June 22, 2021 at 09:00 #2660
OTHER TESTING NOTES
The little manual that came with the charger stated that alkaline batteries can be charged to 75 – 90% of their original capacity. It further said that the batteries should be fully drained before recharging but I found that as a practical matter, the fully drained batteries came up “BAD” and could not be recharged at all.
There is one more rather important note that I want to make. I checked on my recharged batteries about two weeks after storing them in a box in my utility cupboard. Much to my surprise there were some new leakers. Now whether these were overlooked initially (had I wiped away the residue not realizing they were compromised) or a byproduct of the charging process – I do not know. Even more curious, they were all the Costco Kirkland brand batteries.
The manual also indicates that alkaline batteries should not be recharged more than 30 times. 30 times – holy moly – that seems like a lot of charging to me. For now, I will charge them once until I go through the pile and then start anew. But 30 times? I am going to play it safe and protect my devices by recharging no more than a dozen times if that.
SO REALLY, HOW DID THEY WORK?
Using the recharged batteries on low power devices such as mini LED flashlights and of course, my computer mouse, worked great. As a matter of fact, I can not tell the difference between the recharged Alkalines and standard rechargeables. On the other hand, the Survival Husband uses wireless headphones to watch TV and the recharged alkalines only last abut 8 hours as opposed to the typical 15 to 20 hours with standard Ni-mh rechargeables.
STORING THE BATTERIES
I purchased these inexpensive storage cases for my recharged batteries. They are really quite handy and I like that the six individual cases interlock and connect together to make a larger case. The rest of my spares are in the plastic pencil case you see pictured above.
THE FINAL WORD
For a total investment of about $34 I now have what for all intents and purposes, a lifetime of free batteries. The reason I say “lifetime” is that my friends have indicated an willingness to give me all of their dead batteries and you can bet that many will still be either good as is or eligible for recharging.
Would I trust these recycled batteries in a survival situation – sure, if that is all I had, But of course, that is not the case. I still have a large supply of brand new alkaline batteries in all sizes plus a good supply of standard rechargeables (you know how I love those eneloop rechargeables).
But as spare batteries to use day to day, heck, these are great. As my tests have proven, a recharged alkaline battery can keep a small LED flashlight going to over a week and a computer mouse for much longer than that. As a matter of fact, you might say I have become obsessed with using these recharged alkaline batteries. But that, of course, is a story for another time
Treating Skin Blemishes
Hot steaming towels then are applied to the afflicted area at least three times and, afterward, the afflicted area is patted dry.
One tablespoon of common table salt is dissolved in one measuring cup of very hot water having a temperature of not more than 120° F. The salt is completely dissolved in the water and a facecloth is immersed and soaked in the salt water. Sea salt with naturally occurring substances may have antibacterial characteristics that are superior to ordinary table salt and thus, sea salt water may be preferable for use in this step of the treatment process. The salt kills, it is believed, through osmosis.
A towel is folded lengthwise and then across. One side of the towel is thoroughly saturated with ordinary tap water and then folded into thirds in order to keep the wetted side of the towel on the inside of the folds. The towel is heated, preferably in a toaster oven or dryer, for several minute in order to heat the water in the wetted interior until it steams.
With the subject’s head slightly inclined to prevent inhalation of water from the towel (when treating facial acne), the hot salt water soaked facecloth is placed over the affected area, the saline water soaked facecloth then being covered by the unfolded steaming towel. Preferably, a second steaming towel can be placed over the first towel to maintain the facecloth’s heat for a longer period of time. Care must be taken, however, to make sure that these hot applications are sufficiently cool to avoid burning the skin that is undergoing treatment. With respect to this step in the treatment, it is thought that the hot applications help to further open the pores and gravity promotes flow of the saline water from the facecloth into the pores.
The heat, it seems, also promotes an increase in capillary blood flow that not only increases the supply of white and red blood cells to advance the healing process, but also removes waste products from the afflicted area. The effect of the heat applied to the subject’s skin also might destroy at least some of the bacteria that are related to the acne condition. As a general matter, heat may aid in the process in several ways. It may denature proteins, cause membrane damage and also may cause enzymatic cleavage of DNA. Moist heat, moreover, sterilizes at a lower temperature than dry heat because water serves to disrupt noncovalent bonds (hydrogen bonds) which hold protein chains together in their secondary and tertiary structures.
The steaming towel is replaced at least one more time, this replacement being repeated when the subject feels that the applied towel is no longer hot or has a noticeably elevated temperature. In this instance, the towel may actually be very warm, but the subject has at this point in the treatment become so conditioned to heat that the sensation of heat fails to register with the patient as being significant. The process temperatures need to be continually increased to and maintained at the desired levels to cause morbidity of proionibacterium acnes, dilation of the body’s own vascular structures to bring in the body’s own defensive mechanisms, remove waste through the dilated structures, and allow the salt to enter deep into the affected pilosebaceous unit. The salt will, but is not limited to, deprive the bacterium of water needed to survive, and the sodium that enters the bacterium’s membrane can not be excreted (it is a one-way trap for sodium, much like a diode is in electricity), but more water has to be brought into the bacteria to dilute the absorbed sodium until the bacteria actually ruptures.
After the last steaming towel is felt to be cool by the subject, the steaming towels and the underlying saline water soaked facecloth are removed.
The afflicted area then is further treated by being washed with a castile soap. For the purpose of the invention, the soap marketed as “Dr. Bronner’s Magic Soaps 18-in-1 Hemp Peppermint Pure-Castile Soap” has been found to be particularly efficacious. The Dr. Bronner’s soap that is used in the invention is a compound of water, saponified coconut-hemp-olive oils (with retained glycerin), olive fatty acids, peppermint oil (boosted with menthol), vitamin E, and citric acid.
The treated area is rinsed and dried, having on visual inspection a rosy appearance that will disappear in about twelve hours.
Within three days after treatment, the red pustules will be gone from the afflicted area, leaving only a few circular, dark brown freckle-like areas in those portions of the skin that were most seriously afflicted. To post treat these residual marks, a cotton applicator, soaked in either turpentine or in honey, is touched to the dark areas.—
Research suggests that the term “turpentine” is used imprecisely to describe either the oleoresin obtained from the longleaf pine (Pinus palustris Mill.) or the slash pine (P. elliottii Engelm.) along with other Pinus species that yield exclusively terpene oils, or the essential oil obtained from the above oleoresin. More than a half-dozen additional Pinus species have been used in the production of turpentine. The oleoresin is sometimes referred to as “gum turpentine” while turpentine or its oil (also known as spirits of turpentine) are terms for the essential oil. Following steam distillation, gum turpentine yields turpentine oil and a resin called colophony (also known as rosin). Alternately, rosin is collected by scarring the tree trunk, and various grades of material are then refined. Turpentine and rosin also are obtained by the steam distillation of wood chips of that are by-products of the lumber and paper industries, and these sources account for the bulk of the production of these compounds. In terms of volume, turpentine is the largest volume essential oil product in the world, with the bulk of production occurring in the United States. The labor-intensive production of rosin, however, occurs to a greater extent in Spain, Greece, India, and Morocco.—-Turpentine is composed primarily of monoterpene hydrocarbons, the most prevalent of which are the pinenes, camphene, and 3-carene. Rosin contains mostly diterpene resin acids such as abietic acid, dehydroabietic acid, palustric acid and isopimaric acid. Numerous other compounds are present in small quantities in all turpentine products.—Canada turpentine or Canada balsam is an oleoresin obtained from the stems of the balsam fir, Abies balsamae (Family Pinaceae).
Terebinthina.—It is an exudation from different species of Pines. It is a valuable remedy, either externally or internally. Sprinkled on flannel dipped in hot water, wrung out, and then locally applied, it is a powerful counter-irritant, acting like mustard, and even blistering. It is useful for local pains, for lumbago, sciatica, and for inflammatory diseases in the abdomen. As an external application to burns, turpentine has been much used. ———Internally turpentine is anthelmintic[I1] , diaphoretic, diuretic, purgative and stimulant, It is also given as an astringent. As a destroyer of worms, it should be given in combination with Castor Oil, lest failing to purge it should over-stimulate the urinary organs. As a diuretic, it is prescribed in dropsy, and suppression of urine. As a purgative, it is useful in cases of tympanitic distension of the abdomen, and in acute stages of puerperal fever. As a stimulant to the nervous system, in neuralgia and epilepsy.
Uses of Turpentine
Turpentine has been used experimentally in a bath for the treatment of disseminated sclerosis and sexual dysfunction. It also has been studied for its antibacterial activity and inhibition of osteoclast activity. Turpentine is utilized in experimental models of inflammation to induce a systemic inflammatory immune response in animals.
One study from Russia documents the use of turpentine white emulsion baths in patients with sexual dysfunctions, but the safety of this treatment has not been established***** Karpukhin IV, Li AA, Gusev ME. Turpentine white emulsion baths in the rehabilation in patients with sexual dysfunctions [in Russian]. Vopr Kurortol Fizioter Lech Fiz Kult . 2000;32-33.******** –Application of yellow turpentine baths in patients with chronic prostatitis complicated by sexual dysfunctions].
[Article in Russian]-[No authors listed]
Abstract—Patients with chronic prostatitis complicated by sexual dysfunction took turpentine baths with yellow solution in concentration rising from 5 to 55 ml solution per 200 l water, temperature 35-40 degrees C, duration 5-16 min, daily, 12-15 procedures. Yellow turpentine baths raise efficacy of treatment of patients with copulative dysfunction to 64% due to intensive arterial blood inflow to the sexual organs including the cavernous bodies of the penis.
Turpentine white emulsion baths in the rehabilation in patients with sexual dysfunctions].
[Article in Russian]
Karpukhin IV, Li AA, Gusev ME.
100 patients with sexual dysfunction (SD) and 20 SD patients took turpentine white emulsion baths and sodium chloride baths, respectively. The turpentine baths were given with step-by-step rise in turpentine concentration from 20 to 50 ml per 200 l of water, temperature 36-37 degrees C, duration of the procedure 10-15 min. The course consisted of 10-12 procedures which were conducted daily or each other day. The turpentine baths were more effective than sodium chloride baths (85 vs 50%, respectively).-PMID: 11247146 -[PubMed – indexed for MEDLINE]
Use of white turpentine bath emulsion and yellow turpentine solution for the treatment of chronic prostatitis complicated by excretory pathospermia].
[Article in Russian]
Karpukhin IV, Li AA, Gusev MA.
The paper reports a review of up-to-date methods for the use of white turpentine bath emulsion and yellow turpentine solution in the treatment of chronic prostatitis complicated by excretory pathospermia. The results of bath therapy are presented. It is shown that the efficiency of white turpentine bath emulsion amounted to 69.7% compared with 88.3% in patients treated with the use of yellow turpentine solution.—Preliminary reports from Russia suggest that turpentine baths may assist in the treatment of disseminated sclerosis, but the safety of this treatment has not been established.******** . Ludianskii EA. The extension of the use of physical methods of treatment to patients with disseminated sclerosis [in Russian]. Vopr Kurortol Fizioter Lech Fiz Kult . 1992;34-37.*******
Turpentine possesses antibacterial activity in vitro and has been applied topically to debride severe wounds infested with fly larvae***** Leung AY. Encyclopedia of Common Natural Ingredients Used in Food, Drugs, and Cosmetics . New York, NY: John Wiley & Sons, Inc., 1980*******Benign skin tumors have been observed in animal models following chronic topical application of turpentine
[I1]Internal USES— anthelmintic [I1], diaphoretic, diuretic, purgative and stimulant, It is also given as an astringent. As a destroyer of worms- As a diuretic, it is prescribed in dropsy, and suppression of urine. As a purgative, it is useful in cases of tympanitic distension of the abdomen, and in acute stages of puerperal fever. As a stimulant to the nervous system, in neuralgia and epilepsy.
[I2]Turpentine for ED
Johann Wolfgang von Goethe
Speech on Hidden Truth
None are so hopelessly enslaved, as those who falsely believe they are free. The truth has been kept from the depth of their minds by masters who rule them with lies. They feed them on falsehoods till wrong looks like right in their eyes.”
Show of The Month April 20 2013
Myths & Truths About Soy
Doctors Not Informed of Harmful Effects of Medicines During Sales Visits
THE BENFITS OF HIGH CHOLESTEROL
Summary of latest data on antibiotic resistance in the European Union
Recipe for Insulin Regulating
Eggs Prevent Heart Disease and Cancer
Eggs’ Antioxidant Properties May Help Prevent Heart Disease and Cancer,
Myths & Truths About Soy
Myth: Use of soy as a food dates back many thousands of years.
Truth: Soy was first used as a food during the late Chou dynasty (1134-246 BC), only after the Chinese learned to ferment soy beans to make foods like tempeh, natto and tamari.
Myth: Asians consume large amounts of soy foods.
Truth: Average consumption of soy foods in Japan and China is 10 grams (about 2 teaspoons) per day. Asians consume soy foods in small amounts as a condiment, and not as a replacement for animal foods.
Myth: Modern soy foods confer the same health benefits as traditionally fermented soy foods.
Truth: Most modern soy foods are not fermented to neutralize toxins in soybeans, and are processed in a way that denatures proteins and increases levels of carcinogens.
Myth: Soy foods provide complete protein.
Truth: Like all legumes, soy beans are deficient in sulfur-containing amino acids methionine and cystine. In addition, modern processing denatures fragile lysine.
Myth: Fermented soy foods can provide vitamin B12 in vegetarian diets.
Truth: The compound that resembles vitamin B12 in soy cannot be used by the human body; in fact, soy foods cause the body to require more B12
Myth: Soy formula is safe for infants.
Truth: Soy foods contain trypsin inhibitors that inhibit protein digestion and affect pancreatic function. In test animals, diets high in trypsin inhibitors led to stunted growth and pancreatic disorders. Soy foods increase the body’s requirement for vitamin D, needed for strong bones and normal growth. Phytic acid in soy foods results in reduced bioavailabilty of iron and zinc which are required for the health and development of the brain and nervous system[F1]. Soy also lacks cholesterol, likewise essential for the development of the brain and nervous system. Megadoses of phytoestrogens in soy formula have been implicated in the current trend toward increasingly premature sexual development in girls and delayed or retarded sexual development in boys.
Myth: Soy foods can prevent osteoporosis.
Truth: Soy foods can cause deficiencies in calcium and vitamin D, both needed for healthy bones. Calcium from bone broths and vitamin D from seafood, lard and organ meats prevent osteoporosis in Asian countries—not soy foods.
Myth: Modern soy foods protect against many types of cancer.
Truth: A British government report concluded that there is little evidence that soy foods protect against breast cancer or any other forms of cancer. In fact, soy foods may result in an increased risk of cancer.
Myth: Soy foods protect against heart disease.
Truth: In some people, consumption of soy foods will lower cholesterol, but there is no evidence that lowering cholesterol with soy protein improves one’s risk of having heart disease.
Myth: Soy estrogens (isoflavones) are good for you.
Truth: Soy isoflavones are phyto-endocrine disrupters. At dietary levels, they can prevent ovulation and stimulate the growth of cancer cells. Eating as little as 30 grams (about 4 tablespoons) of soy per day can result in hypothyroidism with symptoms of lethargy, constipation, weight gain and fatigue.
Myth: Soy foods are safe and beneficial for women to use in their postmenopausal years.
Truth: Soy foods can stimulate the growth of estrogen-dependent tumors and cause thyroid problems. Low thyroid function is associated with difficulties in menopause.
Myth: Phytoestrogens in soy foods can enhance mental ability.
Truth: A recent study found that women with the highest levels of estrogen in their blood had the lowest levels of cognitive function; In Japanese Americans tofu consumption in mid-life is associated with the occurrence of Alzheimer’s disease in later life.
Myth: Soy isoflavones and soy protein isolate have GRAS (Generally Recognized as Safe) status.
Truth: Archer Daniels Midland (ADM) recently withdrew its application to the FDA for GRAS status for soy isoflavones following an outpouring of protest from the scientific community. The FDA never approved GRAS status for soy protein isolate because of concern regarding the presence of toxins and carcinogens in processed soy.
Myth: Soy foods are good for your sex life.
Truth: Numerous animal studies show that soy foods cause infertility in animals. Soy consumption enhances hair growth in middle-aged men, indicating lowered testosterone levels. Japanese housewives feed tofu to their husbands frequently when they want to reduce his virility.
Myth: Soy beans are good for the environment.
Truth: Most soy beans grown in the US are genetically engineered to allow farmers to use large amounts of herbicides.
Myth: Soy beans are good for developing nations.
Truth: In third world countries, soybeans replace traditional crops and transfer the value-added of processing from the local population to multinational corporations.
Doctors Not Informed of Harmful Effects of Medicines During Sales Visits
Apr. 10, 2013 — The majority of family doctors receive little or no information about harmful effects of medicines when visited by drug company representatives, according to an international study involving Canadian, U.S. and French physicians. —Yet the same doctors indicated that they were likely to start prescribing these drugs, consistent with previous research that shows prescribing behaviour is influenced by pharmaceutical promotion.–The study, which had doctors fill out questionnaires about each promoted medicine following sales visits, was published online today in the Journal of General Internal Medicine. It shows that sales representatives failed to provide any information about common or serious side effects and the type of patients who should not use the medicine in 59 per cent of the promotions. In Vancouver and Montreal, no potential harms were mentioned for 66 per cent of promoted medicines.—“Laws in all three countries require sales representatives to provide information on harm as well as benefits,” says lead author Barbara Mintzes of the University of British Columbia. “But no one is monitoring these visits and there are next to no sanctions for misleading or inaccurate promotion.”—Serious risks were mentioned in only six percent of the promotions, even though 57 per cent of the medications involved in these visits came with US Food and Drug Administration “black box” or Health Canada boxed warnings — the strongest drug warning that can be issued by both countries.—“We are very concerned that doctors and patients are left in the dark and patient safety may be compromised,” says Mintzes, an expert on drug advertising in UBC’s School of Population and Public Health.–Doctors in Toulouse were more likely to be told of a harmful effect in a promotional visit, compared to doctors in Canada and the U.S., according to the study. Researchers suggested that this may reflect stricter regulatory standards for promotion of medicines in France.
About the study
The UBC-led study is the most comprehensive to date of the quality of pharmaceutical sales representative promotions to family physicians.–Researchers recruited physicians to participate using random samples from lists of primary care physicians at four sites — Vancouver, Montreal, Sacramento and Toulouse. Among 704 eligible physicians contacted, 255 (36 per cent) chose to participate. Information was collected on 1,692 drug promotions at sales visits between May 2009 to June 2010.
Doctors were asked to fill out a questionnaire about the information provided for each promoted medicine following each visit they received from pharmaceutical sales representatives. Sales representatives regularly visit doctors’ offices to promote medicines by providing information, free samples and in some cases food and invitations to events. The study focused on how often information was provided about drug safety.–The team includes researchers from UBC, York University, University of Montreal, University of California, Davis and the University of Toulouse.
Story Source-The above story is reprinted from materials provided by University of British Columbia, via EurekAlert!, a service of AAAS. -Journal Reference—Barbara Mintzes, Joel Lexchin, Jason M. Sutherland, Marie-Dominique Beaulieu, Michael S. Wilkes, Geneviève Durrieu, Ellen Reynolds. Pharmaceutical Sales Representatives and Patient Safety: A Comparative Prospective Study of Information Quality in Canada, France and the United States. Journal of General Internal Medicine, 2013; DOI: 10.1007/s11606-013-2411-7
THE BENFITS OF HIGH CHOLESTEROL
People with high cholesterol live the longest. This statement seems so incredible that it takes a long time to clear one´s brainwashed mind to fully understand its importance. Yet the fact that people with high cholesterol live the longest emerges clearly from many scientific papers. Consider the finding of Dr. Harlan Krumholz of the Department of Cardiovascular Medicine at Yale University, who reported in 1994 that old people with low cholesterol died twice as often from a heart attack as did old people with a high cholesterol.1 Supporters of the cholesterol campaign consistently ignore his observation, or consider it as a rare exception, produced by chance among a huge number of studies finding the opposite. But it is not an exception; there are now a large number of findings that contradict the lipid hypothesis. To be more specific, most studies of old people have shown that high cholesterol is not a risk factor for coronary heart disease. This was the result of my search in the Medline database for studies addressing that question.2Eleven studies of old people came up with that result, and a further seven studies found that high cholesterol did not predict all-cause mortality either. Now consider that more than 90 % of all cardiovascular disease is seen in people above age 60 also and that almost all studies have found that high cholesterol is not a risk factor for women.2 This means that high cholesterol is only a risk factor for less than 5 % of those who die from a heart attack. But there is more comfort for those who have high cholesterol; six of the studies found that total mortality was inversely associated with either total or LDL-cholesterol, or both. This means that it is actually much better to have high than to have low cholesterol if you want to live to be very old.
High Cholesterol Protects Against Infection
Many studies have found that low cholesterol is in certain respects worse than high cholesterol. For instance, in 19 large studies of more than 68,000 deaths, reviewed by Professor David R. Jacobs and his co-workers from the Division of Epidemiology at the University of Minnesota, low cholesterol predicted an increased risk of dying from gastrointestinal and respiratory diseases.3 Most gastrointestinal and respiratory diseases have an infectious origin. Therefore, a relevant question is whether it is the infection that lowers cholesterol or the low cholesterol that predisposes to infection? To answer this question Professor Jacobs and his group, together with Dr. Carlos Iribarren, followed more than 100,000 healthy individuals in the San Francisco area for fifteen years. At the end of the study those who had low cholesterol at the start of the study had more often been admitted to the hospital because of an infectious disease.4,5 This finding cannot be explained away with the argument that the infection had caused cholesterol to go down, because how could low cholesterol, recorded when these people were without any evidence of infection, be caused by a disease they had not yet encountered? Isn´t it more likely that low cholesterol in some way made them more vulnerable to infection, or that high cholesterol protected those who did not become infected? Much evidence exists to support that interpretation.
Low Cholesterol and HIV/AIDS
Young, unmarried men with a previous sexually transmitted disease or liver disease run a much greater risk of becoming infected with HIV virus than other people. The Minnesota researchers, now led by Dr. Ami Claxton, followed such individuals for 7-8 years. After having excluded those who became HIV-positive during the first four years, they ended up with a group of 2446 men. At the end of the study, 140 of these people tested positive for HIV; those who had low cholesterol at the beginning of the study were twice as likely to test postitive for HIV compared with those with the highest cholesterol.6 Similar results come from a study of the MRFIT screenees, including more than 300,000 young and middle-aged men, which found that 16 years after the first cholesterol analysis the number of men whose cholesterol was lower than 160 and who had died from AIDS was four times higher than the number of men who had died from AIDS with a cholesterol above 240.7
Cholesterol and Chronic Heart Failure
Heart disease may lead to a weakening of the heart muscle. A weak heart means that less blood and therefore less oxygen is delivered to the arteries. To compensate for the decreased power, the heart beat goes up, but in severe heart failure this is not sufficient. Patients with severe heart failure become short of breath because too little oxygen is delivered to the tissues, the pressure in their veins increases because the heart cannot deliver the blood away from the heart with sufficient power, and they become edematous, meaning that fluid accumulates in the legs and in serious cases also in the lungs and other parts of the body. This condition is called congestive or chronic heart failure. There are many indications that bacteria or other microorganisms play an important role in chronic heart failure. For instance, patients with severe chronic heart failure have high levels of endotoxin and various types of cytokines in their blood. Endotoxin, also named lipopolysaccharide, is the most toxic substance produced by Gram-negative bacteria such as Escherichia coli, Klebsiella, Salmonella, Serratia and Pseudomonas. Cytokines are hormones secreted by white blood cells in their battle with microorganisms; high levels of cytokines in the blood indicate that inflammatory processes are going on somewhere in the body. The role of infections in chronic heart failure has been studied by Dr. Mathias Rauchhaus and his team at the Medical Department, Martin-Luther-University in Halle, Germany (Universitätsklinik und Poliklinik für Innere Medizin III, Martin-Luther-Universität, Halle). They found that the strongest predictor of death for patients with chronic heart failure was the concentration of cytokines in the blood, in particular in patients with heart failure due to coronary heart disease.8 To explain their finding they suggested that bacteria from the gut may more easily penetrate into the tissues when the pressure in the abdominal veins is increased because of heart failure. In accordance with this theory, they found more endotoxin in the blood of patients with congestive heart failure and edema than in patients with non-congestive heart failure without edema, and endotoxin concentrations decreased significantly when the heart’s function was improved by medical treatment.9A simple way to test the functional state of the immune system is to inject antigens from microorganisms that most people have been exposed to, under the skin. If the immune system is normal, an induration (hard spot) will appear about 48 hours later at the place of the injection. If the induration is very small, with a diameter of less than a few millimeters, this indicates the presence of “anergy,” a reduction in or failure of response to recognize antigens. In accordance, anergy has been found associated with an increased risk of infection and mortality in healthy elderly individuals, in surgical patients and in heart transplant patients.10 Dr. Donna Vredevoe and her group from the School of Nursery and the School of Medicine, University of California at Los Angeles tested more than 200 patients with severe heart failure with five different antigens and followed them for twelve months. The cause of heart failure was coronary heart disease in half of them and other types of heart disease (such as congenital or infectious valvular heart disease, various cardiomyopathies and endocarditis) in the rest. Almost half of all the patients were anergic, and those who were anergic and had coronary heart disease had a much higher mortality than the rest.10 Now to the salient point: to their surprise the researchers found that mortality was higher, not only in the patients with anergy, but also in the patients with the lowest lipid values, including total cholesterol, LDL-cholesterol and HDL-cholesterol as well as triglycerides. The latter finding was confirmed by Dr. Rauchhaus, this time in co-operation with researchers at several German and British university hospitals. They found that the risk of dying for patients with chronic heart failure was strongly and inversely associated with total cholesterol, LDL-cholesterol and also triglycerides; those with high lipid values lived much longer than those with low values.11,12 Other researchers have made similar observations. The largest study has been performed by Professor Gregg C. Fonorow and his team at the UCLA Department of Medicine and Cardiomyopathy Center in Los Angeles.13 The study, led by Dr. Tamara Horwich, included more than a thousand patients with severe heart failure. After five years 62 percent of the patients with cholesterol below 129 mg/l had died, but only half as many of the patients with cholesterol above 223 mg/l. When proponents of the cholesterol hypothesis are confronted with findings showing a bad outcome associated with low cholesterol—and there are many such observations—they usually argue that severely ill patients are often malnourished, and malnourishment is therefore said to cause low cholesterol. However, the mortality of the patients in this study was independent of their degree of nourishment; low cholesterol predicted early mortality whether the patients were malnourished or not.
As discussed in The Cholesterol Myths (see sidebar), much evidence supports the theory that people born with very high cholesterol, so-called familial hypercholesterolemia, are protected against infection. But if inborn high cholesterol protects against infections, inborn low cholesterol should have the opposite effect. Indeed, this seems to be true. Children with the Smith-Lemli-Opitz syndrome produce very little cholesterol because the enzyme that is necessary for the last step in the body’s synthesis of cholesterol does not function properly. Most children with this syndrome are either stillborn or they die early because of serious malformations of the central nervous system. Those who survive are imbecile, they have extremely low cholesterol and suffer from frequent and severe infections. However, if their diet is supplemented with pure cholesterol or extra eggs, their cholesterol goes up and their bouts of infection become less serious and less frequent.14
Laboratory studies are crucial for learning more about the mechanisms by which the lipids exert their protective function. One of the first to study this phenomenon was Dr Sucharit Bhakdi from the Institute of Medical Microbiology, University of Giessen (Institut für Medizinsche Mikrobiologie, Justus-Liebig-Universität Gießen), Germany along with his team of researchers from various institutions in Germany and Denmark.15 Staphylococcus aureus a-toxin is the most toxic substance produced by strains of the disease-promoting bacteria called staphylococci. It is able to destroy a wide variety of human cells, including red blood cells. For instance, if minute amounts of the toxin are added to a test tube with red blood cells dissolved in 0.9 percent saline, the blood is hemolyzed, that is the membranes of the red blood cells burst and hemoglobin from the interior of the red blood cells leaks out into the solvent. Dr. Bhakdi and his team mixed purified a-toxin with human serum (the fluid in which the blood cells reside) and saw that 90 percent of its hemolyzing effect disappeared. By various complicated methods they identified the protective substance as LDL, the carrier of the so-called bad cholesterol. In accordance, no hemolysis occurred when they mixed a-toxin with purified human LDL, whereas HDL or other plasma constituents were ineffective in this respect. Dr. Willy Flegel and his co-workers at the Department of Transfusion Medicine, University of Ulm, and the Institute of Immunology and Genetics at the German Cancer Research Center in Heidelberg, Germany (DRK-Blutspendezentrale und Abteilung für Transfusionsmedizin, Universität Ulm, und Deutsches Krebsforschungszentrum, Heidelberg) studied endotoxin in another way.16 As mentioned, one of the effects of endotoxin is that white blood cells are stimulated to produce cytokines. The German researchers found that the cytokine-stimulating effect of endotoxin on the white blood cells disappeared almost completely if the endotoxin was mixed with human serum for 24 hours before they added the white blood cells to the test tubes. In a subsequent study17 they found that purified LDL from patients with familial hypercholesterolemia had the same inhibitory effect as the serum. LDL may not only bind and inactivate dangerous bacterial toxins; it seems to have a direct beneficial influence on the immune system also, possibly explaining the observed relationship between low cholesterol and various chronic diseases. This was the starting point for a study by Professor Matthew Muldoon and his team at the University of Pittsburgh, Pennsylvania. They studied healthy young and middle-aged men and found that the total number of white blood cells and the number of various types of white blood cells were significantly lower in the men with LDL-cholesterol below 160 mg/dl (mean 88.3 mg/l),than in men with LDL-cholesterol above 160 mg/l (mean 185.5 mg/l).18 The researchers cautiously concluded that there were immune system differences between men with low and high cholesterol, but that it was too early to state whether these differences had any importance for human health. Now, seven years later with many of the results discussed here, we are allowed to state that the immune-supporting properties of LDL-cholesterol do indeed play an important role in human health.
The immune systems in various mammals including human beings have many similarities. Therefore, it is interesting to see what experiments with rats and mice can tell us. Professor Kenneth Feingold at the Department of Medicine, University of California, San Francisco, and his group have published several interesting results from such research. In one of them they lowered LDL-cholesterol in rats by giving them either a drug that prevents the liver from secreting lipoproteins, or a drug that increases their disappearance. In both models, injection of endotoxin was followed by a much higher mortality in the low-cholesterol rats compared with normal rats. The high mortality was not due to the drugs because, if the drug-treated animals were injected with lipoproteins just before the injection of endotoxin, their mortality was reduced to normal.19
Dr. Mihai Netea and his team from the Departments of Internal and Nuclear Medicine at the University Hospital in Nijmegen, The Netherlands, injected purified endotoxin into normal mice, and into mice with familial hypercholesterolemia that had LDL-cholesterol four times higher than normal. Whereas all normal mice died, they had to inject eight times as much endotoxin to kill the mice with familial hypercholesterolemia. In another experiment they injected live bacteria and found that twice as many mice with familial hypercholesterolemia survived compared with normal mice.20
Other Protecting Lipids
As seen from the above, many of the roles played by LDL-cholesterol are shared by HDL. This should not be too surprising considering that high HDL-cholesterol is associated with cardiovascular health and longevity. But there is more. triglycerides, molecules consisting of three fatty acids linked to glycerol, are insoluble in water and are therefore carried through the blood inside lipoproteins, just as cholesterol. All lipoproteins carry triglycerides, but most of them are carried by a lipoprotein named VLDL (very low-density lipoprotein) and by chylomicrons, a mixture of emulsified triglycerides appearing in large amounts after a fat-rich meal, particularly in the blood that flows from the gut to the liver. For many years it has been known that sepsis, a life-threatening condition caused by bacterial growth in the blood, is associated with a high level of triglycerides. The serious symptoms of sepsis are due to endotoxin, most often produced by gut bacteria. In a number of studies, Professor Hobart W. Harris at the Surgical Research Laboratory at San Francisco General Hospital and his team found that solutions rich in triglycerides but with practically no cholesterol were able to protect experimental animals from the toxic effects of endotoxin and they concluded that the high level of triglycerides seen in sepsis is a normal immune response to infection.21 Usually the bacteria responsible for sepsis come from the gut. It is therefore fortunate that the blood draining the gut is especially rich in triglycerides.
So far, animal experiments have confirmed the hypothesis that high cholesterol protects against infection, at least against infections caused by bacteria. In a similar experiment using injections of Candida albicans, a common fungus, Dr. Netea and his team found that mice with familial hypercholesterolemia died more easily than normal mice.22 Serious infections caused by Candida albicans are rare in normal human beings; however, they are mainly seen in patients treated with immunosuppressive drugs, but the finding shows that we need more knowledge in this area. However, the many findings mentioned above indicate that the protective effects of the blood lipids against infections in human beings seem to be greater than any possible adverse effects.
Cholesterol as a Risk Factor
Most studies of young and middle-aged men have found high cholesterol to be a risk factor for coronary heart disease, seemingly a contradiction to the idea that high cholesterol is protective. Why is high cholesterol a risk factor in young and middle-aged men? A likely explanation is that men of that age are often in the midst of their professional career. High cholesterol may therefore reflect mental stress, a well-known cause of high cholesterol and also a risk factor for heart disease. Again, high cholesterol is not necessarily the direct cause but may only be a marker. High cholesterol in young and middle-aged men could, for instance, reflect the body’s need for more cholesterol because cholesterol is the building material of many stress hormones. Any possible protective effect of high cholesterol may therefore be counteracted by the negative influence of a stressful life on the vascular system.
Response to Injury
In 1976 one of the most promising theories about the cause of atherosclerosis was the Response-to-Injury Hypothesis, presented by Russell Ross, a professor of pathology, and John Glomset, a professor of biochemistry and medicine at the Medical School, University of Washington in Seattle.23,24 They suggested that atherosclerosis is the consequence of an inflammatory process, where the first step is a localized injury to the thin layer of cells lining the inside of the arteries, the intima. The injury causes inflammation and the raised plaques that form are simply healing lesions. Their idea is not new. In 1911, two American pathologists from the Pathological Laboratories, University of Pittsburgh, Pennsylvania, Oskar Klotz and M.F. Manning, published a summary of their studies of the human arteries and concluded that “there is every indication that the production of tissue in the intima is the result of a direct irritation of that tissue by the presence of infection or toxins or the stimulation by the products of a primary degeneration in that layer.”25 Other researchers have presented similar theories.26 Researchers have proposed many potential causes of vascular injury, including mechanical stress, exposure to tobacco fumes, high LDL-cholesterol, oxidized cholesterol, homocysteine, the metabolic consequences of diabetes, iron overload, copper deficiency, deficiencies of vitamins A and D, consumption of trans fatty acids, microorganisms and many more. With one exception, there is evidence to support roles for all of these factors, but the degree to which each of them participates remains uncertain. The exception is of course LDL-cholesterol. Much research allows us to exclude high LDL-cholesterol from the list. Whether we look directly with the naked eye at the inside of the arteries at autopsy, or we do it indirectly in living people using x-rays, ultrasound or electron beams, no association worth mentioning has ever been found between the amount of lipid in the blood and the degree of atherosclerosis in the arteries. Also, whether cholesterol goes up or down, by itself or due to medical intervention, the changes of cholesterol have never been followed by parallel changes in the atherosclerotic plaques; there is no dose-response. Proponents of the cholesterol campaign often claim that the trials indeed have found dose-response, but here they refer to calculations between the mean changes of the different trials with the outcome of the whole treatment group. However, true dose-response demands that the individual changes of the putative causal factor are followed by parallel, individual changes of the disease outcome, and this has never occurred in the trials where researchers have calculated true dose-response. A detailed discussion of the many factors accused of harming the arterial endothelium is beyond the scope of this article. However, the protective role of the blood lipids against infections obviously demands a closer look at the alleged role of one of the alleged causes, the microorganisms.
Is Atherosclerosis an Infectious Disease?
