for one of the most important tropical crops in the world, appears in ACS’ Journal of Agricultural and Food Chemistry, a bi-weekly publication.–Peas, beans and some other plant seeds contain proteins, called globulins, which ward off insects. Coffee beans contain large amounts of globulins, and Paulo Mazzafera and colleagues wondered whether those coffee proteins might also have an insecticidal effect. The high heat of roasting destroys globulins, so that they do not appear in brewed coffee.—Their tests against cowpea weevil larva, insects used as models for studying the insecticidal activity of proteins, showed that tiny amounts of the coffee proteins quickly killed up to half of the insects. In the future, scientists could insert genes for these insect-killing proteins into important food crops, such as grains, so that plants produce their own insecticides, the researchers suggest. The proteins appear harmless to people.
    Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by American Chemical Society.—Journal Reference–Mirela Batista Coelho, Maria Li%u0301gia Rodrigues Macedo, Se%u0301rgio Marangoni, Desiree Soares da Silva, Igor Cesarino, Paulo Mazzafera. Purification of Legumin-Like Proteins from Coffea arabica and Coffea racemosa Seeds and Their Insecticidal Properties toward Cowpea Weevil (Callosobruchus maculatus) (Coleoptera: Bruchidae). Journal of Agricultural and Food Chemistry, 2010; 58 (5): 3050 DOI: 10.1021/jf9037216
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    HPV Vaccine Works for Boys- Study Shows First Clear Benefits[U2]
    ScienceDaily (Feb. 5, 2011) — The vaccine for human papillomavirus (HPV) can prevent 90 percent of genital warts in men when offered before exposure to the four HPV strains covered by the vaccine, according to a new multi-center study led by H. Lee Moffitt Cancer Center and UCSF.–The four-year, international clinical trial, which also found a nearly 66 percent effectiveness in the general population of young men regardless of prior exposure to these strains, provides the first reported results of using the HPV vaccine as a prophylactic in men.—Initial data from this study informed the Food and Drug Administration’s decision to approve the vaccine for boys in 2009 to prevent warts, while results from a sub study led the FDA to expand approval late last year to prevent anal cancer. Findings can be found in the Feb. 3 issue of the New England Journal of Medicine.–While the HPV vaccine was approved in 2006 for girls to prevent cervical cancer, the vaccine’s benefit for young men was not initially addressed. Yet infection and diseases caused by HPV are common in men, the researchers said, including genital warts, which are one of the leading sexually transmitted diseases (STD) for which treatment is sought nationwide. The Centers for Disease Control and Prevention estimate that half of all sexually active Americans will get HPV at some point in their lives.—“This is an exciting development in the STD world,” said Joel Palefsky, MD, a UCSF professor of medicine who co-led the research along with epidemiologist Anna R. Giuliano, PhD, from the H. Lee Moffitt Cancer Center and Research Institute, in Tampa, FL. “It shows that if we vaccinate males early enough, we should be able to prevent most cases of external genital warts in this population.”—While warts are often considered an annoyance, rather than a life-threatening disease such as cervical cancer, Palefsky noted that warts are a common problem in young people and are often associated with depression, social stigma and loss of self-esteem. Complications of wart treatments are also quite common, he said. The double-blind study included 4,065 healthy men aged 16-26 years, spanning 71 sites in 18 countries. Of those patients, 85 percent reported having exclusively female sexual partners, with the remainder self-identified as having sex with men.–The men were tested at the onset of the trial for previous exposure to each strain and were randomly selected to receive either a placebo or a vaccine that targeted HPV strains 6, 11, 16 and 18. Men with a history of anal or genital warts or lesions were excluded. Participants then received six follow-up examinations over the following three years to assess the vaccine’s effectiveness. In addition to preventing warts, the vaccine also effectively prevented HPV-persistent infection in 86 percent of the participants without previous exposure.”This is the first study to show that this vaccine works in boys,” Giuliano said. “As long as we have a poor record of vaccinations in girls, boys should also be vaccinated.”–Vaccinating boys also should help prevent HPV transmission to women, as well as transmission from men to men, she said, and help reduce the incidence of the virus throughout the general population. This could be particularly significant, Palefsky added, since only 30 to 40 percent of teenage girls in the U.S. have received even one of the three recommended doses of the vaccine.–The authors noted that while they find it likely that the prevention of HPV infection and disease in men will have additional benefits, such as preventing anal, genital and throat cancers, these benefits need to be directly demonstrated through further clinical trials. The international trial included physicians and patients in four U.S. medical centers, as well as in Australia, Brazil, Canada, Germany, Mexico, Norway, Peru and Taiwan.The project was supported by grants from both public and private sources. The trial was designed by the sponsor, Merck & Co, Inc., in collaboration with external investigators and an external data and safety monitoring board.
    Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of California – San Francisco. Journal Reference-Anna R. Giuliano, Joel M. Palefsky, Stephen Goldstone, Edson D. Moreira, Mary E. Penny, Carlos Aranda, Eftyhia Vardas, Harald Moi, Heiko Jessen, Richard Hillman, Yen-Hwa Chang, Daron Ferris, Danielle Rouleau, Janine Bryan, J. Brooke Marshall, Scott Vuocolo, Eliav Barr, David Radley, Richard M. Haupt, Dalya Guris. Efficacy of Quadrivalent HPV Vaccine against HPV Infection and Disease in Males. New England Journal of Medicine, 2011; 364 (5): 401 DOI: 10.1056/NEJMoa0909537
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    Recipe for Bioflavonoid Extract—Take a lemon ( Unsprayed –I get mine from az From friends who have lemon or grapefruit or orange trees) peel the skins off and the whites—Orrrrrr Juice the whole lemon and leave the grinded leftover—what you will do then is take the citrus and begin to add to 1 ½ cup of alcohol –and blend at high speed for 10 minutes—strain –add this fusion back in and add new material and re blend again just adding another 2 oz of alcohol and repeat this 2-3 times—when done you will have extracted the components out of the skins and whites which are the Bioflavonoids of the Citrus.
    ØBioflavonoids—are antioxidants that can increase the strength and absorption of Vitamin C and the ascorbates from C and can prolong the activity of Vitamin C for a longer period of time–Most Bioflavonoids increase the strength and resiliency of Collagen.–Vitamins–Most Bioflavonoids protect and enhance the action of Vitamin C and increase the absorption and bioavailability of orally administered Vitamin C—medicinal properties, especially their putative role in prevention of cancers and cardiovascular diseases. For cancer Cancer–Physiological processing of unwanted flavonoid compounds induces so-called Phase II enzymes that also help to eliminate mutagens and carcinogens, and therefore may be of value in cancer preventionFlavonoids could also induce mechanisms that may kill cancer cells and inhibit tumor invasion. In preliminary studies, UCLA cancer researchers have proposed that study participants who ate foods containing certain flavonoids, such as catechins found in strawberries and green and black teas; kaempferol from brussel sprouts and apples; and quercetin from beans, onions and apples, may have reduced risk of obtaining lung cancer. — Citrus fruits are well known for providing ample amounts of vitamin C. But they also supply bioflavonoids, substances that are not required for life but that may improve health. The major bioflavonoids found in citrus fruits are diosmin, hesperidin, rutin, naringin, tangeretin, diosmetin, narirutin, neohesperidin, nobiletin, and quercetin.– Citrus bioflavonoids and related substances are widely used in Europe to treat diseases of the blood vessels and lymph system, including hemorrhoids, chronic venous insufficiency, leg ulcers, easy bruising, nosebleeds, and lymphedema following breast cancer surgery. These compounds are thought to work by strengthening the walls of blood vessels. Bioflavonoids are also often said to act as antioxidants; however, while they do have antioxidant activity in the test tube, growing evidence suggests that they do not act as antioxidants in people—–
    Hemorrhoids
    A 2-month, double-blind, placebo-controlled trial of 120 individuals with recurrent hemorrhoid flare-ups found that treatment with combined diosmin and hesperidin significantly reduced the frequency and severity of hemorrhoid attacks.21 Another double-blind, placebo-controlled trial of 100 individuals had positive results with the same bioflavonoids in relieving symptoms once a flare-up of hemorrhoid pain had begun. A 90-day, double-blind trial of 100 individuals with bleeding hemorrhoids also found significant benefits for both treatment of acute attacks and prevention of new ones. Finally, this bioflavonoid combination was found to compare favorably with surgical treatment of hemorrhoids. However, less impressive results were seen in a double-blind, placebo-controlled study in which all participants were given a fiber laxative with either combined diosmin and hesperidin or placebo.
    Chronic Venous Insufficiency
    A 2-month, double-blind, placebo-controlled trial of 200 people with relatively severe chronic venous insufficiency found that treatment with diosmin/hesperidin significantly improved symptoms as compared to placebo.
    Another double-blind, placebo-controlled trial of diosmin/hesperidin enrolled 101 people with relatively mild chronic venous insufficiency. The results showed little difference between the two groups; the authors theorize that diosmin/hesperidin might be more effective in severe chronic venous insufficiency.
    ØA 2-month, double-blind, placebo-controlled trial evaluated the effects of diosmin/hesperidin in 107 people with nonhealing leg ulcers (sores) caused by venous insufficiency or other conditions. The results indicated that treatment significantly improved the rate of healing.
    Also, a 3-month, double-blind, placebo-controlled trial of 67 individuals evaluated buckwheat tea (a good source of rutin) for chronic venous insufficiency.27 The results showed less leg swelling in the treated group.
    One study supposedly showed that the supplement oxyrutin is more effective than diosmin/hesperidin for chronic venous insufficiency, but the study was not effective due to procedure
    Easy Bruising
    Some people bruise particularly easily due to fragile capillaries. A 6-week, double-blind, placebo-controlled study of 96 people with this condition found that combined diosmin and hesperidin decreased symptoms of capillary fragility, such as bruising and nosebleeds. –Two studies from the 1960s found benefits with a combination of vitamin C and citrus bioflavonoids for decreasing bruising in collegiate athletes.
    Lymphedema
    Breast cancer surgery sometimes causes persistent swelling of the arm (lymphedema) caused by damage to lymph vessels. Citrus bioflavonoids as well as other natural supplements have shown promise for this condition. In a 3-month, double-blind study, 57 women with lymphedema received either placebo or combination therapy consisting of the modified citrus bioflavonoid trimethylhesperidin chalcone plus the bioflavonoid-rich herb butcher’s broom. The results indicated that use of the bioflavonoid combination resulted in significantly less swelling.
    Cancer
    In a review of 12 studies involving over 5,000 cases, researchers found that people who consumed the highest amounts of flavonoids in their diets had a lower risk of lung cancer than those who consumed less. The significance of these results is weakened by the fact that none of the studies were controlled trials, and the most favorable among them did not account for the quantity of fruits, vegetables, or vitamins in the participants’ diets.
    Some other Uses—
    General Health Benefits of Bioflavonoids
    As noted above, all flavonoids are potentially useful as antioxidants, antivirals, and anti-inflammatories. These properties provide you with the specific potential benefits of bioflavonoids such as:
    • lower cholesterol level
    • stronger blood vessels
    • improved circulation
    • relief from asthma
    • relief from allergy symptoms
    • protection against cancer
    • protection against infections
    • protection against cavities
    • prevention of gum disease
    • improved liver function
    • improved brain function
    • younger-looking skin
    • relief from hemorrhoids
    • treatment and prevention of menstrual disorders
    • improved vision and prevention of eye diseases
    • removal of heavy metals from your body
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    Limonene
    D-limonene is one of the most common terpenes in nature. It is found in
    several citrus oils such as orange, lemon, mandarin, lime, and grapefruit.— Studies have shown that limonene have anti- cancer effects. Limonene increase the levels of liver enzymes involved in detoxifying carcinogens. The Glutathione S-transferase (GST) is a system which eliminates carcinogens. Limonene seems to promote the GST system in the liver and small bowel, thereby decreasing the damaging effects of carcinogens. Animal studies demonstrated that dietary limonene reduced mammary tumor growth—Limonene can dissolve Gallstones.
    Immune System–Limonene can prevent some forms of Cancer
    · Limonene helps to prevent Breast Cancer and also inhibits the progression of established Breast Cancer. research
    · Modification of Limonene by hydroxylation at various positions increases its therapeutic efficiency against Breast Cancer by a factor of 5-10 (for example, the naturally occurring hydroxylated Limonene analogue, Perillyl Alcohol is 5-10 times more potent).
    · Limonene retards the ability of carcinogens to initiate Colon Cancer.
    · Limonene retards the ability of carcinogens to initiate Liver Cancer.
    · Limonene helps to prevent Lung Cancer.
    · Limonene prevents the development of Pancreatic Cancer and causes the regression of existing Pancreatic Cancer.
    · Limonene helps to prevent Skin Cancer:
    · Limonene helps to prevent Squamous Cell Carcinoma.
    · The highest concentrations of Limonene are found in the spongy, white, inner parts of the Fruit of which it (Limonene) is a constituent.
     