For many years scientists have suspected that viruses and bacteria, in particular cytomegalovirus and Chlamydia pneumonia (also named TWAR bacteria) participate in the development of atherosclerosis. Research within this area has exploded during the last decade and by January 2004, at least 200 reviews of the issue have been published in medical journals. Due to the widespread preoccupation with cholesterol and other lipids, there has been little general interest in the subject, however, and few doctors know much about it. Here I shall mention some of the most interesting findings.26 Electron microscopy, immunofluorescence microscopy and other advanced techniques have allowed us to detect microorganisms and their DNA in the atherosclerotic lesions in a large proportion of patients. Bacterial toxins and cytokines, hormones secreted by the white blood cells during infections, are seen more often in the blood from patients with recent heart disease and stroke, in particular during and after an acute cardiovascular event, and some of them are strong predictors of cardiovascular disease. The same is valid for bacterial and viral antibodies, and a protein secreted by the liver during infections, named C-reactive protein (CRP), is a much stronger risk factor for coronary heart disease than cholesterol Clinical evidence also supports this theory. During the weeks preceding an acute cardiovascular attack many patients have had a bacterial or viral infection. For instance, Dr. Armin J. Grau from the Department of Neurology at the University of Heidelberg and his team asked 166 patients with acute stroke, 166 patients hospitalized for other neurological diseases and 166 healthy individuals matched individually for age and sex about recent infectious disease. Within the first week before the stroke, 37 of the stroke patients, but only 14 of the control individuals had had an infectious disease. In half of the patients the infection was of bacterial origin, in the other half of viral origin.27 Similar observations have been made by many others, for patients with acute myocardial infarction (heart attack). For instance, Dr. Kimmo J. Mattila at the Department of Medicine, Helsinki University Hospital, Finland, found that 11 of 40 male patients with an acute heart attack before age 50 had an influenza-like infection with fever within 36 hours prior to admittance to hospital, but only 4 out of 41 patients with chronic coronary disease (such as recurrent angina or pervious myocardial infarction) and 4 out of 40 control individuals without chronic disease randomly selected from the general population.28 Attempts have been made to prevent cardiovascular disease by treatment with antibiotics. In five trials treatment of patients with coronary heart disease using azithromyzin or roxithromyzin, antibiotics that are effective against Chlamydia pneumonia,yielded successful results; a total of 104 cardiovascular events occurred among the 412 non-treated patients, but only 61 events among the 410 patients in the treatment groups.28a-e In one further trial a significant decreased progression of atherosclerosis in the carotid arteries occurred with antibiotic treatment.28f However, in four other trials,30a-d one of which included more than 7000 patients,28d antibiotic treatment had no significant effect. The reason for these inconsistent results may be that the treatment was too short (in one of the trials treatment lasted only five days). Also, Chlamydia pneumonia, the TWAR bacteria, can only propagate inside human cells and when located in white blood cells they are resistant to antibiotics.31 Treatment may also have been ineffective because the antibiotics used have no effect on viruses. In this connection it is interesting to mention a controlled trial performed by Dr. Enrique Gurfinkel and his team from Fundación Favaloro in Buenos Aires, Argentina.32 They vaccinated half of 301 patients with coronary heart disease against influenza, a viral disease. After six months 8 percent of the control patients had died, but only 2 percent of the vaccinated patients. It is worth mentioning that this effect was much better than that achieved by any statin trial, and in a much shorter time.
Does High Cholesterol Protect Against Cardiovascular Disease?
Apparently, microorganisms play a role in cardiovascular disease. They may be one of the factors that start the process by injuring the arterial endothelium. A secondary role may be inferred from the association between acute cardiovascular disease and infection. The infectious agent may preferably become located in parts of the arterial walls that have been previously damaged by other agents, initiating local coagulation and the creation of a thrombus (clot) and in this way cause obstruction of the blood flow. But if so, high cholesterol may protect against cardiovascular disease instead of being the cause! In any case, the diet-heart idea, with its demonizing of high cholesterol, is obviously in conflict with the idea that high cholesterol protects against infections. Both ideas cannot be true. Let me summarize the many facts that conflict with the idea that high cholesterol is bad. If high cholesterol were the most important cause of atherosclerosis, people with high cholesterol should be more atherosclerotic than people with low cholesterol. But as you know by now this is very far from the truth. If high cholesterol were the most important cause of atherosclerosis, lowering of cholesterol should influence the atherosclerotic process in proportion to the degree of its lowering. But as you know by now, this does not happen.If high cholesterol were the most important cause of cardiovascular disease, it should be a risk factor in all populations, in both sexes, at all ages, in all disease categories, and for both heart disease and stroke. But as you know by now, this is not the case I have only two arguments for the idea that high cholesterol is good for the blood vessels, but in contrast to the arguments claiming the opposite they are very strong. The first one stems from the statin trials. If high cholesterol were the most important cause of cardiovascular disease, the greatest effect of statin treatment should have been seen in patients with the highest cholesterol, and in patients whose cholesterol was lowered the most. Lack of dose-response cannot be attributed to the knowledge that the statins have other effects on plaque stabilization, as this would not have masked the effect of cholesterol-lowering considering the pronounced lowering that was achieved. On the contrary, if a drug that effectively lowers the concentration of a molecule assumed to be harmful to the cardiovascular system and at the same time exerts several beneficial effects on the same system, a pronounced dose-response should be seen. On the other hand, if high cholesterol has a protective function, as suggested, its lowering would counterbalance the beneficial effects of the statins and thus work against a dose-response, which would be more in accord with the results from the various trials. I have already mentioned my second argument, but it can’t be said too often: High cholesterol is associated with longevity in old people. It is difficult to explain away the fact that during the period of life in which most cardiovascular disease occurs and from which most people die (and most of us die from cardiovascular disease), high cholesterol occurs most often in people with the lowest mortality. How is it possible that high cholesterol is harmful to the artery walls and causes fatal coronary heart disease, the commonest cause of death, if those whose cholesterol is the highest, live longer than those whose cholesterol is low?
To the public and the scientific community I say, “Wake up!”June 22, 2021 at 09:00 #2661
1. Krumholz HM and others. Lack of association between cholesterol and coronary heart disease mortality and morbidity and all-cause mortality in persons older than 70 years. Journal of the American Medical Association 272, 1335-1340, 1990.
2. Ravnskov U. High cholesterol may protect against infections and atherosclerosis. Quarterly Journal of Medicine 96, 927-934, 2003.
3. Jacobs D and others. Report of the conference on low blood cholesterol: Mortality associations. Circulation 86, 1046–1060, 1992.
4. Iribarren C and others. Serum total cholesterol and risk of hospitalization, and death from respiratory disease. International Journal of Epidemiology 26, 1191–1202, 1997.
5. Iribarren C and others. Cohort study of serum total cholesterol and in-hospital incidence of infectious diseases. Epidemiology and Infection 121, 335–347, 1998.
6. Claxton AJ and others. Association between serum total cholesterol and HIV infection in a high-risk cohort of young men. Journal of acquired immune deficiency syndromes and human retrovirology 17, 51–57, 1998.
7. Neaton JD, Wentworth DN. Low serum cholesterol and risk of death from AIDS. AIDS 11, 929–930, 1997.
8. Rauchhaus M and others. Plasma cytokine parameters and mortality in patients with chronic heart failure. Circulation 102, 3060-3067, 2000.
9. Niebauer J and others. Endotoxin and immune activation in chronic heart failure. Lancet 353, 1838-1842, 1999.
10. Vredevoe DL and others. Skin test anergy in advanced heart failure secondary to either ischemic or idiopathic dilated cardiomyopathy. American Journal of Cardiology 82, 323-328, 1998.
11. Rauchhaus M, Coats AJ, Anker SD. The endotoxin-lipoprotein hypothesis. Lancet 356, 930–933, 2000.
12. Rauchhaus M and others. The relationship between cholesterol and survival in patients with chronic heart failure. Journal of the American College of Cardiology 42, 1933-1940, 2003.
13. Horwich TB and others. Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure. Journal of Cardiac Failure 8, 216-224, 2002.
14. Elias ER and others. Clinical effects of cholesterol supplementation in six patients with the Smith-Lemli-Opitz syndrome (SLOS). American Journal of Medical Genetics 68, 305–310, 1997.
15. Bhakdi S and others. Binding and partial inactivation of Staphylococcus aureus a-toxin by human plasma low density lipoprotein. Journal of Biological Chemistry 258, 5899-5904, 1983.
16. Flegel WA and others. Inhibition of endotoxin-induced activation of human monocytes by human lipoproteins. Infection and Immunity 57, 2237-2245, 1989.
17. Weinstock CW and others. Low density lipoproteins inhibit endotoxin activation of monocytes. Arteriosclerosis and Thrombosis 12, 341-347, 1992.
18. Muldoon MF and others. Immune system differences in men with hypo- or hypercholesterolemia. Clinical Immunology and Immunopathology 84, 145-149, 1997.
19. Feingold KR and others. Role for circulating lipoproteins in protection from endotoxin toxicity. Infection and Immunity 63, 2041-2046, 1995.
20. Netea MG and others. Low-density lipoprotein receptor-deficient mice are protected against lethal endotoxemia and severe gram-negative infections. Journal of Clinical Investigation 97, 1366-1372, 1996.
21. Harris HW, Gosnell JE, Kumwenda ZL. The lipemia of sepsis: triglyceride-rich lipoproteins as agents of innate immunity. Journal of Endotoxin Research 6, 421-430, 2001.
22. Netea MG and others. Hyperlipoproteinemia enhances susceptibility to acute disseminated Candida albicans infection in low-density-lipoprotein-receptor-deficient mice. Infection and Immunity 65, 2663-2667, 1997.
23. Ross R, Glomset JA. The pathogenesis of atherosclerosis. New England Journal of Medicine 295, 369-377, 1976.
24. Ross R. The pathogenesis of atherosclerosis and update. New England Journal of Medicine 314, 488-500, 1986.
25. Klotz O, Manning MF. Fatty streaks in the intima of arteries. Journal of Pathology and Bacteriology. 16, 211-220, 1911.
26. At least 200 reviews about the role of infections in atherosclerosis and cardiovascular disease have been published; here are a few of them: a) Grayston JT, Kuo CC, Campbell LA, Benditt EP. Chlamydia pneumoniae strain TWAR and atherosclerosis. European Heart Journal Suppl K, 66-71, 1993. b) Melnick JL, Adam E, Debakey ME. Cytomegalovirus and atherosclerosis. European Heart Journal Suppl K, 30-38, 1993. c) Nicholson AC, Hajjar DP. Herpesviruses in atherosclerosis and thrombosis. Etiologic agents or ubiquitous bystanders? Arteriosclerosis Thrombosis and Vascular Biology 18, 339-348, 1998. d) Ismail A, Khosravi H, Olson H. The role of infection in atherosclerosis and coronary artery disease. A new therapeutic target. Heart Disease 1, 233-240, 1999. e) Kuvin JT, Kimmelstiel MD. Infectious causes of atherosclerosis. f.) Kalayoglu MV, Libby P, Byrne GI. Chlamydia pneumonia as an emerging risk factor in cardiovascular disease. Journal of the American Medical Association 288, 2724-2731, 2002.
27. Grau AJ and others. Recent bacterial and viral infection is a risk factor for cerebrovascular ischemia. Neurology 50, 196-203, 1998.
28. Mattila KJ. Viral and bacterial infections in patients with acute myocardial infarction. Journal of Internal Medicine 225, 293-296, 1989.
29. The successful trials: a) Gurfinkel E. Lancet 350, 404-407, 1997. b) Gupta S and others. Circulation 96, 404-407, 1997. c) Muhlestein JB and others. Circulation 102, 1755-1760, 2000. d) Stone AFM and others. Circulation 106, 1219-1223, 2002. e) Wiesli P and others. Circulation 105, 2646-2652, 2002. f) Sander D and others. Circulation 106, 2428-2433, 2002.
30. The unsuccessful trials: a) Anderson JL and others. Circulation 99, 1540-1547, 1999. b) Leowattana W and others. Journal of the Medical Association of Thailand 84 (Suppl 3), S669-S675, 2001. c) Cercek B and others. Lancet 361, 809-813, 2003. d) O’Connor CM and others. Journal of the American Medical Association. 290, 1459-1466, 2003.
31. Gieffers J and others. Chlamydia pneumoniae infection in circulating human monocytes is refractory to antibiotic treatment. Circulation 104, 351-356, 2001
32. Gurfinkel EP and others. Circulation 105, 2143-2147, 2002.
About the author
Dr. Ravnskov is the author of The Cholesterol Myths and chairman of The International Network of Cholesterol Skeptics (thincs.org).
There is one risk factor that is known to be certain to cause death. It is such a strong risk factor that it has a 100 percent mortality rate. Thus I can guarantee that if we stop this risk factor, which would take no great research and cost nothing in monetary terms, within a century human deaths would be completely eliminated. This risk factor is called “Life.”
Barry Groves, second-opinions.com
Familial Hypercholesterolemia – Not as Risky as You May Think
Many doctors believe that most patients with familial hypercholesterolemia (FH) die from CHD at a young age. Obviously, they do not know the surprising finding of the Scientific Steering Committee at the Department of Public Health and Primary Care at Radcliffe Infirmary in Oxford, England. For several years, these researchers followed more than 500 FH patients between the ages of 20 and 74 and compared patient mortality during this period with that of the general population.
During a three- to four-year period, six of 214 FH patients below age 40 died from CHD. This may not seem particularly frightening but as it is rare to die from CHD before the age of 40, the risk for these FH patients was almost 100 times that of the general population.
During a four- to five-year period, eight of 237 FH patients between ages 40 and 59 died, which was five times more than the general population. But during a similar period of time, only one of 75 FH patients between the ages of 60 and 74 died from CHD, when the expected number was two.
If these results are typical for FH, you could say that between ages 20 and 59, about 3 percent of the patients die from CHD, and between ages 60 and 74, less than 2 percent die, in both cases during a period of 3-4 years. The authors stressed that the patients had been referred because of a personal or family history of premature vascular disease and therefore were at a particularly high risk for CHD. Most patients with FH in the general population are unrecognized and untreated. Had the patients studied been representative for all FH patients, their prognosis would probably have been even better.
This view was recently confirmed by Dr. Eric Sijbrands and his coworkers from various medical departments in Amsterdam and Leiden, Netherlands. Out of a large group they found three individuals with very high cholesterol. A genetic analysis confirmed the diagnosis of FH and by tracing their family members backward in time, they came up with a total of 412 individuals. The coronary and total mortality of these members were compared with the mortality of the general Dutch population.
The striking finding was that those who lived during the 19th and early 20th century had normal mortality and lived a normal life span. In fact, those living in the 19th century had a lower mortality than the general population. After 1915 the mortality rose to a maximum between 1935 and 1964, but even at the peak, mortality was less than twice as high as in the general population.
Again, very high cholesterol levels alone do not lead to a heart attack. In fact, high cholesterol may even be protective against other diseases. This was the conclusion of Dr. Sijbrands and his colleagues. As support they cited the fact that genetically modified mice with high cholesterol are protected against severe bacterial infections.
“Doctor, don’t be afraid because of my high cholesterol.” These were the words of a 36-year-old lawyer who visited me for the first time for a health examination. And indeed, his cholesterol was high, over 400 mg/dl.
“My father’s cholesterol was even higher,” he added. “But he lived happily until he died at age 79 from cancer. And his brother, who also had FH, died at age 83. None of them ever complained of any heart problems.” My “patient” is now 53, his brother is 56 and his cousin 61. All of them have extremely high cholesterol values, but none of them has any heart troubles, and none of them has ever taken cholesterol-lowering drugs.
So, if you happen to have FH, don’t be too anxious. Your chances of surviving are pretty good, even surviving to old age.
Scientific Steering Committee on behalf of the Simon Broome Register Group. Risk of fatal coronary heart disease in familial hypercholesterolaemia. British Medical Journal 303, 893-896, 1991; Sijbrands EJG and others. Mortality over two centuries in large pedigree with familial hypercholesterolaemia: family tree mortality study. British Medical Journal 322, 1019-1023, 2001.
From The Cholesterol Myths by Uffe Ravnvskov, MD, PhD, NewTrends Publishing, pp 64-65.
See also: Uffe Ravnskov, MD, PhD New cholesterol guidelines for converting healthy people into patients
Summary of latest data on antibiotic resistance in the European Union
Highlights on antibiotic resistance Antibiotic resistance is a major global public health problem and is, for a large part, driven by use of antibiotics. As a result, patients are suffering from infections caused by bacteria that are resistant to antibiotics, sometime to multiple antibiotics. Resistance in bacteria commonly responsible for infections such as Klebsiella pneumoniae and Escherichia coli has been increasing Europe wide for all Antibiotic classes under surveillance. Single and combined resistance to several antibiotics (multidrug resistance) are increasing in these bacteria. The percentage of carbapenem -Resistant lebsiella pneumonia is increasing in the European Union and these resistant bacteria are now spreading to several European countries . Transfer of patients across borders poses a clear risk for the transmission of carbapenem resistant bacteria, especially when patients are transferred from areas with high rates of such bacteria to healthcare facilities in another country or have received medical care abroad in areas with high rates of carbapenem resistant bacteria.Carbapenems are the major last line class of antibiotics to treat infections with multidrug resistant Gram negative bacteria such as K. pneumoniae and E. coli Resistance to carbapenems limits available options for treatment of infected patients to only few antibiotics which often are old antibiotics that were developed several decades ago and often have limitations and side effects . The occurrence of meticillin resistant Staphylococcus aureus (MRSA) shows a decrease in some European countries. However, one fourth of countries are still reporting that of all Staphylococcus aureus invasive infections, more than 25% are MRSA. Antibiotic resistance In the European Union The data presented in this section were collected by the European Antimicrobial Resistance Surveillance Network (EARS Net) which is coordinated by the European Centre for Disease Prevention and Control (ECDC). The maps presented in this summary show the occurrence of antibiotic resistance in selected bacteria causing invasive infections and are based on laboratory results reported by countries participating in EARS Net. 2 Klebsiella pneumoniae ( K. pneumoniae ) K. pneumoniae is one of the common causes of Gram negative urinary and respiratory tract infections. This micro organism can rapidly spread from the gastrointestinal tract of patients and via the hands of the hospital personnel to other patients, leading to nosocomial outbreaks. Importantly, patients with impaired immune defences are at higher risk to acquire these infections. The percentage of carbapenem resistant K. pneumoniae has increased dramatically in the European Union since 2009–In 2009, carbapenem resistance in K. pneumoniae was only established in Greece, although carbapenem resistance in invasive isolates of K. pneumoniae was also found in Italy and Cyprus [F2]. In 2010, an increasing trend of carbapenem –
resistant K. pneumonia e was observed for Austria, Cyprus, Hungary and Italy (Fig. 1). This increasing trend is a particularly worrying phenomenon since carbapenems are last line antibiotics for treatment of infections with multidrug Resistant Gram negative bacteria including those which produce an extended spectrum beta lactamase (ESBL). Treatment options for patients infected with carbapenem resistant K. pneumoniae or other carbapenem resistant bacteria are severely limited [F3]3 Figure 1 Klebsiella pneumoniae : percentage of invasive isolates resistant to carbapenems in 2009 (A) and in 2010 (B) (Data source: EARS Net) Percentage of carbapenem resistant K. pneumoniae in 2009 B:Percentage of carbapenem resistant K. pneumoniae in 2010
4 In addition to resistance to carbapenems, a high frequency of Multidrug resistant K. pneumoniae (resistance to third generation cephalosporins, fluoroquinolones and aminoglycosides ) is evident in Southern, Central and Eastern Europe (Fig.2). Figure 2: Kleb siella pneumoniae percentage of multidrug resistant K. pneumoniae (third generation cephalosporins, fluoroquinolones and aminoglycosides) in 2010 (Data source: EARS Net)
Escherichia coli ( E. coli ) E. coli is the most frequent cause of bacteraemia caused by Gram negative bacteria, as well as community and hospital – acquired urinary tract infections. It is also one of the most common foodborne pathogens worldwide. The occurrence of antibiotic resistance in E. coli continues to increase Europe wide for both multidrug resistance and for single antibiotics under surveillance. For some antibiotics ,the increase is evident even among countries already presenting relatively high levels of resistance. For fluoroquinolones, which are important antibiotics for treatment of E. coli infections, the resistance situation in Europe in 2010 is displayed in
6 Figure 3 Escherichia coli Percentage of invasive isolates with resistance to fluoroquinolones in 2009 and 2010 ( Data source EARS Net)
A:Percentage of E. coli resistant to fluoroquinolones in 2009 B
Percentage of E. coli resistant to fluoroquinolones in 2010 7 Meticillin –
resistant Staphylococcus aureus (MRSA ) MRSA is the most important cause of antibiotic – resistant healthcare -associated infections worldwide. Infections with MRSA may result in prolonged hospital stay and increased mortality rates[F4]. In 2010, seven countries ( Austria, Cyprus, Estonia, France, Greece, Ireland and the UK ) reported decreasing trends for MRSA. This brings hope that national efforts on infection control and containment of resistance may in some cases slow down the development of resistance. Nevertheless , MRSA remains a public health priority, since significantly increasing trend of MRSA was observed in four countries (Italy, Hungary, Germany and Slovenia ) and the proportion of MRSA remains above 25% in more than one fourth of countries (Fig. 4). 8
Figure 4 Staphylococcus aureus Percentage of invasive isolates resistant to meticillin (MRSA) in 2009 A) and in 2010 (B) (Data source EARS Net)
A: Percentage of MRSA in 2009
B: Percentage of MRSA in 2010
Recipe for Insulin Regulating—take Fenugreek and Ginger Powder—equal portions and either make a tea with this or fuse with Honey ( Unpasteurized) –when fused Utilize ¼ tsp of this twice a day or as needed—you will find this will reduce hunger as a direct result of insulin regulating and may find as well the benefits of the components of these herbs—Or brew a tea with this and drink throughout the day – may as well see GH response from this as well Hormonal Levels for regenerating the Body and muscle integrity –potential Testosterone Regulating as well—Estrogen Blocking—Immune Support
Flavonoids Flavone (apigenin, luteolin) glycosides including
orientin and vitexin, quercetin (flavonol).
Saponins 0.6–1.7%. Glycosides yielding steroidal sapogenins
diosgenin and yamogenin (major), with tigogenin, neotigogenin,
gitogenin, neogitogenin, smilagenin, sarsasapogenin, yuccagenin;(
1) fenugreekine, a sapogenin-peptide ester involving diosgenin
and yamogenin;(2) trigofoenosides A–G (furostanol
Other constituents Coumarin,(7) lipids (5–8%),(8) mucilaginous
fibre (50%),(8) vitamins (including nicotinic acid) and minerals
Proteins and amino acids Protein (23–25%) containing high
quantities of lysine and tryptophan. Free amino acids include 4-
hydroxyisoleucine (0.09%), histidine, lysine and arginine
Oleo-resin Gingerol homologues (major, about 33%) including
derivatives with a methyl side-chain,(4) shogaol homologues
(dehydration products of gingerols), zingerone (degradation
product of gingerols), 1-dehydrogingerdione,(5) 6-gingesulfonic
acid(3) and volatile oils.
Volatile oils 1–3%. Complex, predominately hydrocarbons. b-
Bisabolene and zingiberene (major); other sesquiterpenes include
zingiberol, zingiberenol, ar-curcumene, b-sesquiphellandrene, bsesquiphellandrol
(cis and trans); numerous monoterpene hydrocarbons,
alcohols and aldehydes (e.g. phellandrene, camphene,
geraniol, neral, linalool, d-nerol).
Proteins and amino acids
Amino acids (e.g. arginine, aspartic acid,
cysteine, glycine, isoleucine, leucine, serine, threonine and valine),
protein (about 9%), resins, diterpenes (galanolactone),(6) vitamins
(especially nicotinic acid (niacin) and vitamin A), minerals.(2)
The material contains not less than 4.5% of alcohol (90%)-
soluble extractive and not less than 10% of water-soluble
Eggs Prevent Heart Disease and Cancer
New Evidence That Egg White Protein May Help High Blood Pressure
Apr. 9, 2013 — NEW ORLEANS, April 9, 2013 — Scientists reported new evidence today that a component of egg whites –– already popular as a substitute for whole eggs among health-conscious consumers concerned about cholesterol in the yolk –– may have another beneficial effect in reducing blood pressure. Their study was part of the 245th National Meeting & Exposition of the American Chemical Society (ACS), the world’s largest scientific society, which continues here through Thursday.—“Our research suggests that there may be another reason to call it ‘the incredible, edible egg,’” said study leader Zhipeng Yu, Ph.D., of Jilin University. “We have evidence from the laboratory that a substance in egg white –– it’s a peptide, one of the building blocks of proteins –– reduces blood pressure about as much as a low dose of Captopril, a high-blood-pressure drug.”—Yu and colleagues, who are with Clemson University, used a peptide called RVPSL. Scientists previously discovered that the substance, like the family of medications that includes Captopril, Vasotec and Monopril, was an angiotensin-converting-enzyme (ACE) inhibitor. It has a powerful ability to inhibit or block the action of ACE, a substance produced in the body that raises blood pressure.—They set out to further document RVPSL’s effects, using laboratory rats that develop high blood pressure and are stand-ins for humans in such early research on hypertension. The results of feeding the substance were positive, showing that RVPSL did not have apparent toxic effects and lowered blood pressure by amounts comparable to low doses of Captopril.—“Our results support and enhance previous findings on this topic,” Yu said. “They were promising enough to move ahead with further research on the effects of the egg white peptide on human health.”—Yu noted that the research was done with a version of the peptide that was heated to almost 200 degrees Fahrenheit during preparation — less than the temperatures typically used to cook eggs. He cited evidence from other research, however, that egg whites may retain their beneficial effects on blood pressure after cooking.–One, for instance, published in the ACS’ Journal of Agricultural and Food Chemistry, showed that fried egg protein, cooked at high temperatures, actually showed greater ability to reduce blood pressure than eggs boiled at 212 degrees F.–Yu believes that egg white peptides, either in eggs or as a supplement, could become useful as an adjunct to high-blood-pressure medication. For now, he said people with high blood pressure should consult their health care provider before making any changes. –And he noted that findings about egg white and high blood pressure add to the emerging nutritional image of eggs. Once regarded as a food to avoid in a healthy diet, studies in recent years have concluded that many people can eat eggs without raising their blood cholesterol levels, benefiting from an inexpensive food low in calories and rich in protein, vitamins and other nutrients.—The American Chemical Society is a nonprofit organization chartered by the U.S. Congress. With more than 163,000 members, ACS is the world’s largest scientific society and a global leader in providing access to chemistry-related research through its multiple databases, peer-reviewed journals and scientific conferences. Its main offices are in Washington, D.C., and Columbus, Ohio.
Eggs’ Antioxidant Properties May Help Prevent Heart Disease and Cancer, Study
July 6, 2011 — One of nature’s most perfect foods may be even better for us than previously thought.—While eggs are well known to be an excellent source of proteins, lipids, vitamins and minerals, researchers at the University of Alberta recently discovered they also contain antioxidant properties, which helps in the prevention of cardiovascular disease and cancer. –Jianping Wu, Andreas Schieber and graduate students Chamila Nimalaratne and Daise Lopes-Lutz of the U of A Department of Agricultural Food and Nutritional Science examined egg yolks produced by hens fed typical diets of either primarily wheat or corn. They found the yolks contained two amino acids, tryptophan and tyrosine, which have high antioxidant properties. –After analyzing the properties, the researchers determined that two egg yolks in their raw state have almost twice as many antioxidant properties as an apple and about the same as half a serving (25 grams) of cranberries.—However, when the eggs were fried or boiled, antioxidant properties were reduced by about half, and a little more than half if the eggs were cooked in a microwave.[F5] -“It’s a big reduction but it still leaves eggs equal to apples in their antioxidant value,” said Wu.–The findings were published in the peer-reviewed journal Food Chemistry. –The discovery of these two amino acids, while important, may only signify the beginning of finding antioxidant properties in egg yolks, said Wu, an associate professor of agricultural, food and nutritional science.–“Ultimately, we’re trying to map antioxidants in egg yolks so we have to look at all of the properties in the yolks that could contain antioxidants, as well as how the eggs are ingested,” said Wu, adding that he and his team will examine the other type of antioxidant already known to be in eggs, carotenoids, the yellow pigment in egg yolk, as well as peptides. ( see list below )—In previous research, Wu found that egg proteins were converted by enzymes in the stomach and small intestines and produced peptides that act the same way as ACE inhibitors, prescriptions drugs that are used to lower high blood pressure.–That finding defied common wisdom and contradicted the public perception that eggs increased high blood pressure because of their high cholesterol content. Additional research by Wu suggests the peptides can be formulated to help prevent and treat hypertension.—Wu is convinced the peptides also have some antioxidant properties, which leads him to suggest that when he completes the next step in his research, the result will likely be that eggs have more antioxidant properties than we currently know.
Other Antioxidants as well as Fats crucial for regenerating
Eggs Contain these Substances note
(mg of Substance per 100 grams)
Alkaloids: Betaine 0.53
Amino Acids: Methionine Neurine *
Carotenoids: Xanthophylls: * Lutein Zeaxanthin
Lipids: 11,200 Arachidonic Acid Phosphatidylcholine 1,875
Cholesterol 371 -548 Docosahexaenoic Acid ( DHA)
Linoleic Acid Alpha-Linolenic Acid ( Omega 3)
Stearic Acid Sphingomyelin 10.74
Minerals: Calcium 52 Chromium* 16 mcg
Zinc* 1.5 Sodium 140
Potassium 140 Iron 2
Proteins: ** Avidin # Ovalbumin *
Ovomucoid * Ovoglobulin *
Conalbumin * Vitellin*
Vitellenin * Ovomucin #
Levitin Albumin #
Vitamins: Biotin* 25 mcg Choline * 251
Vitamin A 140 mcg Vitamin B5
Vitamin B1 0.09 Vitamin B2 0.47
Vitamin D 50 IU Vitamin E 1.6
Folic Acid (Yolks only) 25 mcg Vitamin B12 1.7 mcg
Vitamin K * Vitamin B3 3.7
* Egg Yolks only
# Egg Whites only
** The biological value of the Proteins in Egg Whites is 88
– The biological value of the Proteins in whole Eggs is 100.
[F1]And then you wonder why kids are spun and grown ups cannot think ir remember anything
[F2]The Mediterranean Region—so this would be what is recorded —the concern would be what is not recorded
[F3]In cases like this then you will utilize iodine –turpentine—essential oils –and minerals such as zinc –copper—salt –and a combinations of salt with other elements to insure penetration and removal—what is going on is that these pathogens are forming a biofilm polymer that will layer inside a body developing a protective shield –to eliminate a immune response while it feeds on the host utilizing the bodies own resources to further cascade the system and take over—a rewriting of the genetic code—which may be tied to direct infiltration through vaccines and Genetically modified foods which are loaded with viral –fungal and bacterial elements not normally seen in the balanced biology of botany—even the soil in which they are grown are being shown to have issues and cause a furthering of genetic distortion in the plants—further exasberating there damage internally—further causing the infections to breakdown the host
[F4]It is completely curable with either essential oils –iodine or turpentine—even botanicals in combination with minerals will remove this
[F5]Never use a Microwave–EVER
Show of the Month April 27 2013
Antibiotic Overload In feed
Caffeine and Exercise May Be Protective Against Skin Cancer Caused by Sun Exposure
Caffeine Shown As Effective At Reducing Exercise-Induced Asthma Symptoms As An Albuterol Inhaler
Immune-modifying and antimicrobial effects of Eucalyptus oil and simple inhalation devices.
Antibiotic Overload In feed
Did you know that each year more antibiotics are fed to food-animals in North Carolina than are given to all Americans? Thanks to this kind of misuse, antibiotic-resistant diseases now kill more Americans than HIV/AIDS.—On March 9, 2011, Rep. Slaughter re-introduced H.R. 965, The Preservation of Antibiotics for Medical Treatment Act (PAMTA), which is designed to end the routine use of antibiotics on healthy animals and curb the growing threat of superbugs.—PAMTA would preserve the effectiveness of medically important antibiotics by phasing out the use of these drugs in healthy food-producing animals, while allowing their use for treatment of sick animals. The legislation also requires the Food and Drug Administration (FDA) to apply the same tough standards to new applications for approval of animal antibiotics.
On February 23, 2011, Slaughter confirmed with the FDA an alarming statistic: 80 percent of all antibiotics used in the United States are used not on humans but on food-animals, most of which are perfectly healthy.—This kind of habitual use of antibiotics has been conclusively linked to the growing risk of antimicrobial-resistant infections in humans. A National Academy of Sciences report stated that, “a decrease in antimicrobial use in human medicine alone will have little effect on the current situation. Substantial efforts must be made to decrease inappropriate overuse in animals and agriculture as well.”—When our limited supply of antibiotics is used indiscriminately and without care, there are public health consequences. It is time to put a stop to big agribusinesses doling out pharmaceuticals to healthy animals just because it is better for their bottom line. Antibiotic use in food-animals must be limited to prevent the inadvertent creation of superbugs that are too powerful for our own medicine. In addition to introducing legislation, Slaughter has called upon the FDA and the United States Department of Agriculture (USDA) to take action to improve regulatory oversight[F1], surveillance, and monitoring of food-animal production and antibiotic resistance. Last year, the Governmental Accounting Agency (GAO) released a report that Rep. Slaughter requested in 2009, which found that federal agencies have made limited progress in combating the growing threat of antibiotic resistance.—On March 11, 2011, Slaughter led a letter to Secretary Sebelius at the Department of Health and Human Services to urge the formation of a national plan to combat antibiotic resistance. Since that letter was written, a national plan to combat antibiotic resistance has been issued.
Slaughter’s work continues to raise awareness of this important issue. On December 13, 2011, she hosted a briefing in which farmers and successful businesses extolled the benefits of tapping into the growing domestic and international demand for antibiotic-free meat.—Slaughter was joined by an impressive panel, including Steve Ells, CEO of Chipotle Mexican Grill, one of the nation’s fastest growing restaurant companies, Stephen McDonnell, CEO of award-winning Applegate Farms, and Paul Willis, President of Niman Ranch, a network of over 650 independent sustainable farms. To learn more about the briefing, click here.—–As businesses such as Applegate Farms, Niman Ranch, the Ozark Mountain Pork Cooperative, and Chipotle Mexican Grill, continue to pioneer affordable, antibiotic-free meat, Slaughter continues to push the federal government to address this looming public health threat.—-Earlier this year, Slaughter sent letters to over 60 fast food companies, producers, processors, and grocery store chains asking them to disclose their policies on antibiotic use in meat and poultry production. Very simply, consumers have a right to know what is in their food. The U.S. is facing a growing public health crisis in the form of antibiotic-resistant bacteria, and information about how companies may or may not be contributing to the problem should be available to consumers. To view Slaughter’s letter, click here.
Edible Parts: Inner bark.
Edible Uses: Condiment.
Inner bark – dried and ground into a powder and used in making bread[2, 66, 105, 177]. It is often mixed with oatmeal. A famine food, it is only used when all else fails. A vanillin flavouring is obtained as a by-product of other resins that are released from the pulpwood.
Antirheumatic; Antiseptic; Aromatherapy; Bach; Balsamic; Diuretic; Expectorant.
Utilizing Scent-or Vapour
The resinous materials
Scot’s pine has quite a wide range of medicinal uses, being valued especially for its antiseptic action and beneficial effect upon the respiratory system. It should not be used by people who are prone to allergic skin reactions whilst the essential oil should not be used internally unless under professional supervision. The turpentine obtained from the resin is antirheumatic, antiseptic, balsamic, diuretic, expectorant, rubefacient and vermifuge[4, 13, 46]. It is a valuable remedy in the treatment of kidney, bladder and rheumatic affections, and also in diseases of the mucous membranes and the treatment of respiratory complaints. Externally it is used in the form of liniment plasters and inhalers. The leaves and young shoots are antiseptic, diuretic and expectorant. They are harvested in the spring and dried for later use. They are used internally for their mildly antiseptic effect within the chest and are also used to treat rheumatism and arthritis. They can be added to the bath water for treating fatigue, nervous exhaustion, sleeplessness, skin irritations. They can also be used as an inhalant in the treatment of various chest complaints. The essential oil from the leaves is used in the treatment of asthma, bronchitis and other respiratory infections, and also for digestive disorders such as wind. An essential oil obtained from the seed has diuretic and respiratory-stimulant properties. The seeds are used in the treatment of bronchitis, tuberculosis and bladder infections. A decoction of the seeds can be applied externally to help suppress excessive vaginal discharge. The plant is used in Bach flower remedies – the keywords for prescribing it are ‘Self-reproach’, ‘Guilt feelings’ and ‘Despondency'. The essential oil is used in aromatherapy. Its keyword is ‘Invigorating'.
Dye; Essential; Fibre; Fuel; Herbicide; Lighting; Packing; Resin; Shelterbelt; Wood.