    Fruits: Cranberry Lemon Peel* Mandarin Orange Peel Grapefruit Peel
    Herbs: Black Cumin (seeds) Buchu Calendula Catnip Coriander Cumin Hops Lavender Lemon Grass Peppermint Star Anise Angelica Horehound Noni
    Seeds: Cardamom Seeds Celery Seeds Caraway Seeds Dill Seeds
    Vegetables: Celery Fennel
     
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    Nobileton
    Health Benefits of Nobiletin
    Nobiletin seems to be a very noble phytochemical with many potential health benefits. The most studied properties of nobiletin are its anti-inflammatory and anti-cancer activities. Nobiletin also helps to lower cholesterol levels and some studies indicate that it may improve impaired memory loss and treat acne.
    Anti-inflammatory
    Numerous in-vivo and in-vitro studies have demonstrated the anti-inflammatory activity of nobiletin and its metabolites. Nobiletin acts directly as an antioxidant but also interferes with biological inflammatory processes. It inhibits the expression of genes involved in inflammation by blocking the binding of NF-kappaB with DNA. One study showed that nobiletin reduced airway inflammation of asthmatic rats. Treatment of skin cells with nobiletin also reduced inflammation caused by ultraviolet-B radiation.
    Anti-cancer
    The anti-cancer properties of nobiletin are supported by many scientific studies. Nobiletin act by its antiproliferation effect without being toxic to normal cells. Favourable results have been obtained on cancer cell lines of the liver, stomach, prostate and colon.
    Cholesterol lowering
    A study with cultured macrophages demonstrated the cholesterol lowering and atherosclerosis inhibiting properties of nobiletin. Nobiletin also inhibits the formation of macrophage foam cells, macrophages loaded wit lipids, which build up on artery walls.
    Memory loss
    One study on a mice model showed that nobiletin my help to improve memory loss. The brains of the mice were chirurgically altered to obtain models of Alzheimer disease. It is not known if nobiletin improves the condition of human Alzheimer patients.
    Acne treatment
    We found one study showing that nobiletin inhibited sebum production on hamsters and inhibited the proliferation of sebocytes, the cells that form the sebaceous gland. This observation led the researchers to conclude that nobiletin could be used as a drug for acne treatment.
     
     
    TOP A
     
    [U1]This is sooooooo Ridiculous—the idea that now they are going after the male population as a means to vaccinate or sterilize is more the term—the idea that having sex is going to cause genital warts must imply strongly that the people engaging are infected with some blood disorder to begin with and the inus is not on vaccination but rather healthy blood—the concepts here on ” giving a Shot or a Vaccine ” is ludicrious–the Medical Field as per usual wil be unable to figure this out and again it will be up to the engineers ( environmental) that will solve the STD issue since a bulk of these issues are a result of the industrial/Tech age Toxins which get into the blood stream and cause these anomalies—as well as poor hygienic practices
    [U2]This is sooooooo Ridiculous—the idea that now they are going after the male population as a means to vaccinate or sterilize is more the term—the idea that having sex is going to cause genital warts must imply strongly that the people engaging are infected with some blood disorder to begin with and the inus is not on vaccination but rather healthy blood—the concepts here on ” giving a Shot or a Vaccine ” is ludicrious–the Medical Field as per usual wil be unable to figure this out and again it will be up to the engineers ( environmental) that will solve the STD issue since a bulk of these issues are a result of the industrial/Tech age Toxins which get into the blood stream and cause these anomalies—as well as poor hygienic practices
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    TOP B
    HOME
     
    Show of the Week February 11, 2011
     
    MR. GORSKY—Humour and why we need it
    Chemtrail Ingredients
     
    PAPAYA
     
    Recipe for Papaya Extract and Ferment
     
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    MR. GORSKY
    In case you didn’t already know this little tidbit of trivia,
    it might make you chuckle when you read it.
    ON JULY 20, 1969, AS COMMANDER OF THE APOLLO 11 LUNAR MODULE, NEIL ARMSTRONG—WAS THE FIRST PERSON TO SET FOOT ON THE MOON.—HIS FIRST WORDS AFTER STEPPING ON THE MOON, “THAT’S ONE SMALL STEP FOR MAN,-ONE GIANT LEAP FOR MANKIND,” WERE TELEVISED TO EARTH AND HEARD BY MILLIONS.*BUT JUST BEFORE HE RE-ENTERED THE LANDER, HE MADE THE ENIGMATIC REMARK “GOOD LUCK, MR.. GORSKY”.MANY PEOPLE AT NASA THOUGHT IT WAS A CASUAL REMARK CONCERNING SOME RIVAL SOVIET COSMONAUT. HOWEVER, UPON CHECKING, THERE WAS NO GORSKY IN EITHER THE RUSSIAN OR AMERICAN SPACE PROGRAMS. OVER THE YEARS MANY PEOPLE QUESTIONED ARMSTRONG AS TO WHAT THE ‘GOOD LUCK, MR. GORSKY’ STATEMENT MEANT, BUT ARMSTRONG ALWAYS JUST SMILED. ON JULY 5, 1995, IN TAMPA BAY, FLORIDA, WHILE ANSWERING QUESTIONS FOLLOWING A SPEECH, A REPORTER BROUGHT UP THE 26- YEAR-OLD QUESTION TO ARMSTRONG. THIS TIME HE FINALLY RESPONDED. MR. GORSKY HAD DIED, SO NEIL ARMSTRONG FELT HE COULD NOW ANSWER THE QUESTION. IN 1938, WHEN HE WAS A KID IN A SMALL MID-WESTERN TOWN, HE WAS PLAYING BASEBALL WITH A FRIEND IN THE BACKYARD. HIS FRIEND HIT THE BALL, WHICH LANDED IN HIS NEIGHBOR’S YARD BY THEIR BEDROOM WINDOW. HIS NEIGHBORS WERE MR. AND MRS. GORSKY. AS HE LEANED DOWN TO PICK UP THE BALL, YOUNG ARMSTRONG HEARD MRS. GORSKY SHOUTING AT MR. GORSKY. “SEX! YOU WANT SEX?! YOU’LL GET SEX WHEN THE KID NEXT DOOR WALKS ON THE MOON!”
    TRUE STORY
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    Chemtrail Poisoning of America report 2010
     
    Look at these pictures posted on Flicker of the skies over Las Vegas. There is a very quick sign up required to view, but well worth it. These are the once deep blue skies of Southern Nevada. Please take a look.
     
    Vegas skies http://www.flickr.com/photos/crime_of_the_century/
     
    These persistent contrails – as the officials like to call them, are filled with dangerous chemicals, heavy metals, polymer fibers, mycoplasmas and biotoxins, greatly effecting the health and quality of life of those who are breathing them; that is you and your family, your friends, your pets, even your plants.
     
    IIThe politicians do nothing. The media remain silent. It is just unbelievable the inattention this is receiving. I
     
    IIIIAccording to a television station which paid to have these chemicals tested, there are many, sometimes thousands times more – than the allowable amount of barium powders and aluminum oxides, plus ethylene dibromide, arsenic, iron, boron, manganese, magnesium, potassium, sodium, copper, zinc, nickel, thorium, zirconium, titanium, chromium, cadmium, strontium, polymers, mycoplasma and biotoxins in these chemical releases. Toxic levels! (a recent Arizona air sample analysis (all elements) showed 12,800,000 PPB for Aluminum.) Incredible. That’s 12 million parts per billion! 6,400 times the toxic limit!
    Analysis report here: http://www.nogw.com/images/052008_chemtrail_lab_result_lg.jpg
    and here: http://www.rense.com/general82/chemit.htm
     
    In the latest Phoenix air-particulate analysis, aluminum particulates in the ambient air have tested at 19,500 times the toxic limit, iron at 28,333 times the toxic limit, barium at 1,050 times the toxic limit, and manganese at 4,870 times the toxic limit, among many other metals at toxic levels.
    New 2009 analysis report here: http://d1027732.mydomainwebhost.com/charts/our%20charts/phx_particulates_2009.htm
     
    There is recent evidence they are using the aerosol disbursement to introduce biological agents over population centers.
     
    Chemtrials in the Sky and the New Microbes
    Pseudomonas fluorescens, streptomyces, brucellosis and mycoplasma found in chemtrails–By WilliamThomas (scroll down to flu epidemics) http://www.consumerhealth.org/articles/display.cfm?ID=20000830164825
    “During this time most of us have been ill with something that doesn’t quite act like the flu because it holds on for weeks and months at a time, seems to get better, and then returns. It is very hard to shake.”
     
    Pneumonic plague following aerial spraying in the Ukraine
    http://www.theflucase.com/index.php?option=com_content&view=article&id=1666%3Amore-reports-of-pneumonic-plague-following-aerial-spraying-in-the-ukraine&catid=41%3Ahighlighted-news&Itemid=105&lang=en
     
    Scientific studies have shown that exposure to barium, a major chemical component in these aerosol releases, breaks down immune systems, producing a receptive environment for any lab-generated recombinant flu-virus which has “accidently” appeared in the population.
     
    Whether intentional or not, the data shows chemtrails and vaccine adjuvants are weakening our immune systems, making us susceptible to illness. Illness delivered in prepared pandemics which keep creeping into the population with slight modifications, each demanding another tainted, untested vaccination: first as swine flu, then SARS, then bird flu, then a recombinant bird/swine/human flu. These vaccines are used as delivery mechanisms, tested on the public tainted with squalene, mercury, aluminum, polysorbate 80 and live viruses. Besides destroying immune systems, these additives often cause illness, paralysis, even death.
     
    Big pharma and the CDC will demand each of you take these untested vaccines. They will work to make them mandatory and the collaborative news media will be echoing the mantra “get the shot, get the shot” – all the while having stripped hapless victims of this vaccine of any legal recourse against the pharmaceutical industry.
     
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    PAPAYA–
    Papain is a protein-cleaving enzyme derived from papaya and certain other plants. Enzymes are complex molecules produced in living organisms to catalyze (speed up) chemical reactions within the cell. A number of digestive enzyme supplements are available. The simple ones are extracted from tropical fruits: bromelain from pineapple and papain from papayas. Papain has a mild, soothing effect on the stomach and aids in protein digestion. It is most often used as a meat tenderizer.
    defibrinating wounds in hospitals
    clotting milk
    shrink proofing of wool
    used in pet food to reduce viscosity and increase palatability
    prevents cornea scar deformation
    used in treatments of jellyfish and insect sings
    used to treat edemas, inflammatory processes, and in the acceleration of wound healing
    it is used as an ingredient in cleaning solutions for soft contact lenses
    in low doses in can be used to as an indigestion medicine.
    As we can see the uses for papain are diverse and thus it makes it an extremely valuable enzyme. Papain has been employed to treat ulcers, dissolve membranes in diphtheria, and reduce swelling, fever and adhesions after surgery. With considerable risk, it has been applied on meat impacted in the gullet. Chemopapain is sometimes injected in cases of slipped spinal discs or pinched nerves. Precautions should be taken because some individuals are allergic to papain in any form and even to meat tenderized with papain. In tropical folk medicine, the fresh latex is smeared on boils, warts and freckles and given as a vermifuge. In India, it is applied on the uterus as an irritant to cause abortion. The unripe fruit is sometimes hazardously ingested to achieve abortion. Seeds, too, may bring on abortion. They are often taken as an emmenagogue ( menustration) and given as a vermifuge( worm expeller). The root is ground to a paste with salt, diluted with water and given as an enema to induce abortion. A root decoction is claimed to expel roundworms. Roots are also used to make salt. Crushed leaves wrapped around tough meat will tenderize it overnight. The leaf also functions as a vermifuge ( worm expeller)and as a primitive soap substitute in laundering. Dried leaves have been smoked to relieve asthma or as a tobacco substitute. Packages of dried, pulverized leaves are sold by “health food” stores for making tea, despite the fact that the leaf decoction is administered as a purgative for horses in Ghana and in the Ivory Coast it is a treatment for genito-urinary ailments. The dried leaf infusion is taken for stomach troubles in Ghana and they say it is purgative and may cause abortion. bacteria. Strong doses are effective against gram-negative bacteria. The substance has protein-like properties. The fresh crushed seeds yield the aglycone of glucotropaeolin benzyl isothiocyanate (BITC) which is bacteriostatic, bactericidal and fungicidal. A single effective does is 4-5 g seeds (25-30 mg BITC). In a London hospital in 1977, a post-operative infection in a kidney-transplant patient was cured by strips of papaya which were laid on the wound and left for 48 hours, after all modern medications had failed. Plant extracts with a high content of proteolytic enzymes have been used for a long time in traditional medicine. Besides proteolytic enzymes from plants, ‘modern’ enzyme therapy additionally includes pancreatic enzymes. The therapeutic use of proteolytic enzymes is partly based on scientific studies and is partly empirical. The aim of the current review is to provide an overview of clinical trials of systemic enzyme therapy in oncology, and to discuss the evidence for their possible mechanisms of action. Clinical studies of the use of proteolytic enzymes in oncology have mostly been carried out on an enzyme preparation consisting of a combination of papain, trypsin and chymotrypsin. This review of these studies owed that enzyme therapy can reduce the adverse effects caused by radiotherapy and chemotherapy. There is also evidence that, in some types of tumours, survival may be prolonged. The beneficial effect of systemic enzyme therapy seems to be based on its anti-inflammatory potential. However, the precise mechanism of action of systemic enzyme therapy remains unsolved. The ratio of proteinases to antiproteinases, which is increasingly being used as a prognostic marker in oncology, appears to be influenced by the oral administration of proteolytic enzymes, probably via an induction of the synthesis of antiproteinases. Furthermore, there are numerous alterations of cytokine composition during therapy with orally administered enzymes, which might be an indication of the efficacy of enzyme therapy.
    Frederick A. Senese. General Chemistry Online! Why is papain an effective meat tenderizer?
    Edgar A. Chavez. PAPAIN-PAPAYA PEPTIDASE I. The Claremont Colleges Joint Science Program
    Morton, J. 1987. Papaya. p. 336�346. In: Fruits of warm climates. Julia F. Morton, Miami, FL.
    Leipner J, Saller R. Systemic enzyme therapy in oncology: effect and mode of action. Drugs 2000 Apr;59(4):769-80
    Health Benefits of Papaya
    Papayas is a pear shaped tropical fruit and than be as long as 7 to 20 inches. This fruit is available in the market throughout the year, but most abundant during the summer.
    Nutritive values : Per 100 gm. Vitamin A : 1,750 I.U Vitamin C : 56 mg. Calcium : 20 mg. Phosphorus : 16 mg. Potassium : 470 mg. Carbohydrates : 10 gm Calories : 39 Also contains Vitamin E and K
    Health Benefits of Papaya :Papaya contains the digestive enzyme papain that is why it is valuable for aiding digestion. The ripe fruit is easily digestible and prevents constipation. It is shown in a study that this fruit when taken alone for 3 days has a beneficial tonic effect in the stomach and intestines. Papaya juice helps in relieving infections of the colon and can help break down pus and mucus. May help prevent cancer in organs and glands with epithelial tissue (ripe papaya). Prevents nausea (includes morning sickness and motion sickness). Taken 230 cc. Of papaya juice or pineapple juice. Slices of green fruits rubbed over meat and boiled with tough meat, make the meat more tender. The seeds are antihelmintic, for expelling worms and they are given with honey. Chew and swallow two teaspoonful of seeds after each principal meal (three times a day).
     

    #2083
    AvatarEK
    Keymaster

    Recipe for Papaya Extract and Ferment
    Take a ripe papaya and peel of outer layer with a potato peeler or thin knife—split the papaya and scoop out the seeds—SAVE THE SEEDS!!!!! Then add 1cup to 1 ½ cup of either a rice wine or a white wine or a clear alcohol of choice ( grappa-spirtus-everclear-gin-vodka) and add cinnamon to this as well about 1 tsp per cycle.
    You will add about 2-3 ounces of papaya per cycle, Meaning you will put this in then strain it and then add the strained fluid back in the blender and add another 2-3 oz and repeat this procedure till you use up all the papaya–You will blend all this for about 5 minutes at high speed and from there proceed to strain through a handkerchief or pillow case ( reason being the cheese cloths will leak this is dense and will require a good grip) pour back into the blender the strained material and repeat the process
     
    For the Ferment you can take the papaya and sprinkle on it cinnamon and tumeric and paprika add vinegar and allow to sit forseveral weeks in fridge and allow for the ferment to take hold and consume as desired especially after a heavy meal of fat or protein—or you can strain it out as previous and add vinegar to this and allow to sit for several days and then use tablespoon amounts again best to be taken after a meal or for healing an issue or imbalance—this will increase the SOD levels as well which is an extremely potent antioxidant that can protect eyes and brain and liver from infectious issues
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    TOP B
     
     
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    TOP C
    HOME
     
    Show of the Week February 14, 2011
     
    Manufacturing of Intravenous Vitamin C Banned
     
    FDA warns niacin marketers about drug claims
    Drinking Wine Lowers Risk Of Barrett’s Esophagus, Precursor To Nation’s Fastest Growing Cancer- Study
     
    Drinking Wine May Increase Survival Among Non-Hodgkin’s Lymphoma Patients
     
    Red Wine Protects The Prostate -Study
     
    Half A Glass Of Wine A Day May Boost Life Expectancy By Five Years
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    Manufacturing of Intravenous Vitamin C Banned
     
    As you may be aware the largest manufacturers of L Ascor 500, McGuff Pharmaceuticals, has received a notice from the FDA to cease manufacturing the product on December 28th 2010.
     
    http://www.fda.gov/ICECI/EnforcementActions/WarningLetters/ucm238251.htm
     
    McGuff Pharmaceuticals is the largest manufacturer of the product and probably the largest supplier to New Zealand. This could mean the product will be harder to source until other manufacturers build their supply.[U1]
     
    We have been in contact with the CEO of McGuff to seek clarification of what has occurred and this was one of the responses: “The FDA told McGuff Pharmaceuticals, Inc. to stop “manufacturing and distribution” of Ascorbic Acid injection. However, the FDA may use its discretionary authority to allow it back into the market[U2]. We just do not know what the FDA’s intentions are as the FDA will not provide policy information.”
     
    You’ll note the date is only a few months after Allen Smith’s story of recovery from Swine Flue and Hairy Cell Leukaemia (using L Ascor 500) went global. We are in the process of assessing the impact to New Zealand and our campaign, so any of your ideas would be appreciated.
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    FDA warns niacin marketers about drug claims
    The Food and Drug Administration has affirmed the principal that references to scientific studies can amount to drug claims under the Federal Food, Drug, and Cosmetic Act (FDCA).
    In a January 19, 2011, warning letter to Minnesota-based Upsher Smith Laboratories, the FDA rapped the company over scientific web references that were linked cardiovascular and cholesterol benefits for the company’s niacin-based dietary supplements. Upsher Smith Laboratories has subsequently taken the website for Slo-Niacin products offline.
    “The therapeutic claims on your website establish that the product is a drug because it is intended for use in the diagnosis, cure, mitigation, treatment, or prevention of disease,” wrote FDA director from the Minneapolis District, Gerald J Berg.[U3]
    Diseased citations
    Berg went on to highlight the problem of companies referencing scientific studies in online marketing materials.
    “Furthermore, the ‘References’ and ‘In the News’ sections of your website for this product cite a number of articles about the usefulness of the ingredient niacin in treating and preventing diseases,” he wrote.
    “When scientific publications are used commercially by the seller of a product to promote the product to consumers, such publications may become evidence of the product’s intended use. For example, under 21 CFR 101.93(g)(2)(iv)(C), [of the FDCA] a citation of a publication or reference in the labeling of a product is considered to be a claim about disease treatment or prevention if the citation refers to a disease use, and if, in the context of the labeling as a whole, the citation implies treatment or prevention of a disease.”
    Claims at stake
    Claims being made on the retracted site included:
    SLO-NIACIN…Reduced risk of cardiovascular events* when combined with a statin
    • SLO-NIACIN…Halted stenosis progression when combined with a statin.
    • Niacin is clinically proven to help manage cholesterol levels, a major factor in heart health
    • SLO-NIACIN Offers an affordable option for Cholesterol Management
    • Niacin is the oldest of today’s commonly used agents for lowering cholesterol
    • See how…niacin complements statin therapy for heart health
    The FDA cited numerous examples of studies that contributed to disease marketing including a 1992 study published in the American Journal of Cardiology titled: ‘Marked benefit with sustained-release niacin therapy in patients with ‘isolated’ very low levels of high-density lipoprotein cholesterol and coronary artery disease.’
    The letter qualified the drug accusations by noting that Slo-Niacin did not have generally recognized as safe (GRAS) status for use as a food ingredient.
    It also highlights several images used on the Slo-Niacin website that can also, “be considered as a claim to diagnose, mitigate, treat, cure, or prevent disease if in the context of the labeling it suggests that the product has an effect on a disease or diseases.”
    Niacin, vitamin B3, has been shown to benefit atherosclerosis at high doses by reducing total cholesterol, triglycerides, very-low-density lipoprotein, and low-density lipoprotein.
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    Drinking Wine Lowers Risk Of Barrett’s Esophagus, Precursor To Nation’s Fastest Growing Cancer- Study
     
    ScienceDaily (Mar. 7, 2009) — Drinking one glass of wine a day may lower the risk of Barrett’s Esophagus by 56 percent, according to a new study by the Kaiser Permanente Division of Research in the March issue of Gastroenterology. Barrett’s Esophagus is a precursor to esophageal cancer, the nation’s fastest growing cancer with an incidence rate that’s jumped 500 percent in the last 30 years.—Barrett’s Esophagus affects 5 percent of the population and occurs when heartburn or acid reflux permanently damages the esophageal lining. People with Barrett’s Esophagus have a 30- to 40-fold higher risk of developing esophageal adenocarcinoma (a type of esophageal cancer) because the Barrett’s Esophagus cells can grow into cancer cells.—Because there are no symptoms or warning signs of Barrett’s Esophagus, people discover they have Barrett’s Esophagus when an endoscopy for anemia, heartburn or a bleeding ulcer reveals esophageal cells that were damaged, then changed form during the healing process. Currently nothing can be done to treat Barrett’s Esophagus; it can only be monitored.
    This is the first and largest population-based study to examine the connection between alcohol consumption and risk of Barrett’s Esophagus. Funded in part by the National Institutes of Health, the Kaiser Permanente study looked at 953 men and women in Northern California between 2002 and 2005 and found that people who drank one or more glasses of red or white wine a day had less than half the risk (or 56 percent reduced risk) of Barrett’s Esophagus. There was no reduction of Barrett’s Esophagus risk among people who drank beer or liquor.–“The rate of esophageal adenocarcinoma in this country is skyrocketing yet very little is known about its precursor, Barrett’s Esophagus. We are trying to figure out how to prevent changes that may lead to esophageal cancer.” said Douglas A. Corley, MD, a Kaiser Permanente gastroenterologist and the study’s principal investigator.
    The study findings are echoed by two other studies published in the same issue of the Gastroenterology journal: Australian researchers found that people who drank wine were at a lower risk of esophageal adenocarcinoma, and Irish researchers found that people who drank wine were at a lower risk for esophagitis, an irritation of the esophagus that follows chronic heartburn and often precedes Barrett’s Esophagus and cancer.—Researchers are not certain why wine reduces the risk of Barrett’s Esophagus and esophageal cancer. One theory is that the wine’s antioxidants neutralize the oxidative damage caused by gastroesophageal reflux disease, a risk factor for Barrett’s Esophagus. Another theory is that wine drinkers typically consume food with their wine as opposed to drinking straight liquor without food, thereby reducing the potentially damaging effect of alcohol on esophageal tissue, said Ai Kubo, MD, an epidemiologist at Kaiser Permanente and lead author on the study. “But we cannot preclude the possibility that wine drinking is a proxy for other ‘health-seeking’ behavior,” Kubo added.
    This study is part of larger, case-controlled Kaiser Permanente study led by Dr. Corley that looked at abdominal obesity and consumption of dietary antioxidants, fruits and vegetables in connection with Barrett’s Esophagus. It found that people can reduce their risk of Barrett’s Esophagus by eating eight servings of fruits and vegetables a day and maintaining a normal body weight.–“My advice to people trying to prevent Barrett’s Esophagus is: keep a normal body weight and follow a diet high in antioxidants and high in fruits and vegetables,” Corley said. “We already knew that red wine was good for the heart, so perhaps here is another added benefit of a healthy lifestyle and a single glass of wine a day.”–Researchers noted, though, that the protective effect of wine in terms of preventing Barrett’s Esophagus was greatest with just one or two glasses a day. The protective effect of wine did not increase with higher consumption.–“It’s not actually clear that treating the acid reflux will necessarily prevent getting someone from getting Barrett’s Esophagus,” said Dr. Corley. “The best way to prevent reflux is to maintain a normal weight.” Authors of the study include: Ai Kubo, T. R. Levin, Gregory Rumore, Charles P. Quesenberry, Jr., of the Kaiser Permanente Division of Research in Oakland, Gladys Block and Patricia Buffler of the School of Public Health, University of California-Berkeley.
    Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Kaiser Permanente, via EurekAlert!, a service of AAAS
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    Drinking Wine May Increase Survival Among Non-Hodgkin’s Lymphoma Patients
     
    ScienceDaily (Apr. 24, 2009) — Pre-diagnostic wine consumption may reduce the risk of death and relapse among non-Hodgkin’s lymphoma patients, according to an epidemiology study presented at the American Association for Cancer Research 100th Annual Meeting 2009.–Xuesong Han, the first author of the abstract and a doctoral candidate at the Yale School of Public Health, said their findings would need to be replicated before any public health recommendations are made, but the evidence is becoming clearer that moderate consumption of wine has numerous benefits.”This conclusion is controversial, because excessive drinking has a negative social and health impact, and it is difficult to define what is moderate and what is excessive,” said Han. “However, we are continually seeing a link between wine and positive outcomes in many cancers.”–This study was the first to examine the link among patients with non-Hodgkin’s lymphoma. Han and her colleagues analyzed data about 546 women with non-Hodgkin’s lymphoma.–They found that those who drank wine had a 76 percent five-year survival compared with 68 percent for non-wine drinkers. Further research found five-year, disease-free survival was 70 percent among those who drank wine compared with 65 percent among non-wine drinkers.–Beer and/or liquor consumption did not show a benefit. The study team at Yale also looked at subgroups of lymphoma patients, and found the strongest link between wine consumption and favorable outcomes among patients with diffuse large B-cell lymphoma. These patients had a 40 to 50 percent reduced risk of death, relapse or secondary cancer.-Researchers then conducted an analysis to examine the effect of wine consumption among those who had drunk wine for at least the previous 25 years before diagnosis. Non-Hodgkin’s lymphoma patients who had been drinking wine for at least this long had a 25 to 35 percent reduced risk of death, relapse or secondary cancer.–Those patients with large B-cell lymphoma had about 60 percent reduced risk of death, relapse or secondary cancer if they had been drinking wine for at least the previous 25 years before diagnosis.–“It is clear that lifestyle factors like alcohol can affect outcome,” said Han. -Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by American Association for Cancer Research
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    Red Wine Protects The Prostate -Study
    ScienceDaily (May 26, 2007) — Researchers have found that men who drink an average of four to seven glasses of red wine per week are only 52% as likely to be diagnosed with prostate cancer as those who do not drink red wine, reports the June 2007 issue of Harvard Men’s Health Watch. In addition, red wine appears particularly protective against advanced or aggressive cancers.–Researchers in Seattle collected information about many factors that might influence the risk of prostate cancer in men between ages 40 and 64, including alcohol consumption. At first the results for alcohol consumption seemed similar to the findings of many earlier studies: There was no relationship between overall consumption and risk. –But the scientists went one step further by evaluating each type of alcoholic beverage independently. Here the news was surprising—wine drinking was linked to a reduced risk of prostate cancer. And when white wine was compared with red, red had the most benefit. Even low amounts seemed to help, and for every additional glass of red wine per week, the relative risk declined by 6%. Why red wine? Doctors don’t know. But much of the speculation focuses on chemicals—including various flavonoids and resveratrol—missing from other alcoholic beverages. These components have antioxidant properties, and some appear to counterbalance androgens, the male hormones that stimulate the prostate.–Many doctors are reluctant to recommend drinking alcohol for health, fearing that their patients might assume that if a little alcohol is good, a lot might be better. The Harvard Men’s Health Watch notes that men who enjoy alcohol and can drink in moderation and responsibly may benefit from a lower risk of heart attack, stroke, diabetes, and cardiac death.
    Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Harvard Men’s Health Watch, via Newswise
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    Half A Glass Of Wine A Day May Boost Life Expectancy By Five Years
     