A tan or green dye is obtained from the needles. The needles contain a substance called terpene, this is released when rain washes over the needles and it has a negative effect on the germination of some plants, including wheat. A reddish yellow dye is obtained from the cones. This tree yields resin and turpentine[64, 66, 100, 171]. Oleo-resins are present in the tissues of all species of pines, but these are often not present in sufficient quantity to make their extraction economically worthwhile. The resins are obtained by tapping the trunk, or by destructive distillation of the wood[4, 64]. In general, trees from warmer areas of distribution give the higher yields. Turpentine consists of an average of 20% of the oleo-resin and is separated by distillation[4, 64]. Turpentine has a wide range of uses including as a solvent for waxes etc, for making varnish, medicinal etc. Rosin is the substance left after turpentine is removed. This is used by violinists on their bows and also in making sealing wax, varnish etc. Pitch can also be obtained from the resin and is used for waterproofing, as a wood preservative etc. An essential oil obtained from the leaves is used in perfumery and medicinally[46, 61]. A fibre from the inner bark is used to make ropes. The roots are very resinous and burn well. They can be used as a candle substitute. The leaves are used as a packing material. The fibrous material is stripped out of the leaves and is used to fill pillows, cushions and as a packing material. Trees are very wind resistant and quite fast growing. They can be planted as a shelterbelt, succeeding in maritime exposure[75, 200]. Wood – light, soft, not strong, elastic, durable, rich in resin. Used in construction, furniture, paper manufacture etc.[13, 46, 100]. A good fuel but it is somewhat smokey[6, 66, 115].
Edible Parts: Inner bark.
Edible Uses: Condiment; Gum; Tea.
Inner bark – cooked. It is usually dried, ground into a powder and then used as a thickening in soups etc or mixed with cereals when making bread[105, 177]. Fir bark is a delight to chew in winter or early spring, slightly mucilaginous and sweetish, better raw than cooked. Another report says that it is an emergency food and is only used when all else fails. An aromatic resinous pitch is found in blisters in the bark. When eaten raw it is delicious and chewy[101, 183]. Another report says that the balsam or pitch, in extreme emergency, forms a highly concentrated, though disagreeable, food. An oleoresin from the pitch is used as a flavouring in sweets, baked goods, ice cream and drinks. Tips of young shoots are used as a tea substitute[177, 183].
Plants For A Future can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.
Analgesic; Antiscorbutic; Antiseptic; Diuretic; Poultice; Stimulant; Tonic; VD.
Cleaner of Wounds
Patch to draw out infections
Energizing or increases Alertness or flow
And Vitality Support
The resin obtained from the balsam fir (see ‘Uses notes’ below) has been used throughout the world and is a very effective antiseptic and healing agent. It is used as a healing and analgesic protective covering for burns, bruises, wounds and sores[213, 222, 226]. It is also used to treat sore nipples and is said to be one of the best curatives for a sore throat. The buds, resin, and/or sap are used in folk remedies for treating cancers, corns, and warts. The resin is also antiscorbutic, diaphoretic, diuretic, stimulant and tonic[4, 171, 222]. It is used internally in propriety mixtures to treat coughs and diarrhoea, though taken in excess it is purgative. A warm liquid of the gummy sap was drunk as a treatment for gonorrhoea. A tea made from the leaves is antiscorbutic[4, 171]. It is used in the treatment of coughs, colds and fevers. The leaves and young shoots are best harvested in the spring and dried for later use. This plant was widely used medicinally by various North American Indian tribes. The resin was used as an antiseptic healing agent applied externally to wounds, sores, bites etc., it was used as an inhalant to treat headaches and was also taken internally to treat colds, sore throats and various other complaints.
Adhesive; Fibre; Gum; Kindling; Microscope; Repellent; Resin; Stuffing; Waterproofing; Wood.
The balsamic resin ‘Balm of Gilead'[11, 46] or ‘Canada Balsam’ according to other reports[64, 226, 238] is obtained during July and August from blisters in the bark or by cutting pockets in the wood. Another report says that it is a turpentine. The term Canada Balsam is a misnomer because balsams are supposed to contain benzoic and cinnamic acids, both absent from the Canada oleoresin. Turpentine is also a misnomer, implying that the oleoresin is entirely steam volatile. Actually it contains 70 – 80% resin, only 16 – 20% volatile oil. Canada Balsam yields 15 – 25% volatile oil, the resin being used for caulking and incense. It is used medicinally and in dentistry, also in the manufacture of glues, candles and as a cement for microscopes and slides – it has a high refractive index resembling that of glass[11, 46, 64, 82, 222, 226, 238]. The pitch has also been used as a waterproofing material for the seams of canoes. The average yield is about 8 – 10 oz per tree. The resin is also a fixative in soaps and perfumery[171, 238]. “Turpentine” is usually collected during July-August by breaking the turpentine blisters into small metal cans with sharp-pointed lids. Trees are then allowed to recuperate for 1 – 2 years before being harvested again. The leaves and young branches are used as a stuffing material for pillows etc – they impart a pleasant scent[46, 61, 257] and also repel moths. The leaves contain an average of 0.65% essential oil, though it can go up to 1.4% or even higher. One analysis of the essential oils reports 14.6% bornyl acetate, 36.1% b-pinene, 11.1% 3-carene, 11.1% limonene, 6.8% camphene, and 8.4% a-pinene. To harvest the oil, it would appear that the branches should be snipped off younger trees in early spring. Fifteen year old trees yield 70% more leaf oil than 110-year-old trees; oil yields are highest in January – March and September, they are lowest from April to August. A thread can be made from the roots. Wood – light, soft, coarse grained, not strong, not very durable. Weighs 24lb per cubic foot. Used mainly for pulp, it is not used much for lumber except in the manufacture of crates etc[46, 82, 226, 229]. The wood is commercially valuable for timber even though it is relatively soft, weak, and perishable. Balsam fir is used in the US for timber and plywood, and is the mainstay of the pulp wood industry in the Northeast. The wood, which is rich in pitch, burns well and can be used as a kindling
Edible Uses: Condiment.
An essential oil from the fresh or dried leaves is used as a flavouring in sweets, baked goods, ice cream etc[177, 183].
Plants For A Future can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.
Antibacterial; Antiperiodic; Antirheumatic; Antiseptic; Antispasmodic; Appetizer; Aromatherapy; Aromatic; Deodorant; Expectorant; Febrifuge;
Stops the flow of menses
Cleaner of Wounds
Stops spasms of muscles and nerves
Desire for food
Remedial use in aromatherapies
Scents Utilize for therapies
Free’s lungs from mucous build up
Sugar regulator for low sugar
Eucalyptus leaves are a traditional Aboriginal herbal remedy. The essential oil found in the leaves is a powerful antiseptic and is used all over the world for relieving coughs and colds, sore throats and other infections. The essential oil is a common ingredient in many over-the-counter cold remedies. The adult leaves, without their petioles, are antiperiodic, antiseptic, aromatic, deodorant, expectorant, febrifuge, hypoglycaemic and stimulant[4, 7, 21, 46]. The leaves, and the essential oil they contain, are antiseptic, antispasmodic, expectorant, febrifuge and stimulant. Extracts of the leaves have antibacterial activity. The essential oil obtained from various species of eucalyptus is a very powerful antiseptic, especially when it is old, because ozone is formed in it on exposure to air. It has a decided disinfectant action, destroying the lower forms of life. The oil can be used externally, applied to cuts, skin infections etc, it can also be inhaled for treating blocked nasal passages, it can be gargled for sore throat and can also be taken internally for a wide range of complaints. Some caution is advised, however, because like all essential oils, it can have a deleterious effect on the body in larger doses. The oil from this species has a somewhat disagreeable odour and so it is no longer used so frequently for medicinal purposes, other members of the genus being used instead. An oleo- resin is exuded from the tree. It can also be obtained from the tree by making incisions in the trunk[4, 152]. This resin contains tannin and is powerfully astringent, it is used internally in the treatment of diarrhoea and bladder inflammation[4, 152, 238], externally it is applied to cuts etc[4, 152]. The essential oil is used in aromatherapy. Its keyword is ‘Respiratory system'.
Cleanser; Deodorant; Dye; Essential; Fuel; Repellent; Wood.
The leaves and the essential oil in them are used as an insect repellent[14, 152, 174, 240]. The trees can also be planted in wet areas where mosquitoes abound. The ground will be dried out by the trees, making it unsuitable for the mosquitoes to breed. A decoction of the leaves is used for repelling insects and vermin. Africans use finely powdered bark as an insect dust. An essential oil is obtained from the leaves[46, 61, 156]. It is used in perfumery and in medicines. The yield is about 0.9% by steam distillation. The essential oil is also in spot removers for cleaning off oil and grease. Yields of 40 to 45 kilos of oil per hectare have been reported. A yellow/brown dye is obtained from the young leaves. It does not require a mordant. Grey and green dyes are obtained from the young shoots. A dark green dye is obtained from the young bark. Wood – heavy[46, 61], (or light according to another report), durable, fire resistant. An important timber species, it is used for various purposes such as carpentry, construction, fences, piles, platforms, plywood, poles, sheds, tool handles and veneer[238, 269]. The oil-rich wood is resistant to termites. This is one of the best eucalypts for pulp production for making paper[152, 269].
Caffeine and Exercise May Be Protective Against Skin Cancer Caused by Sun Exposure– Study Suggests
Apr. 3, 2012 — The combined effects of exercise plus caffeine consumption may be able to ward off skin cancer and also prevent inflammation related to other obesity-linked cancers.—“We found that this combination treatment can decrease sunlight-caused skin cancer formation in a mouse model,” said Yao-Ping Lu, Ph.D., associate research professor of chemical biology and director of skin cancer prevention at the Rutgers Ernest Mario School of Pharmacy in Piscataway, N.J. He presented these findings at the AACR Annual Meeting 2012, held in Chicago March 31 — April 4.–“I believe we may extrapolate these findings to humans and anticipate that we would benefit from these combination treatments as well,” Lu added.—The researchers evaluated the effects of caffeine and exercise on mice at high risk for developing skin cancer. Results showed that mice that took a dose of caffeine and exercised with a running wheel experienced 62 percent fewer skin tumors. The volume of tumors also decreased by 85 percent compared with the mice that did not consume caffeine or exercise.—-Positive effects were found with either caffeine or exercise alone, but to a lesser extent. Researchers observed a 27 percent reduction in tumors in caffeine-only mice and a 61 percent reduction in tumor size. In the exercise-only mice, researchers found that tumor activity decreased by 35 percent and tumor volume decreased by 70 percent.—The researchers also found that exercise and caffeine reduced weight and inflammation. They fed mice a high-fat diet of omega-6 fatty acid-rich foods and measured the volume of the parametrial fat pad (the largest fat pad in a mouse) after two weeks of exercise and/or caffeine treatment.—-Mice that had caffeine and exercised had a fat pad weight decrease of 63 percent. Caffeine-only mice had a 30 percent decrease, and exercise-only mice had a 56 percent decrease. Development and size of cancer decreased as well. The link, Lu believes, is inflammation, which dropped as much as 92 percent in mice that exercised and consumed caffeine.—This research was funded by the National Institutes of Health.- Story Source-The above story is reprinted from materials provided by American Association for Cancer Research (AACR).
Caffeine Shown As Effective At Reducing Exercise-Induced Asthma Symptoms As An Albuterol Inhaler
June 1, 2009 — An Indiana University study found that the ingestion of caffeine within an hour of exercise can reduce the symptoms of exercise induced asthma (EIA).
A large dose — 9 milligrams of caffeine per kilogram of body weight [F2]– was as effective as the use of an albuterol inhaler, which is commonly used to treat or prevent exercise-induced asthma. Smaller amounts of caffeine — for example, 3 and 6 milligrams of caffeine per kilogram of body weight — also reduced the wheezing, coughing and other symptoms of EIA.—Timothy Mickleborough, an associate professor in the Department of Kinesiology and co-investigator of the study, said no additional benefit was found when caffeine was combined with an albuterol inhaler.—Mickleborough and his research colleagues have been investigating the efficacy of a number of nutritional factors, and his research to date has shown that a diet high in fish oil and antioxidants and low in salt has the potential to reduce the severity of EIA and perhaps reduce the reliance on pharmacotherapy. This is especially important since prolonged use of daily medications can result in reduced effectiveness, and there is growing concern about the potential side effects of inhaled corticosteroid use.–*Background: The caffeine study involved 10 asthmatic subjects who also had EIA, in a randomized, double-blind double-dummy crossover study. They ingested 3, 6, or 9 milligrams of caffeine per kilogram of body weight or a placebo an hour before running on a treadmill. Pulmonary function tests were conducted 15 minutes before the a eucapnic voluntary hyperpnea challenge (a surrogate for an exercise challenge) and then again 1, 5, 10, 15 and 30 minutes afterward.–For someone weighing 150 pounds, 3 to 9 milligrams of caffeine per kilogram of body weight equals around 205 to 610 milligrams of caffeine. Earlier research has found that caffeine can reduce the symptoms of EIA. This study extends this earlier work and is the first to examine any synergistic effect of caffeine use along with an albuterol inhaler.—The study, “Comparative and Synergistic Effects of Caffeine and Albuterol on The Severity of Exercise-Induced Bronchoconstriction,” was presented at the American College of Sports Medicine conference during the Respiratory Session on May 29. Co-authors include lead author Timothy A. VanHaitsma, now at the University of Utah; Martin R. Lindley, Loughborough University, United Kingdom; and David Koceja and Joel Stager, IU’s Department of Kinesiology
Immune-modifying and antimicrobial effects of Eucalyptus oil and simple inhalation devices.
Sadlon AE, Lamson DW.
Eucalyptus oil (EO) and its major component, 1,8-cineole, have antimicrobial effects against many bacteria, including Mycobacterium tuberculosis and methicillin-resistant Staphylococcus aureus (MRSA), viruses, and fungi (including Candida). Surprisingly for an antimicrobial substance, there are also immune-stimulatory, anti-inflammatory, antioxidant, analgesic, and spasmolytic effects. Of the white blood cells, monocytes and macrophages are most affected, especially with increased phagocytic activity. Application by either vapor inhalation or oral route provides benefit for both purulent and non-purulent respiratory problems, such as bronchitis, asthma, and chronic obstructive pulmonary disease (COPD). There is a long history of folk usage with a good safety record. More recently, the biochemical details behind these effects have been clarified. Although other plant oils may be more microbiologically active, the safety of moderate doses of EO and its broad-spectrum antimicrobial action make it an attractive alternative to pharmaceuticals. EO has also been shown to offset the myelotoxicity of one chemotherapy agent. Whether this is a general attribute that does not decrease the benefit of chemotherapy remains to be determined. This article also provides instruction on how to assemble inexpensive devices for vapor inhalation.
Self-Constructed Pocket Inhaler
A suitable-sized piece of stockinet or loosely woven fabric can be rolled on a small wooden skewer and twisted into a two-dram glass vial (Patient Handout 2). The fabric easily accepts 70 drops of EO without saturation. Carrying the vial inhaler in a pocket close to the body furnishes heat for satisfactory vaporization. Hold the vial close to or touching the lips or nostrils during inhalation. The amount of vapor intake is regulated by the distance from the vial and the speed of breathing – the closer and slower, the more vapor. Best results seem to be obtained by alternating mouth and nasal inhalation during sessions of 3-5 minutes several times daily
[F1]This is a Joke—asking the wolf to watch the henhouse —there will be no protection at all here on this issue and nothing will be resolved—The FDA has constantly refused to obey congress or any other gov’t in the act of prevention ofr removal of there influence and usually has become more defiant in there quest to control the very fabric of life —and the USDA does absolutely nothing these days to enforce safety in fods and food imports nor do they do anything in regard to Chemtrails or Flouridation of water
[F2]9MG’S/1KG (2.2 LB)
TAKE YOUR weight and divide by 2.2 to get a Kg weight—example 170/2.2= 77kgs approx—then multiply by 9= 693 mgs or 3 mgs /KG— so take as a example 170/2.2===77 X 3 =231mgs
June 22, 2021 at 09:00 #2662
Poland Becomes The 8th EU Nation To Ban Monsanto Maize
Poland has become the eighth EU member state to ban the cultivation of genetically modified (GM) crops. Seven other EU member states have already imposed bans on the cultivation of GM crops approved by the European Food Safety Authority (EFSA) as safe: Austria, France, Germany, Hungary, Luxembourg, Greece and Bulgaria.
The Polish Agriculture Ministry expressed concerns that GM crops may cross-pollinate with non-GM crops and Monsanto’s MON810 maize pollen may find its way into honey. The ministry also pointed to that there exists no scientific assessments confirming that GM crops are safe for the environment and people.
In a statement, Greenpeace Poland’s ‘Stop GMO’ campaign coordinator Joanna Mis said, “The government has delivered on its promise,” and, “We are happy that Poland has joined the club of countries that ban the cultivation of GM crops. However, this is not the end of our campaign. We have to make sure that the European Commission does not manage to lift the ban […]. We also have to make sure that crops will be effectively controlled and the ban on cultivation observed.”
A panel of experts appointed by a Supreme Court has recently recommended a 10-year moratorium on field trials of all genetically modified (GM) food and termination of all ongoing trials of transgenic crops in India. See: http://www.examiner.com/article/a-10-year-cap-on-genetically-modified-foods-recommended-by-supreme-court-panel
The United States, Canada, China, UK, Australia, Mexico, and most of South America, Asia and Africa have no formal GMO-free platforms and their use is typically unrestricted and widespread.
Most Nations in the World Have No GMO-Free Platform To Protect Their Citizens
Unless you live in the dozen or so nations in the world who have declared GMO (genetically modified organism) bans, then you’re likely eating GMO. Eating organic is your best chance of avoiding GM foods, but it’s almost impossible to avoid them entirely, especially if you’re living in a country that doesn’t restrict their cultivation, import or export. The United States, Canada, China, UK, Australia, Mexico, and most of South America, Asia and Africa have no formal GMO-free platforms and their use is typically unrestricted and widespread.
Nations with no formal GMO-Free platform (in red). Nations with laws restricting GMO in most areas (in yellow), and nations who are either GMO-free or have declared national bans (green).
Click for larger imagehttp://preventdisease.com/images/gmo_world_map_large.jpg
The image above is a shocking reminder of how we have allowed biotech industries to take over our planet. It is my dream to one day see this image turn from the majority red to green. Countries represented in red have no enforced national laws banning GMOs, their import, export or cultivation. Most of these nations have a very large percentage of their citizens eating GM foods.
If you’re new to the dangers of GM foods, here are the 7 biggest reasons to avoid them.
The United States is the leader in GM cultivation and now grows mostly GM varieties of corn, canola and soy. Hawaii now grows GM papayas. Approvals have also been given for GM alfalfa, zucchinis, beet sugar and tomato varieties, though not all are currently being grown. In 2010, the US planted 66.8 million hectares of soybean, maize, cotton, canola, squash, papaya, alfalfa and sugarbeet. The largest share of the GMO crops planted globally are owned by Monsanto.
More than 30 of Monsanto’s directors, CEOs, VPs, board members, managers, scientists, attorneys and consultants also hold Federal Positions within the US government.
Canada has widespread GM crop usage. All Canadian canola is GM, as is a large portion of the country’s soy and corn. Prince Edward Island tried to pass a ban on GMO cultivation but failed, and GM crops in the region are currently increasing.
Despite the fact that 233 consumer and farmer groups in 26 countries have joined the “Definitive Global Rejection of GM Wheat”, Canadian MPs voted to reject stronger export rules for crops of genetically modified organisms (GMOs).
Even though GMO continue to face strong resistance in South America, it is widespread. One third of the 134 million hectares of Genetically Modified Organisms (GMO) planted globally in 2009, were in South America. Brazil and Argentina are the main producers, with 21.4 and 21.3 million hectares respectively. Of all the countries in the world that are planting genetically modified crops, seven are in South America. They include Argentina, Bolivia, Brazil, Chile, Columbia, Paraguay and Uruguay. There are also no national restrictions in Guyana, Suriname and French Guiana to the north of the continent. Between 2008 and 2009, world production of GM crops increased eight percent, while in Brazil it rose 35 percent.
Peru, Ecuador and Venezuela have all declared national bans on GM foods. Peru officially passed a law banning genetically modified ingredients anywhere within the country for a full decade before coming up for another review. In 2008 Ecuador declared the country GMO free and will limit its biotechnology. In 2006, Venezuela banned genetically engineered crops thanks to President Hugo Chavez Frias.
In January 2010, Slow Food Tehuacan Mixteca Popoloca convivium launched a campaign to protect traditional varieties of maize after the Mexican government gave the go ahead for the first legal plantings of GM corn following a decade-long battle. The convivium is concerned that modified genes could spread and contaminate genetically valuable native varieties and is working to educate family and farmer organizations about the richness of their country’s biodiversity, encouraging the Mexican community to be proud of their cultural heritage and to work for its revitalization. Mexico does not have any GMO-free zones.
Europe is quickly becoming the most progressive continent in the world to oppose GM foods. France, Italy, Switzerland, Hungary, Bosnia, Serbia, Croatia, Latvia and Albania have all declared many regions to be GMO-free. France made an important step in the no-GMO movement by specifically defining exactly what “GMO-free” means when it comes to food labeling. Spain and Portugal are slowing advancing but they have a long way to go before declaring most of their regions GMO-free. Britain officially supports GM crops and has trials of GMOs like potatoes planted. Austria, Greece and Poland are now completely GMO-free zones thanks to public and government support.
Egypt and Madagascar are the only two countries in Africa to ban GMO. Any agricultural imports to Egypt must have a certificate from the country of origin that the product is not genetically modified and the rule will also apply to Egyptian exports. Madagascar banned growing or importing GMO foods due to concerns over the effect on human health and environment. In Algeria both the planting and distribution of GMO foods is illegal although import laws are lax. Other countries such as Kenya, Lesotho, Ethiopia, Angola, Malawai, Mozambique and Zimbabwe have some limited restrictions in place to ban GMO imports but with many exceptions and critics claim most of the laws are not enforced.
Russia remains GMO-free. According to the official information there is no growing of GMOs in Russia for commercial purposes. So far the Federal Environmental Assessment Commission has not adopted any commercialized GM varieties for agricultural use.
India has widespread GM cotton use. The widespread planting of Monsanto’s GM cotton has led to tragedy throughout India. The Indian government even banned conventional seeds from many government seed banks in an attempt to please Monsanto (in return, the country was given International Monetary Fund loans to help its economy) and slow the nation’s poverty rates.[F1] An estimated 1,000 farmers commit suicide each month in the country as a result of the crop failure and debt caused by planting the GM seeds. Farmers were convinced to spend what was often 1,000 times the cost of conventional seed on the “magic seeds” after listening to Monsanto’s promises of increased yields and resistance to pests. Despite the promises, the crops were often destroyed by bollworms. In addition, the farmers weren’t warned that the crops would require twice as much water as conventional cotton, leading to many crops drying up and dying. The “terminator” seeds also must be purchased again every year. For farmers used to saving seed from year to year, this was often a final financial blow that led to insurmountable debt.
With the exception of Thailand and Sri Lanka who both have GMO-free zones, all of Asia remains resistant to adopting laws to restrict imports, exports and cultivation of GMO.
During the meeting of Nordic countries at Terra Madre 2010, delegates from Sapmi, Sweden, Finland, Denmark, Norway, Faroe Islands, Greenland and Iceland discussed a statement that would describe their united position on GMOs. The result was a declaration against GMOs that the convivium leaders, members and food communities could use to lobby against the introduction of these crops, presenting it to their governments and others. Iceland has declared two GMO-free regions in the country.
AUSTRALIA and NEW ZEALAND
All genetically modified foods intended for sale in Australia and New Zealand must undergo a safety evaluation by Food Standards Australia New Zealand, however there are no national GMO bans in either country. However there are regional bans. Tasmania’s ban on the release of GM organisms to the environment will continue until 2014. South Australia has reaffirmed their commitment to ban all commercial GM crops until 2014.
The status of GM crops is constantly changing, both in the United States and around the world. Public outcry is rising against these largely untested foods and crops. The industry claims of “super yields” and an end to poverty and famine have proven to be dangerously inaccurate. Now, more than ever, is the time when our voices (and purchases) can make a real difference. It’s time to support labeling initiatives which may kickstart global campaigns to slowly change our red planet to green and ban GM foods once and for all.
Natasha Longo has a master’s degree in nutrition and is a certified fitness and nutritional counselor. She has consulted on public health policy and procurement in Canada, Australia, Spain, Ireland, England and Germany.
Henry Kissinger- “If You Can’t Hear the Drums of War You Must Be Deaf”
NEW YORK – USA – In a remarkable admission by former Nixon era Secretary of State, Henry Kissinger, reveals what is happening at the moment in the world and particularly the Middle East. Speaking from his luxurious Manhattan apartment, the elder statesman, who will be 89 in May, is all too forward with his analysis of the current situation in the world forum of Geo-politics and economics.“The United States is bating China and Russia, and the final nail in the coffin will be Iran, which is, of course, the main target of Israel. We have allowed China to increase their military strength and Russia to recover from Sovietization, to give them a false sense of bravado, this will create an all together faster demise for them[F2]. We’re like the sharp shooter daring the noob to pick up the gun, and when they try, it’s bang bang. The coming war will will be so severe that only one superpower can win[F3], and that’s us folks. This is why the EU is in such a hurry to form a complete superstate because they know what is coming, and to survive, Europe will have to be one whole cohesive state. Their urgency tells me that they know full well that the big showdown is upon us. O how I have dreamed of this delightful moment.” “Control oil and you control nations; control food and you control the people.” Mr Kissinger then added: “If you are an ordinary person, then you can prepare yourself for war by moving to the countryside and building a farm, but you must take guns with you, as the hordes of starving will be roaming. Also, even though the elite will have their safe havens and specialist shelters, they must be just as careful during the war as the ordinary civilians, because their shelters can still be compromised.” After pausing for a few minutes to collect his thoughts, Mr Kissinger, carried on: “We told the military that we would have to take over seven Middle Eastern countries for their resources and they have nearly completed their job. We all know what I think of the military, but I have to say they have obeyed orders superfluously this time. It is just that last stepping stone, i.e. Iran which will really tip the balance. How long can China and Russia stand by and watch America clean up? The great Russian bear and Chinese sickle will be roused from their slumber and this is when Israel will have to fight with all its might and weapons to kill as many Arabs as it can. Hopefully if all goes well, half the Middle East will be Israeli. Our young have been trained well for the last decade or so on combat console games, it was interesting to see the new Call of Duty Modern Warfare 3 game, which mirrors exactly what is to come in the near future with its predictive programming. Our young, in the US and West, are prepared because they have been programmed to be good soldiers, cannon fodder, and when they will be ordered to go out into the streets and fight those crazy Chins and Russkies, they will obey their orders. Out of the ashes we shall build a new society, there will only be one superpower left, and that one will be the global government that wins. Don’t forget, the United States, has the best weapons, we have stuff that no other nation has, and we will introduce those weapons to the world when the time is right.” End of interview. Our reporter is ushered out of the room by Kissinger’s minder.
Why Do Governments Recommend This Toxic Food Today When They Didn’t A Decade Ago?
If we analyze the food guide and government advice on nutrition over a decade ago and compare those advisements to what is recommended today, there is one big difference–one specific food crept up onto the radar of public health officials as if it had some kind of miraculous nutritional benefit for the public. The problem is, 80 percent of this food is genetically modified, contains toxic phytochemicals and is linked to digestive distress, immune system breakdown, allergies, ADD and ADHD, higher risk of heart disease and cancer, malnutrition, and loss of libido. Yet, governments seem to think that’s not a problem. You’ve probably already figured out that the food is soy. I’ll get to how deadly soy is shortly, but first let’s backtrack to the year 2000 and analyze the food guides of two countries, namely Canada and The United States. The waybackmachine is a beautiful tool that can show us exactly what a website looked like in the past. So if we plug in both the USDA and Health Canada websites in the year 2000 at about the same period, we can see exactly how each publicly funded message translated to each respective food guide or pyramid.
In the Year 2000
On the Health Canada website, there was absolutely no mention of soy at all. Under milk products, the main message was to choose lower-fat milk products more often. Most people had no idea back then how toxic pasteurized milk was, so it was heavily consumed, much more than it is today. There are currently huge debates throughout the internet as to why humans are drinking milk at all.———-On the USDA website on either the Milk, Yogurt & Cheese page or the main page illustrating the Food Guide Pyramid, there is again no mention of soy. The recommendation was also low dairy.
In the Year 2013
Today, Health Canada promotes fortified soy beverages on their website for those who don’t drink milk. So we go out of the frying pan and into the fire. We go from the recommendation of a dead liquid, namely pasteurized milk to a beverage that may be even more harmful to public health.—“Have milk or fortified soy beverages by the glass or use them in recipes.” “Use milk or fortified soy beverages when preparing scrambled eggs, hot cereal, casseroles and soups.”–“Create smoothies by blending lower fat milk or fortified soy beverage with a combination of fresh or frozen fruits.”–“Try a latte made with low fat milk or fortified soy beverage.” “Use milk or fortified soy beverages to replace some or all of the water when reconstituting canned tomato or cream soups.”
The USDA’s Center for Nutrition Policy and Promotion (CNPP), kicked their message into high gear in 2002 when they started heavily promoting soy across the United States. Their key message is still to switch to fat-free or low-fat milk, however consume calcium-fortified soy milk is a main heading. –“For those who are lactose intolerant… include lactose-reduced or lactose-free milk, yogurt, and cheese, and calcium-fortified soymilk (soy beverage).” Under Tips For Vegetarians –“Sources of protein for vegetarians and vegans include beans, nuts, nut butters, peas, and soy products (tofu, tempeh, veggie burgers).” “Sources of calcium for vegetarians and vegans include calcium-fortified soymilk”—“Calcium-fortified soymilk provides calcium in amounts similar to milk. It is usually low in fat and does not contain cholesterol.” “For breakfast, try soy-based sausage patties or links.”
“try veggie burgers, soy hot dogs, marinated tofu or tempeh, and veggie kabobs.” The site is littered with soy recommendations not only for vegetarians, but also in the promotion of protein foods.——How did this happen? When soy industry lobbyists get together and decide to change the framework of nutrition for the masses, it happens. It’s really that simple.–It’s not only soy. If you care to investigate further, you’ll also notice how three of the most toxic genetically modified oils in the world, canola, corn and soyabean—oil are heavily promoted today on both the Health Canada website and the CNPP website (on behalf of the USDA), and neither agency had those recommendations in 2000.
The USDA had absolutely no mention of any of these oils in 2000.
Health Canada also has no mention of these oils in 2000.
How Deadly is Soy
With Monsanto’s patented genes being inserted into roughly 95 percent of all soybeans and 80 percent of all corn grown in the U.S., the company used its wide reach to control the ability of new biotech firms to get wide distribution for their products, according to a review of several Monsanto licensing agreements and dozens of interviews with seed industry participants, agriculture and legal experts.—-Soy protein is not an effective alternative to any other protein. It is high in allergens (some 28 different proteins present in soy have been found to bind to IgE antibodies). It’s also worth noting that the more soy protein you eat, the more likely you are to develop allergies to it — and the more severe those allergies are likely to become.—As Dr. Spreen has pointed out, phytates in unfermented soy products actually obstruct absorption of protein and four key minerals: calcium, magnesium, iron, and zinc. Even so, the public’s perception of soy as health food got a boost from the FDA with a rule that permits soy beverages, soy-based cheese substitutes, and soy-based butter substitutes to be fortified with vitamin D.[F4] —In their natural form, soybeans contain phytochemicals with toxic effects on the human body. The three major anti-nutrients are phytates, enzyme inhibitors and goitrogens. —These anti-nutrients are the way nature protects the soybean plant so that it can live long enough to effectively reproduce. They function as the immune system of the plant, offering protection from the radiation of the sun, and from invasion by bacteria, viruses, or fungi. They make the soybean plant unappetizing to foraging animals. All plants have some anti-nutrient properties, but the soybean plant is especially rich in these chemicals. If they are not removed by extensive preparation such as fermentation or soaking, soybeans are one of the worst foods a person can eat.—The most common soy (99%) sold at major grocery retailers in soy milks and processed foods is unfermented soy. It is[F5] deadly. Unfermented soy has been linked to digestive distress, immune system breakdown, PMS, endometriosis, reproductive problems for men and women, allergies, ADD and ADHD, higher risk of heart disease and cancer, malnutrition, and loss of libido. The dangers of soy for men are a result of the high levels of the female hormone estrogen that soy and soy-based products contain. Primarily, soy affects the quality and concentration of a male’s sperm, especially if taken in large quantities or if the subject was exposed to high levels in the womb. A study at Harvard University showed that there was a definite correlation between men with low sperm counts and a high intake of soy foods. The study revealed that the average sperm concentration of 80 to 120 million per millimeter of an adult male was more than halved when soy formed part of the diet. The case is more compelling in the study of obese males whose sperm levels are even lower owing to the estrogen making properties of fat tissue.—-When food is eaten, digestive enzymes such as amylase lipase and protease are secreted into the digestive tract to help break it down and free nutrients for assimilation into the body. The high content of enzyme inhibitors in unfermented soybeans interferes with this process and makes carbohydrates and proteins from soybeans impossible to completely digest.[F6] When foods are not completely digested because of enzyme inhibitors, bacteria in the large intestine try to do the job, and this can cause discomfort, bloating, and embarrassment. Anyone with naturally low levels of digestive enzymes such as elderly people would suffer the most from the enzyme inhibiting action of soy.—-Groups most at risk of experiencing negative effects from the anti-nutrient properties of soy are infants taking soy baby formula, vegetarians eating a high soy diet, and mid-life women going heavy on the soy foods thinking they will help with symptoms of menopause.—Soybeans have a high content of goitrogens, substances that can block the production of thyroid hormone as well as cause goiter formation. Low thyroid activity plagues women in America, particularly middle-aged women. Thyroid hormone stokes the cellular furnaces, known as mitochondria. When thyroid production is low, energy levels as well as body heat are also low. Low thyroid level is what makes old people move so slowly and seem like every action is a huge chore. Low thyroid means the action of the heart is reduced, resulting in lack of oxygen to the cells, a prime condition for cancer.—Genistein, an isoflavone found in soybeans, can also block thyroid production. Phytate can accentuate these effects because it binds up zinc and copper, leaving little of these important minerals available to make thyroid hormone.—People filling up their shopping carts with raw or cooked soybeans, soy milk, and other non-fermented soybean products do not realize that the isoflavones they contain will not be available to their bodies. Most of the isoflavones in soy products are bound to carbohydrate molecules called glucosides. In this form genistein is actually called genistin. It is fermentation that transforms genistin into genistein. Many products in the U.S. do not distinguish between genistin and genistein on their labels. Even with fermented soy foods, a little goes a long way. The nutrients found in miso, tempeh, and natto can be beneficial in the moderate amounts found in the typical Asian diet, but have the potential to do harm in higher amounts. In China and Japan, about an ounce of fermented soy food is eaten on a daily basis.[F7]—-
A study in an issue of Indian Journal of Medical Research suggests that eating soybean oil may boost cancer risk compared to eating a type of butter called cow ghee, a type of butter used in South Asian cuisine.[F8]—-
Soy Lecithin has been lingering around our food supply for over a century. It is an ingredient in literally hundreds of processed foods, and also sold as an over the counter health food supplement. Scientists claim it benefits our cardiovascular health, metabolism, memory, cognitive function, liver function, and even physical and athletic performance. However, most people don’t realize what soy lecithin actually is, and why the dangers of ingesting this additive far exceed its benefits.—Soybean lecithin comes from sludge left after crude soy oil goes through a “degumming” process. It is a waste product containing solvents and pesticides and has a consistency ranging from a gummy fluid to a plastic solid. Before being bleached to a more appealing light yellow, the color of lecithin ranges from a dirty tan to reddish brown. The hexane extraction process commonly used in soybean oil manufacture today yields less lecithin than the older ethanol-benzol process, but produces a more marketable lecithin with better color, reduced odor and less bitter flavor.—In theory, lecithin manufacture eliminates all soy proteins, making it hypoallergenic. In reality, minute amounts of soy protein always remain in lecithin as well as in soy oil. Three components of soy protein have been identified in soy lecithin, including the Kunitz trypsin inhibitor, which has a track record of triggering severe allergic reactions even in the most minuscule quantities. The presence of lecithin in so many food and cosmetic products poses a special danger for people with soy allergies.—If you eat soy in any form, you are risking your immediate and long-term health. Check your labels, check your ingredients and most of all stay away from anything that is heavily advertised with soy as being a health food. —If you haven’t already figured it out, your government in not your ally when it comes to your health, so do as much research as possible, and cross reference every detail that comes out of public health education.
A Primer on Propane for Prepping and Survival
INTEGRATING PROPANE INTO YOUR PREPPING STRATEGY – PART ONE
This article covers the basics of propane as an important prepping energy resource. The subjects include safe propane handling, storage, assembling a stockpile of bulk tanks for long term storage at the lowest possible cost and refilling the smaller one pound canisters that are commonly used with portable camping gear, for about 1/5 the cost of new. We’ll also examine a variety of entry-level propane appliances and their suitability in a survival scenario.
For convenience, value, air quality and long term storage stability, nothing beats propane.—Firewood is cheaper, if you have access to it and you don’t count the value of your time. But, when you’re burning all day and every day, it isn’t all that convenient if you have to go out and cut/gather it yourself.–If you’re short on survival labor, which is a fair probability, having the option to cook with propane for an extended time, at least until things settle out, will let you channel that considerable fuel-wood time and energy into other important tasks, such as hunting and growing food, or warding off predators. Also, the ease of using propane allows the delegation of cooking etc. to a lesser-able member of your party, allowing all to contribute to the general welfare.
A Hedge Against Inflation
From an investment standpoint, the prices of both propane and the hardware/appliances that use it are directly tied to monetary price inflation – the “hidden tax” that steadily gnaws away at the purchasing power of your hard-won savings. Inflation is currently being deliberately manipulated to keep it low for now, but some price categories, like food, are still skyrocketing, with wages not keeping up. And, the economic stage is strongly set for hyper-inflation in the not-too-distant future.—So, one of the best investments in pre-inflationary and inflationary times (aka hedges against inflation) is hard goods and consumables, like food, that you’ll be using anyway and that will surely cost even more in the future. In other words, to combat inflation, the best place to store your surplus wealth is in tangible stuff, not pieces of paper, or electrons. For sure, no matter how the future shakes out, the retail prices for propane and propane hardware won’t be getting any cheaper, and they probably will go up by a lot.—Properly stored, both fuel and hardware will last indefinitely without degrading, ready to use on a moment’s notice. For all intents and purposes, unlike food, there’s no limit to its shelf life. –So, you’ll save money in the long run, anyway, by stockpiling consumables. But, if the grid goes down, there won’t be any more propane available to buy at all and the value of your investment will go way up. So, the larger your stockpile and the smaller the appliances that use it, which use less fuel, the longer it will last you–If you play your cards right, you can stockpile at least a year’s worth of cooking and minimal lighting fuel for an average family (10 full five gallon bulk tanks that are equal to 200 small green canisters) for about $200, and do so $20 at a time.
If not handled with respect, Propane is DANGEROUS! As in dynamite dangerous and AK-47 dangerous. As in blowing up your whole house into kindling dangerous. This isn’t vegetable oil. So, treat it with prudent caution and always, always, always read, understand and follow the safety directions that come with every propane product.—Never store tanks of propane indoors or in any other sealed environment. Especially, if you see, smell or hear a gas leak, shut off the source immediately and then fix the problem before you continue. If you can’t shut it off, move away, warn others and cross your fingers. Otherwise, you’ll very likely go up in a cloud of smoke and all your hard-won prepping will have been for naught. On the plus side, propane has been used as a consumer product for nearly 100 years and stringent government regulations require hardware designs that are fairly, if not perfectly, fool-proof. So, it’s not like you’re handling nitroglycerin. But, unless you really know what you’re doing, don’t try to modify or override the hardware safety features. Unlike gasoline vapors, pure propane is nontoxic, though it’s certainly not healthy to breathe it and you should avoid it as you would with wood smoke. However, as with all combustion, there’s a real risk of oxygen depletion in a sealed room and proper ventilation should be an ongoing consideration. If you find yourself starting to gasp for air, triggered by a buildup of carbon dioxide, that’s an early warning sign that the oxygen is running low and it’s time for some fresh air, no matter how cold it might be. Unlike natural gas, propane is heavier than air (1.5 times as dense). In its unburned state, propane vapor sinks and pools at the floor level. So, simply opening a couple of high windows to vent a leak may not be enough: You need to open a door or something else at ground level to let the heavy unburned vapors “drain” outside to disperse. Liquid propane, say from a broken hose or leaking tank valve, will flash to a vapor at atmospheric pressure and the vapor appears white due to moisture condensing from the air. A big cloud of propane in the open air will blow up with the slightest spark, even static. So, you want to avoid coming even close to these.Those basic risks described, however, if you treat propane with respect and understanding, there is no better fuel to stockpile for long term storage and multiple uses in survival situations, at least where you are not on the run, and while it lasts. Especially, when you use propane for just vital things like cooking and night lighting, the use rate is surprisingly low and a little propane will go a long ways.
What is Propane?
Propane is a gaseous byproduct in the refining of both oil and natural gas. It can be compressed into a liquid at relatively low pressures and will readily convert back into a burnable vapor under any conditions which humans can tolerate. First synthesized in 1910, it has been in commercial production since the 1920′s and the technology of using it at the consumer level is very well refined. 90% of all propane used in the US is from US sources, with 70% of the remaining amount coming from Canada. –Propane combustion is much cleaner than gasoline and other liquid hydrocarbons, though not quite as clean as natural gas combustion. Environmentally, it’s usually greener to cook with propane than with electricity. With a perfect burn, attainable only in theory, the only by-products are heat, carbon dioxide and water vapor. So, it can be used for indoor heating applications, but use a stove that provides a very high combustion efficiency and, especially, a low-oxygen sensor that will shut it down if the O2 gets low.
Convenience and Labor Savings
Another way to look at propane is as a serious labor saving device. In a self-subsistence scenario, your greatest critical shortages are going to be labor and the energy to power it. With propane in your resource inventory, the large amount of work that would normally have to be expended gathering and processing fuel for a cooking fire can then redirected into other critical tasks, such as growing food etc. When cooking is less labor-intensive, it can also be assigned to the lessor-able in the party, such as older folks with more enthusiasm than physical stamina, while they simultaneously babysit and teach the young ones how to cook, freeing up the parents to work elsewhere. The easiest way to implement propane into your prepping strategy and to start climbing the learning curve is to start looking for ways to incorporate it into your day-to-day life. It doesn’t much matter where you start, but probably the best place is cooking. So, if you don’t already have one, start shopping for a camping cook stove. Amazon has a good assortment and you can often find propane stuff at very attractive prices at yard sales etc. Generally speaking, you want appliances that use those green 1 pound propane canisters that cost so much new, but that can also be easily refilled at a huge savings.
Combine use of pterostilbene and quercetin for the production of cancer treatment medicaments
Various phenolic compounds, including resveratrol (RESV), display powerful antioxidant effects and may have therapeutic applications in diseases related to oxidative stress such as cancer (1-3). The anticancer activity of RESV was reported for the first time by Jang et al. in 1997 (4). The mechanisms by which RESV exerts its antitumor effects are being actively investigated (3) and may include for example: a) inhibition of ribonucleotide reductase (5), DNA polymerase (6), protein kinase C (7), or cyclooxygenase-2 (8) activities; b) inhibition of carcinogenesis mediated by reactive species of oxygen (4) or of cellular proliferation (9); and c) activation of cell death by apoptosis (10-13). However, the RESV-mediated potential inhibition of cancer growth is severely limited owing to its low bioavailability (14). Consequently, it appears that structural changes are necessary in the RESV molecule in order to increase its bioavailability while preserving its biological activity. The OH of 4′ and the stereoisomerism in its trans conformation are absolutely necessary for inhibition of cellular proliferation (15). Pterostilbene (PTER), a naturally occurring analog, of RESV but approximately 60-100 times more powerful as an antifungal agent, exhibits similar anticancer properties (16). Furthermore, the flavonoids are some of the most powerful antioxidants because they have one or more of the following structural elements: an o-diphenolic group, a double bond at 2-3 conjugated with the 4-oxo function, OH groups in positions 3 and 5. Quercetin (QUER) combines these three properties, and previous studies have confirmed that it also has antitumor properties, probably due to immune stimulation, elimination of free radicals, alteration of the mitotic cycle of the tumor cells, modification of gene expression, antiangiogenic activity, or induction of apoptosis, or a combination of these effects (2, 17). Concretely, QUER has been described (US 2003/0054357) in the treatment of prostate cancer. There are patents (WO02/34262) which highlight the antioxidant effect of certain flavonoids, concretely of a combination of quercetin plus catechin[F9] for use in the treatment and prevention of circulatory or cardiac disorders, by preventing platelet aggregation. — However, since it has not been demonstrated that the potential anticancer effects are effective in systemic administration, we investigated the anticancer properties of combined systemic administration of PTER and QUER at bioavailable concentrations. We found that their combination strongly inhibits the growth of the metastatic malignant melanoma B16-F10 (B16M-F10).
Take Blueberries and wine and fuse in blender for about 10 minutes—1 cup of blue berries 2 cups of wine —after 10 minutes strain this in a owl through a handkerchief And get out as much of the extract as possible —then take the volume put into a pot and Then allow this to simmer down to the bottom of the pot about ½-1/4 inch—allow to cool
Then take garlic or onion and extract in white wine—2 cups of wine 1-2 onions and again run through fro 10 minutes in a blender—then strain—take equal portions 1:1 and mix together in a glass jar and use tsp increments several times a day—this is a simple remedy for boosting anti tumour—antioxidant-anti cancer effect—and permeability in blood vessels
For those with morgellons you may see more of an expediting of polymer biofilm particualtes in feces and urine
[F1]Sounds like prostitution—some body go screwed here
[F2]In Other Words they were sabotage before they even had a chance
[F3]This is an arrogance as well—there will be NO SUPER POWER b the time this is all said and done —just a planet with there defences down due to there constant infighting making Planet earth ripe for take over
[F4]Because Soy Depleted Vitamin D so they had to add it
[F5]This article is way to Generious—None of the soy fermented or otherwise is fit to be consumed AT ALL—it is truly a for of biowarfare—food that taste good but has deadly consequences—and will shorten ones life span
[F6]When they speak of fermentation—the Chinese did this In Lime—not Aluminum and it was done for 2 years not 2 weeks
[F7]In the usa and Canada it exceeds 50 gram—or more considering all the soy being consumed in snacks and even mixed with animal proteins
[F8]FDA study show feeding cows vegetable oil ( soybean oil) caused both stomachs to hemmorhage
[F9]Green tea + Garlic
Show of The Month May 11 2013
Methanolic extract of Nigella sativa seed inhibits SiHa human cervical cancer cell proliferation through apoptosis
Suppression of estrogen biosynthesis by procyanidin dimers in red wine and grape seeds
Grape seed extract is an aromatase inhibitor and a suppressor of aromatase expression
Minced beef is more rapidly digested and absorbed compared with beef steak
Splenda (Sucralose) Found To Have Diabetes-Promoting Effects
Nanoparticles- The Tiniest Toxin
Methanolic extract of Nigella sativa seed inhibits SiHa human cervical cancer cell proliferation through apoptosis.
Nat Prod Commun. 2013 Feb;8(2):213-6
Authors: Hasan TN, Shafi G, Syed NA, Alfawaz MA, Alsaif MA, Munshi A, Lei KY, Alshatwi AA
Nigella sativa (NS), also known as black cumin, has long been used in traditional medicine for treating various cancer conditions. In this study, we sought to investigate the potential anti-cancer effects of NS extract using SiHa human cervical cancer cells. NS showed an 88.3% inhibition of proliferation of SiHa human cervical cancer cells at a concentration of 125 microL/mL methanolic extract at 24 h, and an IC50 value 93.2 microL/mL. NS exposure increased the expression of caspase-3, -8 and -9 several-fold. The analysis of apoptosis by Dead End terminal transferase-mediated dUTP-digoxigenin end labeling (TUNEL) assay was used to further confirm that NS induced apoptosis. Thus, NS was concluded to induce apoptosis in SiHa cell through both p53 and caspases activation. NS could potentially be an alternative source of medicine for cervical cancer therapy.—PMID: 23513732 [PubMed – indexed for MEDLINE]
Suppression of estrogen biosynthesis by procyanidin dimers in red wine and grape seeds.
Eng ET, Ye J, Williams D, Phung S, Moore RE, Young MK, Gruntmanis U, Braunstein G, Chen S.
Source-City of Hope Graduate School of Biological Science,Department of Surgical Research, Beckman Research Institute of the City of Hope, Duarte, California 91010, USA.
In breast cancer, in situ estrogen production has been demonstrated to play a major role in promoting tumor growth. Aromatase is the enzyme responsible for the conversion of androgen substrates into estrogens. This enzyme is highly expressed in breast cancer tissue compared with normal breast tissue. A wine extract fraction was recently isolated from red wine that exhibited a potent inhibitory action on aromatase activity. Using UV absorbance analysis, high-performance liquid chromatography profiling, accurate mass-mass spectrometry, and nanospray tandem mass spectrometry, most of the compounds in our red wine fraction were identified as procyanidin B dimers that were shown to be aromatase inhibitors. These chemicals have been found in high levels in grape seeds. Inhibition kinetic analysis on the most potent procyanidin B dimer has revealed that it competes with the binding of the androgen substrate with a K(i) value of 6 micro M. Because mutations at Asp-309, Ser-378, and His-480 of aromatase significantly affected the binding of the procyanidin B dimer, these active site residues are thought to be important residues that interact with this phytochemical. The in vivo efficacy of procyanidin B dimers was evaluated in an aromatase-transfected MCF-7 breast cancer xenograft model. The procyanidin B dimers were able to reduce androgen-dependent tumor growth, indicating that these chemicals suppress in situ estrogen formation. These in vitro and in vivo studies demonstrated that procyanidin B dimers in red wine and grape seeds could be used as chemopreventive agents against breast cancer by suppressing in situ estrogen biosynthesis.
Grape seed extract is an aromatase inhibitor and a suppressor of aromatase expression.
Kijima I, Phung S, Hur G, Kwok SL, Chen S.
Department of Surgical Research, Beckman Research Institute of the City of Hope, Duarte, California, USA.
Aromatase is the enzyme that converts androgen to estrogen. It is expressed at higher levels in breast cancer tissues than normal breast tissues. Grape seed extract (GSE) contains high levels of procyanidin dimers that have been shown in our laboratory to be potent inhibitors of aromatase. In this study, GSE was found to inhibit aromatase activity in a dose-dependent manner and reduce androgen-dependent tumor growth in an aromatase-transfected MCF-7 (MCF-7aro) breast cancer xenograft model, agreeing with our previous findings. We have also examined the effect of GSE on aromatase expression. Reverse transcription-PCR experiments showed that treatment with 60 mug/mL of GSE suppressed the levels of exon I.3-, exon PII-, and exon I.6-containing aromatase mRNAs in MCF-7 and SK-BR-3 cells. The levels of exon I.1-containing mRNA, however, did not change with GSE treatment. Transient transfection experiments with luciferase-aromatase promoter I.3/II or I.4 reporter vectors showed the suppression of the promoter activity in a dose-dependent manner. The GSE treatment also led to the down-regulation of two transcription factors, cyclic AMP-responsive element binding protein-1 (CREB-1) and glucocorticoid receptor (GR). CREB-1 and GR are known to up-regulate aromatase gene expression through promoters I.3/II and I.4, respectively. We believe that these results are exciting in that they show GSE to be potentially useful in the prevention/treatment of hormone-dependent breast cancer through the inhibition of aromatase activity as well as its expression.— PMID-16740737–[PubMed – indexed for MEDLINE
Minced beef is more rapidly digested and absorbed compared with beef steak, resulting in greater postprandial protein retention in older men1,2,3
Bart BL Groen,
Jan-Willem van Dijk,
Anneke de Lange,
Joan MG Senden, and
Luc JC van Loon
+ Author Affiliations
1. 1From Top Institute Food & Nutrition, Wageningen, Netherlands (BP, BBLG, JMGS, and LJCvL), and the Department of Human Movement Sciences, Nutrition and Toxicology Research Institute Maastricht School for Nutrition, Toxicology and Metabolism, Maastricht University Medical Centre+, Maastricht, Netherlands (BP, BBLG, J-WvD, AdL, AK, MK, JMGS, and LJCvL).
+ Author Notes
· ↵2 This study was not funded by external sources.
· ↵3 Address correspondence to LJC van Loon, Department of Human Movement Sciences, Maastricht University Medical Centre+, PO Box 616, 6200 MD, Maastricht, Netherlands. E-mail: [email protected].
Background: Older individuals generally experience a reduced food-chewing efficiency. As a consequence, food texture may represent an important factor that modulates dietary protein digestion and absorption kinetics and the subsequent postprandial protein balance.
Objective: We assessed the effect of meat texture on the dietary protein digestion rate, amino acid availability, and subsequent postprandial protein balance in vivo in older men.
Design: Ten older men (mean ± SEM age: 74 ± 2 y) were randomly assigned to a crossover experiment that involved 2 treatments in which they consumed 135 g of specifically produced intrinsically l-[1-13C]phenylalanine–labeled beef, which was provided as beef steak or minced beef. Meat consumption was combined with continuous intravenous l-[ring-2H5]phenylalanine and l-[ring-2H2]tyrosine infusion to assess beef protein digestion and absorption kinetics as well as whole-body protein balance and skeletal muscle protein synthesis rates.
Results: Meat protein–derived phenylalanine appeared more rapidly in the circulation after minced beef than after beef steak consumption (P < 0.05). Also, its availability in the circulation during the 6-h postprandial period was greater after minced beef than after beef steak consumption (61 ± 3% compared with 49 ± 3%, respectively; P < 0.01). The whole-body protein balance was more positive after minced beef than after beef steak consumption (29 ± 2 compared with 19 ± 3 μ mol phenylalanine/kg, respectively; P < 0.01). Skeletal muscle protein synthesis rates did not differ between treatments when assessed over a 6-h postprandial period.
Conclusions: Minced beef is more rapidly digested and absorbed than beef steak, which results in increased amino acid availability and greater postprandial protein retention. However, this does not result in greater postprandial muscle protein synthesis rates. This trial was registered at clinicaltrials.gov as NCT01145131.June 22, 2021 at 09:01 #2663
Splenda (Sucralose) Found To Have Diabetes-Promoting Effects
Promoted for decades as a “safe” sugar alternative, presumably to prevent or reduce symptoms of diabetes, Splenda (sucralose) has been found to have diabetes-promoting effects in human subjects.—-The artificial sweetener sucralose, which is approximately 600 times sweeter than sucrose (table sugar), and marketed under a variety of brand names, such as Splenda, Cukren, Nevella and SucraPlus, has recently been found to diabetes-promoting effects in human test subjects, despite containing no calories and being classified as a ‘nonutritive sweetener.’ —A new study published in the journal Diabetes Care, lead by researchers at the Center for Human Nutrition, Washington University School of Medicine, St. Louis, Missouri, set out to test the metabolic effects of sucralose in obese subjects who did not use nonnutritive sweeteners.—Seventeen subjects underwent a 5-hour oral glucose tolerance test on two separate occasions preceded by consuming either sucralose (experimental condition) or water (control condition) 10 min before the glucose load in a randomized crossover design.
The results were reported as follows:
Compared with the control condition, sucralose ingestion caused 1) a greater incremental increase in peak plasma glucose concentrations (4.2 ± 0.2 vs. 4.8 ± 0.3 mmol/L; P = 0.03), 2) a 20 ± 8% greater incremental increase in insulin area under the curve (AUC) (P < 0.03), 3) a 22 ± 7% greater peak insulin secretion rate (P < 0.02), 4) a 7 ± 4% decrease in insulin clearance (P = 0.04), and 5) a 23 ± 20% decrease in SI (P = 0.01).
In other words, a single dose of sucralose lead to a .6 mmol/L increase in plasma glucose concentrations, a 20% increase in insulin levels, a 22+ greater peak insulin secretion rate, and a 7% decrease in insulin clearance, an indication of decreased insulin sensitivity.—These data demonstrate that sucralose affects the glycemic and insulin responses to an oral glucose load in obese people who do not normally consume NNS.
Despite the fact that preapproval research on sucralose found a wide range of adverse health effects in exposed animals [see The Bitter Truth about Splenda], national and international food safety regulatory bodies, including the FDA, now consider it completely safe for daily human consumption.**********The same applies for synthetic sweeteners like aspartame, which despite its well-known link with brain damage and over 40 documented adverse health effects, is safety approved in 90 nations.—-Despite these cozy relationships, the research on sucralose’s adverse health effects continues to accumulate. Some of the more recent research on the chemical indicate that it may contribute to the following health and environmental problems:
Inflammatory Bowel Disease: A researcher from UMDNJ-New Jersey Medical School, Newark NJ, proposes that sucralose may be causing a global increase in cases of IBS, including both ulcerative colitis and Crohn’s disease. In an article titled “What made Canada become a country with the highest incidence of inflammatory bowel disease: could sucralose be the culprit?”, author Xiaofa Qin describes how Canada, which once had one of the lowest rates of IBS in the world, attained the highest levels after being the first country in the world to approve the use of sucralose in thousands of consumer products in 1991.[ii]
Harms Gut Flora and Gastrointestinal Health: A 2008 study found that the administration of sucralose to rats at a dose far below the US FDA Acceptable Daily intake level resulted in: 1) a decrease in the numbers of a wide range of beneficial gut bacteria. 2) “increase in fecal pH.” 3) “enhanced expression levels of P-gp, CYP3A4, and CYP2D1, which are known to limit the bioavailability of orally administered drugs.”[iii]
Migraines: A report was published in the journal Headache in 2006 indicating that physicians should be mindful of the possibility that sucralose can trigger migraines.[iv]
Environmental Persistence: Like many persistent organic pollutants in the pesticide category, sucralose is exceptionally resistant to degradation, both through environmental processes (microbial degradation, hydrolysis, soil sorption) and advanced treatment processes (chlorination, ozonation, sorption to activated carbon, and UV radiation). Sucralose, after all, was discovered accidentally by pesticide researchers, and is chemically related to DDT, a chlorinated hydrocarbon. Some researchers now consider it an ideal “tracer of anthropogenic activity[F1],” which is true also of lethal radioisotopes such as uranium 238 and plutonium 239, due to their resistance to degradation. [v] Indeed, recent research found that sucralose has a low rate of removal (11%) in drinking water tested that presently serves 28 million people.[vi]
Environmental Toxicity: Sucralose was recently found to alter the physiological and behavioral status of crustaceans, leading researchers to warn that the chemical will likely have wider ecological consequences.[vii]
*The FDA approves 1.1 mg/kg bodyweight, or the equivalent of 75 mg a day (about 6 packets) for a 150 lb adult.
Xiaofa Qin. Etiology of inflammatory bowel disease: A unified hypothesis. World J Gastroenterol. 2012 Apr 21 ;18(15):1708-22. PMID: 22553395
[ii] Xiaofa Qin. What made Canada become a country with the highest incidence of inflammatory bowel disease: Could sucralose be the culprit? Can J Gastroenterol. 2011 Sep ;25(9):511. PMID: 21912763
[iii] Mohamed B Abou-Donia, Eman M El-Masry, Ali A Abdel-Rahman, Roger E McLendon, Susan S Schiffman. Splenda alters gut microflora and increases intestinal p-glycoprotein and cytochrome p-450 in male rats. J Toxicol Environ Health A. 2008;71(21):1415-29. PMID:18800291
[iv] Rajendrakumar M Patel, Rakesh Sarma, Edwin Grimsley. Popular sweetner sucralose as a migraine trigger. Headache. 2006 Sep;46(8):1303-4. PMID: 16942478
[v] Lindsay Soh, Kristin A Connors, Bryan W Brooks, Julie Zimmerman. Fate of Sucralose through Environmental and Water Treatment Processes and Impact on Plant Indicator Species. Environ Sci Technol. 2011 Jan 14. [Epub ahead of print]. PMID: 21235203
[vi] Douglas B Mawhinney, Robert B Young, Brett J Vanderford, Thomas Borch, Shane A Snyder. The Artificial Sweetener Sucralose in U.S. Drinking Water Systems. Environ Sci Technol. 2011 Aug 31. Epub 2011 Aug 31. PMID: 21879743
[vii] Ann-Kristin Eriksson Wiklund, Magnus Breitholtz, Bengt-Erik Bengtsson, Margaretha Adolfsson-Erici. Sucralose – An ecotoxicological challenger? Chemosphere. 2012 Jan ;86(1):50-5. Epub 2011 Sep 28. PMID: 21955350
Sayer Ji is an author, researcher, lecturer, and advisory board member of theNational Health Federation.
Sayer Ji is an author, researcher, lecturer, and advisory board member of the National Health Federation.He founded Greenmedinfo.com in 2008 in order to provide the world an open access, evidence-based resource supporting natural and integrative modalities. It is internationally recognized as the largest and most widely referenced health resource of its kind.
[F1] Nanoparticles- The Tiniest Toxin
What exactly is a nanoparticle? As You Sow, a non-profit consumer advocacy group, explains—–Nanomaterials are often heralded as having the potential to revolutionize the food industry – from enabling the production of creamy liquids that contain no fat, to enhancing flavors,improving supplement delivery, providing brighter colors, keeping food fresh longer, or indicating when it spoils. It is reported that nanotechnology is already used in food and food related products, but due to lack of transparency about the issue, concrete information has been difficult to obtain. —Because of their small size, nanoparticles are able to go places in the body that larger particles cannot. Nanoparticles in food or food packaging can gain access via ingestion, inhalation,or skin penetration. When ingested, the nano-sized particles facilitate uptake into cells and can allow them to pass into the blood and lymph where they circulate through the body and reach potentially sensitive target sites such as bone marrow, lymph nodes, the spleen, the brain, the liver, and the heart. Nanoparticles penetrating the skin can distribute through the body via lymphatic channels. Inhaled nano TiO2 has been found to act like asbestos and silicone in that it accumulates in the lung and causes inflammation and can impact DNA proteins and cell membranes. (download the entire report on nanoparticles HERE)—-So, much like GMOs, nanoparticle technology was not properly tested before it entered our food supply.[F2] The few studies that have been done have alarming results. —A Swedish study, Influence of Nanoparticles on Blood-Brain Barrier Permeability and Brain Edema Formation in Rats, revealed that nanoparticles derived from transition metals, silver, copper, aluminum, silicon, carbon and metal oxides easily crossed the blood-brain barrier and produced lasting damage to the barrier, by altering the permeability. Research at Cornell University, led by Dr. Michael Shuler, studied how large doses of polystyrene nanoparticles – a common, FDA-approved substance found in substances ranging from food additives to vitamins – affected the absorption of iron: According to the study, high-intensity, short-term exposure to the particles initially blocked iron absorption, whereas longer-term exposure caused intestinal cell structures to change, allowing for a compensating uptick in iron absorption. –The researchers tested both acute and chronic nanoparticle exposure using human gut cells in petri dishes as well as live chickens and reported matching results. Shuler said the research serves to underscore how such particles, which have been widely studied and considered safe, cause barely detectable changes that could lead to, for example, over-absorption of other, harmful compounds. (source)
One of the major issues with nanoparticles is that when a substance is reduced to nano form (about a billionth in scale) the substance behaves differently. According to experts, the issue is that elements at the nanoscale behave differently from the larger sized particles in which they are normally encountered. [F3]As an example, graphite’s properties are well known: it holds specific position in toxicology guidelines and is not considered a hazardous or reactive material under any normal circumstances. Nobel winning physicist Richard Smalley of Rice University discovered carbon nanotubes and fullerenes (buckyballs) — nanoparticles of carbon — that are categorized as forms of graphite due to the way the carbon atoms are arranged. These particles, however, behave in ways unlike graphite, making their classification a potentially dangerous one. Scientists know that substances become more reactive as their particles get smaller because the surface area is greater relative to the volume, providing a larger surface on which chemical reactions can occur for a given amount of the substance. One example concerns the element iron. An iron nail will not burn, but the same quantity of the element in the form of an extremely fine powder will ignite spontaneously when exposed to air. Similarly, substances that are normally fairly inert may undergo unexpected chemical reactions in the human body or in the environment when in nanoparticle form. (source) Because of this concentrated effect, it isn’t known exactly how nanoparticles will affect humans. They have been shown to cause altered gene markers and brain damage in fish, accumulation in laboratory animals, and at least one type of nanoparticle has been shown to cross the human placenta in preliminary studies.
What is the FDA’s policy on the inclusion of nano-particles in food?[F4]
The European Food Safety Authority requires that foods containing nano-particles be labeled, but in America[F5], the FDA does not require food manufacturers to report the nanoparticles on the label. Ironically, the FDA also refuses to say that the nanoparticles are safe. “At this time, we are not aware of any food ingredient or [food contact substance] intentionally engineered on the nanometer scale for which there are generally available safety data sufficient to serve as the foundation for a determination that the use of a food ingredient or [food contact substance] is GRAS.” (GRAS = Generally Recognized As Safe)—- The FDA allows the control over the use of nanomaterial to be in the hands of food manufacturers. According to the government agency’s website,”Industry remains responsible for ensuring that its products meet all applicable legal requirements, including safety standards. Regardless of whether products are subject to premarket review or authorization, manufacturers are required to ensure that their product satisfies applicable safety standards and complies with other applicable requirements.”—This means that the manufacturers are free to determine what tests should be conducted, whether or not they wish to share the results of those test, and whether or not they opt to label the products that contain nanoparticles. Sebastian Cianci, an FDA spokesperson, said: –Under existing statutory and regulatory provisions, manufacturers are able to voluntarily include information about the use of nanomaterials or nanotechnology in the labeling of products where such information presented in the context of the entire label or labeling is not false or misleading…and does not violate other labeling requirements.–The FDA is apparently not tracking the use of these particles at all, according to an email exchange between EMagazine and the representative for the agency: E Magazine: What can you tell me about the prevalence of nanomaterials in our food supply? Sebastian Cianci: FDA does not have a list of food products that contain nanomaterials. E: Where are nanomaterials most often found within food products? In colorings or additives?
S.C.: FDA does not maintain a list of food products that contain nanomaterials so we cannot reliably answer this question. The FDA is not asking questions because they really just don’t want to know. Big Food has too much influence within the agency for any true oversight to occur. What foods contain nanoparticles?
(1-100 nm) particles derived from transition metals, silver, copper, aluminum, silicon, carbon and metal oxides […] can easily cross the blood-brain barrier (BBB) and/or produce damage to the barrier integrity by altering endothelial cell membrane permeability [… and] induce brain edema formation.16[F6]
One frequently used nano-particle is titanium dioxide. This is included in the powdered-sugar coating on many pastries and lends them a whiter, brighter look. Wondering what to strike from your shopping list? Titanium dioxide nanoparticles have provided a sparkling white appearance to the following, according to EMagazine:
Betty Crocker Whipped Cream Frosting
Jello Banana Cream Pudding
Vanilla Milkshake Pop Tarts
Nestlé Original Coffee Creamer
Because of their small size, nanoparticles are able to go places in the body that larger particles cannot.12 Nanoparticles in food or food packaging can gain access via ingestion, inhalation, or skin penetration. When ingested, the nano-sized particles facilitate uptake into cells and can allow them to pass into the blood and lymph where they circulate through the body and reach potentially sensitive target sites such as bone marrow, lymph nodes, the spleen, the brain, the liver, and the heart.13 Nanoparticles penetrating the skin can distribute through the body via lymphatic channels.14 Inhaled nano TiO2 has been found to act like asbestos and silicone in that it accumulates in the lung and causes inflammation and can impact DNA proteins and cell membranes.15
Because manufacturers are not required to report the inclusion of nanoparticles (trade secrets) and because little analysis of processed food has been done to determine whether or not nanoparticles are present, it’s impossible to provide a comprehensive list of foods that contain nanoparticles.
How can you avoid nanoparticles?
Nanoparticles are impossible to avoid if you eat processed foods – and you won’t know if you are consuming them or not, because they aren’t on the label. More frightening, and potentially much harder to avoid, is the fact that there are some experimental farming methods that use nanoparticles too. The strategies to avoid nanoparticles are identical to the strategies to avoid the other toxins in the grocery store gauntlet.
Grow your own food
Buy from small, local farms (find a farm near you)
Buy organic: The National Organics Standards Board has banned the use of engineered nanoparticles in organic food.
Avoid processed foods (anything with an ingredients list)
The food industry has taken its cue from the backlash against GMOs. If we don’t insist on transparency, they will not provide us with information on what our food contains. If we allow it, they will continue justify their silence by claiming that these tiny toxins are “trade secrets”. Will nanoparticles have negative long-term effects on human health? We honestly don’t know yet, but preliminary research does not look good. Vote with your wallet, and refuse to be a guinea pig for Big Food.
[F1]Referring to things, events, or actions whose origins can be traced to the activities of individual people or human groups.
In 2010, As You Sow, working with several investor firms including Calvert Investments, Domini Social Investments, Green Century Capital Management, and Trillium Asset largest and most iconic food companies in the world including YUM! Brands, PepsiCo, and Whole Foods about Management, contacted some of the [F2]
their use of nanomaterials. Most companies did not know whether or not nanomaterials were used in their
products and had to check with their suppliers. This lack of knowledge about nanomaterials in the supply chain
In 2012, As You Sow surveyed companies in the food industry to better understand the current status of nanomaterial use in the U.S. food market. We sent a survey to 2,500 companies including:
• the 100 largest food processing companies
• the 50 largest food distributors
• the 75 largest food retailers
• the 25 top food packaging companies
• the 50 top fast food companies
• 187 nutritional supplement companies
Nanoparticles are able to penetrate cells and interfere with several subcellular mechanisms. Some studies show that some nanoparticles can penetrate into cell nuclei and may therefore directly interfere with the structure and function of genomic DNA
Titanium Dioxide TiO2 and derived estimates of nano TiO2 in
foods including M&M’s, Betty Crocker Whipped Cream Frosting, Jell-O Banana Cream Pudding, Mentos, Trident and Dentyne gums, Vanilla Milkshake Pop Tarts, and Nestlé Original Coffee Creamer. The authors state that “electron microscopy and stability testing of food-grade TiO2 […] suggests that approximately 36% of the particles are less than 100nm in at least one dimension.”
[F6]Initial studies are already raising health concerns. For instance, nano silver is being used as an antimicrobial for food preparation and packaging materials despite studies showing that it can cause lung toxicity and inflammation in mice
Show of The Month May 18 2013
Preserving Meats and Foods
Colorectal cancer –Dairy Consumption Protection
27 Additional Reasons To Use And Store Salt
Silver nanoparticles induce premutagenic DNA oxidation that can be prevented by phytochemicals from Gentiana asclepiadea.
Brine—- is a solution of salt (usually sodium chloride) in water. In different contexts, brine may refer to salt solutions ranging from about 3.5% (a typical concentration of seawater, or the lower end of solutions used for brining foods) up to about 26% (a typical saturated solution, depending on temperature).—Brining foods in a saltwater mixture before you cook them adds flavor, tenderness, and reduces cooking times. If this sounds like a good thing then its time to learn the basics about brining.
Brining meats is an age-old process of food preservation. Heavy concentrations of salt preserved meats were taken on long ocean voyages and military campaigns before the advent of refrigeration. Today, brining has a new purpose. By using smaller quantities of salt mixed with other spices and herbs, brining can permeate meat with flavor. —-The chemistry behind brining is actually pretty simple. Meat already contains salt water. By immersing meats into a liquid with a higher concentration of salt, the brine is absorbed into the meat. Any flavoring added to the brine will be carried into the meat with the saltwater mixture. Because the meat is now loaded with extra moisture it will stay that way as it cooks. —The process of brining is easy but takes some planning. Depending on the size of what you want to brine it can take up to 24 hours of more. If you are going to be brining whole bird, you will also want an additional 6 to 12 hours between the brining and the cooking. If you want your poultry to have a golden, crispy skin, it needs to sit in the refrigerator for several hours after you remove it from the brine so that the meat can absorb the moisture from the skin. –The most basic process of brining is to take approximately 1 cup of table salt (no iodine or other additives) to 1 gallon of water. Another way to measure this concentration is with a raw egg. The ideal brine has enough salt to float a raw egg. You will need enough brine to completely submerge the meat without any part being out of the liquid. Some items might need to be weighed down to stay under. Brine meat for about a hour per pound. Remove from brine (don’t reuse the brine)and rinse to remove any excess salt before cooking. ——–So what should you brine? Just about an meat you choose. Poultry in particular benefits greatly from brining, regardless of how you plan to cook it. Large roasts, racks of ribs and anything you plan to smoke will be better for having been brined first. But this isn’t just a great barbecue tip but a good idea for meats whether you smoke, grill, roast or fry them.— The typical brine consists of 1 cup of salt for each gallon of water (or other liquids).. Start by determining the amount of liquid you are going to need. To do this take the meat you plan to brine and place it in the container you are going to use. The container can be most anything that will easily fit the meat but isn’t so big that you have to prepare far more brine that you need. Plastic containers, crocks, stainless steel bowls, resealable bags or any non-corrosive material will work— The typical brine consists of 1 cup of salt for each gallon of water (or other liquids). Start by determining the amount of liquid you are going to need. To do this take the meat you plan to brine and place it in the container you are going to use. The container can be most anything that will easily fit the meat but isn’t so big that you have to prepare far more brine that you need. Plastic containers, crocks, stainless steel bowls, resealable bags or any non-corrosive material will work. —- Once you know how much liquid is needed start by boiling 2 cups of water for each cup of salt you will need. Once it boils, add the salt (and sugar if you are going to be using sugar) and stir until dissolved. Add other spices and herbs. Combine with the remaining liquid (should be cold). The brine should always be cold before you add the meat so you should refrigerate it before you add the meat. You don’t want the brine cooking the meat. At this point you can add other brine ingredients like juices or cut up fruit. Submerge the meat into the brine. You can use a plate or other heavy object to keep it down. It is important that no part of the meat be exposed to the air. Saltwater brine will kill bacteria and keep the meat from spoiling but it doesn’t work if part of the meat is sticking out–Brine meats for about 1 hour per pound in the refrigerator. It is important that the whole thing be kept cold. The specific amount of time will vary. Lighter meats like poultry or seafood do not need to be brined as long as denser meats like pork tenderloins. Use the following chart to give you an idea of how long to brine. Remember that the longer you brine the stronger the flavor will be. If you over brine you could end up with some very salty meat– Once the meat is properly brined remove it. You do not need to rinse unless you were using a high salt concentration in the brine or if there is a layer of visible salt on the surface. Otherwise you can take cuts of meat straight to the grill, smoker, or oven. Whole poultry is the exception however. To get a crispy, brown skin, whole birds should be removed from the brine, and contained in a container and put in the refrigerator overnight or for at least 12 hours.