    Researchers found that men who drank only wine, and less than half a glass of it a day, lived around 2.5 years longer than those who drank beer and spirits, and almost five years longer than those who drank no alcohol at all –ScienceDaily (Apr. 30, 2009) — Drinking up to half a glass of wine a day may boost life expectancy by five years—at least in men, suggests research published ahead of print in the Journal of Epidemiology and Community Health. The Dutch authors base their findings on a total of 1,373 randomly selected men whose cardiovascular health and life expectancy at age 50 were repeatedly monitored between 1960 and 2000. The researchers looked into how much alcohol the men drank, what type it was, and over what period, in a bid to assess whether this had any impact on the risks of their dying from cardiovascular disease, cerebrovascular disease, and from all causes.
    They also tracked weight and diet, whether the men smoked, and for how long, and checked for the presence of serious illness.–During the 40 years of monitoring, 1,130 of the men died. Over half the deaths were caused by cardiovascular disease.–The proportion of men who drank alcohol almost doubled from 45% in 1960 to 86% in 2000, with the proportion of those drinking wine soaring from 2% to 44% during that period.–The researchers found that light long term alcohol consumption of all types—up to 20 g a day— extended life by around two extra years compared with no alcohol at all. Extended life expectancy was slightly less for those who drank more than 20 g.–And men who drank only wine, and less than half a glass of it a day, lived around 2.5 years longer than those who drank beer and spirits, and almost five years longer than those who drank no alcohol at all.–Drinking wine was strongly associated with a lower risk of dying from coronary heart disease, cerebrovascular disease, and death from all causes.–These results held true, irrespective of socioeconomic status, dietary and other lifestyle habits, factors long thought to influence the association between wine drinking and better health.
    Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by BMJ-British Medical Journal, via EurekAlert!, a service of AAAS.
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    [U1]You know why they were told to limit Supply to NZ??/ because a family there had the testicular fortitude to MAKE THE MEDICAL system there DO THERE JOB and actually HEAL someone —a Family MAN was Infected with H1N1 and was given Up for DEAD –and the medical People wanted to Pull the plug and the SONS and Mum SAID NOT TIL YOU TRY VITAMIN C so when they did at 50 grams the Father started to Come Around —what did the Senior Medical Doctor do ? He decided to Discontinue the treatments –NOW look at that eh –someone in the Medical field Playing god and deciding who lives or who Dies!!!!!!!!! as it turned out after a discussion with one of the Sons they had the Father move to another fascility and gave a lower dose of the Vitamin C and when he started to be stronger the Mum or wife gave him bigger doses and he made a full recovery—NOWWW they don’t want anyone else to get this due to the fact they wish to spread widespread infection and eliminate a CURE–this should have everybody Spitting FIRE!!!!!!!
    [U2]Sounds Like god playing to me eh!!! must be from the Vatican or the Middle East or even Israel or maybe india or china –this is what happens when you send a religious military wing to control the Access to health and life—-well it appears the FDA has controlled all the High priest ( doctors -since none of them have any testicles) and the heirarchy ( drug companies) are told to bow to the whim of the religious institution –The Father Of Death By Drugs or GMO—Now I wonder who Owns the GMO and Drug Companies?? All roads lead to Rome!! Discretionary??? we will Never See this again–once the EFSA ( FDA ) make article 13 the standard you will see anything of any value will be gone and never to be seen again—and this will be Humanities Lost—the bigger lost is allowing this BS institution have this much Power where even a Gov’t cannot stop them
    WE CAN!!!
    [U3]This is Ludicrious and since this drug company caved this puts out the illusion you can’t beat the FDA –this is a collusion and deception the FDA Violated DSHEA on this because under the DSHEA you can list research to a product no mattter what it’s intended use the only thing you cannot do is proclaim it can heal—this was allllll Bogus and BS!!!
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    Show of the Week February 18, 2011
     
    Confirmed— Mercury Cancels Out the Health Benefits of Fish Oil
     
    Processed Food Diet in Early Childhood May Lower Subsequent IQ
    Nitrates may boost performance by helping cellular efficiency- Study
    EU herbal directive will close health food stores, say owners
    Polyphenol-rich cherry juice may boost exercise recovery
    Recipe for the uses of Both the Beet and Cherry juice extracts
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    Confirmed— Mercury Cancels Out the Health Benefits of Fish Oil
    Submitted by Annie White on January 24, 2011
    http://healthfreedoms.org/2011/01/24/confirmed-mercury-cancels-out-the-health-benefits-of-fish-oil/
    Omega oils have seen a lot of attention for their benefits to brain function and fish oil is a major source of the nutrient. But there is a problem with eating fish as environmental pollutants have caused marine life to become laden with chemicals by the time they reach maturity for fishing.–Mercury is one of the biggest chemical threats to humans as it is incredibly harmful to the brain, causing degradation of the neurons thereby crippling the minds functional capacity.–So the fish have the omega oils that are great for the brain while at the same time containing mercury which will diminish brain function. Thankfully there are alternatives to its sources that don’t rely on fish- check the list at the bottom of the article and encourage environmental practices that reduce mercury pollution.
    ~Health Freedoms
    Lately, a shadow has perched itself over the fish oil industry. While no one disputes the benefits of a diet rich in omega fatty acids, Rachel wrote about a number of studies pointing to myths associated with fish oil. Most recently, a new study was released that confirms that any benefits that come from fish oil are cancelled out but excessive mercury in the system. According to Environmental Health News, methyl mercury is transferred to people who eat the fish, which is especially concerning for pregnant women whose children are exposed while developing in the womb.The study, published in Environmental Research, said this-“Of the five nutritional components evaluated, only the beneficial effects of DHA – as measured by the children’s test scores – were negatively impacted by increased mercury exposure. The nutritional benefits of DHA were significantly reduced and then disappeared altogether at the higher exposures.”–Bottom line: DHA, an omega fatty acid chain has many health benefits but when paired with mercury-laden fish oil, those benefits are cancelled out by the side effects of mercury poisoning.
    Why We Still Need Omega Fatty Acids–No one’s disputing the benefits of the omega fatty acids found in fish oil. These are truly important to the body. They regulate blood clotting, blood pressure, blood lipid levels, and the body’s response to injury and infection. Likely one of the most important benefits is that it greatly reduces inflammation associated with heart disease. But paired with the mercury found in a wide variety of favored fish species like tuna, grouper, or mackerel the mercury exposure makes it not worth the risk.[U1]
    Omega 3 Fatty Acids Minus the Mercury
    Omega 3 is made up of three acids EPA, DHA, and ALA. ALA can be found in hemp seeds, flax seeds, and chia seeds. DHA and EPA are found in algae, spirulina, blue-green algae, and chlorella.
    By Sara Novak
    http://planetgreen.discovery.com/food-health/confirmed-mercury-cancels-out-the-health-benefits-of-fish-oil.html
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    Processed Food Diet in Early Childhood May Lower Subsequent IQ
    ScienceDaily (Feb. 8, 2011) — A diet, high in fats, sugars, and processed foods in early childhood may lower IQ, while a diet packed full of vitamins and nutrients may do the opposite, suggests research published online in the Journal of Epidemiology and Community Health.–The authors base their findings on participants in the Avon Longitudinal Study of Parents and Children (ALSPAC), which is tracking the long term health and wellbeing of around 14,000 children born in 1991 and 1992.–Parents completed questionnaires, detailing the types and frequency of the food and drink their children consumed when they were 3, 4, 7 and 8.5 years old.–Three dietary patterns were identified: “processed” high in fats and sugar intake; “traditional” high in meat and vegetable intake; and “health conscious” high in salad, fruit and vegetables, rice and pasta. Scores were calculated for each pattern for each child.–IQ was measured using a validated test (the Wechsler Intelligence Scale for Children) when they were 8.5 years old. In all, complete data were available for just under 4,000 children.–The results showed that after taking account of potentially influential factors, a predominantly processed food diet at the age of 3 was associated with a lower IQ at the age of 8.5, irrespective of whether the diet improved after that age. Every 1 point increase in dietary pattern score was associated with a 1.67 fall in IQ. On the other hand, a healthy diet was associated with a higher IQ at the age of 8.5, with every 1 point increase in dietary pattern linked to a 1.2 increase in IQ. Dietary patterns between the ages of 4 and 7 had no impact on IQ.–The authors say that these findings, although modest, are in line with previous ALSPAC research showing an association between early childhood diet and later behaviour and school performance. “This suggests that any cognitive/behavioural effects relating to eating habits in early childhood may well persist into later childhood, despite any subsequent changes (including improvements) to dietary intake,” they say.–The brain grows at its fastest rate during the first three years of life, say the authors, by way of a possible explanation for the findings, adding that other research has indicated that head growth at this time is linked to intellectual ability.–“It is possible that good nutrition during this period may encourage optimal brain growth,” they suggest, advocating further research to determine the extent of the effect early diet has on intelligence.–Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by BMJ-British Medical Journal, via EurekAlert!, a service of AAAS.–Journal Reference-K. Northstone, C. Joinson, P. Emmett, A. Ness, T. Paus. Are dietary patterns in childhood associated with IQ at 8 years of age? A population-based cohort study. Journal of Epidemiology & Community Health, 2011; DOI: 10.1136/jech.2010.111955
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    Nitrates may boost performance by helping cellular efficiency- Study
    Dietary inorganic nitrates, as found in green leafy vegetables and beetroot juice, may enhance muscle endurance by boosting mitochondrial efficiency, according to new research.
    The study, published in Cell Metabolism, reports that short term increases in dietary nitrates can boost muscle efficiency during exercise, and finds that the that the improved performance is linked to increased efficiency of the mitochondria that power our cells. —Mitochondrial efficiency is usually measured as the amount of oxygen consumed per adenosine triphosphate (ATP) produced – a measure known as the P/O ratio. The new study shows that nitrate “has profound effects on basal human mitochondrial function as well as whole-body oxygen consumption during exercise.” -“In the current study we found a 19 percent increase in P/O ratio after nitrate supplementation. To our knowledge, there are no other dietary regimes described that have this effect,” said the researchers, led by Professor Eddie Weitzberg from the Karolinska Institutet, Sweden. –“Intriguingly, there was a strong correlation between the reduction in whole-body oxygen consumption during cycling and the increase in P/O ratio, which clearly suggests that a large part of the improved exercise efficiency is taking place at the mitochondrial level,” he added. —Dietary nitrates ——Until recently nitrate – which is abundant in green leafy vegetables – had been considered merely as an inert end product of nitric oxide (NO) metabolism or as a potentially toxic constituent in our diet. –However, the last decade has brought understanding of its importance in biological processes, including regulation of blood flow , blood pressure , cellular signaling, glucose homeostasis, and tissue responses to low oxygen levels (hypoxia). –Previous research has also linked nitrate rich foods and beverages, like beetroot juice , to improvements physiological benefits – including improved endurance . Such benefits have been shown to be, at least in part, due to a reduction in the ‘oxygen cost’ of exercise for healthy individuals after short-term dietary supplementation with nitrate. –Weitzberg and colleagues noted that the findings point toward the mitochondria (the ‘energy factories’ of cells) as a possible target for the effects of nitrate – suggesting that the production of ATP (an important transporter of chemical energy) may become more efficient. However, such mechanisms remain unclear and relatively unknown. –The new study, a double-blind crossover trial, studied the effects of a dietary intervention with inorganic nitrate on mitochondrial function and whole-body oxygen consumption in healthy volunteers. –Improved efficiency –Skeletal muscle biopsies were in conjunction with bicycle exercise tests were used to study the effects of dietary nitrate on mitochondrial efficiency and biochemical parameters, in addition to the expression of proteins involved in energy transfer. –The researchers reported that after taking a small dose of inorganic nitrate for three days volunteers consumed less oxygen while riding an exercise bike. –They said that skeletal muscle mitochondria after nitrate supplementation showed an improvement in efficiency (P/O ratio), which was strongly correlated to the reduction in oxygen cost during exercise. –These effects were found to accompany a reduction in ANT – an important protein involved in mitochondrial energy production. –“The fact that the relatively short term dietary regimen can influence expression of important mitochondrial proteins may have profound impact on exercise physiology,” said Weitzberg. –“Moreover, it may also have implications for metabolic and cardiovascular diseases in which dysfunctional mitochondria play a central role,” he added. –Weitzberg said that the “natural next step” is to repeat the experiment in people with conditions linked to mitochondrial dysfunction, including diabetes and cardiovascular disease, to see if they too enjoy such positive results. –He added that further studies to identify the molecular mechanisms and signaling pathways by which nitrate affects mitochondrial efficiency are clearly needed.
    ROS risk?
    In an accompanying editorial Dr Sreekumaran Nair and his colleagues from the Mayo Clinic, note that the study provides strong evidence that short-term dietary nitrate supplementation enhances mitochondrial efficiency and enhances exercise performance. However they added that future studies should also address the impact of nitrates on mitochondrial reactive oxygen species (ROS) production. –“It is difficult to ignore the possibility that improved coupling may lead to increased ROS production rates, which may have an adverse effect on proteins, DNA, and cellular functions,” they said.
    Research paper:Source: Cell Metabolism
    olume 13, Issue 2 , Pages 149-159, doi: 10.1016/j.cmet.2011.01.004
    “Dietary Inorganic Nitrate Improves Mitochondrial Efficiency in Humans”Authors: F.J. Larsen, T.A. Schiffer, S. Borniquel, K. Sahlin, B. Ekblom, J.O. Lundberg, E. Weitzberg Accompanying editorial:
    Source: Cell Metabolism Volume 13, Issue 2 , Pages 117-118, doi: 10.1016/j.cmet.2011.01.013 “Can Dietary Nitrates Enhance the Efficiency of Mitochondria?” Authors: K.S. Nair, B.A. Irving, I.R. Lanza
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    EU herbal directive will close health food stores, say owners
    Health food store owners are warning they will be forced to close down after April 30 this year, when the European Union Traditional Herbal Medicinal Products Directive (THMPD) kicks in.
    Companies have had a seven year-grace period since the THMPD entered EU law books in 2004, but EU-wide registrations under the regulation have been chronically low, meaning unless thousands of products register in the coming months, they will be stripped from health store and other retail shelves. —Selwyn Soe of the London-based The Herbal Factory told the BBC: “Unfortunately it looks as if we will have to close down because of this legislation.” –“The problem for us is that although we would have to pay many thousands of pounds for a licence to keep making each product, unlike a drug company we would not have a licence to make that product exclusively. It just will not be worth paying out the money.” –The regulation requires all herbal products making health and medicinal claims to be registered. As of December 31, 2010, there had been 187 registrations for individual products in the UK. Eighty four products had been approved and none rejected. The 100 per cent success rate has been attracting the interest of food supplement manufacturers and herbal ingredient suppliers who have struggled to have their science accepted under the 2006 nutrition and health claims regulation (NHCR). But herbal sector observers and associations have been surprised by the lack of THMPD registration applications – with the UK leading the way, followed by Germany, but many member states recording no applications at all. The relative high cost of registrations is deemed as partly to blame for the situation. –The fact that there is a clear discrepancy between the way science is treated under the THMPD and the NHCR has caused some confusion, and was partially responsible for the European Commission recently removing botanicals from the NHCR process to reconsider how science in the sector should be treated.That issue is unlikely to be revisited until the end of 2011.
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    Polyphenol-rich cherry juice may boost exercise recovery
    Juice made from Montmorency cherries may boost the recovery of muscle strength after intensive exercise, says a new study from London. –The benefits of the juice are reportedly related to its antioxidant activity, which may reverse the “oxidative damage induced by the damaging exercise”, report researchers from the Sports and Exercise Science Research Centre at London South Bank University and UK Anti-Doping. –Writing in Medicine & Science in Sports & Exercise, the London-based researchers report that seven days of consumption of the CherryActive-branded cherry juice prior to, and after intensive exercise produced a significant increase in recovery for knee extension maximum voluntary contractions, compared to an isoenergetic fruit concentrate control beverage. —“Montmorency cherry juice consumption improved the recovery of isometric muscle strength after intensive exercise perhaps due to attenuation of the oxidative damage induced by the damaging exercise,” wrote the authors, led by Dr Joanna Bowtell.
    Sports nutrition market –The study supports a number of other studies reporting the potential benefits of cherry, and tart cherries, in particular. Indeed, a previous study (Journal of Nutrition, 2006, Vol. 136, pp. 981-986) reported that daily consumption of 45 cherries could reduce circulating concentrations of inflammatory markers, with the researchers proposing that the flavonoids and anthocyanins in the cherries exert an anti-inflammatory effect and may lessen the damage response to exercise. –Study details –Dr Botwell and her co-workers recruited 10 well-trained men and assigned them to consume the cherry juice or the isoenergetic fruit concentrate beverage for one week before and for two days after a series of single leg knee extensions. –Results showed that the knee extension maximum voluntary contractions (MVC), a measure of muscle function, recovered significantly faster following consumption of the cherry juice, compared with the fruit control. Specifically, the MVC was 90.9 versus 84.9 24 hours after the exercise, and 92.9 versus 88.5 after 48 hours, respectively. No differences between the groups were observed for the activity of the enzyme creatine kinase, which is involved in the production of energy. –According to the researchers, the potential benefits may be linked to the antioxidant activity of the juice. During intense exercise the production levels of oxidative stress are reported to increase and this may damage muscles. However, this may be attenuated with consumption of the antioxidant-rich cherry juice.
    Source: Medicine & Science in Sports & Exercise
    Published online ahead of print, doi: 10.1249/MSS.0b013e31820e5adc
    “Montmorency Cherry Juice Reduces Muscle Damage Caused By Intensive Strength Exercise”–Authors: J.L. Bowtell, D.P. Sumners, A. Dyer, P. Fox, K.N. Mileva
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    Recipe for the uses of Both the Beet and Cherry juice extracts—Juice both the beet and cherry and combo them up or even use the one before a work out ( the beet juice ) and one after the work out the Cherry—explore horizons with this adding creatine powder to either drink before and after say 2.5 grams may make a difference as well—adding them to a yogurt or kefir mix for the protein and enzyme and probiotic effect to enhance restoring of strength and recovery
     
     
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    [U1]In truth these facts as well have been brought to question and have found to be opposite as to what is common knowledge -1) fish oils actually increase Heart failure if used for more then 4 years 2) they oxidize in the body excessively fast causing more damage then the benefit as a result you bring on problems you otherwise would not 3) the mercury has always been a condition that the food pyramid makers and health “scientist” continue to encourage saying eating the fish outweigh the causal problems–today the FAO has stated that the planet is eating more fish which equates to more illness as a result of all the metals and genetics people are consuming from fish farms–not to mention other sewage contaminants—so I would encourage to grow your own fish in a outdoor aquarium or indoor this way you know what your eating!!!!
     
     
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    Show of the Week February 21 2011
     
    Fenugreek have insulinotropic and antidiabetic properties
     
    Recipe for Respitory and Digestion with Fenugreek
     
    Protective effects of fermented ginseng
     
    Fermenting Ginseng
     
    About Organic Produce
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    Fenugreek have insulinotropic and antidiabetic properties
    effect of fenugreek 4-hydroxyisoleucine on liver function biomarkers and glucose in diabetic and fructose-fed rats.
    Phytother Res. 2009 Jan;23(1):61-4
    Authors: Haeri MR, Izaddoost M, Ardekani MR, Nobar MR, White KN
    Fenugreek (Trigonella foenum graecum L) is a plant traditionally used for the treatment of diabetes. It contains an unusual amino acid, 4-hydroxyisoleucine, demonstrated to have insulinotropic and antidiabetic properties in animal models. Here we examine the effect of 4-hydroxyisoleucine on liver function and blood glucose in two rat models of insulin resistance, fructose-fed rats and streptozotocin-induced diabetes type 2. In fructose-fed rats, levels of glucose and liver damage marker aspartate transaminase were markedly (84% and 93%, respectively) and significantly elevated compared with controls (p < 0.001 for both). Alanine transaminase was elevated slightly (18%), and all markers were restored to near control values after treatment with 4-hydroxyisoleucine at 50 mg/kg per day for 8 weeks, the effect being significant (p < 0.01) for all markers. This prolonged exposure to 4-hydroxyisoleucine was well tolerated in control animals and did not alter levels of glucose or liver damage markers significantly. In diabetic rats, treatment with 4-hydroxyisoleucine did not affect glucose or liver damage markers, but did improve HDL-cholesterol levels (31% increase, p < 0.05). These findings indicate 4-hydroxyisoleucine as a useful and well-tolerated treatment for insulin resistance, both directly as a hypoglycaemic and also as a protective agent for the liver.- PMID: 18680121 [PubMed – indexed for MEDLINE
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    Hypocholesterolaemic activity has been reported for fenugreek in rats(9, G41) and alloxan-diabetic dogs.(10) Activity has been attributed to the fibre and saponin fractions, and not to lipid or amino acid fractions.(9, 10) Studies have reported a reduction in cholesterol but not triglyceride concentrations,(9) or in both
    cholesterol and triglyceride concentrations, but without significant alterations in high-density lipoprotein (HDL) and lowdensity lipoprotein (LDL) concentrations.(10) Hypoglycaemic activity has been observed in rabbits, rats and dogs, and attributed to the defatted seed fraction (DSF),(8) trigonelline, nicotinic acid and coumarin.(7, 11) Oral administration of DSF reduced hyperglycaemia in four alloxan-diabetic dogs, and reduced the response to an oral glucose tolerance test in eight normal dogs, whereas the lipid fraction had no effect on serum glucose and insulin concentrations.(8) The high fibre content (50%) of DSF was thought to contribute to its antidiabetic effect although the initial rate of glucose absorption was not affected.(8) Nicotinic acid and coumarin were reported to be the major hypoglycaemic components of fenugreek seeds, following administration to normal and alloxan-diabetic rats.(7) The hypoglycaemic
    action exhibited by coumarin was still significant 24 hours post administration.(7) In addition, a slight antidiuretic action was noted for coumarin.(7) Trigonelline inhibited cortisone-induced hyperglycaemia in rabbits if administered (250 mg/kg) concomitantly or two hours before, but not two hours after, cortisone.(11) In addition, trigonelline exhibited significant hypoglycaemic activity in alloxan-diabetic rats (50 mg/kg), lasting 24 hours.(11) A stimulant action on the isolated uterus (guinea-pig),especially during late pregnancy, has been noted for both aqueous and alcoholic extracts.(G41) An aqueous extract is stated to increase the number of heart beats in the isolated mammalian heart.(G41) In vitro antiviral activity against vaccinia virus has been reported for fenugreekine, which also possesses cardiotonic, hypoglycaemic, diuretic, antiphlogistic and antihypertensive properties.(2)
     
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    The best-documented medical use of fenugreek is to control blood sugar in both insulin-dependent (type 1) and noninsulin-dependent (type 2) diabetics. Some studies also show that serum cholesterol levels in diabetics, and perhaps in others, are reduced by fenugreek. Doses as low as 15 mg per day may produce beneficial effects on fasting blood sugar, elevation of blood sugar after a meal, and overall glycemic control. The use of fenugreek is likely to alter the diabetic patient’s need for insulin or other medications used to control blood sugar. This treatment should be supervised by a health care provider familiar with the use of herbal therapies for diabetes. The recommended
    doses of fenugreek can vary rather widely.- Historically, fenugreek has been used as a topical treatment for abscesses, boils, burns, eczema, gout, and ulceration of the skin as it has an anti-inflammatory effect. It is also reputedly useful for a number of digestive complaints, including gastritis and gastric ulcers. A study published in 2002 found that both an aqueous solution and a gel fraction derived from fenugreek have anti-ulcer effects comparable to those of omeprazole, a standard medication given to reduce gastric secretions. The researchers found that the fenugreek solution protected the gastric mucosa from injury as well as reducing the secretion of gastric acid. Fenugreek reportedly can be helpful in the induction of childbirth, as it is known to stimulate uterine contractions. For this reason it should not be taken during pregnancy. As a gargle, fenugreek may relieve sore throats and coughing. Arthritis, bronchitis, fevers, and male reproductive conditions are other traditional but unsubstantiated indications for this herb.
     
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    Chinese Medicine: The drug is used to treat cold pain in the-lower abdomen, impotence, and hernia (said to be due to cold ‘chi’).
     
    Indian Medicine: The drug is used for fever, vomiting, anorexia, coughs, bronchitis, and colitis.
     
    Recipe for Respitory and Digestion with Fenugreek Complaints take ½ tsp of fenugreek and ½ teaspoon of thyme in a 2 pint pot of boiling water –allow to boil 5 minutes then pour in cup—this will regulate Insulin—assist in the removing of congestion or clogged lungs—absorb and draw out mucous in the lower GI and will remove fungi and bacteria in the system and restore the colon
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    Other tidbits The specific indication for fenugreek is cold damp swelling and stagnation in the legs. It also disperses cold and alleviates pain.
    Use: (a) Demulcent, (b) Anti-inflammatory, (c) Hypolipidemic, (d) Hypoglycemic, (e) Diuretic, (f) Galactagogue, (g) Antiatherosclerotic.
    It is used in gastrointestinal inflammation, coughs, colic, diarrhea in infants, cholesterol level reduction, diabetes and as an eye wash for inflamed eyes. Externally, it can be used as a poultice for inflamed, irritated skin conditions. Research has shown antifertility and antiandrogenic effects on male rats who were fed a fenugreek extract.
    Fenugreek contains alkaloids 0.2-0.36% trigonelline, gentianine, carpaine, 45-60% carbohydrate which is chiefly the mucilage galactomannans, 20-30% protein and 6-10% fixed oil. It is rich in tryptophan There are also several steroidal saponins, flavonoids and 0.015% essential oil.
    Contraindications: Oral drugs or herbs taken at the same time as fenugreek may have delayed absorption due to mucilage content. Since experimental research has shown fenugreek to stimulate animal uteri and due to its emmenagogue and abortifacient effects, it is contraindicated in pre
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    Protective effects of fermented ginseng on streptozotocin-induced pancreatic beta-cell damage through inhibition of NF-kappaB.
    Yuan HD, Chung SH.
    Department of Life and Nanopharmaceutical Science, College of Pharmacy, Kyung Hee University, Seoul 130-701, Korea.
    Abstract
    Ginseng (Panax ginseng C.A. Meyer) is widely used in Asian countries as a traditional medicine for the treatment of various diseases. It is known to have anti-inflammatory effects, although the mechanism is not clear. In this study, preventive effects of fermented ginseng (FG) against streptozotocin (STZ)-induced pancreatic beta-cell death was assessed in RINm5F insulinoma cells. FG markedly inhibited the production of nitrite in a dose-dependent manner. The decrease in nitrite production was found to correlate with reduced inducible nitric oxide (iNOS) protein and mRNA levels. To characterize the anti-inflammatory mechanism of FG at the transcriptional level, we examined effects of FG on the activity of nuclear factor-kappaB (NF-kappaB). FG reduced a translocation of the NF-kappaB subunit and NF-kappaB-dependent transcriptional activity. FG blocked signaling upstream of NF-kappaB activation, such as degradation of inhibitor factor-kappaBalpha (IkappaBalpha ) and phosphorylations of extracellular signal-regulated kinase (ERK) and c-Jun NH2-terminal kinase (JNK). These results suggest that FG protects against STZ-induced pancreatic beta-cell damage by downregulation of iNOS, cyclooxygenase-2 (COX-2), and tumor necrosis factor-alpha (TNF-alpha ) gene expressions by blocking NF-kappaB and mitogen-activated protein kinase activities.-=-PMID: 19956901 [PubMed – indexed for MEDLINE
     