Whole Chicken (4 to 5 pounds)
4 to 5 hours
Turkey (12 to 14 pounds)
Pork Tenderloin (whole)
1 to 2 hours
Now that you know how to brine, it’s time to turn on the imagination. First of all, you don’t need water. What? That’s right the water is optional. Any liquid will do for brining. You can substitute some or all of the water with whatever your heart desires. Wine, beer, fruit juices (especially good is apple), or vinegars all make a good liquid base for your brine. Of course, you might not want to spend the money on a gallon or two of beer or wine for a brine that will just get thrown out when you are done. This is why most people use water for the majority of the brine but add a small quantity of another liquid for flavor. — One thing to remember when putting together a brine, is the chemical state of the liquid. By adding liquids like citrus juices or vinegar you will make the brine acidic. This will tenderize meat but, if it is too strong it can turn the meat to mush. If you do plan on using this kind of brine, reduce the brining time accordingly. — As for spices, imagine that you are going to be using a spice rub, but instead of applying the rub directly to the meat you simply add it to the brine. The brining process works better at pulling the flavors into the meat than applying a rub will. — Once you have the liquid chosen and added the cup of kosher salt per gallon, it’s time to add the flavor. Any herb, spice, sweetener, fruit, or vegetable will work
Preserving Meats and Foods
Raw honey is physically designed to preserve with 95% sugar solids absorbing bacteria through osmosis. The bees themselves add an enzyme to the nectar before it morphs into honey that develops hydrogen peroxide which aids preservation. Honey has a PH of around 3.9 which is similar to a mild vinegar, another reason for edging out the breakdown of foods. In addition, honey is hygroscopic[I1] , which means it absorbs moisture from it’s environment and holds on to it like a sweet sponge, this contributes to it’s moist longevity and preserving abilities.Raw Honey….yum!!!—Apicius, a Roman from the 1st century B.C., used honey religiously in well over half the recipes he compiled. Constantly mixing it with savory items like pepper, herbs, salt and meats. He suggested in his book to, “Cover whatever fresh meat you wish to preserve with a layer of honey.” Pouring Honey –Columella, over 100 years later had a similar suggestion, “Arrange (Quinces) lightly and loosely…in a new flagon…they do not deteriorate any further once they have …(honey) added to them: for such is the nature of honey that it checks any corruption and does not allow it to spread.”–Throughout history food preservation was a common practise. Indians, Romans and Chinese kept their meats sealed in honey from year to following year. Fully submerged meats remained free from spoilage and were delicious when finally eaten. Nuts and fruits maintained freshness and flavor for years and honeyed cakes were lush and long lasting.
If you are interested in giving honey preservation a try I would suggest trying nuts. Make sure your honey is raw and fully submerge the chosen nuts. Try your experiment often and compare your results!
Link of info on where you can find milk in your local area
In other Countries
Many people don’t realize that raw milk has many benefits. It can make you healthier, help you grow properly, and it contains many of the vitamins and enzymes you need to live a healthy life.
Raw milk is rich in enzymes and contains all 22 of the essential amino acids, the building blocks of life1, including phosphate. Phosphate is essential for the absorption of calcium and is plentifully present in raw milk but is completely destroyed by pasteurization. Prosphatase is an essential agent to the proper development of a strong skeletal structure. The enzyme lipase is also present in raw milk. Lipase aids in the digestion of fats. It is destroyed by pasteurization as well. 2. Lactase, an enzyme that helps with milk digestion and in the digestion of lactose, is in raw milk3. “; Raw milk is also an excellent source of vitamins particularly vitamin B12, a necessary vitamin that is difficult to find in non-meat sources1. Johns Hopkins University and the University of Maryland found that raw milk contains 2 1/2 times more of the enzyme lgG than pasteurized milk. This important enzyme inhibits rotavirus organisms that cause diarrhea in infants. Natural vitamin C is 33% higher in fresh raw milk than in pasteurized milk 2. Many people don’t realize that vitamin A, vitamin B, vitamin C, and enzyme inhibitors that kill pathogens are all destroyed by pasteurization. 2 Francis Pottenger, M.D. proved that raw milk reversed scurvy. Raw milk naturally has enzyme-based pathogen killers. These enzymes include lactoferrin, xanthine, oxidase, lactoperoxidase, lysozyme and nisin. Research has shown that when pathogens were added to raw milk they would not grow. The salmonella could not be found after less than 24 hours. The listeria and the E. Coli would not grow either.3 Thus, research has shown that raw milk contains enzymes and antibodies that make milk actually less susceptible to bacterial contamination. Dr. Crew used raw milk therapy on people with advanced cases of pulmonary tuberculosis and they improved rapidly. John Fowler M.D., Worcester Massachusetts, reported relief of muscle cramps in pregnancy. He said that raw milk therapy was very effective, and in no instance where used faithfully, were the muscle cramps in pregnant women a cause of discomfort.
1.Anna Soref: newhope.com
2. William Campbell Douglass, Jr., M.D. & Vonderplanitz, Scientist/Nutrionist
3. Mark McAfee: Founder, Organic Pastures, The Safety of Raw Milk
A lot of people find that raw milk helps them lower weight and blood pressure. One patient went from 325 lbs. to 284 lbs. in two weeks on two qts. of raw milk a day and her blood pressure was reduced from 220 to 170. The worst case of psoriasis Dr. William Douglass had ever seen was when a boy came in and he was covered from head to toe with scales. They put him on a raw milk diet and in less than a month he had skin like a baby’s. Many people are realizing that raw milk has many benefits. Raw milk contains the essential vitamins, enzymes, and amino acids. Raw milk has been a part of our diet all through the millennia and it has just been these last 100 years that we have pasteurized milk. What a coincidence that now we as a nation are sicker than ever. Pasteurization is just an excuse for the sale of dirty milk, it may be used to mask low quality milk. Pasteurization promotes carelessness. People are now realizing that raw milk is now good for you.
Raw milk vs. Pasteurized milk
ENZYMES ALL AVAILIBLE LESS THAN 10% REMAINING
PROTEIN 100% available, all 22 amino acids, including 8 that are essential. Protein-lysine and tyrosine are altered by heat with serious loss of metabolic availability. This results in making the whole protein complex less available for tissue repair and rebuilding
3) Fats: (research studies indicate that fats are necessary to metabolize protein and calcium. All natural protein-bearing foods contain fats.)
All 18 fatty acids metabolically available, both saturated and unsaturated fats. Altered by heat, especially the 10 essential unsaturated fats.
4) Vitamins: All 100% available Among the fat-soluble vitamins, some are classed as unstable and therefore a loss is caused by eating above blood temperature. This loss Vitamin A, D, E and F can run as high as 66%. Vitamin C loss usually exceeds 50%. Losses on water-soluble vitamins are affected by heat and can run from 38% to 80%.
5) Carbohydrates: Easily utilized in metabolism. Still associated naturally with elements. Tests indicate that heat has made some changes making elements less available metabolically.
6) Minerals: All 100% metabolically available. Major mineral components are calcium, chlorine, magnesium, phosphorus, potassium, sodium and sulphur. Vital trace minerals, all 24 or more, 100% available. Calcium is altered by heat and loss in metabolism may run 50% or more, depending on pasteurization temperature. Losses in other essential minerals, because one mineral usually acts synergistically with another element There is a loss of enzymes that serve as leaders in assimilation minerals.
Colorectal cancer –Dairy Consumption Protection
Colorectal cancer is the 3rd most common type of cancer worldwide with about 1.2 million new cases diagnosed in 2008 accounting for 9.7% of all cancers (Ferlay and others 2010). An increased consumption of milk or dairy products is associated with a significant reduction in colon cancer (Elwood and others 2008). Cho and others (2004a) conducted a large pooled analysis of data from 10 cohorts (n = 534, 536) from 5 countries and found 4992 individuals diagnosed with colorectal cancer at follow-up. Individuals who consumed more than a glass of milk (≥250 g)/d had a 15% reduced risk of developing colorectal cancer (relative risk 0.85, 95% CI 0.78 to 0.94), compared to those who consumed <70 g/d. Several mechanisms may explain a protective effect of dairy foods on colorectal cancer risk (Norat and Riboli 2003; Larsson and others 2005).
The results of the large prospective cohort study showed as inverse association of cancers of the digestive system with dairy food and calcium in both men and women, especially with colorectal cancer (Park and others 2009). A variety of studies (epidemiological, animal, laboratory, and clinical trials) indicate that higher ingestions of calcium and/or dairy foods reduce the risk of colon cancer (Cho and others 2004a; Chan and Giovannucci 2010). Calcium intakes of 1200 to 1500 mg/d, or 4 servings of dairy products per day, seem to be the most protective against colon cancer [F2](Holick 2008). Dairy products are one of the main dietary sources of calcium, which has been hypothesized to prevent colon cancer by binding secondary bile acids and ionized fatty acids and thus reducing their proliferative effects in the colonic epithelium (Govers and van der Meet 1993). In addition, it has been shown that calcium can influence multiple intracellular pathways that lead to differentiation in normal cells and apoptosis in transformed cells (Newmark and others 1984; Lamprecht and Lipkin 2001; Fedirko and others 2009) and that calcium can reduce the number of mutations in the K-ras gene in rat colorectal neoplasms (Llor and others 1991; Aune and others 2012).
Several clinical trials have reported reduced cell proliferation in the colon and rectum with intake of calcium and dairy products[F3] (Holt and others 1998; Karagas and others 1998; Holt and others 2001; Ahearn and others 2011). Pooled data of dairy product intake from 10 cohort studies demonstrated a 12% reduction in colon cancer risk with each 500 mL increase in milk intake. There was 17% reduction in colorectal cancer incidence with the ingestion of Ricotta cheese greater than 25 mg/d (Cho and others 2004a, b). Epidemiologic intake and intervention studies have shown that calcium administration lowers colorectal adenomatous polyps as well as cancer rates and this effect may be prolonged (Holt 2008). Furthermore, the results of a case-control study in New Zealand revealed that daily consumption of milk in childhood may reduce colorectal cancer incidence, possibly by the action of calcium on the development of adenoma. Participation in school milk programs was associated with a 2.1% reduction in the odds ratio for colorectal cancer for every 100 half-pint bottles drunk (1 half-pint bottle = 284 mL) (Cox and Sneyd 2011).
Most evidence suggests that the effect of calcium is dependent or partially related to simultaneous vitamin D intake. Vitamin D may also reduce colon cancer risk independent of the presence of increased amounts of calcium or dairy products in the diet (Holt 2008). Vitamin D modulates the effects of calcium on colorectal carcinogenesis (Mizoue and others 2008). The results of a large-scale case-control study among a Japanese population found inverse associations of dietary calcium and vitamin D with colorectal cancer risk that demonstrated dietary modification to increase calcium intake while maintaining adequate vitamin D status through diet and moderate sun exposure had large potential in the prevention of colorectal cancer among Japanese adults (Mizoue and others 2008).
Some fat components of dairy products, including CLA and butyric acid (Liew and others 1995), have been proved to be protective in experimental studies (Hague and Paraskeva 1995; Parodi 1997). The results of a Cohort study in Swedish women showed the women who consumed ≥4 servings of high-fat dairy foods per day (including whole milk, full-fat cultured milk, cheese, cream, sour cream, and butter) had a multivariate rate ratio of colorectal cancer of 0.59 (95% CI: 0.44, 0.79; P for trend = 0.002) when compared to the women who consumed <1 serving/d. Each increment of 2 servings of high-fat dairy foods per day corresponded to a 13% reduction in the risk of colorectal cancer (multivariate rate ratio: 0.87; 95% CI: 0.78, 0.96) (Larsson and others 2005).
Casein, which makes up nearly 80% of the protein in cow milk, has been demonstrated to have anticarcinogenic properties (Goeptar and others 1997). Casein may protect against colon cancer by inhibition of enzymes that are produced by intestinal bacteria and are responsible for deconjugation of procarcinogenic glucuronides to carcinogens. Moreover, casein protects also against colon cancer by its effect on the immune system, especially by its ability to simulate phagocytic activities and increase lymphocytes (Parodi 1998). Other researchers suggest that the molecular structure of casein contributes to its anticarcinogenic properties (MacDonald and others 1994). Moreover, researchers in Australia reported decreased levels of aberrant crypt foci, precancer markers, in the proximal colon of rats fed whey protein concentrate and treated with a chemical carcinogen (Belobrajdic and others 2003).
27 Additional Reasons To Use And Store Salt
Salt Value In Health
On the other hand, salt/sodium is vital to keep our bodies functioning normally. It is a main component of the extracellular fluids in the body and is important for regulating hydration and other body functions such as the transmission of nerve impulses and the contraction and relaxation of muscles.
How Much Salt Is Needed For Survival And Why?
For most of us, the amount of salt needed to stay healthy can be found in processed, packaged or canned foods. Let us hope that we have stored enough of these foods – especially the canned items – that we will never need to worry about adding more.
Which leads me to the next point: what are the uses of salt in an emergency situation?
Tips for SaltUsage:
1. Preserving meat, fish and game that is caught in the wild
2. Providing the nominal amount of dietary sodium once the canned and processed foods are gone
3. Taste enhancement (perhaps this should be number one!)
4. As a natural cleaner
5. First aid & personal care (i.e bee stings,mosquito bites, gargle for sore throats, teeth cleaning)
Having gone most of my adult life avoiding salt, coming to terms with adding salt to my survival pantry was true mind-shift. Not only is there a physiological need for our bodies to ingest salt in one form or another, but there are a multitude of other uses aside from food enhancement and food preservation.
What are some of those uses? This week I am sharing some non-conventional uses of salt compiled by my colleague ‘Above Average’ Joe at SurvivalLife.com.
27 ADDITIONAL REASONS TO USE AND STORE SALT
Salt has been an integral part of civilization dating back as far as 6050 B.C. It has been such an important element of life that it has been the subject of many stories, fables and folktales and is frequently referenced in fairy tales.
It served as currency at various times and places, and it has even been the cause of bitter warfare. Offering bread and salt to visitors, in many cultures, is traditional etiquette.
Aside from all of the uses that salt performs in terms of baking, food flavor and food preservation, salt has a number of other uses that you may never have thought of.
Make a paste using 6 tablespoons of salt and 2 tablespoons lemon juice. Apply paste to rusted area with a dry cloth and rub. Rinse thoroughly and dry.
Perk Up Coffee Flavor
Add a pinch of Salt to the coffee in the basket of your coffeemaker. This will improve the coffee’s flavor by helping to remove some of the acid taste.
Dispose of Disposal Odor
To help remove odors from garbage disposals, pour 1/2 cup of Salt directly into the garbage disposal. By running the disposal following manufacturer’s directions, you’ll send those odors down the drain.
Eliminate Fish Odors
Removing fish odor from your hands is simple with Salt. Just rub your hands with a lemon wedge dipped in salt, then rinse with water.
Cut Cutting Board Odors
To help cut odors off of your wooden cutting board, simply pour a generous amount of Salt directly on the board. Rub lightly with a damp cloth. Wash in warm, sudsy water.
Soothe Sore Throats
To alleviate the discomfort of a mild sore throat, gargle several times daily with a mixture of 1/4 teaspoon Salt and 1/2 cup warm water*. It’s like taking a liquid lozenge.
Treat your Tootsie’s
To prepare a salt water bath, pour 6 quarts (1-1/2 gallons) warm water in a large basin. Mix in 1/4 cup Salt and 1/4 cup baking soda. Soak feet for up to 15 minutes.
Salt added to water makes the water boil at a higher temperature, thus reducing cooking time (it does not make the water boil faster).
Testing egg freshness
Place the egg in a cup of water to which two teaspoonful’s of salt has been added. A fresh egg sinks; if it floats, toss it.
Cleaning greasy pans
The greasiest iron pan will wash easily if you use a little salt in it and wipe with paper towels.
Cleaning stained cups
Rubbing with salt will remove stubborn tea or coffee stains from cups.
Save the bottom of your oven
If a pie or casserole bubbles over in the oven, put a handful of salt on top of the spill. It won’t smoke and smell, and it will bake into a crust that makes the baked-on mess much easier to clean when it has cooled.
Fend Off Fire From A Rogue BBQ
Toss a bit of salt on flames from food dripping in barbecue grills to reduce the flames and calm the smoke without cooling the coals (like water does).
To remove pinfeathers easily from a chicken, rub the chicken skin with salt first.
To prevent mold on cheese, wrap it in a cloth dampened with saltwater before refrigerating.
Keeping milk fresh
Adding a pinch of salt to milk will keep it fresh longer.
Soak fish in salt water before descaling; the scales will come off easier.
Rub salt on your pancake griddle and your flapjacks won’t stick.
Keeping cut flowers fresh
A dash of salt added to the water in a flower vase will keep cut flowers fresh longer.
Keeping patios weed-free
If weeds or unwanted grass come up between patio bricks or blocks, carefully spread salt between the bricks and blocks, then sprinkle with water or wait for rain to wet it down.
Killing poison ivy
Mix three pounds of salt with a gallon of soapy water and apply to leaves and stems with a sprayer.
Sprinkling a little salt in canvas shoes occasionally will take up the moisture and help remove odors.
Relieving bee stings
If stung, immediately wet the spot and cover with salt to relieve the pain.
Sprinkle salt at doorways, window sills and anywhere else ants sneak into your house. Ants don’t like to walk on salt.
Use one part fine salt to two parts baking soda–dip your toothbrush in the mix and brush as usual.
Melt snow and ice
Sprinkle salt on snow or ice to melt away.
Occasionally throw a handful of salt on the flames in your fireplace; it will help loosen soot from the chimney and salt makes a bright yellow flame.
The term “worth one’s weight in salt” means that a person is effective and efficient or deserving of one’s pay.
A little about me (‘Above Average’ Joe): I am just an average guy with a passion for learning. .I am excited to share the things I learn with you but I am most interested in learning from you. Survival Life is more than just one man. It is a growing and living community of individuals; all with the desire to be prepared to survive and thrive no matter what this world throws at us. I want to welcome you to the Survival Lifecommunity and look forward to growing with you! Thank you, Gaye, for inviting me to share the Survival Life with your readers!
Now granted, some of these uses are handy dandy but not applicable to survival. Still, as Joe has demonstrated, there are a ton of day to day uses for salt that will make our lives easier if not more pleasant.
THE FINAL WORD
I checked a number of sources and the consensus is that you should store 5 to 10 pounds of salt per person as a one year supply. This seems like a lot to me but, given that salt is so cheap, there is no harm in stocking some for emergency purposes. And now that I think about it, with so many uses, I would recommend storing a bit more for use as barter currency.
The other thing worth mentioning is that salt is easy to store. You can use Mylar bags, buckets or even re-processed jars or soda bottles. Just remember that you should not use an oxygen absorber because if you do, the salt will turn in to a solid brick!
Silver nanoparticles induce premutagenic DNA oxidation that can be prevented by phytochemicals from Gentiana asclepiadea.
Mutagenesis. 2012 Nov;27(6):759-69
Authors: Hudecová A, Kusznierewicz B, Rundén-Pran E, Magdolenová Z, Hasplová K, Rinna A, Fjellsbø LM, Kruszewski M, Lankoff A, Sandberg WJ, Refsnes M, Skuland T, Schwarze P, Brunborg G, Bjøras M, Collins A, Miadoková E, Gálová E, Dusinská M
Among nanomaterials, silver nanoparticles (AgNPs) have the broadest and most commercial applications due to their antibacterial properties, highlighting the need for exploring their potential toxicity and underlying mechanisms of action. Our main aim was to investigate whether AgNPs exert toxicity by inducing oxidative damage to DNA in human kidney HEK 293 cells. In addition, we tested whether this damage could be counteracted by plant extracts containing phytochemicals such as swertiamarin, mangiferin [F4]and homoorientin[F5] with high antioxidant abilities. We show that AgNPs (20 nm) are taken up by cells and localised in vacuoles and cytoplasm. Exposure to 1, 25 or 100 µg/ml AgNPs leads to a significant dose-dependent increase in oxidised DNA base lesions (8-oxo-7,8-dihydroguanine or 8-oxoG) detected by the comet assay after incubation of nucleoids with 8-oxoG DNA glycosylase. Oxidised DNA base lesions and strand breaks caused by AgNPs were diminished by aqueous and methanolic extracts from both haulm and flower of Gentiana asclepiadea.—PMID: 22940646 [PubMed – indexed for MEDLINE]
Special Comment for thos who are using nano particles of silver you may want to encourage the usage of these plants as teas t remove any excess build up in the cells of silver—nano particles of any metal can be a causative issue with cell death and the restriction of cellular function-so if you utilize these metals/minerals in your system then utilize these herbs as well in reducing the load on the cells—you are as well being loaded with nano particles from the sky —so keep that in mind as well
[I1]Hygroscopy is the ability of a substance to attract and hold water molecules from the surrounding environment. This is achieved through either absorption or adsorption with the absorbing or adsorbing material becoming physically “changed” somewhat, by an increase in volume, stickiness, or other physical characteristic of the material, as water molecules become “suspended” between the material’s molecules in the process
[F2]Dairy Products==Unpasteurized milk—Yogurt-Buttermilk-Kefir—Muslanka-Cottage cheese-riccotta cheese-sour cream—Ice cream made with any of these products
[F3]See Above Comment
[F4]An antioxidant xanthone from mangos
[F5]A flavone that is derived frm passion flower-chaste tree-
Show of The Month May 25 2013
Nanoparticles and Magnetic Current Used to Damage Cancerous Cells in Mice
Using Gold Nanoparticles to Hit Cancer Where It Hurts
Wound-healing potential of an ethanol extract of Carica papaya (Caricaceae) seeds.
The Vaccine Hoax is Over. Documents from UK reveal 30 Years of Coverup
Milk –Breast Cancer Remedy
Promotion of hair growth by Rosmarinus officinalis leaf extract.
Table Reference Of AntiCancer effects of Milk
Nanoparticles and Magnetic Current Used to Damage Cancerous Cells in Mice
Mar. 27, 2012 — Using nanoparticles and alternating magnetic fields, University of Georgia scientists have found that head and neck cancerous tumor cells in mice can be killed in half an hour without harming healthy cells.—The findings, published recently in the journal Theranostics, mark the first time to the researchers’ knowledge this cancer type has been treated using magnetic iron oxide nanoparticle-induced hyperthermia, or above-normal body temperatures, in laboratory mice.–“We show that we can use a small concentration of nanoparticles to kill the cancer cells,” said Qun Zhao, lead author and assistant professor of physics in the Franklin College of Arts and Sciences. Researchers found that the treatment easily destroyed the cells of cancerous tumors that were composed entirely of a type of tissue that covers the surface of a body, which is also known as epithelium.—Several researchers around the globe are exploring the use of heated nanoparticles as a potential cancer treatment. Previous studies also have shown that high temperatures created by combining magnetic iron oxide nanoparticles with strong alternating magnetic currents can create enough heat to kill tumor cells. Zhao said he is optimistic about his findings, but explained that future studies will need to include larger animals before a human clinical trial could be considered.–For the experiment, researchers injected a tiny amount — a tenth of a teaspoon, or 0.5 milliliter — of nanoparticle solution directly into the tumor site. With the mouse relaxed under anesthesia, they placed the animal in a plastic tube wrapped with a wire coil that generated magnetic fields that alternated directions 100,000 times each second. The magnetic fields produced by the wire coil heated only the concentrated nanoparticles within the cancerous tumor and left the surrounding healthy cells and tissue unharmed.—Zhao said the study paves the way for additional research that might investigate how to use a biodegradable nanoparticle material similar to magnetic iron oxide for other roles in fighting cancer, such as carrying and delivering anti-cancer drugs to the tumor site.–“When the cancer cell is experiencing this heated environment, then it becomes more susceptible to drugs,” Zhao said.–Magnetic iron oxide nanoparticles could be useful in improving the contrast in magnetic resonance imaging at a cancer site, he said. In other words, the nanoparticles could help physicians detect cancer even if the cancer is not visible to the naked eye with an MRI scan.–“The reason I am interested in using these magnetic nanoparticles is because we hope to one day be able to offer diagnosis and therapeutics, or theranostics, using a single agent,” Zhao said.–The research was supported by a National Cancer Institute Head and Neck Specialized Program of Research Excellence at Emory University.–Story Source-The above story is reprinted from materials provided by University of Georgia. The original article was written by Kathleen Raven. –Journal Reference-Qun Zhao, Luning Wang, Rui Cheng, Leidong Mao, Robert D. Arnold, Elizabeth W. Howerth, Zhuo G. Chen, Simon Platt. Magnetic Nanoparticle-Based Hyperthermia for Head & Neck Cancer in Mouse Models. Theranostics, 2012; : 113 DOI: 10.7150/thno.3854
Using Gold Nanoparticles to Hit Cancer Where It Hurts
Feb. 18, 2010 — Taking gold nanoparticles to the cancer cell and hitting them with a laser has been shown to be a promising tool in fighting cancer, but what about cancers that occur in places where a laser light can’t reach? Scientists at the Georgia Institute of Technology have shown that by directing gold nanoparticles into the nuclei of cancer cells, they can not only prevent them from multiplying, but can kill them where they lurk.–The research appeared as a communication in the February 10 edition of the Journal of the American Chemical Society.–“We’ve developed a system that can kill cancer cells by shining light on gold nanoparticles, but what if the cancer is in a place where we can’t shine light on it? To fix that problem, we’ve decorated the gold with a chemical that brings it inside the nucleus of the cancer cell and stops it from dividing,”[F1] said Mostafa El-Sayed, Regents professor and director of the Laser Dynamics Laboratory at Georgia Tech.–Once the cell stops dividing, apoptosis sets in and kills the cell.–“In cancer, the nucleus divides much faster than that of a normal cell, so if we can stop it from dividing, we can stop the cancer,” said El-Sayed.–The team tested their hypothesis on cells harvested from cancer of the ear, nose and throat. They decorated the cells with an arginine-glycine-aspartic acide petipde (RGD) to bring the gold nano-particles into the cytoplasm of a cancer cell but not the healthy cells and a nuclear localization signal peptide (NLS) to bring it into the nucleus.—In previous work they showed that just bringing the gold into the cytoplasm does nothing. In this current study, they found that implanting the gold into the nucleus effectively kills the cell.–“The cell starts dividing and then it collapses,” said El-Sayed. “Once you have a cell with two nuclei, it dies.” The gold works by interfering with the cells’ DNA, he added. How that works exactly is the subject of a follow-up study.–“Previously, we’ve shown that we can bring gold nanoparticles into cancer cells and by shining a light on them, can kill the cells. Now we’ve shown that if we direct those gold nanoparticles into the nucleus, we can kill the cancer cells that are in spots we can’t hit with the light,” said El-Saye–Story Source-The above story is reprinted from materials provided by Georgia Institute of Technology. The original article was written by David Terraso. —Journal Reference–Bin Kang, Megan A. Mackey and Mostafa A. El-Sayed. Nuclear Targeting of Gold Nanoparticles in Cancer Cells Induces DNA Damage, Causing Cytokinesis Arrest and Apoptosis. Journal of the American Chemical Society, 2010; 132 (5): 1517 DOI: 10.1021/ja9102698
Wound-healing potential of an ethanol extract of Carica papaya (Caricaceae) seeds.
Int Wound J. 2012 Dec;9(6):650-5
Authors: Nayak BS, Ramdeen R, Adogwa A, Ramsubhag A, Marshall JR
Carica papaya L. (Linn) (Caricaceae) is traditionally used to treat various skin disorders, including wounds. It is widely used in developing countries as an effective and readily available treatment for various wounds, particularly burns. This study evaluated the wound-healing and antimicrobial activity of C. papaya seed extract. Ethanol extract of C. papaya seed (50 mg/kg/day) was evaluated for its wound-healing activity in Sprague-Dawley rats using excision wound model. Animals were randomly divided into four groups of six each (group 1 served as control, group 2 treated with papaya seed extract, group 3 treated with a standard drug mupirocin and papaya seed extract (1:1 ratio) and group 4 treated with a mupirocin ointment. Rate of wound contraction and hydroxyproline content were determined to assess the wound-healing activity of the seed extract. The group 2 animals showed a significant decrease in wound area of 89% over 13 days when compared with groups 1 (82%), 3 (86%) and 4 (84%) respectively. The hydroxyproline content was significantly higher with the granulation tissue obtained from group 2 animals which were treated with C. papaya seed extract. Histological analysis of granulation tissue of the group 2 animals showed the deposition of well-organized collagen. The extract exhibited antimicrobial activity against Salmonella choleraesuis and Staphylococcus aureus. Our results suggest that C. papaya promotes significant wound healing in rats and further evaluation for this activity in humans is suggested.—-PMID: 22296524 [PubMed – indexed for MEDLINE
Remedy Making—Take the papaya seed and mix it with a clear base alcohol( gin-vodka-tequila-rum) and portion this to a 2:1 or 3:1 ratio of alcohol to seed( 3 parts alcohol-1 part seed) blend in your home made extractor ( blender or mason jar bottle I show on the tube ) http://www.youtube.com/watch?v=zcFG2ckjlPE —when done extracting filter through a handkerchief —and pour into glass—andnowyou have a healant as needed
The Vaccine Hoax is Over. Documents from UK reveal 30 Years of Coverup
VaccinationVaccination Hoax is overVaccines
Andrew Baker ( FFN),- Freedom of Information Act in the UK filed by a doctor there has revealed 30 years of secret official documents showing that government experts have
1. Known the vaccines don’t work
2. Known they cause the diseases they are supposed to prevent
3. Known they are a hazard to children
4. Colluded to lie to the public
5. Worked to prevent safety studies
Those are the same vaccines that are mandated to children in the US.
Educated parents can either get their children out of harm’s way or continue living inside one of the largest most evil lies in history, that vaccines – full of heavy metals, viral diseases, mycoplasma, fecal material, DNA fragments from other species, formaldehyde, polysorbate 80 (a sterilizing agent) – are a miracle of modern medicine.—Freedom of Information Act filed in the US with the CDC by a doctor with an autistic son, seeking information on what the CDC knows about the dangers of vaccines, had by law to be responded to in 20 days. Nearly 7 years later, the doctor went to court and the CDC argued it does not have to turn over documents[F2]. A judge ordered the CDC to turn over the documents on September 30th, 2011.—-On October 26, 2011, a Denver Post editorial expressed shock that the Obama administration, after promising to be especially transparent, was proposing changes to the Freedom of Information Act that would allow it to go beyond declaring some documents secret and to actually allow government agencies (such as the CDC) to declare some document “non-existent[F3].”—–Simultaneous to this on-going massive CDC cover up involving its primary “health” not recommendation but MANDATE for American children, the CDC is in deep trouble over its decades of covering up the damaging effects of fluoride and affecting the lives of all Americans, especially children and the immune compromised. Lawsuits are being prepared. Children are ingesting 3-4 times more fluoride by body weight as adults and “[t]he sheer number of potentially harmed citizens — persons with dental fluorosis, kidney patients tipped into needing dialysis, diabetics, thyroid patients, etc — numbers in the millions.”—The CDC is obviously acting against the health of the American people. But the threat to the lives of the American people posed by the CDC’s behavior does not stop there. It participated in designed pandemic laws that are on the books in every state in the US, which arrange for the government to use military to force unknown, untested vaccines, drugs, chemicals, and “medical” treatments on the entire country if it declares a pandemic emergency.
The CDC’s credibility in declaring such a pandemic emergency is non-existent, again based on Freedom of Information Act. For in 2009, after the CDC had declared the H1N1 “pandemic,” the CDC refused to respond to Freedom of Information Act filed by CBS News and the CDC also attempted to block their investigation. What the CDC was hiding was its part in one of the largest medical scandals in history, putting out wildly exaggerated data on what it claimed were H1N1 cases, and by doing so, created the false impression of a “pandemic” in the US.—The CDC was also covering up financial scandal to rival the bailout since the vaccines for the false pandemic cost the US billions. And worse, the CDC put pregnant women first in line for an untested vaccine with a sterilizing agent, polysorbate 80, in it. Thanks to the CDC, “the number of vaccine-related “fetal demise” reports increased by 2,440 percent in 2009 compared to previous years, which is even more shocking than the miscarriage statistic [700% increase].—-The exposure of the vaccine hoax is running neck and neck with the much older hoax of a deadly 1918-19 flu. It was aspirin that killed people in 1918-19, not a pandemic flu. It was the greatest industrial catastrophe in human history with 20-50 million people dying but it was blamed on a flu. The beginning of the drug industry began with that success (and Monsanto was part of it). The flu myth was used by George Bush to threaten the world with “another pandemic flu that could kill millions” – a terror tactic to get pandemic laws on the books in every state and worldwide. Then the CDC used hoax of the pandemic hoax to create terror over H1N1 and to push deadly vaccines on the public, killing thousands of unborn children and others. (CDC will not release the data and continues to push the same vaccine.)
The hoax of the vaccine schedule is over, exposed by FOIAs in the UK.
The hoax of the CDC’s interest in children’s lives has been exposed by its refusal to respond to a doctor’s FOIAs around its knowledge of vaccine dangers.
The 1918-19 pandemic hoax has been exposed by Dr. Karen Starko’s work on aspirin’s role in killing people.
And despite refusing to respond to FOIAS, the CDC’s scandalous hoax of a 2009 flu pandemic and its part in creating it, was exposed by CBS NEWS.
And the Obama administration, in attempting to salvage the last vestige of secrecy around what is really happening with vaccines, by declaring agency documents non-existent, has made its claim of transparency, non-existent.
But pandemic laws arranging for unknown vaccines to be forced on the entire country are still in place with HHS creating a vaccine mixture that should never be used on anyone and all liability for vaccines having been removed. Meanwhile, a Canadian study has just proven that the flu vaccine containing the H1N1 vaccine which kills babies in utero, actually increases the risk of serious pandemic flu.—Americans who have been duped into submitting their children to the CDC’s deadly vaccines, have a means to respond now. People from every walk of life and every organization, must
1. take the information from the UK FOIAs exposing 30 years of vaccine lies, the refusal of the CDC to provide any information on what it knows about those lies, and the Obama Administration’s efforts to hide the CDC’s awareness of those lies, and go to their state legislatures, demand the immediate nullification of the CDC vaccine schedule and the pandemic laws.
2. inform every vet. active duty military person, law enforcement people, DHS agents and medical personnel they know, of the vaccine hoax, for their families are deeply threatened, too, but they may not be aware of it or that they have been folded into agency structures by the pharmaceutical industry (indistinguishable from the bankers and oil companies) that would make them agents of death for their country with the declaration of a “pandemic” emergency or “bio-terrorist” attack. It is completely clear now that he terrorism/bioterrorism structures are scams so that any actions taken to “protect” this country using those laws would in fact be what threatens the existence of Americans.
It was aspirin that killed millions in 1918-19. Now it is mandated and unknown, untested vaccines with banned adjuvants in them that threaten the country with millions of deaths. [F4] At the same time, the CDC is holding 500,000 mega-coffins, built to be incinerated, on its property outside Atlanta. Not to put to fine a point on this, but it’s clear now that the CDC should not be involved in any way with public health.—Thanks to the Freedom of Information Act (FOIA), we know that vaccines are not a miracle of modern medicine. Any medical or government authority which insists vaccines prevent diseases is either ignorant of government documents (and endless studies) revealing the exact opposite or of the CDC’s attempts to hide the truth about vaccines from the public, or means harm to the public.