    Fermenting Ginseng—take any type of ginseng and allow to sit in vinegar for at least 6 weeks or longer —when done blend all the components and then strain through filtering materials and use 1 small tsp at a time
     
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    About Organic Produce
    WHAT MAKES PRODUCE “ORGANIC”?
    Contrary to what most people believe, “organic” does not automatically mean “pesticide-free” or “chemical-free”. In fact, under the laws of most states, organic farmers are allowed to use a wide variety of chemical sprays and powders on their crops. So what does organic mean? It means that these pesticides, if used, must be derived from natural sources, not synthetically manufactured. Also, these pesticides must be applied using equipment that has not been used to apply any synthetic materials for the past three years, and the land being planted cannot have been treated with synthetic materials for that period either.
    Most organic farmers (and even some conventional farmers, too) employ mechanical and cultural tools to help control pests. These include insect traps, careful crop selection (there are a growing number of disease-resistant varieties), and biological controls (such as predator insects and beneficial microorganisms).
    ORGANIC PRODUCE AND PERSONAL HEALTH
    When you test synthetic chemicals for their ability to cause cancer, you find that about half of them are carcinogenic.
    Until recently, nobody bothered to look at natural chemicals (such as organic pesticides), because it was assumed that they posed little risk. But when the studies were done, the results were somewhat shocking: you find that about half of the natural chemicals studied are carcinogenic as well. -This is a case where everyone (consumers, farmers, researchers) made the same, dangerous mistake. We assumed that “natural” chemicals were automatically better and safer than synthetic materials, and we were wrong. It’s important that we be more prudent in our acceptance of “natural” as being innocuous and harmless.
    ORGANIC PESTICIDES VERSUS SYNTHETIC PESTICIDES
    3Clearly, the less we impact our environment, the better off we all are. Organic farming practices have greatly advanced the use of non-chemical means to control pests, as mentioned earlier. Unfortunately, these non-chemical methods do not always provide enough protection, and it’s necessary to use chemical pesticides. How do organic pesticides compare with conventional pesticides? A recent study compared the effectiveness of a rotenone-pyrethrin mixture versus a synthetic pesticide, imidan. Rotenone and pyrethrin are two common organic pesticides; imidan is considered a “soft” synthetic pesticide (i.e., designed to have a brief lifetime after application, and other traits that minimize unwanted effects). It was found that up to 7 applications of the rotenone- pyrethrin mixture were required to obtain the level of protection provided by 2 applications of imidan. It seems unlikely that 7 applications of rotenone and pyrethrin are really better for the environment than 2 applications of imidan, especially when rotenone is extremely toxic to fish and other aquatic life. It should be noted, however, that we don’t know for certain which system is more harmful. This is because we do not look at organic pesticides the same way that we look at conventional pesticides. We don’t know how long these organic pesticides persist in the environment, or the full extent of their effects. When you look at lists of pesticides allowed in organic agriculture, you find warnings such as, “Use with caution. The toxicological effects of [organic pesticide X] are largely unknown,” or “Its persistence in the soil is unknown.” Again, researchers haven’t bothered to study the effects of organic pesticides because it is assumed that “natural” chemicals are automatically safe.
    WHY HAVEN’T WE HEARD THIS BEFORE?
    For obvious reasons, organic farmers have done little, if anything, to dispel the myth that “organic = chemical/pesticide-free”. They would only stand to lose business by making such a disclosure.
    Pesticide manufacturers have little concern in the matter. To them, “synthetic pesticides sold” and “organic pesticides sold” are both “pesticides sold”.
    As for conventional farmers, they are not really in a position to be critical. It would not be in their interest to draw attention to chemical and pesticide use.
    WHAT DOES ALL OF THIS MEAN?
    Unless you know your grower personally, there is no guarantee that your produce has been grown without pesticides or other chemicals. It’s a point to consider, given the substantially higher cost of organic foods.
    There are many choices and decisions that we, as consumers, are asked to make. Hopefully, this has provided some new information that you will find helpful.
    * * * * * * *
    A formatted MS Word version of this document may be downloaded at:
    http://www.ocf.berkeley.edu/~lhom/organics.doc
    The data describing the carcinogenicity of natural and synthetic compounds are referenced in Gold, L.S., et al. (1992) _Science_ Vol. 258, pp. 261-265.
    Many thanks go to the Organic Crop Improvement Association for their cooperation in this study. The OCIA has chapters in AZ, AR, CA, CO, FL, IL, IN, IA, KS, MD, MI, MN, MO, MT, NE, NM, NC, ND, OH, PA, SD, UT, and WI. Thanks are also extended to the California Certified Organic Farmers, the Ohio Ecological Food and Farm Association, and Oregon Tilth Certified Organic. (The appropriate information has not yet been obtained from the Natural Organic Farmers Association (NOFA), but it is almost certain that all facts stated here apply to their products as well.) The following state Departments of Agriculture have also been very helpful: AL, AK, AZ, CA, CO, DE, FL, HI, IA, LA, MD, MI, MS, MO, ND, OK, SC, TN, VA, and WA. States with no laws governing organic products include Alabama, Delaware, Hawaii, Mississippi, and Tennessee.
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    Show of the Week February 25 1011
    Online supplements retailer heeds FDA warning letter advice, but fears business impact
    WARNING LETTER
    Monsanto and FDA move toward healthier GM soybean oil
    Restricted diet’ linked to reduction in ADHD
     
    Alternative Energy Ideas
     
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    Online supplements retailer heeds FDA warning letter advice, but fears business impact
    “You gotta do what you gotta do,” an exhausted Cathy Keys told NutraIngredients-USA today after spending the best part of two months completely renovating her family-run online supplements business, NutriHerb.net, after receiving an FDA warning letter in December last year.
    Keys, the firm’s owner and managing director, said all therapeutic claims that the company had happily been making since building the website in 2001 – and for seven years before that via labelling and print – had been removed, an action she feared could put a severe dent in its profitability. —
    “Not being able to make claims could well run us out of business,” she said. “We were surprised to receive this letter as we did not know the claims were illegal but took steps immediately to comply.”
    The FDA warning letter, sent December 2 and which can be found Below , highlighted unauthorized drug claims for three products – Custom Advantage Chamomile; Custom Advantage CoQ10 and Custom Advantage Omega-3 Fatty Acids – and gave NutriHerb 15 days to take corrective action.
    “We rang them and after discussing it decided to remove all therapeutic claims from our site,” Keys said. “Because there was so much to do they gave us a 60-day extension and we have just written to them to notify them that the work has been completed.” –Keys said she had scanned the FDA-approved list of qualified and unqualified health claims, but said the process was so elongated and expensive that NutriHerb would not be pursuing it. –The firm does also have the option to make structure/function claims provided they can substantiate them. —“Being a small family business, we have a small marketing budget, so I have no idea how we are going to market our products now,” she said. —Another warning letter dated December 21 sent to another Florida-based company regarding unauthorized drug claims for a range of sea algae derived calcium-osteoarthritis supplement products had not as yet been heeded. –Natural Option USA Corporation continues to market OsteOrganiCAL with testimonials and references to studies the FDA has deemed amount to drug claims on its website. –However the company said it was, “in the process of removing the claims”. –That letter can be found here.
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    WARNING LETTER–FLA-11-11–December 21, 2010
    Claudia Muharram, President/Director
    Natural Option USA Corporation
    4957 SW 74th Court
    Miami, FL 33155
    Dear Ms. Muharram:
    This is to advise you that the Food and Drug Administration (FDA) reviewed your website at the Internet address http://www.natural-option.com/ in December 2010 and has determined that the product OsteOrganicCAL (a sea algae calcium compound packaged with Vitamin D3) is promoted for conditions that cause the product to be a drug under section 201(g)(1) of the Federal Food, Drug, and Cosmetic Act (the Act) [21 U.S.C. § 321(g)(1)]. The therapeutic claims on your website establish that the product is a drug because it is intended for use in the cure, mitigation, treatment, or prevention of disease. The marketing of this product with these claims violates the Act.
    Examples of some of the claims observed on your web site include:
    ðClaims in the form of personal testimonials on the OsteOrganiCAL webpage:
    • “My clinical results in reversing osteopenia and osteoporosis have been better than those obtained using Fosamax.”
    • “[My bone density test showed considerable improvement. My first bone density test indicated I was at risk for traumatic fracture. After I began the OsteOrganiCAL program, my follow-up test showed considerable improvement.”
    • “April of 1995, I had a bone scan. The result: I was diagnosed with severe
    osteoporosis. In May I found out about a product called OsteOrganiCAL, I took OsteOrganiCAL for three months, went and had another bone scan. My overall bone density was increased 3.9%. I have never felt better.”
    • ” had a hard fall, my hip was black and purple. My chiropractor said she had never seen any bruise that bad, without a broken hip. I am sure it was the Natural Option product [OsteOrganiCAL] that saved me from a break. Thank you so much for stronger bones.”
    • “[Seven months my wife has been on OsteOrganiCAL. During this time she has not fractured any bones.”
    Claims made under the heading “OsteOrganiCAL® CASE STUDY ANALYSIS”:
    • “The fall of 2003 the Osteoporosis Education Project completed a year long pilot study with the sea algae calcium and vitamin D product known as “OsteOrganiCAL®”. The study included eleven postmenopausal women, six of whom experienced impressive gains in bone mineral density from use of the product.”
    • “[A]nalysis of these cases documents the potential of this novel Calcium and Vitamin D product [OsteOrganiCAL] to halt and reverse the osteoporosis process in postmenopausal women.”
    • “Using OsteOrganiCAL however, the few women in the sample with only moderate bone loss (osteopenia) also benefited significantly from this therapy.”
    Your product is not generally recognized as safe and effective for the above referenced uses and, therefore, the product is a “new drug” under section 201[U1](p) of the Act [21 U.S.C. § 321(p)]. New drugs may not be legally marketed in the U.S. without prior approval from FDA as described in section[U2] 505(a) of the Act [21 U.S.C. § 355(a)]. FDA approves a new drug on the basis of scientific data submitted by a drug sponsor to demonstrate that the drug is safe and effective. Furthermore, your product OsteOrganiCAL is misbranded within the meaning of section 502(f)(1) of the Act [21 U.S.C. § 352(f)(1)] because the labeling for this drug fails to bear adequate directions for use.
    This letter is not intended to be an all-inclusive review of your website and the products your firm markets. It is your responsibility to ensure that all products marketed by your firm comply with the Act and its implementing regulations. We advise you to review your website, product labels, and other labeling and promotional materials for your products to ensure that the claims you make for your products do not cause them to violate the Act. You should take prompt action to correct the violations described above and prevent their future recurrence. Failure to do so may result in enforcement action without further notice. The Act authorizes the seizure of illegal products and injunctions against manufacturers and distributors of those products [21 U.S.C. §§ 332 and 334].
    Please notify this office, in writing, within fifteen (15) working days from your receipt of this letter as to the specific steps you have taken to correct the violations noted above and to ensure that similar violations do not recur. Your response should include any documentation necessary to show that correction has been achieved. If you cannot complete all corrections before you respond, please explain the reason for the delay and the date by which the corrections will be completed.
    Your response should be directed to Winston Alejo, U.S. Food and Drug Administration, Florida District Office, 555 Winderley Place, Suite 200, Maitland, Florida 32751. If you have any questions about this letter, please contact Mr. Alejo at (407) 475-4731.
    Sincerely, /S/
    Emma R. Singleton
    Director, Florida District
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    Monsanto and FDA move toward healthier GM soybean oil
    Monsanto and the Food and Drug Administration (FDA) have completed a biotechnology consultation process for a key component of the company’s high-oleic soybeans, the biotech giant has said.
    The company said completion of the consultation process is a significant first step toward commercialization of Monsanto’s Vistive Gold soybeans[U3], which are low in trans and saturated fats, enabling food manufacturers to improve the health profile of foods made with soybean oil.[U4] The crop represents one of the first to be developed through a combination of biotechnology and traditional breeding methods to provide health benefits for consumers. –Monsanto’s global technology lead for food quality traits Joe Cornelius said that the product has been developed with input from the food and nutrition industry. The soybeans have 60 percent less saturated fat than conventional soybean oil, and 70 percent less saturated fat than fry shortening, the company said. –“Vistive Gold soybeans will give farmers an opportunity to produce nutritionally improved soybean oil for food companies and consumers,” Cornelius said. “Monsanto is continuing to provide innovations to the soybean oil market and help growers create additional value within the food supply chain.” –The Vistive Gold soybeans will be combined with the company’s Genuity Roundup Ready 2 Yield soybeans, to increase yield for farmers, as well as providing nutritionally improved oil.
    Trans fat substitution
    There is a large body of evidence suggesting that trans fatty acids raise levels of LDL-cholesterol (so-called ‘bad’ cholesterol) and reduce levels of HDL-cholesterol (or ‘good’ cholesterol, and can influence other risk factors for cardiovascular disease. This has led to trans fat bans in many parts of the United States.
    In the food industry this has been mirrored by an increase the in pressure on manufacturers to eliminate trans fat from their products. But food manufacturers have faced a number of technical difficulties in removing trans fat from their products, and some have expressed concern that trans fats could end up being replaced by saturated fats, which are nearly as unhealthy as the fats they replace, prompting companies to look for healthier replacements. [U5]
    Trans fats formed by partial hydrogenation of vegetable oil are attractive to the food industry due to their extended shelf life, flavor and heat stability, and have displaced natural solid fats and liquid oils in many areas of food processing.
    “Food companies [U6]continue to look for ways to eliminate trans fats and reduce saturated fat, and Vistive Gold soybean oil is an excellent option that is more economical, sustainable and nutritionally desirable than current solutions,” Cornelius said.
    Monsanto’s Vistive Gold soybeans are subject to a number of further regulatory hurdles with both the FDA and the US Department of Agriculture before they can be fully commercialized.
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    Restricted diet’ linked to reduction in ADHD–
    A restrictive diet, which bans processed foods, may reduce the symptoms of attention-deficit hyperactivity disorder (ADHD) in young children, according to a new study.
    New research published in The Lancet, suggests that a diet restricted to just a few basic ingredients could be “a valuable instrument to assess whether ADHD is induced by food.” In the new study, children with ADHD were put on a ‘restricted elimination diet’ – containing only rice, meat, vegetables, pears and water – for five weeks. The authors found that ADHD symptoms were reduced in 78 per cent of children placed on the diet. —“A strictly supervised restricted elimination diet is a valuable instrument to assess whether ADHD is induced by food,” wrote the researchers, led by senior author Professor Jan Buitelaar from Radboud University, The Netherlands. –“We think that dietary intervention should be considered in all children with ADHD, provided parents are willing to follow a diagnostic restricted elimination diet for a five-week period, and provided expert supervision is available,” they said. -However, writing in an accompanying comment piece for The Lancet, Dr Jaswinder Ghuman from the University of Arizona said that in her opinion[U7], the diet should not be continued for more than five weeks, “because long-term effects of dietary elimination on the child’s nutritional status are not known.”
    ADHD development