Milk –Breast Cancer Remedy—
Breast cancer is the most common cancer with an expected 1.4 million females being diagnosed with breast cancer in 2010 (Dong and others 2011; Duarte and others 2011). Experimental studies in animals and in vitro have shown protective effects of CLA against carcinogenesis in the mammary gland, potentially by inhibiting the cyclooxygenase-2 or the lipo-oxygenase pathway or by inducing the expression of apoptotic genes (Kelley and others 2007). In the Nurses’ Health Study II (Cho and others 2003), women with a high consumption of low-fat dairy products during their premenopausal years had a nonsignificant negative association with breast cancer risk. The findings of a meta-analysis of prospective cohort studies indicated that low-fat, but not high-fat, dairy consumption is associated with a reduced risk of breast cancer and is broadly in line with current evidence (Dong and others 2011).
Calcium intake has been inversely correlated with reducing the risk of breast cancer in some prospective studies (Knekt and others 1996; McCullough and others 2005). The finding of a population-based prospective cohort study showed a negative association between pre- and postmenopausal breast cancer risk and calcium intake. The results also demonstrated a 50% decreased risk for premenopausal breast cancer among women consuming 25 g of white cheese per day compared to women consuming less than 6 g/d (Hjartåker and others 2010). Calcium may exert its anticarcinogenic properties through several mechanisms: a) decreasing cell proliferation and inducing differentiation of mammary cells (Cui and Rohan 2006), b) probably binding and neutralizing fatty acids and mutagenic bile acids (Parodi 2005), and c) decreasing fat-induced epithelial hyperproliferation in rodent mammary glands.—-Metabolically, calcium is closely related to vitamin D, which also has been shown to influence breast carcinogenesis, and it has been suggested that some of the anticarcinogenic effect of calcium may be mediated through vitamin D (Cui and Rohan -). For instance, calcium may play an important role in 1, 25(OH)2D (the active form of vitamin D) induced apoptosis (Sergeev 2005). A recent meta-analysis has provided evidence that vitamin D and calcium intakes protect against breast cancer, particularly in premenopausal women (Chen and others 2010). The results of a large cohort study showed women with the highest intake of dietary calcium (>1250 mg/d) were at a lower risk of breast cancer than those reporting ≤500 mg/d [RR, 0.80; 95% confidence interval (95% CI), 0.67 to 0.95; P = 0.02] (McCullough and others 2005). The results of latter study support the theory that dietary calcium and/or some other components in dairy products may reduce the risk of postmenopausal breast cancer.—-Proteins and peptides existing in milk have been reported to be cancer preventive agents (Knekt and others 1996; Tsuda and others 2000; Wakabayashi and others 2006; Rodrigues and others 2008). For example, lactoferrin (LF) that is also known for its inhibitory action on cell proliferation as well as for its anti-inflammatory and antioxidant abilities (Tsuda and others 2002; Ward and others 2005; Rodrigues and others 2008; Iigo and others 2009. LF is an iron-binding glycoprotein from the transferrin family. In vivo studies showed that oral administration of bovine LF to rodents significantly reduces chemically induced carcinogenesis in different organs (breast, esophagus, tongue, lung, liver, colon, and bladder) and inhibits angiogenesis (Tsuda and others 2002; Iigo and others 2009). Although the mechanisms of LF action are still not fully understood, there is evidence representing its ability to interact with some receptors, as well as to modulate genetic expression of several molecules that are vital to the cell cycle and apoptosis machinery.
Promotion of hair growth by Rosmarinus officinalis leaf extract.
Phytother Res. 2013 Feb;27(2):212-7
Authors: Murata K, Noguchi K, Kondo M, Onishi M, Watanabe N, Okamura K, Matsuda H
Topical administration of Rosmarinus officinalis leaf extract (RO-ext, 2 mg/day/mouse) improved hair regrowth in C57BL/6NCrSlc mice that experienced hair regrowth interruption induced by testosterone treatment. In addition, RO-ext promoted hair growth in C3H/He mice that had their dorsal areas shaved. To investigate the antiandrogenic activity mechanism of RO-ext, we focused on inhibition of testosterone 5α-reductase, which is well recognized as one of the most effective strategies for the treatment of androgenic alopecia. RO-ext showed inhibitory activity of 82.4% and 94.6% at 200 and 500 µg/mL, respectively. As an active constituent of 5α-reductase inhibition, 12-methoxycarnosic acid was identified with activity-guided fractionation. In addition, the extract of R. officinalis and 12-methoxycarnosic acid inhibited androgen-dependent proliferation of LNCaP cells as 64.5% and 66.7% at 5 µg/mL and 5 μM, respectively. These results suggest that they inhibit the binding of dihydrotestosterone to androgen receptors. Consequently, RO-ext is a promising crude drug for hair growth.—PMID: 22517595 [PubMed – indexed for MEDLINE
Table Reference Of AntiCancer effects of Milk ( real Milk Upasteurized)
Table 1. Selected publications on cancer prevention of indigenous compounds of milk and milk product
Type of cancer
Type of product
Women and men who consumed ≥2 servings of cultured milk per day had a 38% lower risk of bladder cancer than did those who never consumed cultured milk.
A significantly (53%) decreased risk of death due to bladder cancer among Japanese men and women who consumed milk almost every day in comparison with those who consumed 2 servings milk/month.
Protective effect of natural fermented milk (NFM) containing Lactobacillus spp. and Bifidobacterium spp. against cancer of the liver.
Regular daily consumption of milk in childhood may reduce colorectal cancer incidence. Participation in school milk programs in New Zealand was associated with a 2.1% reduction (95% C I: 0 .7, 3. 5) in the odds ratio for colorectal cancer for every 100 half-pint bottles drunk (1 half-pint bottle = 284 mL).
Milk and dairy foods
Swedish women who consumed ≥4 servings of high-fat dairy foods per day (including whole milk, full-fat cultured milk, cheese, cream, sour cream, and butter) had a multivariate rate ratio of colorectal cancer of 0.59 (95% CI: 0.44, 0.79; P for trend = 0.002) when compared with the women who consumed <1 serving per day. Each increment of 2 servings of high-fat dairy foods per day corresponded to a 13% reduction in the risk of colorectal cancer.
The reduced risk was most pronounced at the higher levels of intake (equivalent to 2–3 glasses of milk per day).
Consumption of less than a quarter of a glass of milk every day had 15% more risk of colorectal cancer than those who consumed ≥1 glass of milk a day.
12% reduction in colon cancer risk with each 500-mL increase in milk intake.
17% reduction in colorectal cancer incidence with the ingestion of ricotta cheese greater than 25 mg per day.
The probiotic strains of E. faecium RM11 and L. fermentum RM28 also triggered antiproliferation of colon cancer cells at the rates of 21% to 29% and 22% to 29%, respectively.
Fermented dairy milks
Each increment of 2 servings of high-fat dairy foods per day corresponded to a 13% reduction in the risk of colorectal cancer (multivariate rate ratio: 0.87; 95% CI: 0.78, 0.96).
High-fat dairy foods
Consumption of more than a glass of milk (≥250 g) per day had a 15% reduced risk of developing colorectal cancer (relative risk 0.85, 95% CI 0.78 to 0.94), compared to those who consumed <70 g/d.
Calcium intakes of 1200 to 1500 mg/d, or 4 servings of dairy per day, seem to be the most protective against colon cancer.
A 50% decreased risk for premenopausal breast cancer among women consuming 25 g of white cheese per day compared to women consuming less than 6 g/d. In the analysis, corrected for measurement errors, an increase of 5 g cheese decreased the risk 24%.
Women with the highest intake of dietary calcium (>1250 mg/d) were at a lower risk of breast cancer than those reporting ≤500 mg/d.
[F1]Nanoparticles stop any cell replication or cellular respiration
[F2]DID NOT WANT TO RELEASE THE BIOWARFARE AGENTS IN THERE VACCINES
[F3]FULL DISCLOSURE—WOW DOES THIS MAKE YOU FEEL ALL COMFORTABLE
[F4]And I would question this finding as well —if they are using adjuvants now they may have then as well or a solution that people had reacted toJune 22, 2021 at 09:01 #2664
Bacteria Adapt and Evade Nanosilver’s Sting
May 8, 2013 — Researchers from UNSW have cautioned that more work is needed to understand how micro-organisms respond to the disinfecting properties of silver nano-particles, increasingly used in consumer goods, and for medical and environmental applications.—Although nanosilver has effective antimicrobial properties against certain pathogens, overexposure to silver nano-particles can cause other potentially harmful organisms to rapidly adapt and flourish, a UNSW study reveals.–This result, published in the journal Small, could have wide-reaching implications for the future use of nanosilver as an antimicrobial agent with biomedical and environmental applications.—“We found an important natural ability of a widely occurring bacteria to adapt quite rapidly to the antimicrobial action of nanosilver. This is the first unambiguous evidence of this induced adaptation,” says co-author Dr Cindy Gunawan, from the UNSW School of Chemical Engineering.—Using an experimental culture, UNSW researchers observed that nanosilver was effective in suppressing a targeted bacteria (Escherichia coli), but that its presence initiated the unexpected emergence, adaptation and abnormally fast growth of another bacteria species (Bacillus).—The efficacy of nanosilver to suppress certain disease-causing pathogens has been well-documented, and as a result, it has become widely used in medicine to coat bandages and wound dressings. It also has environmental uses in water and air purification systems, and is used in cosmetics and detergents, and as a surface coating for things like toys and tupperware.—But the researchers say this exploitation of nanosilver’s antimicrobial properties have “gained momentum due in part to a lack of evidence for the potential development of resistant microorganisms.”—“Antimicrobial action of nanosilver is not universal and the widespread use of these products should take into consideration the potential for longer-term adverse outcomes,” says Gunawan.—The researchers say these adverse impacts could be more pronounced given the near-ubiquitous nature of the Bacillus bacteria, which originate from airborne spores, and because the resistance trait can potentially be transferred to the genes of other micro-organisms.—“For the medical use of nanosilver, this implies the potential for reduced efficacy and the development of resistant populations in clinical settings,” says co-author Dr Christopher Marquis, a senior lecturer from the UNSW School of Biotechnology and Biomolecular Sciences.—“This work suggests caution in the widespread use of nanosilver and the requirement for much deeper research into the antimicrobial mechanisms, the extent of adaptability and the molecular basis or genetics of cell defence against the antimicrobial activity.”—–Story Source-The above story is reprinted from materials provided by University of New South Wales. —Journal Reference–Cindy Gunawan, Wey Yang Teoh, Christopher P. Marquis, Rose Amal. Induced Adaptation ofBacillus sp.to Antimicrobial Nanosilver. Small, 2013; DOI: 10.1002/smll.201300761
Whole Walnuts and Their Extracted Oil Improve Cardiovascular Disease Risk
Walnuts. Consumption of whole walnuts or their extracted oil can reduce cardiovascular risk through a mechanism other than simply lowering cholesterol, according to new research. —May 8, 2013 — Consumption of whole walnuts or their extracted oil can reduce cardiovascular risk through a mechanism other than simply lowering cholesterol, according to a team of Penn State, Tufts University and University of Pennsylvania researchers.–“We already know that eating walnuts in a heart-healthy diet can lower blood cholesterol levels,” said Penny Kris-Etherton, Distinguished Professor of Nutrition, Penn State. “But, until now, we did not know what component of the walnut was providing this benefit. Now we understand additional ways in which whole walnuts and their oil components can improve heart health.”—-In a randomized-controlled trial, the researchers gave 15 participants with elevated blood cholesterol one of four treatments — either 85 grams of whole walnuts, 6 grams of skin, 34 grams of defatted nutmeat, or 51 grams of oil. The team evaluated biochemical and physiological responses in the participants before the treatments were administered and again 30 minutes, one hour, two hours, four hours and six hours after administering the treatments. The researchers repeated this process for each of the remaining three treatments.—-Results — which will appear in the June 1 issue of the Journal of Nutrition and are now online — showed that a one-time consumption of the oil component in walnuts favorably affected vascular health. In addition, consumption of whole walnuts helped HDL — good cholesterol — perform more effectively in transporting and removing excess cholesterol from the body.–“Our study showed that the oil found in walnuts can maintain blood vessel function after a meal, which is very important given that blood vessel integrity is often compromised in individuals with cardiovascular disease,” said Claire Berryman, graduate student in nutritional sciences, Penn State. “The walnut oil was particularly good at preserving the function of endothelial cells, which play an important role in cardiovascular health.”—According to the researchers, walnuts contain alpha-linolenic acid, gamma-tocopherol and phytosterols, which may explain the positive effects of the walnut oil treatment.–“Implications of this finding could mean improved dietary strategies to fight heart disease,” said Berryman. “The science around HDL functionality is very new, so to see improvements in this outcome with the consumption of whole walnuts is promising and worth investigating further.” Further studies are needed to determine the mechanisms that account for cardiovascular disease risk reduction with walnut consumption, according to Kris-Etherton “Our study indicates that simple dietary changes, such as incorporating walnuts and/or their oil in a heart healthy diet, may reduce the risk of heart disease,” she said Other authors on the paper include Jessica Grieger and Sheila West of Penn State, Oliver Chen and Jeffrey Blumberg of Tufts University, and George Rothblat and Sandhya Sankaranarayanan of the University of Pennsylvania. The California Walnut Board funded this research— Story Source-The above story is reprinted from materials provided by Penn State. The original article was written by Sara LaJeunesse.
Adding Walnuts To Good Diet May Help Older People Improve Motor And Behavioral Skills
Adding a moderate, but not high, amount of walnuts to an otherwise healthy diet may help older individuals improve performance on tasks that require motor and behavioral skills.—Apr. 25, 2009 — Adding a moderate, but not high, amount of walnuts to an otherwise healthy diet may help older individuals improve performance on tasks that require motor and behavioral skills, according to an animal model study by Agricultural Research Service (ARS)-funded scientists. Walnuts contain polyphenols and other antioxidants and essential fatty acids.–The study was conducted by researchers with the Jean Mayer USDA Human Nutrition Research Center on Aging (HNRCA) at Tufts University in Boston, Mass.—Neuroscientist James Joseph, psychologist Barbara Shukitt-Hale and coauthors Lauren Willis and Vivian Cheng reported the study in the British Journal of Nutrition. They are with the HNRCA’s Neuroscience Laboratory. ARS is the principal intramural scientific research agency of the U.S. Department of Agriculture.—The aging brain undergoes many changes that can result in altered or impaired neuronal functioning. Such disruption can be attributed in part to alterations in “synaptic plasticity,” or the ability of the connections between neurons to change in strength and function, and also by increased oxidative damage to neural tissue. In aged rodents, these impairments are seen as poor performance on age-sensitive tests of balance, coordination, and “spatial” working memory. –For the study, weight-matched, aged rats were randomly assigned to one of four diet groups. For eight weeks, the rats were fed special chow mixes that contained either 2 percent, 6 percent or 9 percent walnuts-or no walnuts-before undergoing motor and memory tests. For comparison, the 6 percent walnut study diet is equivalent to a human eating 1 ounce, or about 7 to 9 walnuts, a day. That counts as both a 2-ounce equivalent from the “meat and beans group” and 2 teaspoons toward a daily allowance of dietary oil, as described at MyPyramid.gov.–The study found that in aged rats, the diets containing 2 percent or 6 percent walnuts were able to improve age-related motor and cognitive shortfalls, while the 9 percent walnut diet impaired reference memory. Walnuts, eaten in moderation, appear to be among other foods containing polyphenols and bioactive substances that exhibit multiple effects on neural tissue, according to the researchers. –The above story is reprinted from materials provided by USDA/Agricultural Research Service. —
Basic Anti-Biofilm and Anti NanoParticle Approach-Morgellons
ANTI-BIOFILM PROTOCOL KEYS
Humans, as a rule, are a host to various “friendly” bacteria and viruses. We carry them around with us in tissues and biofilms and they normally exist in balance within our bodies. The body is made of an estimated 100 trillion cells. However, humans host between 1.5-2 quadrillion and approximately 1,500 different types of bacteria. “The number of bacteria living within the body of the average healthy adult human are estimated to outnumber human cells 10 to 1!”
Although more research is needed as to everything we get from this arrangement, we know that:
We need their enzymes for various body processes
They communicate with the immune system
They prevent growth of harmful species
They regulate the development of the gut
They produce vitamins (such as biotin and vitamin K)
They ferment unused energy substrates
They produce needed hormones
They assist us in the biofilms in the gut, respiratory system, urinary tract, mucus membranes of the reproductive tract.
Is it realistic or even possible to try and remove all biofilm from the body, when we have always hosted biofilm communities? No, because we are designed to live in harmony with one another, unless infection and other problems create an imbalance. Humans are “symbiotes” with various organisms.
It is when Lyme Disease and co-infections and strong antibiotics enter the picture that the normal, symbiotic biofilm arrangement in the body can most likely be tipped over the edge into more pathogenic (“bad”) biofilm communities.
Is it possible that the good guy, friendly biofilms can be “brought to the dark side,” and go from good to pathogenic? We think this is certainly possible, particularly in chronic illness, and especially in the theory of “Pleomorphism.” Pleomorphists believe that internal microorganisms can change from avirulent, benign forms into a potentially virulent, pathogenic, forms.” Click for article and reference. The goal then, is to re-establish the healthy balance and symbiotic relationship to the natural biofilm and organisms in the body.
Think of it this way: When you are healthy, your body exists as a balanced series of communities of human cells carrying thousands of species of organisms that help you and one another survive. When you begin to kill off these species, especially with toxic chemicals like antibiotics, they will in effect fight back to survive, turning against the host or one another in chronic diseasJune 22, 2021 at 09:01 #2665
Scientists Uncover How Grapefruits Provide a Secret Weapon in Medical Drug Delivery
May 21, 2013 — Lipids (right panel first three tubes) derived from grapefruit. GNVs can efficiently deliver a variety of therapeutic agents, including DNA, RNA (DIR-GNVs), proteins and anti-cancer drugs (GNVs-Drugs) as demonstrated in this study.—Grapefruits have long been known for their health benefits, and the subtropical fruit may revolutionize how medical therapies like anti-cancer drugs are delivered to specific tumor cells.—University of Louisville researchers have uncovered how to create nanoparticles using natural lipids derived from grapefruit, and have discovered how to use them as drug delivery vehicles. UofL scientists Huang-Ge Zhang, D.V.M., Ph.D., Qilong Wang, Ph.D., and their team today (May 21, 2013), published their findings in Nature Communications.—-“These nanoparticles, which we’ve named grapefruit-derived nanovectors (GNVs), are derived from an edible plant, and we believe they are less toxic for patients, result in less biohazardous waste for the environment, and are much cheaper to produce at large scale than nanoparticles made from synthetic materials,” Zhang said.—-The researchers demonstrated that GNVs can transport various therapeutic agents, including anti-cancer drugs, DNA/RNA and proteins such as antibodies. Treatment of animals with GNVs seemed to cause less adverse effects than treatment with drugs encapsulated in synthetic lipids.—“Our GNVs can be modified to target specific cells — we can use them like missiles to carry a variety of therapeutic agents for the purpose of destroying diseased cells[F1],” he said. “Furthermore, we can do this at an affordable price.”—The therapeutic potential of grapefruit derived nanoparticles was further validated through a Phase 1 clinical trial for treatment of colon cancer patients. So far, researchers have observed no toxicity in the patients who orally took the anti-inflammatory agent curcumin encapsulated in grapefruit nanoparticles.——–The UofL scientists also plan to test whether this technology can be applied in the treatment of inflammation related autoimmune diseases like rheumatoid arthritis.—A Common Sense Approach—–Zhang said he began this research by considering how our ancestors selected food to eat. –“The fruits and vegetables we buy from the grocery today were passed down from generation to generation as favorable and nutritious for the human body. On the flip side, outcomes were not favorable for our ancestors who ate poisonous mushrooms, for example,” he said. “It made sense for us to consider eatable plants as a mechanism to create medical nanoparticles as a potential non-toxic therapeutic delivery vehicle.”[F2]—In addition to grapefruit, Zhang and his team analyzed the nanoparticles from tomatoes and grapes. Grapefruits were chosen for further exploration because a larger quantity of lipids can be derived from this fruit.–Story Source–The above story is reprinted from materials provided by University of Louisville.
Herbals and Foods For Anti Cancer for Intestines
Water extracts of tree Hypericum sps. protect DNA from oxidative and alkylating damage and enhance DNA repair in colon cells.
Food Chem Toxicol. 2013 Jan;51:80-6
Authors: Ramos AA, Marques F, Fernandes-Ferreira M, Pereira-Wilson C
Diet may induce colon carcinogenesis through oxidative or alkylating DNA damage. However, diet may also contain anticarcinogenic compounds that contribute to cancer prevention. DNA damage prevention and/or induction of repair are two important mechanisms involved in cancer chemoprevention by dietary compounds. Hypericum sps. are widely used in traditional medicine to prepare infusions due to their beneficial digestive and neurologic effects. In this study, we investigated the potential of water extracts from three Hypericum sps. and some of their main phenolic compounds to prevent and repair oxidative and alkylating DNA damage in colon cells. The results showed that water extracts of Hypericum perforatum-( St John’sWort), Hypericum androsaemum- (Tutsan), Hypericum undulatum- ( Wavy St John’s Wort ), quercetin and rutin have protective effect against oxidative DNA damage in HT29 cells. Protective effect was also observed against alkylating DNA damage induced by methyl-methanesulfonate ( MSM), except for H. androsaemum. With regard to alkylating damage repair H. perforatum, H. androsaemum and chlorogenic acid increased repair of alkylating DNA damage by base excision repair pathway. No effect was observed on nucleotide excision repair pathway. Antigenotoxic effects of Hypericum sps. may contribute to colon cancer prevention and the high amount of phenolic compounds present in Hypericum sps. play an important role in DNA protective effects.-PMID: 23000446 [PubMed – in process]
Remedy—Simplest way of utilizing any form of St John’s would be in tea form —after a meal—would increase protection and assist in the reversing of damage to colon area—in making the tea use 1 tablespoon in a 2-3 pint pot of distilled water—bring to a boil —rolling boil is good or when starting shut off stove after the boiling occurs this will reduce heat and allow to simmer—drank before bed with other herbs like lemon balm or Melissa or lavender may for some induce relaxation and sleep
Apigenin and its impact on gastrointestinal cancers.
Mol Nutr Food Res. 2013 Jan;57(1):126-44
Authors: Lefort ÉC, Blay J
Apigenin (4′,5,7-trihydroxyflavone, 5,7-dihydroxy-2-(4-hydroxyphenyl)-4H-1-benzopyran-4-one) is a flavonoid found in many fruits, vegetables, and herbs, the most abundant sources being the leafy herb parsley and dried flowers of chamomile. Present in dietary sources as a glycoside, it is cleaved in the gastrointestinal lumen to be absorbed and distributed as apigenin itself. For this reason, the epithelium of the gastrointestinal tract is exposed to higher concentrations of apigenin than tissues at other locations. This would also be true for epithelial cancers of the gastrointestinal tract. We consider the evidence for actions of apigenin that might hinder the ability of gastrointestinal cancers to progress and spread. Apigenin has been shown to inhibit cell growth, sensitize cancer cells to elimination by apoptosis, and hinder the development of blood vessels to serve the growing tumor. It also has actions that alter the relationship of the cancer cells with their microenvironment. Apigenin is able to reduce cancer cell glucose uptake, inhibit remodeling of the extracellular matrix, inhibit cell adhesion molecules that participate in cancer progression, and oppose chemokine signaling pathways that direct the course of metastasis into other locations. As such, apigenin may provide some additional benefit beyond existing drugs in slowing the emergence of metastatic disease.–PMID: 23197449 [PubMed – in process]
Herbal Extract Rhodiola rosea Boosts Fruit Fly Lifespan by Nearly 25 Percent
June 18, 2013 — The herbal extract of a yellow-flowered mountain plant long used for stress relief was found to increase the lifespan of fruit fly populations by an average of 24 percent, according to UC Irvine researchers.—But it’s how Rhodiola rosea, also known as golden root, did this that grabbed the attention of study leaders Mahtab Jafari and Sam Schriner. They discovered that Rhodiola works in a manner completely unrelated to dietary restriction and affects different molecular pathways.—This is significant, said Jafari, associate professor of pharmaceutical sciences, because dietary restriction is considered the most robust method of improving lifespan in laboratory animals, and scientists have been scrambling to identify compounds that can mimic its effects.—“We found that Rhodiola actually increases lifespan on top of that of dietary restriction,” Jafari said. “It demonstrates that Rhodiola can act even in individuals who are already long-lived and healthy. This is quite unlike resveratrol, which appears to only act in overfed or unhealthy individuals.”—The researchers proved this by putting flies on a calorie-restricted diet. It has been shown that flies live longer when the amount of yeast they consume is decreased. Jafari and Schriner expected that if Rhodiola functioned in the same manner as dietary restriction, it would not work in these flies. But it did. They also tested Rhodiola in flies in which the molecular pathways of dietary restriction had been genetically inactivated. It still worked.[F3]—-Not only did Rhodiola improve lifespan an average of 24 percent in both sexes and multiple strains of flies, but it also delayed the loss of physical performance in flies as they aged and even extended the lives of old flies. Jafari’s group previously had shown that the extract decreased the natural production of reactive oxygen species molecules in the fly mitochondria and protected both flies and cultured human cells against oxidative stress.—Jafari and Schriner, an assistant project scientist in Jafari’s laboratory, are not claiming that Rhodiola supplements will enable humans to live longer, but their discovery is enhancing scientific understanding of how supplements believed to promote longevity actually work in the body.–Rhodiola has already shown possible health benefits in humans, such as decreasing fatigue, anxiety and depression; boosting mood, memory and stamina; and preventing altitude sickness. Grown in cold climates at high elevations, the herb has been used for centuries by Scandinavians and Russians to reduce stress. It’s also thought to have antioxidant properties.—Jafari’s research group is currently exploring the plant’s potential to kill cancer cells, improve Alzheimer’s disease and help stem cells grow.—Rhodiola is readily available online and in health food stores. Jafari, though, has analyzed several commercial products and found them to not contain sufficient amounts of the reputed active compounds — such as rosavin and salidroside — that characterize high-quality products.—Story Source-The above story is reprinted from materials provided by University of California – Irvine. –Journal Reference—Samuel E. Schriner, Kevin Lee, Stephanie Truong, Kathyrn T. Salvadora, Steven Maler, Alexander Nam, Thomas Lee, Mahtab Jafari. Extension of Drosophila Lifespan by Rhodiola rosea through a Mechanism Independent from Dietary Restriction. PLoS ONE, 2013; 8 (5): e63886 DOI: 10.1371/journal.pone.0063886
Testosterone Therapy May Help Improve Pain in Men With Low Testosterone
June 17, 2013 — Testosterone therapy is associated with decreased pain perception in men with low testosterone levels related to opioid (narcotic) pain relievers (analgesics), a new study finds. —The results were presented Monday at The Endocrine Society’s 95th Annual Meeting in San Francisco.—“In this study, we attempted to determine whether testosterone replacement improves pain perception and tolerance, and quality of life in men with low testosterone levels due to narcotic analgesics,” said the study’s lead author Shehzad Basaria, MD, Medical Director, Section of Men’s Health, Aging, & Metabolism at Brigham & Women’s Hospital, Harvard Medical School, in Boston, MA. “We found that testosterone administration in these men was associated with a greater reduction in several measures of pain sensitivity during laboratory pain testing compared with men who were on placebo.”—Opioids belong to a class of pain-reducing drugs that are used to relieve chronic pain from injuries, surgery and cancer treatment. These drugs include morphine, codeine, fentanyl and oxycodone, and are among the most frequently prescribed medications in the United States today.–In addition to being highly addictive, opioid use is associated with a number of side effects, including suppression of the hypothalamic-pituitary-gonadal axis in both women and men, resulting in decreased testosterone production. Low testosterone, in turn, can result in sexual dysfunction, decreased muscle mass, increased fat mass and decreased quality of life.–Previous animal research has demonstrated that castration of rodents is associated with increased pain perception while testosterone replacement reduces pain perception, suggesting an analgesic effect of this sex steroid. Whether these beneficial effects can be replicated in humans, however, remained unclear.—In this study, investigators found that, compared to placebo, testosterone therapy significantly improved pain perception and tolerance during laboratory pain testing. Testosterone therapy also improved some aspects of quality of life.—“If larger studies confirm these findings, testosterone therapy in this patient population may be beneficial in improving pain perception,” Basaria said.—The study included 84 men ages 18-64 years old with opioid-induced testosterone deficiency. Their average age was 49 years. Of this group, 65 participants completed the study. Investigators randomly assigned participants to receive either testosterone gel, applied to the skin, or placebo, for 14 weeks. Thirty-six men received the testosterone gel, and 29 received a placebo.—At the beginning of the study, and then again at 14 weeks, the investigators assessed pain measures and quality-of-life parameters.—Solvay (now Abbott) Pharmaceuticals, Inc. funded the study.
#1. Biological Mechanisms Behind Statin Side Effects: Duane Graveline, the M.D. who goes by the nickname “Spacedoc” has done everything he can to inform the public of the dangers of statin drugs. Dr. Graveline is a former NASA astronaut and Air Force flight surgeon. His reasons for taking on the challenge of debunking statins are very personal, because he developed global transient amnesia while taking Lipitor, an unnerving experience that deprives you of all memory of events in your life looking back several decades.
Since then, he has conducted extensive research on the biological mechanisms behind the diverse severe side effects of statin drugs. The link above is but one branch of a vast network of frightening yet informative information about the potentially devastating long-term effects of statin therapy.
#2. Statins and Nerve Damage: The People’s Pharmacy is a wonderful forum that allows patients to share their experience. This is one of many examples of adverse side effects of statin drugs available on that site.
#3. Statins and Pregnancy: Like thalidomide, statin drugs are a class X drug with regard to pregnancy, meaning they are contraindicated and should NOT be taken by pregnant women. They can cause significant damage to the nervous system of a developing embryo.
#4. New York Times Exposes Potential for Misdiagnosis Caused by Statin Use: This article points out some of the severe side effects statins can cause, and illustrates with a poignant story about a woman from Kansas. She had been taking a statin for years to reduce her cholesterol. Over that same time period, she experienced chronic muscle pain which neither she nor her doctor attributed to the statin therapy. It even led to a useless shoulder operation.—Her problem eventually escalated into skin lesions caused by a reaction to toxic protein by-products released by her disintegrating muscles. She was given an antifungal to treat the skin lesions, another misdiagnosis. But the antifungal interacted with the statins to further increase the severity of her muscle disorders. Three months later, she could barely stand, and her pulmonary muscles were so weak she couldn’t breathe. She died shortly thereafter.
#5. The International Network of Cholesterol Skeptics (THINCS): Dr. Uffe Ravnskov has campaigned tirelessly to fight the myth that high cholesterol is damaging to your health. This web page contains valuable information about all the ways in which cholesterol keeps you healthy.
#6. The ASEPSIS Trial: The statin industry continues to claim that statins protect against sepsis because of several retrospective studies that show that those who take statins have less risk than those who don’t.—What these studies are proving is that cholesterol protects from sepsis. The media keep saying that what is needed is a double-blind placebo controlled study, but they already have one they could talk about. It’s just that you can’t find out anything about it except that it was completed in January, 2008. You can read my take on statins and sepsis here.
#7. Statins Increase Pneumonia Risk: Just as for sepsis, the statin industry likes to claim that statins improve your chances against pneumonia. But a double-blind placebo controlled study proved them wrong. The risk for pneumonia that required hospitalization was increased by 61 percent in the statin group compared to the controls.
#8. WebMD Article on Muscle Pain and Weakness: This relatively benign article on WebMD provoked a firestorm of responses; each comment tells the story of another tragedy unfolding. To understand the biological mechanism behind the process by which statin drugs destroy muscles, see this link.—- Here’s a typical comment from that site—-“I was prescribed Crestor 20mg 2 weeks ago with cholesterol level 7.6. First time on any medication. After approx. 4 days I started to experience severe muscle pain, thigh, buttocks, arms, legs to the extent that I can hardly get out of bed in the morning. Have been back to Dr. who advised stopping tablets. Have been off them for 3 days, very little difference. I am hoping these pains will go away soon. I will never take a statin drug again – would rather take the healthy option, diet and exercise and take the risk. Have never felt so bad. Usually very healthy, fit person.”
#9. Statins and Heart Failure: Dr. Peter Langsjoen believes that statin drugs greatly increase your risk of heart failure. I have argued why this might be true here.
#10. JUPITER Trial and Diabetes: The JUPITER trial, which was terminated prematurely after less than two years, was widely heralded for showing that statins reduce the risk of heart attacks in the short term for people with high levels of C-reactive protein but without high cholesterol.—-However, little note was made of the fact that the JUPITER trial also showed a 25 percent increased risk to new-onset diabetes in the treatment group. Since diabetes is a strong risk factor for heart disease, one wonders how the trial would have turned out if it had been allowed to run to completion.
#11. High Cholesterol and Alzheimer’s: This is the article that inspired the Newsweek article, #12 below, with the lead story that statins “protect from” Alzheimer’s — which is the exact opposite of the truth about statins and Alzheimer’s.—-The only relationship between high cholesterol and Alzheimer’s the authors could find was if they looked back 30 years. What they’re not saying is that, in the intervening years, cholesterol levels fell for those who later developed Alzheimer’s. While no one has said exactly why their levels might have fallen, statin drugs are a good bet.
#12. Glowing Newsweek Article on Statins:This article illustrates how thoroughly the statin industry has succeeded in brainwashing the media into believing that black is white. The lead story is that statins protect against Alzheimer’s. If you have read my essay on statins and Alzheimer’s, you will think otherwise.
Script of the Show July 13 2013
PICA—EATING DISORDER FROM MINERAL DEFICIENCY
Formula-feeding linked to metabolic stress and increased risk of later disease
Zanthoxylum Avicennae-Prickly Ash Leaves or Seeds
Getting Enough Sleep Could Help Prevent Type 2 Diabetes
Timing of Calcium and Vitamin D Supplementation May Affect How Bone Adapts to Exercise
Aspirin May Fight Cancer by Slowing DNA Damage
PICA—EATING DISORDER FROM MINERAL DEFICIENCY
A person afflicted with pica has a persistent craving for a substance that is not commonly considered to be food. The substances that are craved and ingested tend to vary with the person’s age. Children with pica may eat glue, animal droppings, sand, insects, leaves, or gravel. Adolescents and adults may consume clay, laundry detergent, or soil.
Although the cravings associated with pica may be related to vitamin or mineral deficiencies, ingesting non-foods can cause serious medical complications such as intestinal obstruction, intestinal perforation, infections, or lead poisoning
Pica (/ˈpaɪkə/ PY-kə) is characterized by an appetite for substances largely non-nutritive, such as clay, chalk, dirt, or sand. For these actions to be considered pica, they must persist for more than one month at an age where eating such objects is considered developmentally inappropriate. There are different variations of pica, as it can be from a cultural tradition, acquired taste or a neurological mechanism such as an iron deficiency, or chemical imbalance. It can lead to intoxication in children which can result in an impairment in both physical and mental development. In addition, it can also lead to surgical emergencies due to an intestinal obstruction as well as more subtle symptoms such as nutritional deficiencies and parasitosis. Pica has been linked to mental disorders and they often have psychotic comorbidity. Stressors such as maternal deprivation, family issues, parental neglect, pregnancy, poverty, and a disorganized family structure are strongly linked to pica.
Signs and symptoms
Chalky stone composed of kaolinite with traces of quartz ingested by a person with pica.
Pica is the consumption of substances with no significant nutritive value such as earth or ice. Subtypes are characterized by the substance eaten for example:
Amylophagia (consumption of starch)
Coprophagy (consumption of feces)
Geophagy (consumption of soil, clay, or chalk)
Hyalophagia (consumption of glass)
Consumption of dust or sand has been reported among iron-deficient patients.
Lithophagia (a subset of geophagia, consumption of pebbles or rocks)
Mucophagia (consumption of mucus)
Odowa (soft stones eaten by pregnant women in Kenya)
Consumption of paint.
Pagophagia (pathological consumption of ice)
Self-cannibalism (rare condition where body parts may be consumed; see also Lesch-Nyhan syndrome)
Trichophagia (consumption of hair or wool)
Urophagia (consumption of urine)
Xylophagia (consumption of wood or paper)
This pattern of eating should last at least one month to fit the diagnosis of pica.
There is no single test that confirms pica. However, because pica can occur in people who have lower than normal nutrient levels and poor nutrition (malnutrition), the health care provider should test blood levels of iron and zinc. Hemoglobin can also be checked to test for anemia. Lead levels should always be checked in children who may have eaten paint or objects covered in lead-paint dust. The health care provider should test for infection if the person has been eating contaminated soil or animal waste
People who consume animal products usually do not get afflicted with this unless there are other imbalances —this is usually those on vegetarian Diet —will consume things that are unusal—this is again a sign of starvation—lack of nutrients or minerals—so one way to avoid this is to ge adequate nutrients—if not in Diet then in supplementation—or in the consumption of veges or fruits that are fermented to yield a higher mineral and nutrient uptake due to the fibre being broken down releasing the vitality of it’scompliment—People who crave clay usually are trying to fulfill mineral loss—In some cases a Iron ( gotten from meats) and zinc ( animal Protein) restored to the diet will sometimes restore a person to normal—those who consume ice ( pagophagia)—are usually iron deficient—In some cases cannibalism has ben recorded as well or different animals from cats and dogs to mice ( while they are living)—The asian Diet is low in calcium and as a result leach the bones with vinegar to obtain there calcium levels—Magnesium deficiency will result in higher salt use —and Potassium deficiency will be a result of diuretic use –so if you utilize diuretics then make sure you use a dandelion tea it puts potassium back into the system—Minerals Like chromium if there is a deficiency the cravings will be for a pop ( soft drink)and high consumption of grain related carbs—
Cannibalism —would be an extreme case of pica as result of high mineral deficiency
Animals can show signs of this by consuming bones –wood-and cattle will sometimes gnaw on each others horns as well especially if they have a phosphorus need—
The term pica originates from the Latin word for magpie, a bird that is reputed for its unusual eating behaviors where they are known to eat almost anything. In 13th century Latin work, pica was referenced by the Greeks and Romans; however, it was not addressed in medical texts until 1563. In the southern United States in the 1800s, geophagia was a common practice among the slave population.[Geophagia is a form of pica in which the person consumes earthly substances such as clay, and is particularly prevalent to augment a mineral-deficient diet. Research on eating disorders in the 16th century to the 20th century suggests that during that time in history, pica was regarded more as a symptom of other disorders rather than its own specific disorder. Even today, what can be classified as pica behavior is a normative practice in some cultures as part of magical beliefs, healing methods, or religious ceremonies.
Formula-feeding linked to metabolic stress and increased risk of later disease
June 5th, 2013 in Chemistry / Biochemistry
New evidence from research suggests that infants fed formula, rather than breast milk, experience metabolic stress that could play a part in the long-recognized link between formula-feeding and an increased risk of obesity, type 2 diabetes and other conditions in adult life. —New evidence from research suggests that infants fed formula, rather than breast milk, experience metabolic stress that could play a part in the long-recognized link between formula-feeding and an increased risk of obesity, type 2 diabetes and other conditions in adult life. Credit: iStockphoto/Thinkstock—New evidence from research suggests that infants fed formula, rather than breast milk, experience metabolic stress that could play a part in the long-recognized link between formula-feeding and an increased risk of obesity, type 2 diabetes and other conditions in adult life.[F4] The study appears in ACS’ Journal of Proteome Research.—Carolyn Slupsky and colleagues explain that past research showed a link between formula-feeding and a higher risk for chronic diseases later in life. Gaps exist, however, in the scientific understanding of the basis for that link. The scientists turned to rhesus monkeys, stand-ins for human infants in such research, that were formula-fed or breast-fed for data to fill those gaps.
Their analysis of the monkeys’ urine, blood and stool samples identified key differences between formula-fed and breast-fed individuals. It also produced hints that reducing the protein content of infant formula might be beneficial in reducing the metabolic stress in formula-fed infants[F5]. “Our findings support the contention that infant feeding practice profoundly influences metabolism in developing infants and may be the link between early feeding and the development of metabolic disease later in life,” the study states.— The article is titled “Early Diet Impacts Infant Rhesus Gut Microbiome, Immunity, and Metabolism.”–More information: Early Diet Impacts Infant Rhesus Gut Microbiome, Immunity, and Metabolism, J. Proteome Res., Article ASAP. DOI: 10.1021/pr4001702—- Abstract
Epidemiological research has indicated a relationship between infant formula feeding and increased risk of chronic diseases later in life including obesity, type-2 diabetes, and cardiovascular disease. The present study used an infant rhesus monkey model to compare the comprehensive metabolic implications of formula- and breast-feeding practices using NMR spectroscopy to characterize metabolite fingerprints from urine and serum, in combination with anthropometric measurements, fecal microbial profiling, and cytokine measurements. Here we show that formula-fed infants are larger than their breast-fed counterparts and have a different gut microbiome that includes higher levels of bacteria from the Ruminococcus genus and lower levels of bacteria from the [F6]Lactobacillus genus. In addition, formula-fed infants have higher serum insulin coupled with higher amino acid levels, while amino acid degradation products were higher in breast-fed infants. Increases in serum and urine galactose and urine galactitol were observed in the second month of life in formula-fed infants, along with higher levels of TNFα, IFN-γ, IL-1β, IL-4, and other cytokines and growth factors at week 4. These results demonstrate that metabolic and gut microbiome development of formula-fed infants is different from breast-fed infants and that the choice of infant feeding may hold future health consequences.—-Provided by American Chemical Society
Zanthoxylum Avicennae-Prickly Ash Leaves or Seeds
[Study on the chemical components, antimicrobial and antitumor activities of the essential oil from the leaves of Zanthoxylum avicennae].
Zhong Yao Cai. 2012 Aug;35(8):1263-7
Authors: Zhang DS, Zhong QX, Song XM, Liu WJ, Wang J, Zhang QY
OBJECTIVE: To study the chemical constituents, antimicrobial activity and antitumor activity of the essential oil from Zanthoxylum avicennae.
METHODS: The essential oil from the leaves of Zanthoxylum avicennae was extracted by steam distillation. The components of the essential oil were separated and identified by GC-MS.–RESULTS: 72 components were identified and accounted for 98.15% of the all peak area. The essential oil exhibited strong antitumor activity against K-562 human tumor cell lines with IC50 of 1.76 microg/mL. It also exhibited moderate antimicrobial activity against three bacteria.—CONCLUSION: The essential oil of Zanthoxylum avicennae contains various active constituents. This result provides scientific reference for the pharmacological further research of Zanthoxylum avicennae.–PMID: 23320360 [PubMed – indexed for MEDLINE]
Getting Enough Sleep Could Help Prevent Type 2 Diabetes
June 18, 2013 — Men who lose sleep during the work week may be able to lower their risk of developing Type 2 diabetes by getting more hours of sleep, according to Los Angeles Biomedical Research Institute (LA BioMed) research findings presented today at The Endocrine Society’s 95th Annual Meeting in San Francisco.—The study by Peter Liu, MD, PhD, an LA BioMed lead researcher, found that insulin sensitivity, the body’s ability to clear glucose (blood sugar) from the bloodstream, significantly improved after three nights of “catch-up sleep” on the weekend in men with long-term, weekday sleep restrictions.—“We all know we need to get adequate sleep, but that is often impossible because of work demands and busy lifestyles,” said Dr. Liu. “Our study found extending the hours of sleep can improve the body’s use of insulin, thereby reducing the risk of Type 2 diabetes in adult men. –Insulin is a hormone that regulates a person’s blood sugar level. The body of a patient with Type 2 diabetes cannot effectively use the insulin it produces, or it becomes “resistant” to insulin. Retaining the body’s sensitivity to insulin reduces the risk of developing Type 2 diabetes, a chronic illness that is the seventh leading cause of death in the U.S.–Other research had demonstrated the harmful effects of experimental sleep restriction on insulin sensitivity in healthy, normal sleepers. The new study provides information about people who lose sleep during the week — often because of jobs and busy lifestyles — but “catch up” on their sleep on the weekends.-“The good news is that by extending the hours they sleep, adult men — who over a long period of time do not get enough sleep during the working week — can still improve their insulin sensitivity,” Liu said.-Liu and researchers from the University of Sydney in Australia studied 19 non-diabetic men, with an average age of 28.6 years, who for six months or longer (average, 5.1 years) self-reported inadequate sleep during the workweek. On average, the men received only 6.2 hours of sleep each work night. But they regularly caught up on their sleep on the weekends, sleeping an extra 37.4 percent, or 2.3 hours, per night, the authors reported. Their reported sleep times were verified by actigraphy, in which each man wore a small device on his wrist that monitored sleep-wake cycles.—The men spent three nights in a sleep lab on each of two separate weekends. The researchers randomly assigned the men to two of three sleep conditions: (1) 10 hours of sleep, (2) six hours of sleep or (3) 10 hours in bed, in which noises during deep sleep aroused them into shallow sleep without waking them. The six hours of sleep tested persistent sleep restriction.—On the fourth morning, the research staff drew the men’s blood to measure their blood sugar and insulin levels to calculate insulin sensitivity. Each individual had the same food intake during the study visits, so that diet would not influence the results, Liu said.—When the men slept 10 hours a night on each of three nights of catch-up sleep, their insulin sensitivity was much better than when they had persistent sleep restriction, the scientists found. Their insulin resistance test score also improved (decreased) with sleep extension.—Story Source-The above story is reprinted from materials provided by Los Angeles Biomedical Research Institute at Harbor-UCLA Medical Center (LA BioMed).
Timing of Calcium and Vitamin D Supplementation May Affect How Bone Adapts to Exercise
June 18, 2013 — Taking calcium and vitamin D before exercise may influence how bones adapt to exercise, according to a new study. The results will be presented on Tuesday at The Endocrine Society’s 95th Annual Meeting in San Francisco.—“The timing of calcium supplementation, and not just the amount of supplementation, may be an important factor in how the skeleton adapts to exercise training,” said study lead author Vanessa D. Sherk, PhD, postdoctoral research fellow at the University of Colorado Anschutz Medical Campus. “Further research, however, is needed to determine whether the timing of calcium supplementation affects the skeletal adaptations to exercise training.”—Previous research has shown that a year of intense training is associated with substantial decreases in bone mineral density among competitive road cyclists. Experts believe that this kind of exercise-induced bone loss could be related to the loss of calcium during exercise. As blood calcium levels drop, the parathyroid gland produces excess parathyroid hormone, which can mobilize calcium from the skeleton.—In this study, investigators found that an exercise-induced decrease in blood calcium occurred whether calcium supplements were taken before or after exercising. Pre-exercise supplementation, however, resulted in less of a decrease. Although not statistically significant, parathyroid hormone levels increased slightly less among cyclists who took calcium before exercising.—“These findings are relevant to individuals who engage in vigorous exercise and may lose a substantial amount of calcium through sweating,” Sherk said. “Taking calcium before exercise may help keep blood levels more stable during exercise, compared to taking the supplement afterwards, but we do not yet know the long-term effects of this on bone density.”—The timing of calcium supplementation did not cause a difference in blood levels of a compound that is a biological indicator of bone loss. Both the before- and after-exercise groups exhibited 50-percent increases in the level of this compound, called CTX, for collagen type-1 C-telopeptide.—Study participants included 52 men aged 18 to 45 years. Investigators randomly assigned participants to take 1,000 milligrams of calcium and 1,000 international units of vitamin D either 30 minutes before or one hour after exercise. The exercise comprised a simulated 35-kilometer time trial, and participants wore skin patches to absorb sweat.—Investigators measured blood levels of calcium and parathyroid hormone before and immediately after exercise. They also measured CTX before and 30 minutes after exercise. They used pre- and post-body weight, adjusted for fluid intake, combined with the calcium measured in the sweat from the skin patches, to estimate the amount of calcium lost through the skin during exercise.–The National Institutes of Health and the American College of Sports Medicine funded the study.—Story Source-The above story is reprinted from materials provided by Endocrine Society, via Newswise.
Special Note— if anyone was going to try this then you would not use 1000mgs of Calcium but rather 300mmgs with magnesium 150mgs—or you would utilize a creatine with the calcium or even a carbonated calcium —this would insure absorption and usage —there test were flawed because there deliver system would not work due to the overloading of calcium—even comboing it with gelatin as well—then you would be chelating the minerals into the system
Aspirin May Fight Cancer by Slowing DNA Damage
June 18, 2013 — Aspirin is known to lower risk for some cancers, and a new study led by a UC San Francisco scientist points to a possible explanation, with the discovery that aspirin slows the accumulation of DNA mutations in abnormal cells in at least one pre-cancerous condition.—-“Aspirin and other non-steroidal anti-inflammatory drugs, which are commonly available and cost-effective medications, may exert cancer-preventing effects by lowering mutation rates,” said Carlo Maley, PhD, a member of the UCSF Helen Diller Family Comprehensive Cancer Center, and an expert on how cancers evolve in the body over time. In the study, published June 13 in the online journal PLOS Genetics, Maley, working with gastroenterologist and geneticist Brian Reid, MD, PhD, of the Fred Hutchinson Cancer Research Center, analyzed biopsy samples from 13 patients with a pre-cancerous condition called Barrett’s esophagus who were tracked for six to 19 years. In an “observational crossover” study design, some patients started out taking daily aspirin for several years, and then stopped, while others started taking aspirin for the first time during observation. The goal was to track the rate of mutations in tissues sampled at different times.—The researchers found that biopsies taken while patients were on an aspirin had on average accumulated new mutations about 10 times more slowly than biopsies obtained during years when patients were not taking aspirin.[F7]—“This is the first study to measure genome-wide mutation rates of a pre-malignant tissue within patients for more than a decade, and the first to evaluate how aspirin affects those rates,” Maley said.—Gender and ethnic distribution of study patients reflected the known demographics of esophageal cancer, which predominantly affects, white, middle-aged and elderly men, he said. Barrett’s esophagus only occasionally progresses to esophageal cancer.—Cancers are known to accumulate mutations over time much more rapidly than normal tissue, and different mutations arise in different groups of cells within the same tumor. The acquisition of key mutations ultimately allows tumor cells to grow out of control, and diversity within a tumor may foster drug resistance, a phenomenon that is a major focus of Maley’s research.—-Maley plans to test a hypothesis that may explain the results — that aspirin’s lowering of mutation rates is due to the drug’s effect of reducing inflammation. Inflammation, a response of the immune system, in recent years has been recognized as a hallmark of cancer. Maley said that less inflammation may result in less production within pre-cancerous tissue of oxidants known to damage DNA, and may dampen growth-stimulating signaling.—For the duration of the study, the rate of accumulation of mutations measured in the biopsied tissue between time points was slow, even when patients were not taking aspirin, with the exception of one patient. While mutations accumulated at a steady rate, the vast majority of mutations arose before the abnormal tissue was first detected in the clinic, the researchers concluded.—These findings are consistent with the fact that although Barrett’s esophagus is a significant risk factor for esophageal cancer, the vast majority of cases do not progress to cancer, Maley said.
In the one patient who later went on to develop cancer, a population of cellular “clones” with a great number of mutations emerged shortly before he started taking aspirin.—More studies are needed to further explore the link between non-steroidal anti-inflammatory drugs, mutation rates and the development of invasive cancer, Maley said. He plans to continue studying Barrett’s esophagus and esophageal cancer, and to expand his research to investigate lung cancer.–Rather than aiming to kill the most tumor cells, it may be better to try to halt or slow growth and mutation. Current drug treatments for cancer may in many cases hasten the emergence of cancer that is more difficult to eradicate, according to Maley. The capability to mutate frequently allows tumors to become resistant to drug treatment, he said. A better-adapted mutant can begin to spin off a population of genetic clones that survives and grows, while poorly adapted tumor cells die off.[F8]–Additional authors from the Fred Hutchinson Cancer Research Center include Xiaohong Li, PhD, Carissa Sanchez, PhD, Patricia Galipeau, PhD, Thomas Paulson, PhD, Patricia Blount, PhD, Thomas Vaughan, PhD, and Cassandra Sather, PhD. Amitabh Srivastava, MD, and Robert Odze, MD, from Harvard University; Rumen Kostadinov, PhD, from the University of Pennsylvania; and Mary Kuhner, PhD from the University of Washington also were members of the research team and authors of the study.—Story Source-The above story is reprinted from materials provided by University of California, San Francisco (UCSF). The original article was written by Jeffrey Norris. –Journal Reference-Rumen L. Kostadinov, Mary K. Kuhner, Xiaohong Li, Carissa A. Sanchez, Patricia C. Galipeau, Thomas G. Paulson, Cassandra L. Sather, Amitabh Srivastava, Robert D. Odze, Patricia L. Blount, Thomas L. Vaughan, Brian J. Reid, Carlo C. Maley. NSAIDs Modulate Clonal Evolution in Barrett’s Esophagus. PLoS Genetics, 2013; 9 (6): e1003553 DOI: 10.1371/journal.pgen.1003553
% Average Human Body Content
(70 kg person)
Oxygen 65.4 43 kg
Carbon 18.2 12 kg
Hydrogen 9.5 6.3 kg
Nitrogen 3 2 kg
Calcium 1.67 1.1 kg
Phosphorus 1.14 750 grams
Potassium 0.342 225 grams
Sulfur 0.228 150 grams
Chloride 0.152 100 grams
Sodium 0.137 90 grams
Magnesium 0.053 35 grams
Silicon 0.046 30 grams
Iron 0.00638 4,200 mg Essential
Fluoride 0.00395 2,600 mg Possibly Essential
Zinc 0.00365 2,400 mg Essential
Rubidium 0.000532 350 mg Probably Essential
Strontium 0.000486 320 mg Possibly Essential
Lead 0.000243 160 mg Toxic
Copper 0.000137 90 mg Essential
Aluminium 0.0000988 65 mg Toxic
Cadmium 0.0000608 40 mg Toxic
Barium 0.0000344 22 mg Possibly Essential
Cobalt 0.0000304 20 mg Essential
Vanadium 0.0000304 20 mg Probably Essential
Iodine 0.0000228 15 mg Essential
Tin 0.0000228 15 mg Possibly Essential
Selenium 0.0000228 15 mg Essential
Arsenic 0.0000228 15 mg Possibly Essential
Manganese 0.0000198 13 mg Essential
Mercury 0.0000190 12.5 mg Toxic
Nickel 0.0000167 11 mg Possibly Essential
Molybdenum 0.0000122 8 mg Essential
Chromium 0.0000091 6 mg Essential
Bismuth 0.0000045 3 mg Probably Toxic
Lithium 0.0000038 2.5 mg Probably Essential
Uranium 90 mcg Toxic
Boron Probably Essential
Germanium Probably Essential
Bromine Possibly Essential
Gold Probably Non Essential
Silver Probably Non Essential
Beryllium Probably Toxic
Antimony Slightly Toxic
Forms of Minerals (Generally)
Mineral Form Mineral Bonded With Comments
Inorganic Salts: Phosphates-Sulfates-Chlorides Important for Electrolyte balance.
Organic Acid Salts-Ascorbates-Acetates-Citrates Natural and more effectively absorbed by the Digestive System than Inorganic Salts
Amino Acid Chelates-Amino Acids The best form of absorption of Minerals. Specific Minerals chelate best with specific Amino Acids.
[F1]Methods such as these can be done with almost any fat ( lipid) —and directing them would be what you would sompile in the lipid ( fat)
[F2]Creating a Nanoparticle delivery or liposomal delivery to get things into the cells would be a good concept—with cells salt is one way and mixing a salt with some of these herbals may in fact potentiate them better
[F3]Rhodiola Rosea acts as an Antioxidant and Life Extending even on a restricted diet
[F4]This would have to do with the soy proteins in the formulas which are low on the protein scale for uptake and digestibility—the strain and stress on the digestive system would cause the colon to become corrupt with bad bacteria
[F5]Would be better to give a child a higher profile of protein then the soy and rice in the formulas—they are low on the bv and are excessively difficult t break down even for an adult so the excretment would be a direct result of the proteins passing through thedigestive system and causing damage topancreas-liver and stomach
[F6]Gut flora affects brain functionality in the utmost way—so damaging this will effect emotional and mental up the way
[F7]Make sure if you use an aspirin it does not have yellow coluring agents-synthetic flavours—natural or artificial flavours or polymers—if necessary buy a a supply for animals will be cleaner—and mix this with magnesium and choline as well to insure effect and safety
[F8]This is why today you need more then one remedy and a lifestyle change due to the genetics of cancer and capacity to mutate and adapt—this is how morgellons works as well they are finding more and more that cancer tumours are carrying crystal like components and by offsetting the inflammation this can allow the body to remove the nano particles out through antioxidant free radical scavenging or by chelation
Script of the Show July 20 2013
Curcuminoids and resveratrol as anti-Alzheimer agents.
RUSSIA Suggests that WAR May Be Necessary To Stop MONSANTO
Over 30 Million Bees Found Dead In Elmwood Canada
Monsanto to pull the plug on GM lobbying and activities in Europe
Curcuminoids and resveratrol as anti-Alzheimer agents.
Taiwan J Obstet Gynecol. 2012 Dec;51(4):515-25
Authors: Villaflores OB, Chen YJ, Chen CP, Yeh JM, Wu TY
Alzheimer disease (AD) is by far the most common cause of dementia globally. This neurodegenerative disorder of the brain is chronic and progressive, characterized clinically by the deterioration in the key symptoms of behavioral and cognitive abilities. Treatment options for this disease currently are limited. Deposition of amyloid-β and tau hyperphosphorylation are cardinal pathologic features of AD that lead to the formation of neuronal plaques and neurofibrillary tangles, respectively. In addition to mounting research on herbal compounds for the treatment of AD, curcuminoids and resveratrol appear to be beneficial as anti-AD agents. Curcuminoids (curcumin and demethoxycurcumin) and resveratrol possess unique properties that make them especially worthy of further studies. This review article revisits and presents the current research done on the potential of the curcuminoids curcumin and demethoxycurcumin and the polyphenolic compound resveratrol as anti-AD compounds.—PMID: 23276553 [PubMed – indexed for MEDLINE]
Let’s Make our own version of this—Lets take some wine – 1 .5 cups-some tumeric -1/4 cup and pepper ( black) 1/8 cup and some deep bluish purple raisons -1/2 cup and 2 cups of water—put everything in either a high speed blender or a pressure cooker— and proceed to process—high speed blender 15 minutes at high speed—pressure cooker 1 hour —-when done stop devices ( either one) allow pressure cooker to cool down before opening—take either extract from either device and pour through a filter and then wring out what is in the filter ( sheet—handkerchief-pillow case) then take the filtered stuff and put in pot and simmer down to about ½-1/4 above the bottom—when cooled off the add to container and use ¼ tsp increments several times a day
Will get the benefits for the liver-blood-brain—circulation—antioxidants-anti fungal –antibacterial-immune enhancing—
RUSSIA Suggests that WAR May Be Necessary To Stop MONSANTO
Posted on June 13, 2013
Russian President Vladimir Putin commented that war might be a necessary result of the Monsanto Protection Act. did a little digging around, and it turns out the story checks out… The shocking minutes relating to President Putin’s meeting last month with US Secretary of State John Kerry reveal the Russian leaders “extreme outrage” over the Obama regimes continued protection of global seed and plant bio-genetic giants Syngenta and Monsanto in the face of a growing “bee apocalypse” that the Kremlin warns “will most certainly” lead to world war. -According to these minutes, released in the Kremlin today by the Ministry of Natural Resources and Environment of the Russian Federation (MNRE), Putin was so incensed over the Obama regimes refusal to discuss this grave matter that he refused for three hours to even meet with Kerry, who had traveled to Moscow on a scheduled diplomatic mission, but then relented so as to not cause an even greater rift between these two nations.—At the center of this dispute between Russia and the US, this MNRE report says, is the “undisputed evidence” that a class of neuro-active insecticides chemically related to nicotine, known as neonicotinoids, are destroying our planets bee population, and which if left unchecked could destroy our world’s ability to grow enough food to feed its population.—So grave has this situation become, the MNRE reports, the full European Commission (EC) this past week instituted a two-year precautionary ban (set to begin on 1 December 2013) on these “bee killing” pesticides following the lead of Switzerland, France, Italy, Russia, Slovenia and Ukraine, all of whom had previously banned these most dangerous of genetically altered organisms from being used on the continent.–Two of the most feared neonicotinoids being banned are Actara and Cruiser made by the Swiss global bio-tech seed and pesticide giant Syngenta AG which employs over 26,000 people in over 90 countries and ranks third in total global sales in the commercial agricultural seeds market.–Important to note, this report says, is that Syngenta, along with bio-tech giants Monsanto, Bayer, Dow and DuPont, now control nearly 100% of the global market for genetically modified pesticides, plants and seeds.—Also to note about Syngenta, this report continues, is that in 2012 it was criminally charged in Germany for concealing the fact that its genetically modified corn killed cattle, and settled a class-action lawsuit in the US for $105 million after it was discovered they had contaminated the drinking supply of some 52 million Americans in more than 2,000 water districts with its “gender-bending” herbicide Atrazine.—To how staggeringly frightful this situation is, the MNRE says, can be seen in the report issued this past March by the American Bird Conservancy (ABC) wherein they warned our whole planet is in danger, and as we can, in part, read: “As part of a study on impacts from the world’s most widely used class of insecticides, nicotine-like chemicals called neonicotinoids, American Bird Conservancy (ABC) has called for a ban on their use as seed treatments and for the suspension of all applications pending an independent review of the products’ effects on birds, terrestrial and aquatic invertebrates, and other wildlife.—“It is clear that these chemicals have the potential to affect entire food chains. The environmental persistence of the neonicotinoids, their propensity for runoff and for groundwater infiltration, and their cumulative and largely irreversible mode of action in invertebrates raise significant environmental concerns,” said Cynthia Palmer, co-author of the report and Pesticides Program Manager for ABC, one of the nation’s leading bird conservation organizations.—ABC commissioned world renowned environmental toxicologist Dr. Pierre Mineau to conduct the research. The 100-page report, “The Impact of the Nation’s Most Widely Used Insecticides on Birds,” reviews 200 studies on neonicotinoids including industry research obtained through the US Freedom of Information Act. The report evaluates the toxicological risk to birds and aquatic systems and includes extensive comparisons with the older pesticides that the neonicotinoids have replaced. The assessment concludes that the neonicotinoids are lethal to birds and to the aquatic systems on which they depend.—“A single corn kernel coated with a neonicotinoid can kill a songbird,” Palmer said. “Even a tiny grain of wheat or canola treated with the oldest neonicotinoid — called imidacloprid — can fatally poison a bird. And as little as 1/10th of a neonicotinoid-coated corn seed per day during egg-laying season is all that is needed to affect reproduction.”—The new report concludes that neonicotinoid contamination levels in both surface- and ground water in the United States and around the world are already beyond the threshold found to kill many aquatic invertebrates.”
Quickly following this damning report, the MRNE says, a large group of group of American beekeepers and environmentalists sued the Obama regime over the continued use of these neonicotinoids stating: “We are taking the EPA to court for its failure to protect bees from pesticides. Despite our best efforts to warn the agency about the problems posed by neonicotinoids, the EPA continued to ignore the clear warning signs of an agricultural system in trouble.”—And to how bad the world’s agricultural system has really become due to these genetically modified plants, pesticides and seeds, this report continues, can be seen by the EC’s proposal this past week, following their ban on neonicotinoids, in which they plan to criminalize nearly all seeds and plants not registered with the European Union, and as we can, in part, read:—“Europe is rushing towards the good ol days circa 1939, 40… A new law proposed by the European Commission would make it illegal to “grow, reproduce or trade” any vegetable seeds that have not been “tested, approved and accepted” by a new EU bureaucracy named the “EU Plant Variety Agency.”[F1]–It’s called the Plant Reproductive Material Law, and it attempts to put the government in charge of virtually all plants and seeds. [F2]Home gardeners who grow their own plants from non-regulated seeds would be considered criminals under this law.”—This MRNE report points out that even though this EC action may appear draconian, it is nevertheless necessary in order to purge the continent from continued contamination of these genetically bred “seed monstrosities.”–[F3]-Most perplexing in all of this, the MRNE says, and which led to Putin’s anger at the US, has been the Obama regimes efforts to protect pesticide-producer profits over the catastrophic damaging being done to the environment, and as the Guardian News Service detailed in their 2 May article titled “US rejects EU claim of insecticide as prime reason for bee colony collapse” and which, in part, says:–“The European Union voted this week for a two-year ban on a class of pesticides, known as neonicotinoids, that has been associated with the bees’ collapse. The US government report, in contrast, found multiple causes for the collapse of the honeybees.”-To the “truer” reason for the Obama regimes protection of these bio-tech giants destroying our world, the MRNE says, can be viewed in the report titled “How did Barack Obama become Monsanto’s man in Washington?” and which, in part, says:—“After his victory in the 2008 election, Obama filled key posts with Monsanto people, in federal agencies that wield tremendous force in food issues, the USDA and the FDA: At the USDA, as the director of the National Institute of Food and Agriculture, Roger Beachy, former director of the Monsanto Danforth Center. As deputy commissioner of the FDA, the new food-safety-issues czar, the infamous Michael Taylor, former vice-president for public policy for Monsanto. Taylor had been instrumental in getting approval for Monsanto’s genetically engineered bovine growth hormone.”—Even worse, after Russia suspended the import and use of an Monsanto genetically modified corn following a study suggesting a link to breast cancer and organ damage this past September, the Russia Today News Service reported on the Obama regimes response:
“The US House of Representatives quietly passed a last-minute addition to the Agricultural Appropriations Bill for 2013 last week – including a provision protecting genetically modified seeds from litigation in the face of health risks.–The rider, which is officially known as the Farmer Assurance Provision, has been derided by opponents of biotech lobbying as the “Monsanto Protection Act,” as it would strip federal courts of the authority to immediately halt the planting and sale of genetically modified (GMO) seed crop regardless of any consumer health concerns.—The provision, also decried as a “biotech rider,” should have gone through the Agricultural or Judiciary Committees for review. Instead, no hearings were held, and the piece was evidently unknown to most Democrats (who hold the majority in the Senate) prior to its approval as part of HR 993, the short-term funding bill that was approved to avoid a federal government shutdown.”—On 26 March, Obama quietly signed this “Monsanto Protection Act” into law thus ensuring the American people have no recourse against this bio-tech giant as they fall ill by the tens of millions, and many millions will surely end up dying in what this MRNE report calls the greatest agricultural apocalypse in human history as over 90% of feral (wild) bee population in the US has already died out, and up to 80% of domestic bees have died out too.
Over 30 Million Bees Found Dead In Elmwood Canada
July 1, 2013 by Arjun
Shortly after 50,000 bees were found dead in an Oregon parking lot (read more here), a staggering 37 million bees have been found dead in Elmwood, Ontario, Canada. Dave Schuit, who runs a honey operation in Elmwood has lost 600 hives. He is pointing the finger at the insecticides known as neonicotinoids, which are manufactured by Bayer CropScience Inc. This also comes after a recent report released by the British Beekeepers Association (BBKA) that recorded its largest loss of honeybees ever. You can read more about that here. The European Union has stepped forward, having banned multiple pesticides that have been linked to killing millions of bees. You can view the studies and read more about that here.—The loss comes after the planting of corn. Neonicotinoid pesticides are used to coat corn seed with air seeders. This results in having the pesticide dust blown into the air when planted. The death of millions of pollinators was studied by Purdue University. They discovered that Bees exhibited neurotoxic symptoms. They analyzed dead bees and found that traces of thiamethoxam/clothiandin were present in each case. The only major source of these compounds are seed treatments of field crops. You can view that study here (1).—Bee deaths are increasing exponentially. An international team of scientists led by Holland’s Utrecht University has concluded that, “large scale prophylaxic use in agriculture, their high persistence in soil and water, and their uptake by plants and translocation to flowers, neonicotinoids put pollinators at risk. This is some of the research that led to the European Unions ban of the pesticides, as mentioned and referenced earlier.—Can we really debate this much longer? The evidence linking pesticides to bee deaths is overwhelming. It’s not only bees, but an array of other insects as well. The last thing we need is for Monsanto and other corps to use another reason to manufacture and develop fake food! One reason that has been used for justification of GMO’s is a food shortage, and we all know how critical bees are to our food supply. There is a huge conflict of interest here, the pesticides used to spray the crops that are killing the bees are developed by biotech corporations like Monsanto.
Monsanto to pull the plug on GM lobbying and activities in Europe
Posted on03 June 2013.
Monsanto plans to halt lobbying activity for its genetically modified (GM) crop varieties in Europe, the agriculture and biotechnology giant has confirmed.
US-based biotechnology company Monsanto will drop lobbying activities in Europe, and does not plan any new applications for the approval of genetically modified seeds in the EU, a spokesperson has said.–The moves comes on the back of low demand from farmers and ‘stiff opposition’ from the general public, according to the German spokeswoman Ursula Luettmer-Ouazane.—“As long as there’s not enough demand from farmers for these products and the public at large doesn’t accept the technology, it makes no sense to fight against windmills,” she confirmed.—Indeed, Monsanto officials added that the GM giant will only focus on expansion and market penetration of its GM products in areas that provide broad support – meaning that activities will continue in Portugal, Spain, and Eastern Europe where its MON810 maize is more widely grown and accepted.—Monsanto’s confirmation comes almost 18 months after an announcement that German chemical and biotech firm BASF would be pulling out of the European market due to a lack of consumer and industry acceptance. Other companies including Bayer AG’s CropScience unit and Syngenta have also largely moved out of the European market.–Monsanto is also currently under growing pressure in the USA and Asia, after the discovery of a non-approved, experimental wheat strain was found growing in Oregon.–The finding of the experimental wheat, which the company said it finished trials of several years previously, has led to uncertainty in the wheat export market with Japan imposing an outright ban , while a number of other markets – including the EU – said to be closely monitoring and assessing the situation.
[F1]This legislation is a set up to further control access to foods and eventually will lead to wars as well due to the restrictions of what can and cannot be grown—the legislation should have regulated the Seeds of Biotech not the seeds that have been utilized historically
[F2]Unbelievable this will be europes undoing since corpations run Gov’t this will be just another way for the agro globalist to run a nation by either starvation or regulation—this will cause a shortage and a hike in cost to eat
[F3]This legislation has taken the power of the seed out of the farmers hands into the gov’t –the farming industry has now lost it’s only leverage
SCRIPT OF THE SHOW JULY 27 2013
Fighting Cancer with Fats
Ketogenic diet effective against metastatic cancer
Long-term effects of a ketogenic diet in obese patients
Morgellons-Biowarefare-with Nano Technolgy
The Global Threat of Persistent Organic Pollutants
Fighting Cancer with Fats
Barrow researcher explores innovative cancer treatment
For Barrow Researcher Adrienne C. Scheck, PhD, diets are far more than a health fad: Dr. Scheck is researching the potential of a therapy known as the ketogenic diet to complement traditional treatments for brain cancer such as radiation and chemotherapy.—Since the 1920s, the ketogenic diet has been used to control epilepsy, but its popularity has waxed and waned. It is now used almost exclusively in children whose seizures do not respond to conventional drug therapies. The highly regimented diet calls for nearly 80 percent of a patient’s calorie intake to come from fat and less than 1 percent from carbohydrates, with the remainder coming from protein.—-Researchers were spurred to investigate the ketogenic diet as a cancer therapy because cancer cells rely primarily on glucose to fuel their metabolism. The diet causes the body to enter a state called ketosis, where fat-derived chemicals called ketones are burned in response to the absence of carbohydrates. It has been theorized that because tumor cells cannot use ketones for energy, this metabolic change would starve tumor tissue, stunting its growth and improving survival rates for cancer patients.–While not ruling out the metabolic explanation, Dr. Scheck’s research has uncovered other ways that the diet acts on tumors.–Dr. Scheck and her team in the Barrow Neuro-Oncology Research Laboratory observed that live tumor models subjected to the ketogenic diet had fewer reactive oxygen species present in and around the cancerous tissue.–“Reactive oxygen species are created during normal cellular metabolism and are important in the control of many aspects of cell growth—too many or too few are bad for the cell,” says Dr. Scheck. “The metabolism of tumors is higher than that of normal tissue—a feature that may allow them to adapt to increases in reactive oxygen species and contribute to their resistance against chemotherapy.”—Dr. Scheck hypothesizes that by helping to reduce the number of reactive oxygen species tumor cells are accustomed to, the diet may enhance the effectiveness of chemotherapy.[F1]—The results of Dr. Scheck’s latest study also suggest that the diet affects gene expression in tumor cells. “We are seeing that the genetic profile of the tumor cells exposed to ketone bodies is shoved more toward the gene expression of normal cells,” says Dr. Scheck.—How the above mechanisms work together to slow cancer growth is not yet clearly defined, but the results of their combined action are reason to take notice. Dr. Scheck’s team reports that a ketogenic diet, combined with traditional therapies such as radiation and chemotherapy, has the potential to slow the growth of living cancer cells and significantly increase survival time.[F2]—Nonetheless, the ketogenic diet has its drawbacks. Dr. Scheck cautions that this treatment is very different from diets that people manage on their own. Rather, she says, it is a therapy that requires total compliance
Ketogenic diet effective against metastatic cancer
A research team from the Hyperbaric Biomedical Research Laboratory at the University of South Florida has finally found what the nutritional healing community has been crying hoarse about – that a ketogenic diet kills cancer cells. The study, “The Ketogenic Diet and Hyperbaric Oxygen Therapy Prolong Survival in Mice with Systemic Metastatic Cancer,” was published online on June 5, 2013 in PLOS ONE.—-In the study, mice with advanced metastatic cancer were fed either a standard high carbohydrate diet or carbohydrate-restricted ketogenic diet. Mice on both diets also received hyperbaric oxygen therapy, which uses a special chamber to increase the amount of oxygen in the tissues.—The ketogenic diet forces a physiological shift in substrate utilization from glucose to fatty acids and ketone bodies for energy. Normal healthy cells readily adapt to using ketone bodies for fuel, but cancer cells lack this metabolic flexibility, and thus become selectively vulnerable to reduced glucose availability. Solid tumors also have areas of low oxygen, which promotes tumor growth and metastatic spread. –Hyperbaric oxygen therapy involves breathing 100 percent oxygen at elevated barometric pressure, saturating the tumors with oxygen. When administered properly, both the ketogenic diet and hyperbaric oxygen therapy are non-toxic and even protect healthy tissues while simultaneously damaging cancer cells.–The research shows the effects of combining two nontoxic adjuvant cancer therapies, the ketogenic diet and hyperbaric oxygen therapy, in a mouse model of late-stage, metastatic cancer. While both therapies slowed disease progression independently, animals receiving the combined ketogenic diet and hyperbaric oxygen therapy lived 78 percent longer than mice fed a standard high-carbohydrate diet.—Metastasis, the spreading of cancer from the primary tumor to distant spots, is responsible for over 90 percent of cancer-related deaths in humans. A lack of available therapies effective against metastatic disease remains the largest obstacle in finding a cure for cancer.