    #2084
    AvatarEK
    Keymaster

    The authors said that several genetic and environmental factors are involved in the development of ADHD[U8]. One of the many environmental factors proposed to have an effect on ADHD development is diet. –According to the National Institute of Mental Health (NIMH), parents have long suspected that sugary foods, additives, and colours, may play a role in inducing symptoms, however there is very little evidence to support such theories. –Prof. Buitelaar and colleagues noted that some children have negative physical reactions to certain foods, and suggested that foods may also affect the brain in ways that result in changes in behaviour. –They explained that ADHD may be triggered by a hypersensitivity reaction to certain foods, adding that some complementary medicine practices test for the antibody IgG (immunoglobulin G) and recommend eliminating foods high in IgG for ADHD. However, they noted that IgG testing is unproven to work, and remains controversial among mainstream physicians.
    Study details
    The new research investigated the link between ADHD and diet by recruiting 100 children diagnosed with ADHD (between the ages of 4 and 8), and putting them on a restricted food (hypoallergenic) diet. After this a ‘food challenge’ was given to the children to test whether foods high in IgG re-triggered ADHD symptoms. Of the children to complete the restrictive diet phase of the trial, 78 percent had reduced ADHD symptoms, compared with no improvement in the controls.
    After five weeks, children on the restricted diet received food challenge diets, in which certain foods (either high or log in IgG) were re-introduced into the diet. Buitelaar and co workers reported “a substantial relapse in behaviour in 63 per cent of children” when previously restricted processed foods were put back in the diet. However they said that no differences in relapse or behavioural effects were seen between high- and low- IgG foods. –Commenting on the study Dr Ghuman said: “IgG levels against foods did not predict which foods might lead to a negative effect on behaviour because an equal number of low and high IgG food challenges resulted in relapse of ADHD symptoms.”
    Unanswered questions
    Ghuman warned that the new study does not answer a number of questions, such as whether or not the elimination diet reduces symptoms long-term, and said that clinical practice should not be changed based on the results of one study. ( C-Y-A )~~~~ Means Cover your @$$~~~The authors also noted that the study had potential limitations, due to the fact that it was open-label.
    “Expectations of the parents cannot be fully ruled out as a possible cause of the behavioural improvements … We [also] cannot rule out that the behavioural improvements during the first phase might have been caused by increased attention for the child in the diet group,” they said.
    Source: The Lancet -Volume 377, Issue 9764 , Pages 494-503, doi: 10.1016/S0140-6736(10)62227-1 “Effects of a restricted elimination diet on the behaviour of children with attention-deficit hyperactivity disorder (INCA study): a randomised controlled trial”Authors: L. Pelsser, K. Frankena, J. Toorman, H.F. Savelkoul, A.E. Dubois, R.R. Pereira, et al
    Source: The LancetVolume 377, Issue 9764 , Pages 446-448, doi: 10.1016/S0140-6736(11)60133-5 “Restricted elimination diet for ADHD: the INCA study”
    Author: J.K. Ghuman
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    Alternative Energy Ideas
    Permanent Magnetic Generators:
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    Permanent Magnetic Motor:
    http://www.rexresearch.com/johnson/1johnson.htm#scimech
    Micro-Hydro Water Turbine Generators:
     
    Here is a one stop web site for all the latest innovations out there in free and subsidized energy sources: http://peswiki.com/index.php/Main_Page
    Brown’s gas or HHO:
    http://peswiki.com/index.php/Directory:Brown’s_Gas
    http://www.cool-flame.com/index.htm
    Hydrogen: http://peswiki.com/index.php/Directory:Hydrogen
     
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    TOP E
    [U1]This can be challenged the FDA is off way off here but this person here does not know her rights under the DSHEA act which is still applicable under grandfather clauses–they cannot legally make the statements they have made and what they have said is ILLEGAL–BUTTTT because she is an independent and has no oney to back her–she has to cave in—and this is why a network is so important—the legalities or rather I should say the ILLEGALITIES of this is going to be more and more of a target to those who cannot afford to take legal action–this women will be done –an another access will be removed for peoples health –they are goingto go after the independents so then you will be directed to a drug store or drug company for your supplements which will be filled with unwanted contaminats and mimimal potency if any!!
    [U2]WHO says this is a Drug—SO now are we going to remove oranges and Cherries from the diet and apples and limes and papaya and pineapple because they can do what drugs do not—see the Difference between a Vitamin and a food as Opposed to drugs Drugs Do not heal and No Drug can make this claim butttt Foods do and the claim that can be made is that with right nutrition you have health so there is no comparison with this supplement and the drug definition!!
    [U3]WOW I can’t wait New Poison YUM YUM–Bad enough soy causes cancer —now we are taking the cholesterol out of it—the very thing that protects us against cancer—with out the cholesterol this fat oxidizes at a super fast rate causing cellular decay and carcinogen breeding ground—Good Bye Long Life in the Future—it is going to be more then anything sustained life and then termination when they have gotten or have profited all that they can
    [U4]Increase the efficacy in breaking down a human being this is industrial oil again should not be consumed
    [U5]WHAT A CROCK OF %$#@—the food industry was manuevored into this position giving a reason to sell theis GMO product–saturated fats are the safest fats in the planet—GLOBALLY it is the fat of choice—Only in areas where Monsanto has a stronghold is this not the case—Total deception and Marketing
    [U6]MORE LIKE GE or GMO producers of toxic foods–it would appear that this is a battle that will never cease–the earth’s existence as well as ours will depend on this–
    [U7]The article is good up to here—“in her opinion”–opinions are like backsides–everyone has one–what is so incredible here is that you see a reduction up to 78%—“78%” that is huge and if then there was a observation diet ( lets give everything a title shall wee–then we can sound medically stupid as well ) then by observing the intake we can best determine the foods that are causing this issue and not get a kid on drugs because it is convienent—then the food can be traced right to where it is produced and then can be examined to see what is being done to the food–Genteically—Chemically–or even- Processed
    [U8]NOWWWWW PAY ATTENTION HERE!!!!!!!””””Environmental and Genetic Factors”””””what environment and what genetics—see the obvious is presented foreign or damaged Genetics and environments—toxic chemicals from the production of foods from seed to harvest–we would all think in terms our living environments–factories–exhaust from cars–toxic waste in the rivers–prescription drug and plastics exposure -and this will have a role—but the real factor that is obviously overlooked is the genetics that is being introduced to the foods andd the genetic impact that it is having on the human genome–when the genetics are being introduced this will target everyone differnt due to the genetic matrix of each individual ” Perfect Race” would appear to be on this agenda–if you cannot utilize this waste then you are not the human genome they want to be alive–Something to consider
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    Show of the Week February 28-2011
    Round UP GENETIC DAMAGE
     
    Asthma Tied to Bacterial Communities in the Airway
     
    Bacteria to Blame in Asthma Attacks in Children, Research Suggests
     
    Blocking Effects Of Viral Infections May Prevent Asthma In Young Children
     
    Monsanto’s Technology Stewardship Agreement shifts responsibility to growers
     
    Recipe For Anti Bacterial Resistance
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    Round UP GENETIC DAMAGE
    Dear Secretary Vilsack:
    A team of senior plant and animal scientists have recently brought to my attention the discovery of an electron microscopic pathogen that appears to significantly impact the health of plants, animals, and probably human beings. Based on a review of the data, it is widespread, very serious, and is in much higher concentrations in Roundup Ready (RR) soybeans and corn – suggesting a link with the RR gene or more likely the presence of Roundup. This organism appears NEW to science!- This is highly sensitive information that could result in a collapse of US soy and corn export markets and significant disruption of domestic food and feed supplies. On the other hand, this new organism may already be responsible for significant harm (see below). My colleagues and I are therefore moving our investigation forward with speed and discretion, and seek assistance from the USDA and other entities to identify the pathogen’s source, prevalence, implications, and remedies.
    We are informing the USDA of our findings at this early stage, specifically due to your pending decision regarding approval of RR alfalfa. Naturally, if either the RR gene or Roundup itself is a promoter or co-factor of this pathogen, then such approval could be a calamity. Based on the current evidence, the only reasonable action at this time would be to delay deregulation at least until sufficient data has exonerated the RR system, if it does.
    For the past 40 years, I have been a scientist in the professional and military agencies that evaluate and prepare for natural and manmade biological threats, including germ warfare and disease outbreaks. Based on this experience, I believe the threat we are facing from this pathogen is unique and of a high risk status. In layman’s terms, it should be treated as an emergency.
    A diverse set of researchers working on this problem have contributed various pieces of the puzzle, which together presents the following disturbing scenario:
    Unique Physical Properties
    This previously unknown organism is only visible under an electron microscope (36,000X), with an approximate size range equal to a medium size virus. It is able to reproduce and appears to be a micro-fungal-like organism. If so, it would be the first such micro-fungus ever identified. There is strong evidence that this infectious agent promotes diseases of both plants and mammals, which is very rare.
    Pathogen Location and Concentration
    It is found in high concentrations in Roundup Ready soybean meal and corn, distillers meal, fermentation feed products, pig stomach contents, and pig and cattle placentas.
    Linked with Outbreaks of Plant Disease
    The organism is prolific in plants infected with two pervasive diseases that are driving down yields and farmer income – sudden death syndrome (SDS) in soy, and Goss’ wilt in corn. The pathogen is also found in the fungal causative agent of SDS (Fusarium solani fsp glycines).
    Implicated in Animal Reproductive Failure
    Laboratory tests have confirmed the presence of this organism in a wide variety of livestock that have experienced spontaneous abortions and infertility. Preliminary results from ongoing research have also been able to reproduce abortions in a clinical setting.–The pathogen may explain the escalating frequency of infertility and spontaneous abortions over the past few years in US cattle, dairy, swine, and horse operations. These include recent reports of infertility rates in dairy heifers of over 20%, and spontaneous abortions in cattle as high as 45%- For example, 450 of 1,000 pregnant heifers fed wheatlege experienced spontaneous abortions. Over the same period, another 1,000 heifers from the same herd that were raised on hay had no abortions. High concentrations of the pathogen were confirmed on the wheatlege, which likely had been under weed management using glyphosate.
    Recommendations
    In summary, because of the high titer of this new animal pathogen in Roundup Ready crops, and its association with plant and animal diseases that are reaching epidemic proportions, we request USDA’s participation in a multi-agency investigation, and an immediate moratorium on the deregulation of RR crops until the causal/predisposing relationship with glyphosate and/or RR plants can be ruled out as a threat to crop and animal production and human health.
    It is urgent to examine whether the side-effects of glyphosate use may have facilitated the growth of this pathogen, or allowed it to cause greater harm to weakened plant and animal hosts. It is well-documented that glyphosate promotes soil pathogens and is already implicated with the increase of more than 40 plant diseases; it dismantles plant defenses by chelating vital nutrients; and it reduces the bioavailability of nutrients in feed, which in turn can cause animal disorders. To properly evaluate these factors, we request access to the relevant USDA data.
    I have studied plant pathogens for more than 50 years. We are now seeing an unprecedented trend of increasing plant and animal diseases and disorders. This pathogen may be instrumental to understanding and solving this problem. It deserves immediate attention with significant resources to avoid a general collapse of our critical agricultural infrastructure.
    Sincerely,
    COL (Ret.) Don M. Huber
    Emeritus Professor, Purdue University
    APS Coordinator, USDA National Plant Disease Recovery System (NPDRS)
     