How you can incorporate this into your lifestyle to not let cancer cells grow:
Restrict carbohydrates and replace them with healthy fats available in fish oil, nuts, seeds, extra virgin olive oil (raw) and healthy protein upasteurized milk, yogurt with fat ,kefir with fat,nuts, eggs, fish ghee-coconut oils-mct oils-tallow-lard. Concentrate on low glycemic index, high fibre carbohydrates in small quantities like apples-peaches-plums –pears-apricots.
When eating fruit, combine with spices-such as cinnamon-clove-cardamon-tumeric-galangal-ginger, as examples to avoid fructose related sugar spikes in the blood. The vitamins and minerals in fruits are anti-cancer hence fruits and fruit fibre are very important. Go for low GL foods like apple, papaya and fruits like pineapples with anti-inflammatory qualities. Consume raw, consume juices made from fresh veges home made and unpasteurized-if you use fruit juices then add the minerals zinc or chromium or manganese-or magnesium to them—or a full spectrum of minerals
To adapt hyperbaric oxygen therapy for prevention and to oxygenate your organs, practice deep breatheing techniques every day for 20-30 min. This will flush out the toxins from the blood and re-energise you while increasing oxygen in the blood flow, hence killing tumour cells in your body.
Remember, a strong immune system will fight cancer and tumour cells, and a ketogenic diet combined with oxygenating your body goes a long way in building immunity.
Long-term effects of a ketogenic diet in obese patients
Hussein M Dashti, MD PhD FICS FACS,1 Thazhumpal C Mathew, MSc PhD FRCPath,4 Talib Hussein, MB ChB,5 Sami K Asfar, MB ChB MD FRCSEd FACS,1 Abdulla Behbahani, MB ChB FRCS FACSI PhD FICS FACS,1 Mousa A Khoursheed, MB ChB FRCS FICS,1 Hilal M Al-Sayer, MD PhD FICS FACS,1 Yousef Y Bo-Abbas, MD FRCPC,2 and Naji S Al-Zaid, BSc PhD3
Author information ► Copyright and License information ►
This article has been cited by other articles in PMC.
Although various studies have examined the short-term effects of a ketogenic diet in reducing weight in obese patients, its long-term effects on various physical and biochemical parameters are not known.
To determine the effects of a 24-week ketogenic diet (consisting of 30 g carbohydrate, 1 g/kg body weight protein, 20% saturated fat, and 80% polyunsaturated and monounsaturated fat) in obese patients.
PATIENTS AND METHODS:
In the present study, 83 obese patients (39 men and 44 women) with a body mass index greater than 35 kg/m2, and high glucose and cholesterol levels were selected. The body weight, body mass index, total cholesterol, low density lipoprotein (LDL) cholesterol, high density lipoprotein (HDL) cholesterol, triglycerides, fasting blood sugar, urea and creatinine levels were determined before and after the administration of the ketogenic diet. Changes in these parameters were monitored after eight, 16 and 24 weeks of treatment.
The weight and body mass index of the patients decreased significantly (P<0.0001). The level of total cholesterol decreased from week 1 to week 24. HDL cholesterol levels significantly increased, whereas LDL cholesterol levels significantly decreased after treatment. The level of triglycerides decreased significantly following 24 weeks of treatment. The level of blood glucose significantly decreased. The changes in the level of urea and creatinine were not statistically significant.
CONCLUSIONS:–The present study shows the beneficial effects of a long-term ketogenic diet. It significantly reduced the body weight and body mass index of the patients. Furthermore, it decreased the level of triglycerides, LDL cholesterol and blood glucose, and increased the level of HDL cholesterol. Administering a ketogenic diet for a relatively longer period of time did not produce any significant side effects in the patients. Therefore, the present study confirms that it is safe to use a ketogenic diet for a longer period of time than previously demonstrated.
Keywords: Diet, Ketosis, Obesity
Obesity has become a serious chronic disease in both developing and developed countries. Furthermore, it is associated with a variety of chronic diseases (
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Therefore, the purpose of the present study was to investigate the long-term effects of a ketogenic diet on obesity and obesity-associated risk factors in a large population of obese patients.
PATIENTS AND METHODSJune 22, 2021 at 09:02 #2666
Patients and biochemical analysis
The prospective study was carried out at the Academic Department of Surgery, Consultation and Training Centre, Faculty of Medicine, Kuwait University (Jabriya, Kuwait) in 83 obese subjects (39 men and 44 women). The body mass index (BMI) of men and women was 35.9±1.2 kg/m2 and 39.4±1.0 kg/m2, respectively. The mean age was 42.6±1.7 years and 40.6±1.6 years for men and women, respectively. The mean age, initial height, weight and BMI for all patients are given in Table 1. Fasting blood tests were carried out for all of the subjects. Initially, all patients were subjected to liver and renal function tests, and glucose and lipid profiles, using fasting blood samples, and a complete blood count. Thereafter, fasting blood samples were tested for total cholesterol, high density lipoprotein (HDL) cholesterol, low density lipoprotein (LDL) cholesterol, triglycerides, blood sugar, urea and creatinine levels at the eighth, 16th and 24th week. In addition, weight and height measurements, and blood pressure were monitored at each visit.
Patient data at baseline before treatment with the ketogenic diet
Protocol for ketogenic diet-induced body weight reduction
All 83 subjects received the ketogenic diet consisting of 20 g to 30 g of carbohydrate in the form of green vegetables and salad, and 80 g to 100 g of protein in the form of meat, fish, fowl, eggs, shellfish and cheese. Polyunsaturated and monounsaturated fats were also included in the diet. Twelve weeks later, an additional 20 g of carbohydrate were added to the meal of the patients to total 40 g to 50 g of carbohydrate. Micronutrients (vitamins and minerals) were given to each subject in the form of one capsule per day (Table 2).
Composition of the capsule*
Statistical analysis —Statistical differences between body weight, total cholesterol, HDL cholesterol, LDL cholesterol, triglycerides, level of fasting blood sugar, and urea and creatinine levels before and after the administration of the ketogenic diet were analyzed using a paired Student’s t test using the Stat-view version 4.02 (Abacus Concepts Inc, USA). Weight, BMI and all biochemical parameters are expressed as mean ± SEM.
The mean initial weight of the subjects was 101.03±2.33 kg. The weight decreased significantly during all stages of the treatment period. The body weights at the eighth, 16th and 24th week were 91.10±2.76 kg, 89.39±3.4 kg and 86.67±3.70 kg, respectively (Figure 1). Similar to the loss in body weight, a significant decrease was observed in the BMI of the patients following the administration of the ketogenic diet. The initial BMI, and the BMI after the eighth, 16th and 24th week were 37.77±0.79 kg/m2, 33.90±0.83 kg/m2, 33.24±1.00 kg/m2 and 32.06±1.13 kg/m2, respectively
The level of total cholesterol showed a significant decrease from week 1 to week 24 (Figure 3). The level of HDL cholesterol significantly increased (Figure 4), whereas LDL cholesterol levels significantly decreased with treatment (Figure 5). The level of triglycerides decreased significantly after 24 weeks of treatment. The initial level of triglycerides was 2.75±0.23 mmol/L, whereas at week 24, the level decreased to 1.09±0.08 mmol/L (Figure 6). The level of blood glucose significantly decreased at week 24. The initial blood glucose level and its level at the eighth, 16th and 24th week were 7.26±0.38 mmol/L, 5.86±0.27 mmol/L, 5.56±0.19 mmol/L and 5.62±0.18 mmol/L, respectively (Figure 7). The changes in the levels of urea (Figure 8) and creatinine (Figure 9) were not statistically significant.
Until recently, ketosis was viewed with apprehension in the medical world; however, current advances in nutritional research have discounted this apprehension and increased public awareness about its favourable effects. In humans, ketone bodies are the only additional source of brain energy after glucose (
The following popper user interface control may not be accessible. Tab to the next button to revert the control to an accessible version.- Thus, the use of ketone bodies by the brain could be a significant evolutionary development that occurred in parallel with brain development in humans. Hepatic generation of ketone bodies during fasting is essential to provide an alternate fuel to glucose. This is necessary to spare the destruction of muscle from glucose synthesis.—A ketogenic diet is clinically and experimentally effective in antiepileptic and antiobesity treatments; however, the molecular mechanisms of its action remain to be elucidated. In some cases, a ketogenic diet is far better than modern anticonvulsants
Recently, it has been shown that a ketogenic diet is a safe potential alternative to other existing therapies for infantile spasms It was further shown that a ketogenic diet could act as a mood stabilizer in bipolar illness –Beneficial changes in the brain energy profile have been observed in subjects who are on a ketogenic diet ( This is a significant observation because cerebral hypometabolism is a characteristic feature of those who suffer from depression or mania It has also been found that a ketogenic diet affects signal transduction in neurons by inducing changes in the basal status of protein phosphorylation. In another study (it was shown that a ketogenic diet induced gene expression in the brain. These studies provide evidence to explain the actions of a ketogenic diet in the brain. –One of the mechanisms of a ketogenic diet in epilepsy may be related to increased availability of beta-hydroxybutyrate, a ketone body readily transported through the blood-brain barrier. In support of this hypothesis, it was found that a ketogenic diet was the treatment of choice for glucose transporter protein syndrome and pyruvate dehydrogenase deficiency, which are both associated with cerebral energy failure and seizures. One argument against the consumption of a high fat diet is that it causes obesity. The major concern in this regard is whether a high percentage of dietary fat promotes weight gain more than a low percentage of fat intake. Because fat has a higher caloric density than carbohydrate, it is thought that the consumption of a high fat diet will be accompanied by a higher energy -On the contrary, recent studies from our laboratory and many other laboratories of various chronic diseases of the body. A recent showed that sugar can accelerate aging. Several recent studies have pointed to the fact that a diet with a high glycemic load is independently associated with the development of cardiovascular diseases, type II diabetes and certain forms of cancer. Glycemic load refers to a diet of different foods that have a high glycemic index. Glycemic index is a measure of the elevation of glucose levels following the ingestion of a carbohydrate. The classification of a carbohydrate based on its glycemic index provided a better predictor of risk for coronary artery diseases than the traditional method of classification of carbohydrate into simple or complex forms In other studies it was shown that the risk of dietary glycemic load from refined carbohydrates was independent of other known risk factors for coronary diseases. It is now evident that high carbohydrate diets increase fasting plasma triglyceride concentrations and decrease HDL cholesterol concentrations These changes are associated with enhanced atherogenesis However, it has been shown that short-term ketogenic diets improve the lipid disorders that are characteristic of atherogenic It has also been found that sugary drinks decreased blood levels of vitamin E, thus reducing the amount of antioxidants in the body. It has been proven, beyond a doubt, that disrupting the oxidant-antioxidant status of the cell will lead to various diseases of the body – The relation between a high fat diet and cancer is not conclusive. Recent epidemiological studies
could not explain a specific causal relationship between dietary fat and cancer. It has been found that altered energy metabolism and substrate requirements of tumour cells provide a target for selective antineoplastic therapy. The supply of substrates for tumour energy metabolism can be reduced by dietary manipulation (eg, ketogenic diet) or by pharmacological means at the cellular level (eg, inhibitors of glycolysis or oxidative phosphorylation). Both of these techniques are nontoxic methods for controlling tumour growth in vivo . Sugar consumption is positively associated with cancer in humans and test animals This observation is quite logical because tumours are known to be enormous sugar absorbers. It has also been found that the risk of breast cancer decreases with increases in total fat intake Further studies on the role of a ketogenic diet in antineoplastic therapy are in progress in our laboratory.- A link between low fat diets and osteoporosis has been suggested. Very low fat diets are considered to be low in calcium content. Women on low fat diets excrete most of the calcium they consume; therefore, they are more prone to osteoporosis. However, a high fat diet can rectify this situation In the present study, a control population on a low fat diet was not included due to the difficulties in recruiting subjects for a control group. However, several studies with appropriate control groups that compared the effect of a low fat diet with a low carbohydrate ketogenic diet have recently been published. In this regard, these two recent studies are comparable with the present study. Brehm et al showed that obese women on a low carbohydrate ketogenic diet lost 8.5 kg over six months compared with 4.2 kg lost by those in the low fat diet group (P<0.001). Twenty-two subjects from the low carbohydrate ketogenic diet and 20 subjects from the low fat diet completed the study, with both groups reducing their energy intake by approximately 450 kcal from the baseline level. In another study performed in 132 severely obese subjects for six months there was greater weight loss in the low carbohydrate ketogenic diet group than in the low fat diet group (5.8 kg versus 1.9 kg, P=0.002). Both of these studies support the findings presented in the present paper.
The data presented in the present study showed that a ketogenic diet acted as a natural therapy for weight reduction in obese patients. This is a unique study monitoring the effect of a ketogenic diet for 24 weeks. There was a significant decrease in the level of triglycerides, total cholesterol, LDL cholesterol and glucose, and a significant increase in the level of HDL cholesterol in the patients. The side effects of drugs commonly used for the reduction of body weight in such patients were not observed in patients who were on the ketogenic diet. Therefore, these results indicate that the administration of a ketogenic diet for a relatively long period of time is safe. Further studies elucidating the molecular mechanisms of a ketogenic diet are in progress in our laboratory. These studies will open new avenues into the potential therapeutic uses of a ketogenic diet and ketone bodies.
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Morgellons-Biowarefare-with Nano Technolgy
In 1999 the world completely changed. The money trust”Before 1999 vs. after 1999. The money in your wallet is not money. The money trust probably collapsed.”, the law (commerical code/contract law”As of 1999 it is unconditionally impossible to state the truth on an affidavit in a court of law and thereby all applicable law in any realm is void & null. aka Ruby Sparks (2012).”, applicability of public/statutory presumptions, etc.), technology (nano-tech), and the general direction of the planet completely changed. In order to even remotely grasp what is happening you must disconnect your mind from the last century’s way of thinking. Failure to change the way you think will preclude you from seeing reality accurately. There is no money anymore, taxes are a thing of the past and no written law applies to you.
So what we now have is a real brave new world. The purpose of this site is to simply show the extent at which they have leveraged nano-technology directly on you, how they control everyone on demand with it, and end the confusion around the subject of chemtrails and how they fit into the larger picture while showing how the media and movies are conditioning the mind of the general public. This web site is my account of the entire last decade. I’m sure I’m missing something here or there, but this is 98% accurate and applies to every single person on the planet today in one way (typically via phase 1) or another.
The Reason for Everything
Let’s just get right to it. Forget everything you know. Here it is, the epitome of reality. This video is the best example that summarizes what’s happening or has happened in your body already. This is the most prevalent secret in the whole world because it has been forced onto everyone unknowingly.
What you see here is a nano-bot encapsulating a neuron or synapse (for example your purkinje neurons) or other nerve ending/bridge. It’s only a simulation, but accurately summaries everything that has happened in the past decade. This allows complete control of the host (your body) remotely as demonstrated repeatedly in the movies (for example Metropia (2009), Ultrasonic (2012)). A more sophisticated set of nano-bots would and very well has subsequently allowed for a complete and full BioAPI to be installed without the host (you) even knowing it. If you want to get technical your neurons have been encapsulated, your synapses have been bridged.
The basic idea consists of a set of nano-wires tethered to electronics in the main catheter such that they will spread out in a “bouquet” arrangement into a particular portion of the brain’s vascular system. Such arrangement could support a very large number of probes (in the millions). Each n-wire would be used to record, very securely, electrical activity of a single or small group of neurons without invading the brain parenchyma.
What is the Purpose of Chemtrails
Chemtrails are a huge logistical operation. Larger than the hoover damn, trans-alaska pipeline or moon landing. It’s large. And expensive. The biggest mistake one can make is assuming there is only one reason for chemtrails. There are about five or six reasons and possibly more. The top six are listed below with a brief summary. This web site is concerned with the last. A visual overview may help by reviewing a flowchart here.
· Blocking the Sun: This is the standard reason given to fools in the government. We need to secretly stop global warming, so keep it a secret that we’re spraying. Global warming is the catch all con for everyone in the government. If you’re smarter than this they’ll give you a better reason.
· Blocking the Sun (Again): A reduction in sun light across the planet works well to decrease or manipulate crop yields slightly. This is part of the requirement to engineer a food crisis and bring in a famine. You can dismiss this.
· Superheating the Atmosphere: In order to create earthquakes and steer hurricanes (for example hurricane Katrina in New Orleans) the atmosphere needs to be more conductive for electricity so installations such as HAARP (HAARP is just what they want you to see, HAARP has nothing to do with anything) can work their magic. So the chemtrails spray barium and aluminum among other things to create a more conductive upper atmosphere.
· Health Erosion: As a side effect everyone’s health and immune systems become slightly compromised. This is usually not an issue for most healthy people. Older people on average will now die sooner and any health complication is slightly more likely to be fatal. This is both a side effect of spraying and intentional.
· Climate Modification: To help or hurt crops, keep skies clear for a major event (like the Olympics), cause a typhoon, steer super storms, etc.
· Nano-fiber Propagation: To universally install a BioAPI in everyone they need to spray nano-fibers. These fibers cannot be put into the food supply or given in some other way, the uptake across the population would take forever and not propagate very effectively. It’s much easier just to spray everyone like an insect; and because it’s happening to everyone the universal herd mentality of the unwashed masses then justifies it.
Nano-fibers specifically are a transport mechanism. Nothing more. They hold a payload for delivery. A payload that would otherwise be compromised by the sun or atmosphere or not make it to its destination (your body). Such as viral RNA code, metals such as aluminum, nano-components, etc. The fibers are (surprisingly) quite harmless as everyone has them. Examples of these fibers can be found all over the internet or in the physical examples section of this site. The fibers must be independently sprayed, if they we’re added to the jet fuel the extreme heat would destroy the payload.
So it’s not the fiber that is critical, it’s the payload.
This is a complicated question. The people creating and doing this are trying to force biblical principles onto the populace (including themselves) through technology. For example the seven deadly sins. They take a basic human requirement (food, sex, a specific emotion) and quantify it (within the BioAPI). If the result is to extreme (for example you eat too much) or you do something not approved of then they decide that you’re not worthy of life or judge you accordingly. In the alternative your are added to a program. There is a good or beneficial angle to it as mentioned on the chemtrails flowcart page or even in this partially out of context clip for the The Simpsons. —-Note the possibilities for the BioAPI or nano-tech in general is endless. Therefore you should not focus on any one reason as being the end all purpose. It’s too dynamic. It’s to complex. As I mentioned on the top of page 5 of the media references – ‘the BioAPI is the greatest revelation in human history’. For example see the last paragraph of the description for Vexille (2007), specifically the trailer for H+ mentioned in it. —Also see question #9 of the frequently asked questions for a brief explanation on how this (and chemtrails) are (mostly) lawful.
The same group of people that brought humanity HIV in the late 1970’s.
There are essentially two phases involved with the installation of the BioAPI. I categorize it as phase 1 and phase 2. If you can imagine a new laptop computer, all it has is the operating system like Windows, so it’s kind of useless. This would be the equivalent to phase 1. So a new computer can be remotely controlled (aka phase 1, see Surrogates (2009)) by your IT tech support guy, but that is all. There are no programs installed (provided by phase 2) to do much else with it. These names of a phase 1 and 2 are not necessarily just random nonsense I made up, see the clip and movie for Control Factor (2003) in which they use these exact names in the exact same context; because they are telling you everything.
· Phase 1: Everyone on the planet is affected and involved in this phase. Everyone to some extent has the nano-fibers within their body cavity, and therefore wired [‘I’m wired too.’ – Michael Hall, Gamer (2009)]. Side effects include a clicking sound from within the skull and basic annoying body complications like aching joints. This phase provides complete remote control of your speech and thought patterns through suggestion (partially subconsciously). I guess about 99% of the populace of the entire planet has this phase complete.
Phase 1 could be construed as positive and beneficial to you, at least in the future. See John Hodgman (2012) for more information. You should also see question 9 of the FAQs.
· Phase 2: This phase must be triggered (by nano-trigger-bots) and is extreme. It completely compromises your health and can do anything from kill you to simply monitor you. This phase cannot be forced onto you like phase 1 (technically it can but they don’t do that yet). This involves multiple nano-sensors from ocular to heart and everything in between. I figure about 2% of the population has gone through this phase. If this phase is triggered in you they consider you evil as shown within the media examples page of this site. You must do something to trigger this phase, including eating cheap red meat, kissing specific people, using specific corporate health care/beauty products, etc. The objective they are (partially) reaching for here is to connect each event with a deadly sin of some sort. For example morgellons would be connected with vanity because your skin goes to hell. Ultimately this phase provides complete remote control of your body and mind, including the monitoring of your emotions, thoughts, body functions and everything in between. Phase 2 then can be considered a nano-tech disease (as clearly shown in the Family Guy clip) in which the contagious aspect can be switched on and off. For example I have phase 2, but I am not contagious, but I can be if they decide to make me contagious in some way – typically kissing. This allows them to completely control the transmission/vector or spreading of the nano-disease. If you want to get specific, the nano-tech or nano-implants that compose phase 2 of the BioAPI is actually just the vehicle they use to monitor, torment, test and hurt people. The disease itself is actually one of dishonor. The more dishonor you demonstrate, the more they hate you, the worse things get for you. They do not want people to figure that out. See Meeting Evil (2012) for clear details.
Phase 2 can or is definitely detrimental to your life. That is the point of it. Thoughts on a cure can be found in the review for Rise of the Zombies (2012).
Nano-fiber and BioAPI Side Effects
Of course with something as extreme as nanotech being installed within people’s body’s you would assume there would be health implications and side effects. This is correct and covered on this site. The approach to handle these side effects has been one of “embrace and extend” it’s called. There are several examples in the media section that show how the specific side effects listed below are recognized and then associated with something ridiculous or stupid which then discounts the authenticity in the mind of the viewer. In effect convincing the viewer to dismiss a real side effect as being something that’s too crazy to be real. Each side effect is dealt with in a media example. Additional technical possibilities are also talked about in the BioAPI details section.
· Phase 1 & 2 – Cranium Clicking/Screeching: A phase 1 side effect goes back as early as 2001. Exactly what is happening is not completely known but involves some sort of nano-chip being installed/operated in the cranium (your head) of the host. This is probably the equivalent of a CPU of some sort. The actual clicking/screeching sound observed is usually at night on average once a month and only lasts for a few seconds. Completely painless and easily ignored or passed off by the person. The entire purpose of the movie Shutter Island (2010) is to discount this. The nano-implant that is specifically and clearly responsible for this side effect is symbolically referenced in the second clip for Surrogates (2009). I suspect over time they have improved this side effect.
· Phase 1 & 2 – Aching Joints, Headaches, Fatigue, etc.: The saturation of nano-fibers has different effects on different people. The sheer numbers involved results is a random combination of health implications. Most people will not notice anything, or pass any slight symptom off as getting older. Other people who have more of a reaction will go to the doctor and get diagnosed with fibromyalgia. Fibromyalgia is a catch all disease that was created about a decade ago to give doctors something to tell the patient when they complained. The doctors can’t accurate diagnose or understand what or why a patient is feeling a certain way, so the corrupt medical establishment gives them this nonsense to spew. These side effects are primarily phase 1 but are a constant problem across the board. Notice the root word of fibromyalgia is fib[e]r, it’s not a coincidence. This Family Guy clip indirectly references Fibromyalgia.
· Phase 2 – Itching: For whatever reason they may force harsh itching on you when they do not agree with what you are doing or how you are behaving. You probably will have no idea it is phase 2 at the beginning. This is shown in Flash of Genius (2008) when they show her typing and zoom in on her hand (@ 44:50 in the movie) when she itches it. She’s presumed to be a bad wife for leaving her husband (no clip is provided; screenshot here”Flash of Genius (2008); They show her with an itching problem in the movie for no relevant reason. A reference to phase 2 but with no applicability to the movie.”; you’ll have to read this whole site to understand this). The exact same concept is shown in Lay the Favorite (2012) where Bruce Willis itches his forearm clearly and intentionally after referencing it a few seconds earlier (screenshot here””). Why? Why would they put that in? I mean millions of dollars are spent on these scripts and production thereto. This happens in real life to countless people around the world all day long, he’s being warned. Why? Because in the movie he’s thinking about cheating on his wife with the hot blonde that just walked in. In people with phase 2, the BioAPI is monitoring thought and emotional patterns which if conflict triggers an itch; it’s automated. For example lust + guilt (because he’s married) do not go together. Think Pontypool (2008). So they are judging you (or more accurately people with phase 2 who are pre-targeted). Itching is also shown in Fast Zombies with Guns (2011), as they turn into zombies [contract phase 2 in real life] they itch a lot”Fast Zombies with Guns (2011)”. Again, why show this? Because it’s real. Most targeted individuals will understand the extremely itchy forearm”Fast Zombies with Guns (2011)”. So itching is not a side effect in the common sense of the term; it is instead intentionally inflected via the BioAPI as reflected in the aforementioned references as well as loosely shown in A Scanner Darkly (2006) @ 0:44 in the clip/trailer.
· Phase 2 – Burning Smell: Phase 2 encapsulates the person’s ability to smell, so they can read/write scents. It’s used to help warp the reality of someone they have specifically targeted (aka Black Limousine (2010)). When inhaling or specifically exhaling quickly its often a burning/smoke smell that is noticed. This is an unwanted side effect – or more accurately to encapsulate any neuron in the body involved in sensing (for example, smell, taste, etc.) there ends up being be some minor side effect. Interestingly when I cry the smell is amplified and it smells like buttered popcorn of all things. An example of how the media discounts this is demonstrated in the movie Bandits (2001).
· Phase 2 – The Left Eye: One of the concepts they push in the movies is the left eye is evil for some reason. Or to a lesser extent use the eye as a gateway to demonstrate functionality such as with Technotise (2009) or Gamer (2009). In phase 2 an actual nano-camera will be installed in the left eye. People with this might comment on how they feel like there’s a small bump in their eye under slightly drier conditions such as when going to sleep at night. This is documented in the physical example page. Note if you figure out you have a camera in the left eye they will probably install something in the right eye too. Clips referencing this concept are now available here, here and here and now also Doomsday Book (2012).
· Phase 2 – Permanent Metallic Taste: Some people will comment on a metallic taste in the mouth. Typically when going to sleep it becomes prevalent. This is mostly a direct response to be monitored. The more they monitor you or your BioAPI the more of a metallic taste you’ll receive. It is not a side effect of medication, that’s the typical response a doctor will give you. If you are not on medication and otherwise completely healthy and all of a sudden have a permanent metallic taste in your mouth, you are being recorded (but not watched) 24/7 as per the trailer for A Scanner Darkly (2006).
· Phase 2 – Morgellons: Morgellon’s can strike anyone. It’s a direct problem from the nano-fibers, whether intentional or accidental. The body’s immune system can’t see or recognize the fibers at all. So when the body can’t accept the fibers anymore it beings to push them out through the skin. But the skin is a barrier because the fibers are too large. So the skin breaks up which is why people get lesions. Note that technically everyone has morgellons (nano-fibers), the actual mogellon’s symptoms are when the person’s body tries to get rid of them the only way possible. Some more conclusions can be seen here and examples within media references including this.
Chemtrail Nano-fiber Examples & Evidence
Ultimately you need some proof. This is very difficult, as we all don’t exactly have nano-tech labs in our basements. The only thing possible at this point in time is to put out the physical evidence that is known and back it up with media/movie supporting clips. A complete list of unbelievable things this technology can do is listed here, also make sure you see the real life body & mind control examples in Media References.
The Global Threat of Persistent Organic Pollutants
There is a connection between pesticides, both general and POPs (Persistent Organic Pollutants), to a myriad of adverse health effects such as cancer and neurological disorders, particularly Parkinson’s Disease. For these reasons alone, it is important to look at the standards currently set for such chemicals by the international standard-setting organization known as Codex Alimentarius.—-As mentioned in the first article in this series, There will be a comparison between two different types of pesticides for the sole purpose of the topic of discussing Codex Alimentarius guidelines – general pesticides and POPs. General pesticides should be recognized as, quite simply, those pesticides not considered a Persistent Organic Pollutant by the Stockholm Convention. General pesticides are those which are most widely used since the Stockholm Convention actually banned the use of POPs altogether. Nevertheless, it is important to make the distinction for the purpose of clearly understanding Codex guidelines in this regard.– The term Persistent Organic Pollutant applies to specific types of pesticides and chemicals. These substances have been used mostly in pest control, “disease control,” agriculture, and other different industries. The EPA distinguishes between two different types of POPs in terms of their production – intentional and unintentional. Intentionally produced POPs are those which are produced for the purpose of being used in manufacturing, agriculture, pest/disease control, or other industrial uses. To put it quite simply, intentionally produced POPs are those that are produced intentionally.
Unintentionally produced POPs are those that are essentially byproducts of industrial processes or combustion (like the incineration of waste) etc.
The chemical in question has to meet fairly stringent requirements in order to obtain the status of POP, however. Additionally, the label “organic” means the chemicals must be carbon-based (organic) substances.
According the website of the Stockholm Convention on Persistent Organic Pollutants, they must also meet the following requirements.
They possess a particular combination of physical and chemical properties such that, once released into the environment, they:
Remain intact for exceptionally long periods of time (many years);
Become widely distributed throughout the environment as a result of natural processes involving soil, water and, most notably, air;
Accumulate in the fatty tissue of living organisms including humans, and are found at higher concentrations at higher levels in the food chain; and
Are toxic to both humans and animals. 
Initially, the Stockholm Convention, the United Nations treaty that requires nations to cease or reduce the production, application, and release of POPs, distinguished 12 substances as those that should be eliminated. This treaty places the POPs into 3 different categories: pesticides, industrial chemicals, and by-products. POPs and their categories are listed below:
Pesticides: Aldrin; Chlordane; Dichlorodiphenyl tricholoreothane (DDT); Dieldrin; Endrin; Heptachlor; Hexachlorobenzene; Mirex; Toxaphene;
Industrial Chemicals: Hexachlorobenzene; Polychlorinated biphenyls (PCBs)
By-Products: Hexachlorobenzene; Polychlorinated dibenzo-p-dioxins; Polychlorinated Dibenzofurans; PCBs
In May 2009, the Conference of the Parties to the Stockholm Convention agreed to add 9 new chemicals to the list of POPs. They are as follows:
Pesticides: Chlordecone; Alpha Hexcholorocyclohexane; Beta Hexachlorocyclohexane; Lindane; Pentachlorobenzene;
Industrial Chemicals: Hexabromobiphenyl; Hexabromodiphenyl ether/Heptabromodiphenyl ether; Pentachlorobenzene; Pefluorooctane Sulfonic Acid, its salts, and Perfluorooctane Sulfonyl Fluoride; Tetrabromodiphenyl ether, Pentabromodiphenyl ether
By-Products: Alpha Hexachlorocyclohexane; Beta Hexachlorohexane; Pentachlorobenzene; 
The major difference between general pesticides and POPs is not necessarily in their level of danger but the fact that they meet the guidelines listed above and that they remain intact for long periods of time, become widely distributed, and accumulate in fatty tissue.
The accumulation of POPs in fatty tissue is at the heart of the problem with these chemicals due to the fact that they can pose an even more serious threat to predators at the top of the food chain than those at the bottom. This is due to the process known as biomagnification, where POPs accumulate in the fatty tissue of organisms and become more and more concentrated as they move from one organism to the other. As they work their way through the food chain, becoming more and more contaminated as they move along, those organisms at the very top of the food chain will be ingesting the largest amounts of the chemicals. It is for this reason that even small releases of POPs can be disastrous, especially for localized ecosystems that dine on local game/fish.
An example cited on the EPA’s website reflects on a study conducted by the Arctic Monitoring and Assessment Programme, where it was found that caribou in the Northwest Territories of Canada had as much as 10 times the amounts of PCBs as the lichen that they ate. Not only that, but the wolves that dined on the caribou contained 60 times the amount of PCBs as the lichen. The adverse effects of POPs on wildlife are very similar to those on humans. Studies conducted on wildlife communities showed “reproductive, developmental, endocrine, immunologic, and carcinogenic effects.” Other studies have shown further connections between thyroid and estrogen disorders. Marine animals warrant a particular concern due to the fact that POPs have low water solubility and therefore bond easily with particulate matter in sediment. This gives them the ability to lay dormant for a long time in the water supply, becoming active again when disturbed years later. Not only that, but the fact that POPs do not have to be directly applied to an area or species to be present or have an effect on these organisms creates a pollution problem that knows no boundaries[F3]. The discovery of POPs in the Alaskan Arctic, thousands of miles away from any known source, is a prime example of this. It is well-known that POP compounds can attach themselves not only to aquatic sediment material, but also to airborne particles. Along with the ability of most POPs to exist as gases, this allows them to have increased ability to travel long distances and potentially pollute the entire globe.[F4]
Some of these chemicals are able to evaporate from land and water sources into the air, and return to the earth in rain, snow, and mist. Add to this the transportability of the POPs by airborne and aquatic means, as well as by the very human and animal organisms being contaminated by them, and one can easily see the problem posed here. However, it should be noted that although only 21 of these substances are listed as POPs, the potential for any of the thousands more combinations of chemicals to do the same is still very real.
 “Persistant Organic Pollutants: A Global Issue, A Global Response.” Environmental Protection Agency. December 17, 2009.http://www.epa.gov/international/toxics/pop.htm
 “What are POPs?” Stockholm Convention on persistant organic pollutants. Accessed March 23, 2010. http://chm.pops.int/Convention/ThePOPs/tabid/673/language/en-US/Default.aspx
 “What are POPs?” Stockholm Convention on persistant organic pollutants. http://chm.pops.int/Convention/ThePOPs/tabid/673/language/en-US/Default.aspx Accessed March 23, 2010.
 “Persistant Organic Pollutants: A Global Issue, A Global Response.” Environmental Protection Agency. December 17, 2009.http://www.epa.gov/international/toxics/pop.htm
 Abelsohn, Alan., Gibson, Brian L., Sanborn, Margaret D., Weir, Erica. “Identifying and managing adverse environmental health effects:5. Persistant Organic Pollutants.” Canadian Medical Association Journal. June 11, 2002. 166 (12). http://www.cmaj.ca/cgi/content/full/166/12/1549 Accessed May 24, 2010.
[F1]In this case the chemo preferred would be herbals and botanicals and foods using chemistry that id by design to assist people in restoration and renewal
[F2]So By incorporating a ketone type diet and the use of supplements –herbs and foods we should see a improvement
[F3]Consider the GMO’s and there by products how much have they spread in 40 years and with the increased levels of these organisms being planted and the reduction of the traditional foods and the added chemicals and mixes —how long will they lay dormant?? And how much as people eating the foods fed the gmos ho much more are we getting??
[F4]With harp this is more then likely
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