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    Asthma Tied to Bacterial Communities in the Airway
    ScienceDaily (Feb. 19, 2011) — Asthma may have a surprising relationship with the composition of the species of bacteria that inhabit bronchial airways, a finding that could suggest new treatment or even potential cures for the common inflammatory disease, according to a new UCSF-led study.
    Using new detection methods, researchers learned that the diversity of microbes inside the respiratory tract is far vaster than previously suspected — creating a complex and inter-connected microbial neighborhood that appears to be associated with asthma, and akin to what has also been found in inflammatory bowel disease, vaginitis, periodontitis, and possibly even obesity.[U1]—Contrary to popular belief, the scientists also learned that the airways are not necessarily entirely sterile environments, even in healthy people, while the airways of asthmatics are infected by a richer, more complex collection of bacteria. These findings could improve understanding of the biology of asthma, and potentially lead to new and much-needed therapies.-“People thought that asthma was caused by inhalation of allergens but this study shows that it may be more complicated than that — asthma may involve colonization of the airways by multiple bacteria,” said study co-author Homer Boushey, MD, a UCSF professor of medicine in the division of Pulmonary and Critical Care Medicine.–The study is published online in the Journal of Allergy and Clinical Immunology.–Asthma is one of the most common diseases in the world, with approximately 300 million asthmatics globally, including 24 million in the United States, according to the Centers for Disease Control. The disease has been on the rise for the last 60 years.–“It has gone from 3 percent of the population to slightly more than 8 percent of the population in the U.S.,” said Boushey. “It is most prevalent in western, developed nations — and we don’t know why.”[U2]—In recent years, scientists began studying communities of mixed-species microorganisms (microbiome) found in both diseased and healthy people to better understand their role in a variety of diseases. But research on the microbiome in respiratory disease is relatively uncharted terrain.–“We know fairly little about the diversity, complexity and collective function of bacteria living in the respiratory tract, and how they might contribute to diseases like asthma,” said Yvonne J. Huang, MD, the paper’s first author. She is a research fellow and clinical instructor in the UCSF Pulmonary Division.–“Traditionally, the airways have been thought to be sterile. However, this study suggests this is not the case. Certain asthma patients who require inhaled corticosteroid therapy possess a great abundance of bacteria compared to healthy individuals, and have an increased relative abundance of specific organisms that is correlated with greater sensitivity of their airways.”In their three-year pilot project, the scientists collected samples from the airway linings of 65 adults with mild to moderate asthma and 10 healthy subjects. Then, using a tool that can identify approximately 8,500 distinct groups of bacteria in a single assay, the scientists profiled the organisms present in each sample to look for relationships between bacterial community composition and clinical characteristics of the patients’ asthma.–The researchers found that bronchial airway samples from asthmatic patients contained far more bacteria than samples from healthy patients. The scientists also found greater bacterial diversity in the asthmatic patients who had the most hyper-responsive or sensitive airways (a feature of asthma).–“People have viewed asthma as a misdirected immune reaction to environmental exposures, but few have thought of it in the context of airway microbiota composition,” said senior author Susan Lynch, PhD, an assistant professor of medicine and director of the UCSF Colitis and Crohn’s Disease Microbiome Research Core in the division of gastroenterology.—“We took an ecological approach, considering the bacteria in the context of their microbial neighborhoods to identify relationships between characteristics of these communities and features of the disease…This new approach will help us to better understand the microbiota-host relationships that define human health.”–The authors say that further studies are needed to determine how these specific bacteria identified in the study may influence the cause and development of asthma.
    ###
    The study was supported by the National Heart, Lung and Blood Institute and by the Strategic Asthma Basic Research Center at UCSF, supported by the Sandler Family Foundation. Huang was funded by a National Institutes of Health grant and by a UC Tobacco-related Disease Research Program award; Lynch receives research support from the NIH; Boushey is an ad-hoc consultant for KaloBios Pharmaceuticals, Inc., is on the advisory committee for Pharmaxis, is on ad-hoc advisory committees for GlaxoSmithKline and Merck, and receives research support from GlaxoSmithKline.
    UCSF is a leading university dedicated to promoting health worldwide through advanced biomedical research, graduate-level education in the life sciences and health professions, and excellence in patient care.
    Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of California – San Francisco, via EurekAlert!, a service of AAAS.–Journal Reference-Yvonne J. Huang, Craig E. Nelson, Eoin L. Brodie, Todd Z. DeSantis, Marshall S. Baek, Jane Liu, Tanja Woyke, Martin Allgaier, Jim Bristow, Jeanine P. Wiener-Kronish. Airway microbiota and bronchial hyperresponsiveness in patients with suboptimally controlled asthma. Journal of Allergy and Clinical Immunology, 2011; 127 (2): 372 DOI: 10.1016/j.jaci.2010.10.048
     
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    Bacteria to Blame in Asthma Attacks in Children, Research Suggests
    ScienceDaily (Oct. 7, 2010) — Doctors have long known that viral infections can bring about asthma attacks and the shortness of breath, coughing, and wheezing associated with them. But while viral infections cannot be treated, scientists at the Danish Paediatric Asthma Centre (DPAC) at the University of Copenhagen and Gentofte Hospital have discovered that treatable bacterial infections can also cause asthma attacks. The discovery could revolutionize treatment.—“We found a significant relationship between bacterial infections and acute asthma attacks — above and beyond the expected relationship between viral infections and attacks,” says Hans Bisgaard, a professor of pediatrics at the DPAC.–The study examined 361 children between the ages of four weeks and three years to determine the presence of viral and bacterial infections during severe asthma attacks. The results conclude that the number of attacks was just as high in children with bacterial respiratory infections as in those with viral infections.—
    Using antibiotics to treat asthma attacks?–“This indicates that bacteria can exacerbate asthma symptoms even if they aren’t infected with a virus,” Professor Bisgaard says. “The findings open up an entirely new method for treating severe asthma attacks. We can’t treat viral infections, but scientists will now look into whether treatment with antibiotics can help children when they have an asthma attack if they are also suffering from a bacterial infection.”–“Being able to use antibiotics to treat asthma attacks in children would be revolutionary,” Professor Bisgaard says.–The effects of antibiotics in treating asthma attacks will now be examined in large-scale, clinical study by the DPAC. The research has been published in British Medical Journal on 4 October 2010.
    Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of Copenhagen.
    Journal Reference-H. Bisgaard, M. N. Hermansen, K. Bonnelykke, J. Stokholm, F. Baty, N. L. Skytt, J. Aniscenko, T. Kebadze, S. L. Johnston. Association of bacteria and viruses with wheezy episodes in young children: prospective birth cohort study. BMJ, 2010; 341 (oct04 1): c4978 DOI: 10.1136/bmj.c4978
     
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    Blocking Effects Of Viral Infections May Prevent Asthma In Young Children
    ScienceDaily (Nov. 9, 2007) — Babies who get severe respiratory viral infections are much more likely to suffer from asthma as they get older. Now researchers at Washington University School of Medicine in St. Louis have pinpointed a key step in the development of asthma in mice after a severe respiratory infection. They suggest that medications designed to interfere with this mechanism could potentially prevent many cases of childhood asthma. –“A severe respiratory infection in infancy greatly increases the risk of developing asthma,” says the study’s lead author Mitchell Grayson, M.D., assistant professor of medicine in the Division of Allergy and Immunology. “Less than one in 30 people who don’t suffer a severe respiratory infection as a baby develop asthma, but of those who do get these infections, one in five goes on to have asthma.” Grayson and colleagues recently published their research in the Journal of Experimental Medicine. They found that mice that developed asthma-like symptoms after a severe respiratory viral infection had an unusual immune reaction. During the infection, the mice produced antibodies and immune signals similar to those produced during an allergic response, instead of those typically made in response to infection. That started a chain reaction that led to asthma. The researchers propose that a similar reaction occurs in some people who suffer severe respiratory viral infections. –“We think genetically predisposed [U3]individuals will tend to have this kind of immune reaction to a severe respiratory viral infection,” Grayson says. “In those people an allergic-type response could be part of their antiviral immune response. That sets them up to make antibodies against a lot of environmental substances, like pet dander or pollen, and they can go on to develop allergies or asthma.” –Reports by the Centers for Disease Control and Prevention indicate that the number of people with asthma in the United States rose from approximately 7 million in 1980 to about 20 million in 2003. The reasons for this trend are unclear, Grayson indicates. But he suggests that a growing population density and the resulting increase in transmission of respiratory viral infections might be a cause. —Respiratory syncytial virus (RSV) is a common source of respiratory infections. In the United States nearly all children have been infected with RSV by two or three years of age. Severe RSV infections, typified by persistent coughing, wheezing and gasping for breath, send many thousands of children to the hospital each year. –To investigate the connection between severe respiratory viral infections and subsequent asthma, the researchers used mice genetically selected to have an asthma susceptibility and infected them with a virus similar to RSV. They found that severe respiratory infections in the mice induced an allergic-type immune response and ultimately caused long-term changes in the airways of the lungs that are hallmarks of chronic asthma. —The researchers discovered that certain immune cells in the mouse lungs reacted to severe viral infections by releasing compounds that instigated an inflammatory response. That in turn induced many lung airway cells to transform into mucus-producing cells, which can cause the obstruction of lung passages and shortness of breath characteristic of asthma. —The researchers found that interfering with this process by altering the immune cells or removing the inflammatory compounds they secreted prevented overgrowth of mucus-producing cells. –The findings promise a new approach to asthma prevention, according to Grayson. “This offers a different way of thinking about what happens in the development of asthma,” Grayson says. “It may be possible to prevent many cases of asthma and other chronic inflammatory airway diseases by stopping allergic-type antibody production after a severe viral infection in infants.”
    Journal reference: Grayson MH, Cheung D, Rohlfing MM, Kitchens R, Spiegel DE, Tucker J, Battaile JT, Alevy Y, Yan L, Agapov E, Kim EY, Holtzman MJ. Induction of high-affinity IgE receptor on lung dendritic cells during viral infection leads to mucous cell metaplasia. Journal of Experimental Medicine 2007 Oct 29;204(11):2759-69. –Funding from the National Institute of Allergy and Infectious Disease, the National Heart, Lung and Blood Institute, Genentech Inc., Novartis International AG, the Martin Schaeffer Fund and the Alan A. and Edith L. Wolff Charitable Trust supported this research.–Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Washington University in St. Louis, via EurekAlert!, a service of AAAS.
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    Monsanto’s Technology Stewardship Agreement shifts responsibility to growers
    for any and all losses, injury or damages resulting from the use of Monsanto seeds. There is no expiration date on the contract. The grower may terminate the contract, but: “Grower’s responsibilities and the other terms herein shall survive…”
    This includes contamination of other farms. Growers are purchasing seed for Spring planting right now. Alfalfa, America’s 4th largest crop, is a particular problem because it is a perennial plant and the seeds may lie dormant in the ground for 10-20 years, and WILL contaminate non-GM plants. Contaminated alfalfa cannot be recalled from the environment. The liability burden can follow the grower for decades. Farmers must be made aware of the danger of being sued before they plant GM crops (especially alfalfa because it is used for cattle feed and will affect dairy farmers).
    Currently, Australian organic farmer Steve Marsh, who lost his organic certification due to contamination, is suing his GM crop-growing neighbor for the GM contamination.
    Contamination of processing equipment is another risk.
    There is evidence from India that GM crops are linked to livestock deaths. The Monsanto Technology Stewardship Agreement contract holds growers responsible for injuries, so this is another potential consequence for farmers planting Monsanto GM crops to consider.
    The Monsanto Technology Stewardship Agreement has another clause that farmers will find disturbing:
    it appears that the growers agree that in order to sell their farm, the new purchaser must also sign a Monsanto Technology Stewardship Agreement. According to a top real estate broker, the contract places a covenant, condition or restriction (CCR) on the farmer’s land:
    “GROWER AGREES: To accept and continue the obligations of this Monsanto Technology/Stewardship Agreement on any new land purchased or leased by Grower that has Seed planted on it by a previous owner or possessor of the land; and to notify in writing purchasers or lessees of land owned by Grower that has Seed planted on it that the Monsanto Technology is subject to this Monsanto Technology/Stewardship Agreement and they must have or obtain their own Monsanto Technology/Stewardship Agreement.”
    Environmental attorney Anthony Patchett further elaborated on Monsanto’s contract in a letter that states “Monsanto’s agreement shifts all liability to the growers, including contamination issues or any potential future liability. All the grower receives is the price of the seed.” He further stated that this contract appears to be “Unconscionable”. Click here to view the letter.
    For more information about the perils of contamination, please go to MorphCity.com to read the interview with alfalfa seed grower Phil Geertson who opposed Monsanto in the GM case heard in the Supreme Court last summer. Geertson said that Monsanto’s GM seeds are more expensive and after a few years, weeds can become tolerant to Roundup Ready and other glyphosate herbicides so farmers must return to conventional farming practices anyway. Therefore, there is no benefit to planting GM crops.
    You can alert farmers to the hazard of growing GM crops and how growers can be hurt by Monsanto’s contract, if you would like to take action in opposing GM crops. Please share this article and video.
     
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    Recipe For Anti Bacterial Resistance
    Antimicrobials: Silver (and Copper) Bullets to Kill Bacteria
    ScienceDaily (Nov. 10, 2009) — Dana Filoti of the University of New Hampshire is presenting thin films of silver and copper she has developed that can kill bacteria and may one day help to cut down on hospital infections.The antimicrobial properties of silver and copper have been known for centuries — last year, the U.S. Environmental Protection Agency officially registered copper alloys, allowing them to be marketed with the label “kills 99.9% of bacteria within two hours.” Copper ions are known to penetrate bacteria and disrupt molecular pathways important for their survival.–Using zeolite ceramic structures, Filoti is testing the hypothesis that the combination of silver and copper might work synergistically to better kill bacteria, work that she will present on November 12 at a meeting of the scientific society AVS in San Jose. “The hard ceramic structure looks like Swiss cheese and inside the holes there are ions of silver and copper,” says Filoti.–By experimenting with the ratio of the two metals and the texture of the thin films, she has been able to reduce the amount of microbes present on the surface by 99 percent. One application of these antimicrobials, which Filoti is developing in partnership with a company in New Hampshire, is an antimicrobial face mask designed to protect against pathogens that cause many hospital-acquired infections.
    Adapted from materials provided by American Institute of Physics
     
    øøAn Idea to try—
    Make a green drink with parsley and dandelion—add ¼ dropper of colloidal silver with this—should see an impact The mix will not be as refined— but you may still see results—-again this is something to try Dandelion and Parsley are loaded with copper—and enter the blood stream very efficiently—-colloids are extremely penetrating due to the way our system absorbs them—
     
    TOP F
    [U1]LEAVE STARCHY foods –grains–pastas–rice–corn–polentas–cakes–pastries–bananas–grapes–watermelon-raisons–soy–soy derivatives–tvp-hvp-avp-hpp msg autolysed yeast—all Problematic
    [U2]GMO and SOY might be the problem anddd the lack of nutrients in the soil
    [U3]Genetically altered food would cause a predispposition to having asthma!!??? is this what they are saying!!!

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